ライフサイエンスコーパス: PSC

1 PSC has only been used to explain analytically solvable

2 PSC patients show increased Th17 differentiation already

3 PSC with lower proportion of component-shared alteration

4 PSC-derived cardiomyocytes can be generated routinely wi

5 PSCs can be stabilized in the naive state by blocking ex

6 In 6 of 12 PSC surveys, the estimated prevalence of zinc deficiency

7 e transcriptome analysis was performed on 14 PSC samples, evaluated also for the presence of the main

8 h-volume sites, PSC at low-volume sites; (4) PSC only; (5) DBS only.

9 imary end point, bezafibrate led in 45% (41% PSC, 55% PBC) and placebo in 11% to >=50% reduction of s

10 ed patients, 70 completed the trial (95%; 44 PSC, 24 PBC, 2 SSC).

11 In the 6 PSC surveys in which adjustment methods were compared, a

12 13 nationally representative surveys (18,859 PSC, 22,695 WRA) from the Biomarkers Reflecting Inflamma

13 Twenty-seven (87%) PSCs had component-private alterations.

14 t the rational design of 1D ETMs can achieve PSCs that perform as well as typical mesoscopic and plan

15 Subjects with advanced PSC or cholangiocarcinoma (CCA) at baseline were exclude

16 n the PSC, or how their organization affects PSC function.

17 All PSC cases were investigated for PD-L1 expression.

18 In addition, the PTPBT-ET(0.3) -based all-PSC also exhibits long-term photostability over 300 hour

19 And the all-polymer solar cell (all-PSC) based on PBDB-T:PTPBT-ET(0.3) achieved a high power

20 performance of all-polymer solar cells (all-PSCs).

21 s with donor polymer (PBDB-T) can enable all-PSCs with significantly increased PCE (up to 8.5%).

22 tor polymers to achieve high-performance all-PSCs with optimal active layer film morphology.

23 on surveys conducted in WRA (n = 32,588) and PSC (n = 8,256) from the Biomarkers Reflecting Inflammat

24 A pathway were differentiated into ESPCs and PSC-derived epidermal organotypic rafts (PSC-EORs).

25 Effect estimates for PLC and PSC remained similar after adjustment for PM and gaseous

26 thin the size range of 10 - 100 nm, PLC, and PSC were associated with an increase of MI 6 h later by

27 Liver transplantation and PSC-related events accounted for approximately 75% of cl

28 heles mosquitoes collected by light trap and PSC in all sites.

29 nts between CRP and vitamin B-12 for WRA and PSC ranged from -0.25 to 0.16, and correlations between

30 assivated MAPbI(3) perovskite thin films and PSCs were significantly enhanced, retaining about 90 % o

31 ivation of the various interfaces of RBs and PSCs.

32 umber (PNC), length (PLC), and surface area (PSC) concentrations, and myocardial infarction (MI) at a

33 The anatase TiO(2) nanopyramid array-based PSCs deliver a champion power conversion efficiency of a

34 far above the most efficient fullerene-based PSCs.

35 have been achieved in the field of TDM-based PSCs.

36 The relationship between PSC and inflammatory bowel disease has inspired theories

37 ures of a large panel of PSC-associated BTC (PSC-BTC).

38 nts with compensated liver disease caused by PSC were randomized 1:1:1 to receive weekly subcutaneous

39 accounted for 86% of Anopheles collected by PSC and 66% by CDC light trap in the sprayed sites while

40 n the numbers of An. arabiensis collected by PSC were 69% in the intervention compared to the non-int

41 Pulmonary sarcomatoid carcinoma (PSC) contains carcinomatous component (CaC) and sarcomat

42 Pulmonary sarcomatoid carcinomas (PSC) are a rare group of lung cancer with a median overa

43 luripotent stem cell-derived cardiomyocytes (PSC-CMs) for both patient health and scientific investig

44 luripotent stem cell-derived cardiomyocytes (PSC-CMs) hold great promise for disease modeling and dru

45 Recently, plasma separation cards (PSCs) have become available and, though more expensive,

46 rate that C(60) is applicable in hot carrier PSCs as it is capable of extracting hot carriers generat

47 oor CDC light trap, pyrethrum spray catches (PSC) and human landing collection (HLC) before and after

48 ly alterations in perisynaptic Schwann cell (PSC), glial cells at this synapse, may impact their abil

