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1 ortunity to study the immune response during Plasmodium falciparum infection.
2 regarding fulminant disease associated with Plasmodium falciparum infection.
3 group affects host susceptibility to severe Plasmodium falciparum infection.
4 oimmun; no association was found with active Plasmodium falciparum infection.
5 erebral malaria (CM) is the severest form of Plasmodium falciparum infection.
6 inks between kidney and brain dysfunction in Plasmodium falciparum infection.
7 roduction by CD4+ T cells during blood-stage Plasmodium falciparum infection.
8 atest risk for severe disease resulting from Plasmodium falciparum infection.
9 vivo efficacy in a humanized mouse model of Plasmodium falciparum infection.
10 ral malaria (CM) is a severe complication of Plasmodium falciparum infection.
11 gulated in vaccinees who were protected from Plasmodium falciparum infection.
12 l malaria (HCM) is a serious complication of Plasmodium falciparum infection.
13 which targets the pre-erythrocytic stage of Plasmodium falciparum infection.
14 is a severe and often fatal complication of Plasmodium falciparum infection.
15 production were found in women with current Plasmodium falciparum infection.
16 antibodies associated with the prevention of Plasmodium falciparum infection.
17 tion is central to the pathogenesis of acute Plasmodium falciparum infection.
18 are important in the control of blood stage Plasmodium falciparum infection.
19 is a common and often fatal complication of Plasmodium falciparum infection.
20 th infections may increase susceptibility to Plasmodium falciparum infection.
21 al attention is being paid to submicroscopic Plasmodium falciparum infections.
22 nancy, mainly with respect to submicroscopic Plasmodium falciparum infections.
23 ive US adults, potentially due to concurrent Plasmodium falciparum infections.
24 h genomic regions to characterize polyclonal Plasmodium falciparum infections.
25 h genomic regions to characterize polyclonal Plasmodium falciparum infections.
26 c, detection measures the real prevalence of Plasmodium falciparum infections.
27 chieving parasite densities similar to fatal Plasmodium falciparum infections.
28 malaria (CM) is a leading cause of death in Plasmodium falciparum infections.
29 s were more likely to be submicroscopic than Plasmodium falciparum infections (3.69, 2.45-5.54; p<0.0
30 ter the report of three cases of complicated Plasmodium falciparum infection acquired by transfusion,
33 l malaria is the most severe complication of Plasmodium falciparum infection and accounts for a large
34 ys leading to pathology and morbidity during Plasmodium falciparum infection and has been termed a ma
35 l malaria is a common presentation of severe Plasmodium falciparum infection and remains an important
36 nd without inflammation and with and without Plasmodium falciparum infection and to assess the effect
37 Ap) represents a potential strategy to clear Plasmodium falciparum infections and reduce the human pa
39 um discoideum development and sexual cycles, Plasmodium falciparum infection, and the nervous systems
40 from an Antarctic lake, plastids from mixed Plasmodium falciparum infections, and virulent Neisseria
44 n highly endemic regions, where asymptomatic Plasmodium falciparum infections are present across all
46 rile controls revealed spatial clustering of Plasmodium falciparum infections around patients with ma
47 e was high; 70% (n = 1,138) had PCR-positive Plasmodium falciparum infection at least once over the c
48 y seen an increase in the number of reported Plasmodium falciparum infections believed to be imported
49 ceptibility of the mosquito to bacterial and Plasmodium falciparum infection, but not to infection wi
50 ral malaria (CM) is a deadly complication of Plasmodium falciparum infection, but specific interactio
51 e Dantu blood group variant protects against Plasmodium falciparum infections, but its wider conseque
52 jor malaria vector Anopheles gambiae promote Plasmodium falciparum infection by maintaining midgut ep
53 parous women were tested for first-trimester Plasmodium falciparum infection by quantitative polymera
57 l malaria (CM) is a life-threatening form of Plasmodium falciparum infection caused by brain inflamma
73 The effect of timing of exposure to first Plasmodium falciparum infections during early childhood
75 etocytes were quantified in 161 PCR-positive Plasmodium falciparum infections from a cross-sectional
77 ccine development against the blood-stage of Plasmodium falciparum infection has focused on recombina
78 apacity of antibodies induced during natural Plasmodium falciparum infection has not been evaluated.
