ライフサイエンスコーパス: PPS

1 PPS and heparin also decreased MMP-9 (P < 0.001) after t

2 PPS by itself, or CCh and NE in the absence of synaptic

3 PPS encodes MAX2/ORE9 (for MORE AXILLARY BRANCHES2/ORESA

4 PPS enhanced fear reactions to a conditioned stimulus (C

5 PPS is a promising new therapy for alphavirus-induced ar

6 PPS is an excellent permeation enhancer which provides n

7 PPS was found to improve sensitivity in MALDI analyses o

8 PPS was injected intramuscularly weekly for 3 weeks.

9 PPS was tested for dose- and time-dependent cytotoxicity

10 PPS, including sugar beet pectin/arabinan, apple/citrus

11 PPSs identified the highest prevalence of C. auris colon

12 ylene glycol)(9) methyl ether acrylate](17) (PPS(135)-b-POEGA(17)).

13 est response being to the PPS of serotype 3 (PPS 3).

14 NF-A and conducted 7 C. auris PPSs; during 4 PPSs, we also performed CPO screening and environmental

15 ses, the percentage of patients who filled a PPS prescription and were diagnosed later with a maculop

16 d with a 23-valent pneumococcal vaccine or a PPS 3-bovine serum albumin conjugate revealed that they

17 ulin loci (XenoMouse mice) vaccinated with a PPS-3-tetanus toxoid conjugate and their molecular genet

18 There is more ethical consensus about PPS than PSU.

19 ospice programs develop clear policies about PPS and PSU, including mechanisms for training and ensur

20 hCD19Tg mice lacked B-1b cells and adaptive PPS-specific antibody responses.

21 generated B-1b cells and protective adaptive PPS-specific antibody responses, whereas hCD19Tg mice la

22 nt jumping reads information across adjacent PPSs and implement it in the HapSeq program.

23 lities conditional on the states of adjacent PPSs.

24 All PPS-containing preparations induced IL-6 and TNF-alpha f

25 as degraded more rapidly and that PB-145 and PPS inhibited the degradation of both proteins.

26 uctions in joint destruction with PB-145 and PPS treatments (P < 0.01) compared with buffer control.

27 rface in the animals treated with PB-145 and PPS.

28 The avidities of PPS 6B- and PPS 23F-specific IgG2 antibodies ranged from 6 to 31 nM-

29 ntly higher than both unselected B cells and PPS-specific B cells.

30 duced calcium responses, PD-1 induction, and PPS-3-specific IgG3 responses and restored protection du

31 a negative correlation between HIV load and PPS response for the groups receiving HAART.

32 ] is also an independent predictor of OS and PPS in patients with radiologic progression.

33 Both PLGA and PPS MPs enabled sustained release of EPO-R76E, providing

34 puborectalis muscles between the PPS(+) and PPS(-) groups were compared.

35 s sequence to analyse the effect of VWS- and PPS-associated mutations in the DNA-binding domain of IR

36 ference between young and older adults, anti-PPS IgA or IgM antibodies were removed from immune sera

37 All anti-PPS levels were at or below prevaccination baselines by

38 Thus, functionally disparate anti-PPS antibodies can arise within individuals both by acti

39 In both age groups, anti-PPS IgA or IgM antibody levels were much lower than anti

40 enC resulted in a boosted secondary IgG anti-PPS response to S. pneumoniae.

41 IgG anti-PPS responses to PPS3, PPS14, and C-polysaccharide (C-PS

42 in IL-7Ralpha are severely impaired in anti-PPS responses and do not survive Streptococcus pneumonia

43 In order to determine the effects of anti-PPS IgA or IgM antibodies on the functional difference b

44 IgM antibody levels, the low levels of anti-PPS IgM antibody alone can explain the functional differ

45 enhanced both the primary and secondary anti-PPS responses in mice, especially the type 1 IgG isotype

46 Serum anti-PPS levels and antibodies specific for capsular types 3

47 gM antibody levels were much lower than anti-PPS IgG antibody levels.

48 In conclusion, even though anti-PPS IgG antibody levels are high compared with anti-PPS

49 antibody levels are high compared with anti-PPS IgM antibody levels, the low levels of anti-PPS IgM

50 rmal unfolding and the stabilization by APS, PPS-1 behaved like the unstable human PAPSS2 protein sug

51 Using multiplex bead arrays, PPS treatment was found to have significantly increased

52 rly post-CPB neither prevents nor attenuates PPS in children.