49 Our knowledge of pluripotent stem cell (PSC) biology has advanced to the point where we now can

50 Variability among pluripotent stem cell (PSC) lines is a prevailing issue that hampers not only e

51 iblast and mammalian pluripotency stem cell (PSC) models, we show that PSCs undergo a mesenchymal-epi

52 Dynamic pluripotent stem cell (PSC) states are in vitro adaptations of pluripotency con

53 ecent advent of human pluripotent stem cell (PSC)-derived 3D brain organoids has opened a window into

54 Perisynaptic Schwann cells (PSCs) are specialized, non-myelinating, synaptic glia of

55 oltage hysteresis in perovskite solar cells (PSCs) and, in turn, to impact the interfacial carrier in

56 Metal-halide perovskite solar cells (PSCs) are one of the most promising photovoltaic materia

57 ort-layer (ETL)-free perovskite solar cells (PSCs) are still inferior to ETL-containing devices.

58 Perovskite solar cells (PSCs) composed of organic polymer-based hole-transportin

59 rt, high-performance perovskite solar cells (PSCs) contain a large amount of iodine to realize smalle

60 dation mechanisms of perovskite solar cells (PSCs) containing unreacted PbI(2) has been still insuffi

61 oltaic efficiency of perovskite solar cells (PSCs) depends drastically on the charge-carrier dynamics

62 nmental stability of perovskite solar cells (PSCs) has been improved by trial-and-error exploration o

63 ional instability of perovskite solar cells (PSCs) is known to mainly originate from the migration of

64 rface passivation of perovskite solar cells (PSCs) using a low-cost industrial organic pigment quinac

65 llowing us fabricate perovskite solar cells (PSCs) with a champion reverse scan power conversion effi

66 -based devices, the perovskite solar cells (PSCs) with acid-treated TiO(2) ETL exhibit an enhanced s

67 rently, blade-coated perovskite solar cells (PSCs) with high power conversion efficiencies (PCEs), th

68 sion efficiencies of perovskite solar cells (PSCs).

69 ized and employed in perovskite solar cells (PSCs).

70 in triple cation 2D perovskite solar cells (PSCs).

71 materials (ETMs) for perovskite solar cells (PSCs); however, experimental evidence is lacking.

72 co-cultured with pancreatic stellate cells (PSCs) they are significantly more resistant to gemcitabi

73 ted platelets and pancreatic stellate cells (PSCs), the two major players in the TME, we can effectiv

74 erentiation of human pluripotent stem cells (PSCs) and create inference networks that integrate gene

75 Reprogrammed pluripotent stem cells (PSCs) are valuable for research and potentially for cell

76 ies, patient-derived pluripotent stem cells (PSCs) conditional for the FA pathway were differentiated

77 eer AEC2s from human pluripotent stem cells (PSCs) in vitro and use time-series single-cell RNA seque

78 Pluripotent stem cells (PSCs) transition between cell states in vitro, reflectin

79 from mouse and human pluripotent stem cells (PSCs), as well as strategies to characterize and mature

80 a major property of pluripotent stem cells (PSCs).

81 ely referred to as 'pluripotent stem cells' (PSCs)) can acquire genetic changes, which commonly inclu

82 rvations that, in contrast to somatic cells, PSCs are programmed to die in response to genomic damage

83 PZC and inflammation in preschool children (PSC; 6-59 mo) and nonpregnant women of reproductive age

84 age (WRA; 15-49 yr) and preschool children (PSC; 6-59 mo).

85 e effects of primary sclerosing cholangitis (PSC) in patients with inflammatory bowel disease (IBD).

86 Primary sclerosing cholangitis (PSC) is a cholestatic liver disease characterised by chr

87 omy, whereas primary sclerosing cholangitis (PSC) may be protective.

88 cer (BTC) in primary sclerosing cholangitis (PSC) may exceed 20%, and BTC is currently the leading ca

89 atients with primary sclerosing cholangitis (PSC) show a prominent IL-17 response upon stimulation wi

90 atients with primary sclerosing cholangitis (PSC) show a prominent interleukin (IL)-17 response upon

91 hogenesis of primary sclerosing cholangitis (PSC), a progressive biliary tract disease without approv

92 is (PBC) and primary sclerosing cholangitis (PSC), are frequently associated with damage to the barri

93 atients with primary sclerosing cholangitis (PSC), must be accompanied by functional changes.

