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1 omosome mec (SCCmec) typing, and PCR for the Panton-Valentine leukocidin.
2 es encoding toxic shock syndrome toxin 1 and Panton-Valentine leukocidin.
3 mplexes, the junkyard regions as well as the Panton-Valentine leukocidin.
4 resistance, as well as toxic shock toxin and Panton-Valentine leukocidin.
5 A) to SEH, toxic shock syndrome toxin 1, and Panton-Valentine leukocidin.
6 but lacked all common toxin genes, including Panton-Valentine leukocidin.
7 were less diverse (DI = 0.566), positive for Panton-Valentine leukocidin (96.3%), and resistant to er
8 e properties of CA-MRSA, with an emphasis on Panton-Valentine leukocidin, alpha-hemolysin, and the re
9 n transcription and translation of genes for Panton-Valentine leukocidin, alpha-hemolysin, and toxic-
10  of nafcillin induced and prolonged mRNA for Panton-Valentine leukocidin, alpha-toxin, and toxic-shoc
11 trains did differ, however, in expression of Panton-Valentine leukocidin and in the degree of inflamm
12  strains express a number of toxins, such as Panton-Valentine leukocidin and LukAB, that have specifi
13 s aureus (MRSA) strain with genes coding for Panton-Valentine leukocidin and the arginine catabolic m
14  of the Rams' MRSA; all carried the gene for Panton-Valentine leukocidin and the gene complex for sta
15                The vaginal isolate was mecA, Panton-Valentine leukocidin, and staphylococcal enteroto
16 avoiding barotrauma to lungs made friable by Panton-Valentine leukocidin expressing S. aureus infecti
17 ere were four patients with sputum-confirmed Panton-Valentine leukocidin expressing S. aureus pneumon
18 available on the management of patients with Panton-Valentine leukocidin expressing S. aureus pneumon
19 we reviewed our experience and outcomes with Panton-Valentine Leukocidin expressing S. aureus pneumon
20                                              Panton-Valentine leukocidin expressing S. aureus pneumon
21                                              Panton-Valentine leukocidin expressing Staphylococcus au
22 teremia were less likely to be infected with Panton-Valentine leukocidin gene (pvl)-constitutive MRSA
23 sessed different exotoxin gene profiles (eg, Panton Valentine leukocidin genes) compared with health
24 ec (SCCmec) type IV, 145 (35.9%) carried the Panton-Valentine leukocidin genes (PVL+), and 162 (40.1%
25 eus isolates from Cape Verde showed that (i) Panton-Valentine leukocidin genes were present in 35% of
26                        Staphylococcus aureus Panton-Valentine leukocidin is a pore-forming toxin targ
27 aphylococcal virulence factors, particularly Panton-Valentine leukocidin, is common in CA-MRSA, empha
28 ibody levels against leukotoxin E (LukE) and Panton-Valentine leukocidin (LukS-PV), but not alpha-hem
29 aphylococcal bicomponent pore-forming toxins Panton-Valentine leukocidin LukSF-PV (PVL) and gamma-hem
30                                      Neither Panton-Valentine leukocidin nor protein A expression was
31 of CA-MRSA isolates tested were positive for Panton-Valentine leukocidin, of which 90% carried staphy
32  gel electrophoresis (PFGE), spa typing, and Panton-Valentine leukocidin PCR.
33 e of ST5, 2 carried arcA and opp3, and 1 was Panton-Valentine leukocidin positive (PVL(+)).
34 of the 63-wound isolates belonged to the CC8/Panton-Valentine leukocidin-positive (PVL(+)) group of S
35 assette chromosome mec (SCCmec) type IV, and Panton-Valentine leukocidin-positive clustered separatel
36 otype (complicated or uncomplicated SAB) and Panton-Valentine leukocidin-positive isolate.
37 colonization rate of ciprofloxacin-sensitive Panton-Valentine leukocidin-positive methicillin-resista
38                                              Panton-Valentine leukocidin-positive USA300 strains caus
39 t two cases of invasive infections caused by Panton-Valentine leukocidin-positive, community-associat
40                              The role of the Panton Valentine leukocidin (PVL) in CA-MRSA pathogenesi
41 al cassette (SCC)mec type IV, and 61.5% were Panton Valentine leukocidin (PVL) positive.
