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1                                              PRRS is one of the most important diseases in swine indu
2 ates that vaginal microbiota collected after PRRS vaccination could be potentially used to classify s
3 l protein 11 (NSP11) of the causative agent, PRRS virus (PRRSV), contains a highly conserved nidovira
4 rm that CD163 is the likely receptor for all PRRS viruses.
5 rus derived from VR-2332, the North American PRRS virus prototype.
6 nd respiratory syndrome virus (PRRSV) causes PRRS and is known to effectively suppress host innate im
7 ental viruses have been detected in clinical PRRS cases, raising the need for a better differential t
8      MLV vaccines are widely used to control PRRS; however, there have been serious concerns regardin
9                                      Current PRRS inactivated vaccine provides only a limited protect
10 ted under the conditions of this experiment, PRRS virus was shown to evolve continuously in infected
11                          High-risk areas for PRRS were best-predicted by pig density and climate seas
12 hylodynamic models to generate risk maps for PRRS outbreaks and reconstructed the evolutionary histor
13 l immunity and leading to novel vaccines for PRRS prevention.
14                                 Furthermore, PRRS viruses were characterized by typical seasonality i
15 his study was to describe ongoing changes in PRRS virus during replication in pigs under experimental
16                          B cell expansion in PRRS was not associated with preferential VH gene usage
17 ntal step in elucidating the role of nsp2 in PRRS pathogenesis and provides an important insight in f
18 ferent secondary bacterial diseases occur in PRRS virus (PRRSV)-infected pigs.
19 - 0.0%), 98-37120 (0.8% +/- 0.42%), Ingelvac PRRS MLV (0.9% +/- 0.46%), and negative controls (2.3% +
20 ted vaccines, Ingelvac PRRS MLV and Ingelvac PRRS ATP, derived from VR2332 and JA142, respectively, h
21 ccine strains Ingelvac PRRS MLV and Ingelvac PRRS ATP, respectively.
22 son with modified live virus (MLV) (Ingelvac PRRS MLV).
23 isolate 98-38803 is a derivative of Ingelvac PRRS MLV and that the isolate is pneumovirulent.
24 ates and the VR2332 parent virus of Ingelvac PRRS MLV, which were all highly related to Ingelvac PRRS
25 orm homoduplexes with the reference Ingelvac PRRS MLV.
26 ensed low-virulence vaccine strain, Ingelvac PRRS modified live virus (MLV), activated expression of
27 s infection by an avirulent strain, Ingelvac PRRS modified live virus (MLV), did not.
28 h the HMA reference vaccine strains Ingelvac PRRS MLV and Ingelvac PRRS ATP, respectively.
29 nificantly more severe lesions than Ingelvac PRRS MLV (0.7 +/- 0.17) and controls (0.7 +/- 0.15).
30 V, which were all highly related to Ingelvac PRRS MLV with </=2% nucleotide sequence divergence as de
31            Two attenuated vaccines, Ingelvac PRRS MLV and Ingelvac PRRS ATP, derived from VR2332 and
32 g field isolate was identified when Ingelvac PRRS ATP was used as the HMA reference except for its pa
33 roup 2 (n = 19) was inoculated with Ingelvac PRRS MLV vaccine, group 3 (n = 20) was inoculated with t
34                       Compared with Ingelvac PRRS((R)) MLV strain, A2MC2-P90 elicits higher virus neu
35 ates to those of a modified live virus (MLV) PRRS vaccine and its parent strain.
36 uvant candidate for the development of novel PRRS inactivated vaccine.
37 ent may have contributed to the emergence of PRRS.
38 ed the relations between the spatial risk of PRRS outbreaks and its phylodynamic history in the U.S d
39 rcine respiratory and reproductive syndrome (PRRS) vaccination.
40 rcine reproductive and respiratory syndrome (PRRS) and bovine coronavirus viruses.
41 rcine reproductive and respiratory syndrome (PRRS) caused by the PRRS virus (PRRSV) is an important s
42 rcine reproductive and respiratory syndrome (PRRS) consistently elevates the frequency of disease and
43 rcine reproductive and respiratory syndrome (PRRS) has a heritable component, yet little is known abo
44 rcine reproductive and respiratory syndrome (PRRS) is one of the most significant swine diseases.
45 rcine reproductive and respiratory syndrome (PRRS) remains a major threat to animal health and causes
46 rcine reproductive and respiratory syndrome (PRRS) virus nonstructural protein 2 (nsp2) contains a cy
47 rcine reproductive and respiratory syndrome (PRRS) virus.
48 rcine reproductive and respiratory syndrome (PRRS) viruses are recognized as possessing a high degree
49 rcine reproductive and respiratory syndrome (PRRS), one of the most persistent global livestock disea
50 rcine reproductive and respiratory syndrome (PRRS).
51 ate immune evasion properties encoded by the PRRS viral genome.
52 nd respiratory syndrome (PRRS) caused by the PRRS virus (PRRSV) is an important swine disease worldwi
53 as reported to be present in only one of the PRRS modified live virus vaccine strains (Ingelvac) and
54  and genomic recombination properties of the PRRS virus (PRRSV) have not been completely elucidated.
55 -edited pigs that are fully resistant to the PRRS virus have raised expectations for eliminating this
56 lop production animals that are resistant to PRRS, the costliest viral disease to ever face the swine
57 chitecture of gene expression in response to PRRS virus (PRRSV) infection.