94 ce of sex on primary sclerosing cholangitis (PSC), pre and post-liver transplantation (LT) is unclear

95 In primary sclerosing cholangitis (PSC), the focus of this review, the cholangiocyte respon

96 atients with primary sclerosing cholangitis (PSC).

97 atients with primary sclerosing cholangitis (PSC).

98 affected by primary sclerosing cholangitis (PSC).

99 primary or secondary sclerosing cholangitis (PSC, SSC) and primary biliary cholangitis (PBC).

100 0% under 1 sun illumination for blade-coated PSCs is demonstrated with an open-circuit voltage of 1.1

101 a high efficiency of 21.9% for blade-coated PSCs with an open-circuit voltage of 1.20 V, correspondi

102 interact with the Rad51 presynaptic complex (PSC).

103 The resulting post-synaptic complex (PSC) has a surprisingly short mean lifetime.

104 menon is due to parameter space compression (PSC), in which a subset of parameters emerges as "stiff"

105 Conclusion: PSC patients show increased Th17 differentiation already

106 By contrast, PSC-CMs fail to fully undergo these developmental proces

107 In contrast, PSC-RANTES induces CCR5 hyperphosphorylation, driving en

108 y Cs(3)Bi(2)I(9) perovskite single crystals (PSCs).

109 Analysis of the ELTR database (PSC patients registered before 2018), including baseline

110 Here we derived PSCs from distinct mouse strains under naive conditions

111 eta (TGF-beta), and WNT pathways, we derived PSCs from mice, horses, and humans (designated as XPSCs)

112 ationwide; of these, 2588 patients developed PSC.

113 im was to quantify the effects of developing PSC in patients with all subtypes of IBD and evaluate it

114 cating that it is a marker of differentiated PSCs.

115 ned the developmental potential of the donor PSCs and were associated with global DNA hypomethylation

116 n of H2HR signaling ameliorates large ductal PSC-induced damage.

117 wing the efficiency gap with high-efficiency PSCs employing highly crystalline TiO(2) ETLs.

118 e discussed for the fabrication of efficient PSCs.

119 FA PSC-EORs harbored diminished cell-cell junctions and inc

120 -associated platelet regulation and 16 h for PSC regulation.

121 r conversion efficiency reported to date for PSCs consisting of 1D ETMs.

122 tent candidates for a design of new HTMs for PSCs with improved longevity.

123 ency of 21.1 % for n-i-p structured ETL-free PSCs, outperforming their ETL-containing metal oxide cou

124 uence of commitment and differentiation from PSC-derived embryonic mesoderm through hematopoietic spe

125 on stimulation with microbes, monocytes from PSC patients produced significantly more IL-1beta and IL

126 whose expression differed in organoids from PSC patients compared to non-PSC controls, including hum

127 ion has changed recently, with non-fullerene PSCs developing very rapidly.

128 wer conversion efficiencies of non-fullerene PSCs have now reached over 15 %, which is far above the

129 Furthermore, PSCs exhibit high X-ray imaging capability thanks to its

130 Furthermore, PSCs from both muscles extended disorganized processes f

131 demonstration of >20% efficient low-band gap PSCs.

132 we are still far from being able to generate PSC-derived cardiomyocytes with adult-like phenotypes in

133 her caspase-substrate, such as filamin, in H-PSCs suggested that molecular events related to early ap

134 However, PSC-CMs exhibit immature phenotypes in culture, and the

135 ts, supporting the consistency between human PSC-derived and mouse primary spinal motor neuron models

136 In human PSC, inflammatory and stromal cells trigger PBG activati

137 city of already-established murine and human PSCs.

138 uss the types of mutations acquired by human PSCs and the mechanisms that lead to their accumulation.

139 vitamin B6 and branched-chain amino acids in PSC (P < .0001), which strongly associated with reduced

140 Genomic alterations in PSC-BTC include a significant number of putative actiona

141 5HT levels increase in Mdr2(-/-) mice and in PSC human patients compared to their controls and decrea

142 Thirty-eight genes deregulated in PSC were identified, among these IGJ and SLMAP were conf

143 ation, potentially overcoming differences in PSC line differentiation potential.

144 and progression of hepatobiliary fibrosis in PSC.

145 ity, confirming a major role for genetics in PSC phenotypic variability.