42 lpha-hemolysin (Hla), delta-hemolysin (Hld), Panton Valentine leukocidin (PVL), staphylococcal entero
43   The gamma-hemolysins (HlgAB and HlgCB) and Panton-Valentine leukocidin (PVL or LukSF) were shown to
44 aturity (P = 0.02) and isolates positive for Panton-Valentine leukocidin (PVL) (P = 0.008).
45 f potent staphylococcal exotoxins, including Panton-Valentine leukocidin (PVL) and alpha-hemolysin (H
46                                              Panton-Valentine leukocidin (PVL) and alpha-toxin are ex
47 ate isolates showed all strains were USA300, Panton-Valentine leukocidin (PVL) and arginine catabolic
48 reus protein A (spa) typing and detection of Panton-Valentine leukocidin (PVL) and scn genes.
49 ant Staphylococcus aureus strains expressing Panton-Valentine leukocidin (PVL) are associated with se
50  Staphylococcus aureus (MRSA) expressing the Panton-Valentine leukocidin (PVL) are rampant, but the c
51 s, the most prominent CA-MRSA strain encodes Panton-Valentine leukocidin (PVL) cytotoxin genes, belon
52 l cassette chromosome mec (SCCmec) types and Panton-Valentine leukocidin (PVL) gene carriage were com
53                                The lukF/lukS Panton-Valentine leukocidin (PVL) genes did not directly
54 testing by broth microdilution, detection of Panton-Valentine leukocidin (PVL) genes, arginine catabo
55 nce typing (MLST), as well as assays for the Panton-Valentine leukocidin (PVL) genes, the protein A g
56 dulins, and acquisition of the phage-encoded Panton-Valentine leukocidin (PVL) genes.
57 isolates were tested for the presence of the Panton-Valentine leukocidin (PVL) genes.
58 ons (SSTI) are associated with SCCmec IV and Panton-Valentine leukocidin (PVL) genes.
59                                  The role of Panton-Valentine leukocidin (PVL) in determining the sev
60                                 sThe role of Panton-Valentine leukocidin (PVL) in Staphylococcus aure
61                                  The role of Panton-Valentine leukocidin (PVL) in Staphylococcus aure
62                                          The Panton-Valentine leukocidin (PVL) is a cytotoxin express
63 lly, many workers have hypothesized that the Panton-Valentine leukocidin (PVL) is a key virulence det
64                                              Panton-Valentine leukocidin (PVL) is a pore-forming toxi
65                    The Staphylococcus aureus Panton-Valentine leukocidin (PVL) is a pore-forming toxi
66                                              Panton-Valentine leukocidin (PVL) is a two-component cyt
67                                              Panton-Valentine leukocidin (PVL) is common in African S
68                          The S. aureus toxin Panton-Valentine leukocidin (PVL) is most likely causati
69 CA-MRSA) can harbor a bacteriophage encoding Panton-Valentine leukocidin (PVL) lysogenized into its c
70       Antimicrobial susceptibility patterns, Panton-Valentine leukocidin (PVL) occurrence, and staphy
71                                          The Panton-Valentine leukocidin (PVL) of Staphylococcus aure
72                                The impact of Panton-Valentine leukocidin (PVL) on the outcome in Stap
73                                              Panton-Valentine leukocidin (PVL) production by methicil
74                All strains were subjected to Panton-Valentine leukocidin (PVL) screening, and SCCmec,
75                              USA300 secretes Panton-Valentine leukocidin (PVL) toxin, which is associ
76                                              Panton-Valentine leukocidin (PVL) was present in 21.9%,
77   The genes lukS-PV and lukF-PV encoding the Panton-Valentine leukocidin (PVL) were present in all CA
78                                              Panton-Valentine leukocidin (PVL), a bacteriophage encod