53 ed effort at vSNF-A and conducted 7 C. auris PPSs; during 4 PPSs, we also performed CPO screening and

54 ntramyocardial injection of Rg3-loaded PEG-b-PPS nanoparticles improved the cardiac function and redu

55 generate ROS-responsive nanoparticles (PEG-b-PPS) via the self-assembly of diblock copolymers of poly

56 e showed no significant associations between PPS exposure and diagnosis of drusen, nonexudative AMD,

57 e analysis, no association was found between PPS exposure and subsequent diagnosis of maculopathy.

58 e was no dose-dependent relationship between PPS exposure and diagnosis of any maculopathy.

59 tcome measure was association between binary PPS exposure and any maculopathy.

60 independent and failed to generate a boosted PPS-specific secondary IgG response.

61 ion, the severity of which was alleviated by PPS therapy during RRV and CHIKV clinical disease.

62 The LTD induced by PPS in the presence of NE or CCh is of comparable magnit

63 Induction of LTD by PPS was inhibited by NMDA receptor blockers (completely

64 At ambient temperature (25 degrees C), PPS-b-PDMA-b-PNIPAAM assembled into 66 +/- 32 nm micelle

65 Once the proteins are free from the cell, PPS also assists in protein solubilization by shielding

66 al variability (ITV) in brain regions coding PPS predicts individual differences of its boundary at t

67 methylprednisolone treatment may complicate PPS.

68 Complicated PPS--noncomplicated PPS plus hospital readmission +/- pe

69 rginally significant increase in complicated PPS (p = 0.05).

70 In conclusion, PPS alters extracellular matrix turnover through the ind

71 throughout IRF6 may cause VWS; in contrast, PPS-causing mutations are highly associated with the DNA

72 mester the PPS was larger than the controls' PPS and the shift between representation of near and far

73 acrylamide)-block-(N-isopropylacrylami de)] (PPS-b-PDMA-b-PNIPAAM) that forms physically cross-linked

74 In total, 39/246 children (16%) developed PPS (noncomplicated: n = 30, complicated: n = 9).

75 duced AHN and hippocampal function following PPS in a rodent model.

76 e during recall of the CS stimulus following PPS.

77 g identified binding sites on PrP 23-106 for PPS, which include the octarepeat histidine and an N-ter

78 bs were found to have similar affinities for PPS-3 but different epitope specificities and CDR3 regio

79 V(H) was associated with lower affinity for PPS 14, a result suggesting that somatic mutation could

80 nd-alone version of the PPS and guidance for PPS users are being developed.

81 psular polysaccharide (PPS) are required for PPS-based vaccine-mediated protection against Streptococ

82 tion of anti-dsDNA IgA, but a major role for PPS-associated TLR2 agonists was also revealed.

83 analyses of seven Fab fragments specific for PPS serotype 6B, 14, or 23F.

84 all patients with WS undergoing therapy for PPS from 1984 to 1999 were reviewed.

85 tory cytokines in the bITON model, drug-free PPS MPs have innate antioxidant properties that provide

86 t classic disease; and clearly distinct from PPS maculopathy.

87 ed evidence of disease clearly distinct from PPS maculopathy.

88 ic geranyl(geranyl)diphosphate synthase [G(G)PPS] is involved in myrcene biosynthesis in hop trichome

89 , we also assessed peripersonal space, e.g., PPS, the area around the body used to act on nearby obje

90 The patient was also given PPS for a short period by peripheral infusion and there

91 unrestricted funding support for the Global-PPS.

92 dataset were correctly categorized as having PPS maculopathy.

93 osure were categorized incorrectly as having PPS maculopathy.

94 o catalyzes depupylation, it was unclear how PPS function could be maintained without Dop and PafA ca

95 +/- 32 nm micelles comprising a hydrophobic PPS core and PNIPAAM on the outer corona.

96 s to identify some of the recent advances in PPS and to describe progress towards greater standardiza

97 proposed as a mechanism of Peters anomaly in PPS.

98 rget proteins is functionally compromised in PPS is unknown.

99 This was explained by >10-fold increases in PPS-specific immunoglobulin G (IgG) levels in Pneumovax-

100 purified and/or contaminating TLR ligands in PPS vaccine preparations.

101 molecular basis for the defects observed in PPS and potential targets that contribute to the patholo

102 observation that B3GLCT mutant phenotypes in PPS patients are less severe than embryonic lethal pheno

103 function of the disaccharide and its role in PPS remain unexplored.