146 tely predicts hepatic decompensation (HD) in PSC and exceeds the performance among other widely avail

147 s may be relevant for the disease process in PSC.

148 this dominant T-helper 17 (Th17) response in PSC are not clear.

149 e reasons for this dominant TH17 response in PSC are not clear.

150 subsides over time, ultimately resulting in PSC-derived AEC2s that exhibit a stable phenotype and ne

151 e-based personalized treatment strategies in PSC-BTC.

152 nd senescent cholangiocyte subpopulations in PSC.

153 In PSCs with lower differentiation potential, DNA replicati

154 Short exposure to miR-203 in PSCs (miPSCs) induces a transient expression of 2C marke

155 arrays and demonstrate their application in PSCs.

156 gins and a higher frequency of DNA breaks in PSCs with incompletely reprogrammed DNA replication were

157 ation of additional recombination centers in PSCs facilitated by elevated temperature and excess char

158 ile method to reset the epigenetic memory in PSCs, and improve their effectiveness in regenerative me

159 uggests that the underlying mutation rate in PSCs is low, although they also seem to be particularly

160 cludes genes known to play critical roles in PSCs and synapses.

161 oduction of an MHC class I-restricted TCR in PSCs produced naive, antigen-specific CD8alphabeta(+) T

162 or management of cholangiopathies, including PSC.

163 on with phorbol myristate acetate/ionomycin, PSC patients showed significantly increased numbers of I

164 wing ex vivo stimulation with PMA/Ionomycin, PSC patients showed significantly increased numbers of I

165 h, we visualized DNA replication in isogenic PSCs generated by different reprogramming approaches, ei

166 To date, however, no known PSCs have demonstrated dual competency for chimera forma

167 Regardless of the anatomical localization, PSC-BTC exhibited a uniform molecular and histological c

168 ges in incidence, profile and outcome in LT- PSC patients in Europe with specific emphasis on sex.

169 bility to isolate and genetically manipulate PSCs in a cell specific manner.

170 veloping a robust strategy to produce mature PSC-CMs.

171 and present the current approaches to mature PSC-derived cardiomyocytes.

172 (C-statistic, 0.72; 95% CI, 0.57-0.84), Mayo PSC risk score (C-statistic, 0.85; 95% CI, 0.77-0.92), a

173 mice and performed Ca(2+)-imaging to monitor PSC activity and used immunohistochemistry to analyze th

174 We conclude that most PSCs originate from a monoclone accompanied by genomic I

175 most highly expressed miRNAs in naive mouse PSC.

176 atures of the naive state in human and mouse PSCs can be recapitulated without affecting FGF-MEK sign

177 nsensus that naive hPSC resemble mouse naive PSC, showing that the absence of miR-371-373 in conventi

178 organoids from PSC patients compared to non-PSC controls, including human leukocyte antigen DM alpha

179 Notably, PSC-BTC shows a distinct extrahepatic morpho-molecular p

180 We propose that numerical PSC has the potential to identify the low-dimensional st

181 his was accompanied by a failure of numerous PSCs to upregulate galectin-3 (MAC-2), a marker of glial

182 stem cells can be isolated from the bile of PSC patients undergoing endoscopic retrograde cholangiop

183 sis, hPFCs harbor the clonogenic capacity of PSC cultures and emerge prior to commitment events or ph

184 arcinoma (LUAD), adenocarcinoma component of PSC showed lower EGFR incidence.

185 Development of PSC also increased risks of cholangiocarcinoma (HR, 28.4

186 Development of PSC was associated with increased risk of death and CRC

187 l aberrations might drive the development of PSC.

188 events when patients received a diagnosis of PSC at an age younger than 40 years vs 31% of patients w

189 Patients who receive a diagnosis of PSC at an age younger than 40 years, men, and patients o

190 ve as a springboard for revealing drivers of PSC differentiation and function.

191 visualized the epithelial-damaging effect of PSC-derived K. pneumoniae that was associated with bacte

192 whether it translates into an impairment of PSC-dependent repair mechanisms.

193 bean heritage have an increased incidence of PSC-related events.

194 origin and intratumor heterogeneity (ITH) of PSC.