79                                              Panton-Valentine leukocidin (PVL), a pore-forming cytoto
80 SA300, like other CA-MRSA strains, expresses Panton-Valentine leukocidin (PVL), a pore-forming toxin
81 of LukGH in vivo were compared with those of Panton-Valentine leukocidin (PVL), a well-characterized
82 f staphylococci, detection of genes encoding Panton-Valentine leukocidin (PVL), and antimicrobial res
83 he arginine catabolic mobile element (ACME), Panton-Valentine leukocidin (PVL), and other toxins that
84 e chromosome (SCC)mec type IV, the genes for Panton-Valentine leukocidin (PVL), and the enterotoxin Q
85            Most SSTI CA-MRSA strains produce Panton-Valentine leukocidin (PVL), but its contribution
86  for disease and whether a virulence factor, Panton-Valentine leukocidin (PVL), could account for the
87                                              Panton-Valentine leukocidin (PVL), encoded by the lukSF-
88                              A single toxin, Panton-Valentine leukocidin (PVL), has been linked by ep
89      Exchangeable virulence factors, such as Panton-Valentine leukocidin (PVL), have been proposed to
90 are pore-forming cytolytic toxins, including Panton-Valentine leukocidin (PVL), leukotoxin GH (LukGH;
91 some mec type IV [SCCmecIV]) and carried the Panton-Valentine leukocidin (pvl), lukD, and lukE genes,
92 essively acquired the USA300 characteristics Panton-Valentine leukocidin (PVL), SCCmec IVa, the argin
93 filed gel electrophoresis (PFGE) and PCR for Panton-Valentine leukocidin (PVL), the arginine cataboli
94 resence of genes mecA and mupA and those for Panton-Valentine leukocidin (PVL), USA300, and USA400.
95 on of the lukF-PV gene, encoding part of the Panton-Valentine leukocidin (PVL), was observed in the c
96 n in large part by alpha-hemolysin (Hla) and Panton-Valentine leukocidin (PVL), we evaluated whether
97 tis caused by CA-MRSA LAC(WT) USA300 and its Panton-Valentine leukocidin (PVL)- and alpha-hemolysin (
98  of methicillin resistance, SCCmec type, and Panton-Valentine leukocidin (PVL)-producing genes on an
99 cluding alpha toxin, secreted proteases, and Panton-Valentine leukocidin (PVL).
100 ciated with strains harboring genes encoding Panton-Valentine leukocidin (PVL).
101 ing agr, saeRS, sarA, alpha-toxin (hla), and Panton-Valentine leukocidin (pvl).
102 ec but lacked genes for the virulence factor Panton-Valentine leukocidin (PVL).
103 ococcal exotoxins, alpha-hemolysin (Hla) and Panton-Valentine leukocidin (PVL; LukF-PV/LukS-PV subuni
104 phoresis clonal type, toxin genes (e.g., for Panton-Valentine leukocidin [PVL]), and staphylococcal c
105 cilitates hemolysis, but the closely related Panton-Valentine leukocidin S (LukS-PV) does not bind to
106 occal cassette chromosome mec (SCCmec) type, Panton-Valentine leukocidin status, and multilocus seque
107 reus produce toxins such as leukocidins (eg, Panton-Valentine leukocidin, toxic shock syndrome toxin
108 assays were used to detect mecA, mecC, vanA, Panton-Valentine Leukocidin toxin (PVL), and toxic shock
109                       SCCmec type IV and the Panton-Valentine leukocidin toxin gene were detected in
110    All USA300 isolates were positive for the Panton-Valentine leukocidin toxin genes and agr I.
111  staphylococcal cassette chromosome mec, and Panton-Valentine leukocidin typing.
112 maximally expressed 4 h after infection, and Panton-Valentine leukocidin was maximally expressed 72 h
113                        The bicomponent toxin Panton-Valentine leukocidin was not produced during intr
114 ce genes encoding secreted toxins, including Panton-Valentine leukocidin, were highly expressed durin
115 xin; however, carriage of the genes encoding Panton-Valentine leukocidin, while common among MRSA of

 
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