104 ng of trunk-centered multisensory signals in PPS is of particular relevance for theoretical models an

105 efficacy of specific pharmaceuticals used in PPS.

106 rs of a frameshift ABCG8 mutation increasing PPS levels in carriers by 50%.

107 al models using a binary variable indicating PPS exposure showed no significant associations between

108 rioception, body-related visual information, PPS, and embodiment) and argue that the fronto-parietal

109 Subcutaneously injected PPS-b-PDMA-b-PNIPAAM polymer solutions formed stable hyd

110 Fluorescently labeled PPS were used in FACSAria flow cytometry to characterize

111 The labeled PPS were capable of inhibiting binding of Ab to the nati

112 e oxidants were found to oxidize the micelle PPS core, making it more hydrophilic and triggering mice

113 Most residents screened during multiple PPSs remained persistently colonized with C. auris.

114 Reads that span multiple PPSs (jumping reads) can provide additional haplotype in

115 Polyphenylsilsesquioxane [PhSiO(1.5)](n) (PPS) and polyvinylsilsesquioxane [vinylSiO(1.5)](n) (PVS

116 Similarly, the native PPS were able to inhibit binding of PPS-specific B cells

117 le of inhibiting binding of Ab to the native PPS.

118 Noncomplicated PPS--temperature >100.5 degrees F, pericardial friction

119 Complicated PPS--noncomplicated PPS plus hospital readmission +/- pericardiocentesis or

120 on of certain V(H)3 genes used in the normal PPS response.

121 The added advantage of PPS over conventional detergents such as sodium dodecyl

122 The avidities of PPS 6B- and PPS 23F-specific IgG2 antibodies ranged from

123 e native PPS were able to inhibit binding of PPS-specific B cells in a flow cytometric assay demonstr

124 te dissociation, modifying the boundaries of PPS, but leaving IPS distance unaltered.

125 d fMRI to capture the individual boundary of PPS and examine its neural underpinnings.

126 A major characteristic of the boundary of PPS in humans is the extremely high variability of its l

127 In this presentation, we report a case of PPS with homozygous pathogenic variant in B3GLCT who pre

128 The imaging characteristics of PPS maculopathy allow for differentiation from hereditar

129 Direct characterization of PPS-specific B cells has not been performed.

130 d-type C57BL/6 (Wt) mice with a conjugate of PPS of serotype 3 and tetanus toxoid (PPS3-TT) and deter

131 se response, we calculated the total days of PPS prescriptions filled and created a categorical varia

132 eport expands the phenotypic descriptions of PPS by characterizing posterior segment findings.

133 Graphical display of PPS rules, a stand-alone version of the PPS and guidance

134 confirmed the reversibility of the effect of PPS.

135 Our results demonstrate lasting effects of PPS on the hippocampus and highlight the utility of EE d

136 However, neither the efficacy of PPS vaccines in immunocompromised individuals nor the ho

137 ns show high variability in the extension of PPS across individuals, but there is a lack of evidence

138 stimuli predicts the individual extension of PPS.

139 ens-cornea adhesion), which is a hallmark of PPS.

140 and 99.6% specificity for identification of PPS maculopathy by masked review of fundus imaging in th

141 , and results in a striking 100% increase of PPS in carriers.

142 , we directly characterized the phenotype of PPS-specific B cells before and after vaccination with P

143 w cytometry to characterize the phenotype of PPS-specific B cells obtained from 18 young adults pre-

144 n infection and investigate the potential of PPS to treat disease.

145 nd transcellular pathways in the presence of PPS.

146 nown effect on the incidence and severity of PPS in children undergoing surgery with CPB.

147 eration, CPB time, incidence and severity of PPS.

148 ized as having findings highly suggestive of PPS maculopathy; 25 patients showed some features resemb

149 ient images as follows: highly suggestive of PPS maculopathy; some features resembling PPS maculopath

150 TLR2 activity was distinct from that of PPS, in that it was phenol extractable.

151 he TLR2/4 antagonist, OxPAPC, at the time of PPS immunization completely blocked the production of an

152 ective data demonstrate the potential use of PPS-b-PDMA-b-PNIPAAM as an injectable, cyto-protective h

153 n was inhibited by the addition of PB-145 or PPS.

154 ere was no inter-group difference in overall PPS incidence (p = 0.73).