195 nocytes and T cells from blood and livers of PSC patients and controls were analyzed ex vivo and in v

196 nocytes and T cells from blood and livers of PSC patients and controls were analyzed ex vivo and in v

197 date, the greatest success in maturation of PSC-derived cardiomyocytes has been with transplantation

198 gene 2 knockout (Mdr2(-/-) ) mouse model of PSC, in vitro studies were performed in cell lines of ch

199 an increasing number of PSC patients, particularly women, are being transplanted

200 939609 was associated with increased odds of PSC (OR: 2.14; 95% CI: 1.27, 3.60) in the lowest quintil

201 nd pathological features of a large panel of PSC-associated BTC (PSC-BTC).

202 In populations of PSC that exhibit a significant negative association betw

203 uch changes raise concerns for the safety of PSC-derived cellular therapies for regenerative medicine

204 cribed in genome-wide association studies of PSC.

205 roved biochemical parameters and symptoms of PSC with oral antibiotics, ostensibly through manipulati

206 These findings advance our understanding of PSC-associated cholangiocarcinogenesis and provide stron

207 This system is expected to facilitate use of PSC-CMs in a variety of scientific and medical investiga

208 Here, it is shown that degradation of PSCs is hastened by unreacted PbI(2) crystals under cont

209 reatly impact the charge carrier dynamics of PSCs.

210 how that the photoconversion efficiencies of PSCs increase by ~40%, for applied pressures between 0 a

211 ity frequently decreases during expansion of PSCs in vitro.

212 The majority of PSCs (97%) had component-shared alterations.

213 Previous studies have generated a number of PSCs with distinct properties.

214 ilms and affect the long-term performance of PSCs.

215 rs (mAChRs) regulate the repair phenotype of PSCs and are overactivated at disease-resistant NMJs [so

216 integrity and differentiation proficiency of PSCs, analyzing DNA replication may be a useful quality

217 nt prognostic factor, and more proportion of PSCs may be beneficial from immune checkpoint inhibitors

218 tion energetics and operational stability of PSCs is investigated.

219 which limit the efficiency and stability of PSCs.

220 effective route to improve the stability of PSCs.

221 al contact in the multilayered structures of PSCs.

222 The study of PSCs has relied on an anatomy-based approach, as the ide

223 nt role in driving phenotypic variability of PSCs.

224 We find that X-ray detectors based on PSCs exhibit high sensitivity of 1652.3 muC Gy(air)(-1)

225 overall prevalence or score for cortical or PSC cataracts between exposed and control animals.

226 tly higher prevalence or risk of cortical or PSC cataracts compared to control animals.

227 no correlation between dose and cortical or PSC score.

228 are useful to differentiate iCCA from HCC or PSC, and could help in the early diagnosis of these dise

229 increased risks of liver transplantation or PSC-related death compared with white race (HR, 2.05; P

230 itamin B-12 or folate deficiencies in WRA or PSC.

231 lear pattern of association in either WRA or PSC.

232 ptake/correct VL result using either DBSs or PSCs to increase VL access on equipment available in-cou

233 orm as well as typical mesoscopic and planar PSCs.

234 at $30.90 compared to $31.62 in our plasma + PSC scenario.

235 This appears to prevent PSCs from adopting a repair phenotype, resulting in a ma

236 tive stress in E. granulosus protoescoleces (PSCs) to identify molecular pathways and antioxidant res

237 identified incident cases of IBD, with PSC (PSC-IBD) and without, from April 2006 to April 2016 and

238 h halide perovskite thin films coated by QA, PSCs based on methylammonium lead iodide (MAPbI(3) ) sho

239 and PSC-derived epidermal organotypic rafts (PSC-EORs).

240 However, only a fraction of reprogrammed PSCs are developmentally competent.

241 e differentiation potentials of reprogrammed PSCs.

242 We collected 31 resected PSC tumors and obtained CaC and SaC by laser capture mic

243 Myom2-RFP(+) PSC-CMs exhibited more mature phenotypes than RFP(-) cel

244 2) as a unique molecular marker of S100beta+ PSCs in skeletal muscle.

245 dium, patient age, and number of years since PSC was diagnosed.

246 lume sites; (3) plasma at high-volume sites, PSC at low-volume sites; (4) PSC only; (5) DBS only.

247 edial control, and under SPL7, somatosensory PSC, ventral LOC and cerebellar control.SIGNIFICANCE STA

248 rmal organ size and specification from SOX2+ PSCs.

249 approach, as the identities of cell-specific PSC molecular markers have remained elusive.

