ライフサイエンスコーパス: PNES

1 PNES carried a higher risk of natural (HR 8.1, 95% CI 4.

2 PNES showed a comparable mean brain-PAD (10.6 years) to

3 m correctly classified 24 GTCSs (96%) and 18 PNESs (95%).

4 Of 45 video-documented signs, only 3 PNES signs ("preserved awareness," "eye flutter," and "b

5 PNES vs. FE vs. GE (3 x 2 chi(2)), P = 0.30; PNES vs. epilepsy (2 x 2 chi(2)), P = 0.14).

6 We analyzed 120 seizures (36 PNES, 84 ES) from 35 consecutive subjects.

7 e individuals with FE (3.05%) or GE (1.82%) (PNES vs. FE vs. GE (3 x 2 chi(2)), P = 0.30; PNES vs. ep

8 of epilepsy, PNES or concurrent epilepsy and PNES attending between 1 January 2007 and 18 June 2021.

9 le with concurrent diagnosis of epilepsy and PNES or PNES alone have significantly increased odds of

10 le with concurrent diagnosis of epilepsy and PNES, the odds for suicide attempt-related admissions we

11 The association between PNES diagnosis and all-cause mortality varied with age a

12 We reviewed the literature comparing PNES with other functional neurological symptoms.

13 m 11 patients, and 19 episodes of convulsive PNES from 13 patients.

14 ntaneous mouse model of autoimmune diabetes, PNES inhibited disease progression in diabetic mice.

15 semiological signs that reliably distinguish PNES and ES, and found that eyewitness reports of these

16 ations to identify reliable video-documented PNES and ES signs, and we compared eyewitness reports wi

17 th seizures included a diagnosis of epilepsy+PNES (OR 5.7, p=0.009), work status (OR 3.9, p=0.027) an

18 Like epilepsy, PNES carries a higher than expected risk of both natural

19 primary or secondary diagnosis of epilepsy, PNES or concurrent epilepsy and PNES attending between 1

20 ic acid sodium salt)}naphthylene]ethynylene (PNES), which helically wraps the nanotube surface with p

21 T(1)-state formation and decay dynamics for PNES-SWNTs in aqueous and DMSO solvents, as well as thos

22 psychiatric disorders as candidate genes for PNES.

23 ptoms and global functioning with CBT-ip for PNES without and with sertraline.

24 orts the use of manualized psychotherapy for PNES and successful training of mental health clinicians

25 This pilot randomized clinical trial for PNES revealed significant seizure reduction and improved

26 available for 172 patients, of whom 154 had 'PNES only'.

27 er corroborate these interesting findings in PNES.

28 genetic factors are likely to play a role in PNES or its comorbidities in a subset of individuals.

29 Outcome in PNESs is poor but variable.

30 comes were less encouraging, some aspects of PNES outcome may be better than previously thought.

31 ified individuals with incident diagnosis of PNES, epilepsy and conversion disorder with motor sympto

32 nifestation and 4.1 years after diagnosis of PNES.

33 serve in this first genetic investigation of PNES that six (5.88%) individuals with PNES without coex

34 le is known about the molecular pathology of PNES, much less about possible underlying genetic factor

35 death was the patient age at presentation of PNES.

36 concurrent diagnosis of epilepsy and PNES or PNES alone have significantly increased odds of hospital

37 nd video-documented semiology for predicting PNES, and we measure accuracy of eyewitness reports.

38 EEG monitoring, to inquire about 48 putative PNES and ES signs.

39 uced proliferation in pLNs of mice receiving PNES as compared to sham controls.

40 sts that psychogenic non-epileptic seizures (PNES) are associated with increased mortality.

41 Psychogenic non-epileptic seizures (PNES) are classified with other functional neurological

42 nts with psychogenic non-epileptic seizures (PNES), 5-10 years after diagnosis.

43 tcome in psychogenic non-epileptic seizures (PNES), and none of long-term healthcare utilization.

44 Psychogenic nonepileptic seizures (PNES) are diagnosed in approximately 30% of patients ref

45 delay of psychogenic nonepileptic seizures (PNES) requires prompt referral for video electroencephal

46 known as psychogenic nonepileptic seizures (PNES).

47 TLE), (2) psychogenic nonepileptic seizures (PNESs) from MRI-negative epilepsies, and (3) progressive

48 utcome in psychogenic nonepileptic seizures (PNESs) patients is limited; we know less still about fac

49 eptic and psychogenic nonepileptic seizures (PNESs).

50 Pancreatic nerve electrical stimulation (PNES) resulted in beta-adrenergic receptor-mediated-accu

51 Spectroscopic interrogation of such PNES-SWNT samples in aqueous and DMSO solvents using E(2

52 make VEEG referrals when semiology suggests PNES, although few semiological signs are supported by w

53 ogenic (LP) variants in 102 individuals with PNES and 448 individuals with focal (FE) or generalized

54 nd psychiatric disorders in individuals with PNES shows that genetic factors are likely to play a rol

55 of P/LP variants among the individuals with PNES was similar and not significantly different to the

56 on of PNES that six (5.88%) individuals with PNES without coexistent epilepsy carry P/LP variants (de

57 Included were 885 individuals with PNES, 50 663 with epilepsy and 1057 with conversion diso

58 ound 32 (3.6%) deaths among individuals with PNES, compared with 46 (0.5%) deaths in controls, giving

59 py or psychopharmacology to outpatients with PNES.

60 Suicide ranked high in patients with PNES (18.8%) and conversion disorder with motor symptoms

61 le with concurrent diagnosis and people with PNES alone (OR 0.75; 95% CI 0.41 to 1.48; p=0.40).

62 alyses revealed that the odds of people with PNES alone were 1.93 times the odds of people with epile

63 a cohort of 260 patients who presented with PNES between 1999 and 2004.

64 One hundred sixty-four adult patients with PNESs (66.7%) responded to outcome, personality, and psy