155 thylene glycol)-poly(propylene sulfide) (PEG-PPS) and poly(ethylene glycol)-oligo(ethylene sulfide) (

156 t anti-pneumococcal capsular polysaccharide (PPS) antibody avidity can influence protective efficacy.

157 ies to pneumococcal capsular polysaccharide (PPS) are a critical component of vaccine-mediated immuni

158 ies to pneumococcal capsular polysaccharide (PPS) are required for PPS-based vaccine-mediated protect

159 Pneumococcal capsular polysaccharide (PPS) vaccines are less immunogenic in immunocompromised

160 ies to pneumococcal capsular polysaccharide (PPS), we studied the response of transgenic mice reconst

161 ies to pneumococcal capsular polysaccharide (PPS).

162 . pneumoniae type 3 capsular polysaccharide (PPS-3) and other strong TI-2 Ags were significantly impa

163 type 3 pneumococcal capsular polysaccharide (PPS-3) were generated from transgenic mice reconstituted

164 th the purified pneumococcal polysaccharide (PPS) has been a topic of debate.

165 By contrast, pneumococcal polysaccharide (PPS) immunization protected CD19(-/-) mice during lethal

166 Remarkably, pneumococcal polysaccharide (PPS) vaccination alone induced C4(-/-) mice to produce i

167 The current pneumococcal polysaccharide (PPS) vaccine is highly effective in young adults; howeve

168 o the 23-valent pneumococcal polysaccharide (PPS) vaccine, Pneumovax, such as children <2 y, the aspl

169 ibodies against pneumococcal polysaccharide (PPS), older adults had lower IgA and IgM antibody levels

170 ning to isolate pneumococcal polysaccharide (PPS)-specific Fab fragments from two vaccinated adults.

171 I) Ags, such as pneumococcal polysaccharide (PPS).

172 zed with native pneumococcal polysaccharide (PPS; Pneumovax23) or protein-PPS conjugate (Prevnar-13)

173 (PPT) with selected pectic polysaccharides (PPS) and modulation of the conjugation in order to obtai

174 es to isolated pneumococcal polysaccharides (PPS) required TLR signaling in vivo.

175 d to determine whether pentosan polysulfate (PPS) inhibited proliferation and altered extracellular m

176 Pentosan polysulfate (PPS) is a glycan derivative that is orally bioavailable,

177 an (GAG)-like molecule pentosan polysulfate (PPS) to alleviate virus-induced arthritis.

178 thereof when bound to pentosan polysulfate (PPS).

179 ombin III (n = 9); and pentosan polysulfate (PPS; n = 7).

180 The reactivity of PPT/PPS and their ratio were determinants for their heteroco

181 Dimethyl palmitoyl ammonio propanesulfonate (PPS), with excellent enhancement potential and minimal t

182 e key population found to produce protective PPS-specific antibody in both wild-type and CD19(-/-) mi

183 polysaccharide (PPS; Pneumovax23) or protein-PPS conjugate (Prevnar-13) vaccines.

184 gion genes to form pneumococcal capsular PS (PPS) 6B-specific paratopes.

185 ed a rapid primary pneumococcal capsular PS (PPS) response in mice that was dependent on CD4(+) T cel

186 Repeat PPSs at vSNF-A in 2018 identified increasing C. auris pr

187 costs of HAIs and AMU in Singapore, repeated PPSs over the next decade will be useful to gauge progre

188 PD-1 blockade also selectively rescued PPS-3-specific IgG3 responses in CD21/35(-/-) mice.

189 25 patients showed some features resembling PPS maculopathy but not classic disease; and 1091 patien

190 of PPS maculopathy; some features resembling PPS maculopathy, but not classic disease; and clearly di

191 n response to reactive oxygen species (ROS), PPS becomes hydrophilic and degrades to enable drug rele

192 Proportionate palliative sedation (PPS) uses the minimum amount of sedation necessary to re

193 atically, and pediatric procedural sedation (PPS) is increasingly performed by practitioners who are

194 s at individual potential polymorphic sites (PPSs).

195 Peripersonal space (PPS) is a multisensory and sensorimotor interface mediat

196 sorimotor interface, the peripersonal space (PPS), mediates every physical interaction between our bo

197 pace surrounding it: the peripersonal space (PPS).

198 ry bodily stimuli within peripersonal space (PPS).

199 n dilation of peripheral pulmonary stenosis (PPS) in Williams syndrome (WS) is limited.

200 n and isolated peripheral pulmonic stenosis (PPS).