250 able fabrication of efficient and air-stable PSCs composed of an invasive plant (Eichhornia crassipes

251 uclear, cortical, and posterior subcapsular (PSC) cataracts in vivo and photographically.

252 We show that PSC mAChR-dependent activity was transiently increased a

253 PSCs are specifically labeled, we show that PSCs have a unique molecular signature that includes gen

254 potency stem cell (PSC) models, we show that PSCs undergo a mesenchymal-epithelial transition (MET) p

255 The PSC risk estimate tool (PREsTo) consists of nine variabl

256 greatest difference in mortality between the PSC-IBD alone group vs the IBD alone group was for patie

257 In the PSC-IBD group, diagnosis of IBD at age younger than 40 y

258 ably, transcription effectively resolves the PSC, and we predict that this is a conserved mechanism t

259 ttle about how they are organized within the PSC, or how their organization affects PSC function.

260 must bind to a specific location within the PSC, whereas Rdh54 retains its function in the repair of

261 Rdh54 have distinct binding sites within the PSC, which allow these proteins to act cooperatively as

262 distributions of Rad54 and Rdh54 within the PSC.

263 The PSCs are then fabricated before applying a range of pres

264 so improves the operational stability of the PSCs, retaining 92% efficiency after constant illuminati

265 Finally, differences in these PSC-dependent NMJ repair mechanisms were MU type depende

266 he key to this striking performance of these PSCs is the formation of a full-coverage, stable native-

267 result by developing a numerical approach to PSC that can be applied to any computational model.

268 5 of 10 on visual analog scale [VAS]) due to PSC, PBC, or SSC were recruited for this double-blind, r

269 The H2HR axis may be targeted in treating PSC.

270 a newly generated transgenic mouse in which PSCs are specifically labeled, we show that PSCs have a

271 2, 95% CI 0.07-0.21, p < 0.001) and 93% with PSC collections (RR = 0.07, 0.03-0.17, p < 0.001).

272 We identified incident cases of IBD, with PSC (PSC-IBD) and without, from April 2006 to April 2016

273 rman), in total comprising 136 patients with PSC (58% with IBD), 158 HCs, and 93 patients with IBD wi

274 sma from an independent set of patients with PSC and controls found reduced concentrations of vitamin

275 pneumonia in the microbiota of patients with PSC and demonstrate that K. pneumoniae disrupts the epit

276 moderate to severe pruritus in patients with PSC and PBC.

277 In livers of patients with PSC cirrhosis, CD14(hi) CD16(int) and CD14(lo) CD16(hi)

278 In livers of patients with PSC cirrhosis, CD14(hi)CD16(int) and CD14(lo)CD16(hi) mo

279 ntial of the gut microbiome in patients with PSC compared with healthy controls (HCs) and patients wi

280 The gut microbiome in patients with PSC exhibits large functional differences compared with

281 Patients with PSC had fewer microbial genes compared with HCs (P < .00

282 ociated microbial studies that patients with PSC harbor an abnormal enteric microbiome.

283 ation of biliary strictures in patients with PSC is especially challenging, with imaging and endoscop

284 nd effectiveness of its use in patients with PSC remain uncertain, with only a few studies performed

285 Compared with HCs, patients with PSC showed enrichment and increased prevalence of Clostr

286 Patients with PSC showed marked differences in the abundance of genes

287 ferences were detected between patients with PSC with and without IBD.

288 of bezafibrate on pruritus in patients with PSC, PBC, and SSC.

289 e of peroral cholangioscopy in patients with PSC, with a focus on its use in the evaluation and manag

290 embedded tumor tissue from 186 patients with PSC-BTC from 11 centers in eight countries with all anat

291 ed to patients with IBD alone, patients with PSC-IBD had a 4-fold higher risk of CRC if they received

292 nwide study, we confirmed that patients with PSC-IBD have increased risks of CRC, hepatopancreatobili

293 Among patients with PSC-IBD we observed 173 first liver transplants.

294 -related death was lower among patients with PSC-IBD who received annual imaging evaluations before t

295 ody directed against LOXL2, in patients with PSC.

296 ed transplant free-survival in patients with PSC.

297 the leading cause of death in patients with PSC.

298 athogenic role of monocytes in patients with PSC.

299 ), 158 HCs, and 93 patients with IBD without PSC, were subjected to metagenomic shotgun sequencing, g

300 n age at CRC diagnosis (59 y vs 69 y without PSC; P < .001).