79 Adoption of molecular techniques to detect Plasmodium falciparum infection has revealed many previo
80 of neuroimaging techniques to patients with Plasmodium falciparum infection has uncovered a wide ran
81 The widespread use of chloroquine to treat Plasmodium falciparum infections has resulted in the sel
82 chloroquine (CQ) as first-line treatment of Plasmodium falciparum infections has typically led to th
83 CM), a severe encephalopathy associated with Plasmodium falciparum infection, has a 20-30% mortality
84 udy found that severely anemic children with Plasmodium falciparum infection have low plasma TNF leve
85 nonymous single-nucleotide polymorphisms and Plasmodium falciparum infection identified three natural
87 that was shown to protect against controlled Plasmodium falciparum infection in a phase 1 clinical tr
90 most common life-threatening complication of Plasmodium falciparum infection in African children.
91 REP1) gene are significantly associated with Plasmodium falciparum infection in Anopheles gambiae, an
93 obium was associated with protection against Plasmodium falciparum infection in children who were 4 t
94 describe a mechanistic within-host model of Plasmodium falciparum infection in humans and pathogen t
97 previously identified to be associated with Plasmodium falciparum infection in natural Anopheles gam
101 relationship between placental pathology and Plasmodium falciparum infection in the placenta with PE
102 emiology of malaria, we intensively followed Plasmodium falciparum infections in a cohort in a malari
103 c test and microscopy - for the detection of Plasmodium falciparum infections in endemic populations
104 xist on the incidence or duration of natural Plasmodium falciparum infections in high-transmission se
105 a prevention primarily address prevention of Plasmodium falciparum infections in short-term travelers
108 acteristics of clinical illness accompanying Plasmodium falciparum infection induced by controlled ex
111 susceptibility to febrile malaria following Plasmodium falciparum infection is associated with the c
114 Severe malarial anemia (SMA) resulting from Plasmodium falciparum infection is one of the leading ca
120 aria (CM) is a severe complication caused by Plasmodium falciparum infection, leading to persistent n
121 bpopulation of individuals with asymptomatic Plasmodium falciparum infection may play a significant r
122 SMA (n = 17) and community children without Plasmodium falciparum infection (n = 12) and determined
123 fitted to experimental data to describe how Plasmodium falciparum infection of the African malaria v
126 bral malaria (CM) is a major complication of Plasmodium falciparum infection, particularly in childre
127 33%), 172 (20%), 333 (43%), and 17 (2%) with Plasmodium falciparum infection, Plasmodium vivax infect
133 How antibodies naturally acquired during Plasmodium falciparum infection provide clinical immunit
136 Factors governing the clinical trajectory of Plasmodium falciparum infection remain an important area
139 ral malaria (CM) is a severe complication of Plasmodium falciparum infection responsible for thousand
141 ia (CM), a severe and fatal form of clinical Plasmodium falciparum infection, results from a damaging
144 ircumsporozoite protein to determinate their Plasmodium falciparum infection status and assessed the
145 eted antihelminth therapy and its effects on Plasmodium falciparum infection status, hemoglobin level
146 t sickle-cell heterozygotes have less severe Plasmodium falciparum infections than do children with n
147 ew axis of mosquito resistance to monoclonal Plasmodium falciparum infections that includes the AP-1
150 We therefore studied spatial distribution of Plasmodium falciparum infections to compare simulated ef
154 homozygous primiparae, the odds of placental Plasmodium falciparum infection were reduced by 64%.
155 estigated how new, recurrent, and persistent Plasmodium falciparum infections were associated with th
156 to evaluate the epidemiology and control of Plasmodium falciparum infections, where the parasite, ra
157 ebral malaria is a dangerous complication of Plasmodium falciparum infection, which takes a devastati
158 udy involved 226 patients with uncomplicated Plasmodium falciparum infection who had successfully com
159 ia is a severe neurovascular complication of Plasmodium falciparum infection, with high mortality rat
160 herapies are the first line of treatment for Plasmodium falciparum infections worldwide, but artemisi
161 mechanisms of naturally acquired immunity to Plasmodium falciparum infections would be highly valuabl