201 We analyzed plasma plant sterol (PPS) levels, a surrogate measure of cholesterol absorpti

202 ng SE induced by perforant path stimulation (PPS) at the ages of postnatal day 21 (P21) and P35 were

203 we discovered that paired-pulse stimulation (PPS) elicits a form of homosynaptic long-term depression

204 ction and AHN following pre-pubertal stress (PPS) is unknown.

205 Here we demonstrate that random-structured PPS and PVS rearrange in the presence of catalytic amoun

206 glycol) (PEG) and poly (propylene sulfide) (PPS) and use them for Rg3 encapsulation and delivery.

207 es (ROS)-degradable poly(propylene sulfide) (PPS).

208 yloxyethyl)pyridin-1-yl]propane-1-sulfonate (PPS).

209 sDNA IgA in C4(-/-) mice without suppressing PPS-specific Ab production.

210 onducted a national point prevalence survey (PPS) to determine the prevalence of healthcare-associate

211 We performed point prevalence surveys (PPSs) to identify patients colonized with C. auris and i

212 ed to characterize postprogression survival (PPS) and assess with time-dependent covariates analysis

213 and severity ofpostpericardiotomy syndrome (PPS) in children after cardiac surgery with cardiopulmon

214 Peters plus syndrome (PPS) is a combination of congenital Peters anomaly and s

215 Peters Plus syndrome (PPS), a congenital disorder of glycosylation, results fr

216 ns in B3GLCT result in Peters plus syndrome (PPS), an autosomal recessive disorder characterized by e

217 rome (VWS) and popliteal pterygium syndrome (PPS) are autosomal dominant disorders characterized by c

218 Popliteal pterygium syndrome (PPS; OMIM 119500) is a disorder with a similar orofacial

219 assessing paradoxical puborectalis syndrome (PPS) in patients with obstructive defecation syndrome (O

220 Its Pathway Pre & QJ1;diction System (PPS) is now an internationally recognized, open system f

221 he Medicare ESRD prospective payment system (PPS) and changes to dosing guidelines for erythropoiesis

222 quitin-like protein (Pup)-proteasome system (PPS), the bacterial equivalent of the eukaryotic ubiquit

223 Caco-2 studies revealed that PPS is an effective enhancer of macromolecular transport

224 aumatic optic neuropathy (bITON) showed that PPS and PLGA MP-mediated delivery of EPO-R76E provided t

225 Histological studies showed that PPS does not induce damage to the intestine.

226 In vivo studies in rats showed that PPS enhanced relative bioavailability of sCT by 45-fold

227 Our data suggest that PPS-3 consists of epitopes that can elicit both highly p

228 Our findings suggest that PPS-based vaccines can be effective in the setting of CD

229 ammation and joint swelling, suggesting that PPS is a promising candidate for drug repurposing for th

230 The PPS-b-PDMA-b-PNIPAAM micelles were preloaded with the mo

231 The PPS-based MP platform is especially promising for furthe

232 4 vs 0.27 +/- 0.17, P = 0.009) analysis, the PPS(+) patients displayed a lower absolute ADC differenc

233 ches of the puborectalis muscles between the PPS(+) and PPS(-) groups were compared.

234 In contrast, the PPS-specific B cells obtained postimmunization were pred

235 a lower absolute ADC difference than did the PPS(-) patients.

236 this prediction, we cloned and expressed the PPS-1 protein from the roundworm Caenorhabditis elegans

237 However, the PPS-responding B-cell population has not yet been identi

238 Likewise, the PPS-specific B cells obtained preimmunization consisted

239 udio-tactile integration task to measure the PPS boundary at different stages of pregnancy.

240 y of PPS rules, a stand-alone version of the PPS and guidance for PPS users are being developed.

241 dividual differences in the extension of the PPS are predicted by variability of BOLD responses in th

242 nization of the neural representation of the PPS occurs.

243 arities resulting from implementation of the PPS or ESA label change.

244 ART may influence qualitative aspects of the PPS response by restoring expression of certain V(H)3 ge

245 nt with the proposed nutritional role of the PPS under starvation conditions.

246 The next decade should see the PPS, and the UM-BBD on which it is based, find increasin

247 died PS-specific responses, we find that the PPS 6B repertoire makes use of a diverse collection of h

248 These findings indicate that the PPS repertoire in the adult derives from memory B-cell p

249 ies, with the greatest response being to the PPS of serotype 3 (PPS 3).

250 However, in the third trimester the PPS was larger than the controls' PPS and the shift betw

251 tween July 2015 and February 2016, using the PPS methodology developed by the European Centre for Dis

252 bservations in healthy young volunteers, the PPS-responding B-cell population consisted primarily of

253 ) emissions were 28% and 17% higher with the PPS-M compared to the SMPS for LSHFO and MGO, respective

254 rtum women did not show differences in their PPS size as compared to the control group (non-pregnant

255 Consistent with this, PPS immunization induced a robust TI response in young I

256 n vitro opsonophagocytic activities of three PPS-specific mouse immunoglobulin G1 monoclonal antibodi

257 Thus, PPS is influenced by progression pattern and this is key

258 ctive, serotype-specific human antibodies to PPS 3, and they lend support to the proposal that these

259 ation immunoglobulin G2 (IgG2) antibodies to PPS serotypes 6B and 23F and examined the relationship b

260 Binding to PPS exposes a hydrophobic surface composed of aligned tr

261 ced fluorescence of PrP 23-106 when bound to PPS, consistent with the alignment of tryptophan side ch

262 and categorical, time-dependent, exposure to PPS and to drusen, nonexudative age-related macular dege

263 pecificity, and efficacy for defined MAbs to PPS may identify antibody features that might be useful

264 sis of three monoclonal antibodies (MAbs) to PPS 3 generated from lymphoid cells from mice vaccinated

265 odel to study the human antibody response to PPS antigens.

266 anifested a V(H)3-(D12)-positive response to PPS, despite a similar IgG response in each group.

267 als to generate a V(H)3-positive response to PPS.

268 not adult mice are impaired in responses to PPS vaccination and to 4-hydroxy-3-nitrophenyl-acetyl-Fi

269 by the monoclonal antibody D12) responses to PPS were determined for first-time recipients of a 23-va

270 A single 526-kb haplotype mapped strongly to PPS levels, dramatically refining the mapped interval.

271 Although only one animal subjected to PPS at P21 developed chronic spontaneous seizures by sev

272 of observation, all the animals subjected to PPS at P35 became epileptic.

273 the hippocampus in P21 animals subjected to PPS, although extensive activation of hippocampal and ex

274 pal structures was seen in pups subjected to PPS-induced self-sustaining SE at P35 or LiPC SE at P21.

275 ith human immunoglobulin loci, XenoMouse, to PPS antigens in a pneumococcal vaccine.

276 mirror website of the UM-BBD, UM-BPT and UM-PPS is being developed at ETH Zurich to improve speed an

277 The new UM-PPS produces a multi-level prediction within an acceptab

278 The original UM-PPS predicted up to two prediction levels at a time.

279 y of Minnesota pathway prediction system (UM-PPS) recognizes functional groups in organic compounds t

280 d a rule-based Pathway Prediction System (UM-PPS) that predicts plausible pathways for microbial degr

281 Currently, the UM-PPS contains 260 biotransformation rules derived from re

282 The UM-PPS is freely available to all without registration.

283 As rules were added to the UM-PPS, more of them were triggered at each prediction step

284 ajority of PrP 23-106 remain disordered upon PPS binding, the octarepeat region adopts a repeating lo

285 -valent pneumococcal polysaccharide vaccine (PPS).

286 ned for first-time recipients of a 23-valent PPS vaccine, both receiving and not receiving HAART, and

287 The commercial 23-valent PPS vaccine, Pneumovax-23 also contained TLR ligands (TL

288 POEGA forms the hydrophilic NP surface while PPS forms the hydrophobic NP core that sequesters GANT58

289 tinct mutations in 13 families affected with PPS.

290 All 10 patients with PPS maculopathy in this dataset were correctly categoriz

291 In the 5 patients treated with PPS survival was 16 months, 45 months, 84 months, 105 mo

292 aximum survival in patients not treated with PPS.

293 ical findings in a case of vCJD treated with PPS.

294 significantly increased after treatment with PPS (P < 0.001) and heparin (P < 0.05).

295 Treatment with PPS and heparin increased TIMP-1.

296 Furthermore, treatment with PPS reduced the severity of both RRV- and CHIKV-induced

297 These data suggest that vaccination with PPS may not be effective for patients during and after l

298 ic B cells before and after vaccination with PPS vaccine (PPV) in elderly adults, using fluorescently

299 Five patients without PPS exposure were categorized incorrectly as having PPS

300 o present, although less pronounced, without PPS.