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1 cus, Finegoldia, Micrococcus, Prevotella and Propionibacterium).
2 ptures skin commensal microbiota, especially Propionibacterium.
5 (10) CFU/g), Bifidobacterium (1 x 10(10)/g), Propionibacterium (3 x 10(10)/g), Acetobacter (1 x 10(6)
6 acteria Staphylococcus, Corynebacterium, and Propionibacterium (6.5, 5.7, 5.4 log10 copies/100 mL, re
7 10.5%), Olsenella (9.4%), Prevotella (8.8%), Propionibacterium (7.2%), Streptococcus (3.9%), Selenomo
8 investigated the acid tolerance mechanism of Propionibacterium acidipropionici at microenvironmental
9 46% vs 80%; 3) the most frequent isolate was Propionibacterium acnes (11/26) vs coagulase-negative St
10 1), Propionibacterium species (n = 15), and Propionibacterium acnes (n = 19) isolates; all of these
11 S (20 microg/mouse) 7 days after heat-killed Propionibacterium acnes (P. acnes) injection into wild-t
12 monstrate that fermentation of glycerol with Propionibacterium acnes (P. acnes), a skin commensal bac
13 ne treatment as a natural antibiotic against Propionibacterium acnes (P. acnes), which promotes folli
14 e investigated the roles of TLR2 and TLR4 in Propionibacterium acnes (P. acnes)-primed, LPS-induced l
17 In this issue, Kistowska et al. confirm that Propionibacterium acnes activates inflammasomes leading
18 asts, 6 nonfermenters, and 1 isolate each of Propionibacterium acnes and coagulase-negative staphyloc
20 olleagues present strain-based resolution of Propionibacterium acnes and its association with the com
21 the presence of the Gram-positive bacterium Propionibacterium acnes and its potential association wi
24 s associated with formation of resistance in Propionibacterium acnes and other bacteria, with clinica
25 ndependent growth, requiring helpers such as Propionibacterium acnes and Prevotella intermedia for st
26 on in common skin commensal bacteria such as Propionibacterium acnes and Staphylococcus epidermitis.
29 ites from the abundant skin-resident microbe Propionibacterium acnes can influence cytokine expressio
31 resulting from the immune response targeting Propionibacterium acnes has a significant role in its pa
34 ase-negative staphylococci in 6.0% (27/448), Propionibacterium acnes in 4.7% (21/448), and Pseudomona
51 stigated the in vitro susceptibilities of 23 Propionibacterium acnes ophthalmic isolates to ertapenem
54 overed, including Escherichia coli phage T3, Propionibacterium acnes phage PA6, and Streptococcus mit
55 on of early childhood acne, the emergence of Propionibacterium acnes resistance, and the rare but ser
56 observed the decline of an early-colonizing Propionibacterium acnes strain similar to SK137 and the
57 ny countries reporting that more than 50% of Propionibacterium acnes strains are resistant to topical
58 es of the human skin microbiome suggest that Propionibacterium acnes strains may contribute different
61 s-wide comparative analysis of 90 genomes of Propionibacterium acnes that represent the known diversi
69 iome at the strain level and genome level of Propionibacterium acnes, a dominant skin commensal, betw
70 mechanism by which S. aureus may commandeer Propionibacterium acnes, a key member of the human skin
72 Bradyrhizobium, Anaerococcus, Peptoniphilus, Propionibacterium acnes, Dorea, and Ruminococcus and red
73 rial pathogens including Bacillus anthracis, Propionibacterium acnes, Enterococcus faecalis, and both
75 e primed 12 days in advance with heat-killed Propionibacterium acnes, IL-18BP-Fc prevents LPS-induced
77 teen of the species or phylotypes, including Propionibacterium acnes, Staphylococcus spp., and the op
78 rect antimicrobial activity in vitro against Propionibacterium acnes, the bacterium linked to the pat
79 ct to sequence multiple clinical isolates of Propionibacterium acnes, we have produced a draft genome
80 teria such as Staphylococcus epidermidis and Propionibacterium acnes, were identified in bacteriologi
81 ulture showed antimicrobial activity against Propionibacterium acnes, whereas the enhanced antimicrob
82 ica charantia Linn. var. abbreviata Ser.) on Propionibacterium acnes-induced inflammation and to iden
83 us studies showed that TLR9 and TLR2 mediate Propionibacterium acnes-induced sensitization to lipopol
90 t contributes to the pathogenesis of acne is Propionibacterium acnes; yet, the molecular mechanism by
91 and four different Cutibacterium (previously Propionibacterium) acnes strains, and compare outcomes w
92 ilar to SK137 and the proliferation of novel Propionibacterium and Peptoniphilus species late in colo
94 e of the genera Enterococcus, Lactobacillus, Propionibacterium, and Streptococcus and two clones were
95 r proportions of Shinella, Terribacillus and Propionibacterium, and these genera are known to have to
96 cus, Finegoldia, Micrococcus, Prevotella and Propionibacterium at all time points, suggesting their p
97 loss of skin commensals (Corynebacterium and Propionibacterium) at the surgical site, which were repl
98 loss of skin commensals (Corynebacterium and Propionibacterium) at the surgical site, which were repl
100 rder Corynebacterineae; revisions within the Propionibacterium, Clostridium, Borrelia, and Enterobact
101 well as bacterial genus-specific probes for Propionibacterium, Corynebacterium, Streptococcus, and S
104 identity to PP(i)-PFKs from two eubacteria, Propionibacterium freudenreichii and Sinorhizobium melil
107 acillus casei, Levilactobacillus brevis, and Propionibacterium freudenreichii resulted in the product
108 human active vitamin B12 in cell extracts of Propionibacterium freudenreichii subsp. shermanii and af
112 s developed as a useful tool for engineering Propionibacterium jensenii, and two key enzymes-glycerol
113 healthy controls vs HS skin, indicating that Propionibacterium may be part of the pathogenesis in HS.
115 gibacterium, Peptostreptococcus, Prevotella, Propionibacterium, Selenomonas, Solobacterium, Streptoco
117 tetradehydrocorrin, factor IV, isolated from Propionibacterium shermanii has been established by mult
119 million Dalton (Da) multienzyme complex from Propionibacterium shermanii that couples two carboxylati
121 in-accepting domain from the 1.3S subunit of Propionibacterium shermanii transcarboxylase (PSBT) is t
122 hift by fusing a compact protein domain, the Propionibacterium shermanii transcarboxylase domain (PST
129 cluded Bacteroides thetaiotaomicron (n = 1), Propionibacterium species (n = 15), and Propionibacteriu
131 uded multiple Actinomyces species in wounds, Propionibacterium species in joints and cerebrospinal fl
132 produced a draft genome sequence of a novel Propionibacterium species that is closely related to, ye
134 phaga species, six Actinomyces species, four Propionibacterium species, and eight Streptococcus speci
138 ertinent to former designations of or within Propionibacterium spp., Corynebacterium spp., Clostridiu
140 ptostreptococcus, Porphyromonas, Provetella, Propionibacterium, Tisierella, and Veillonella and 36 of
141 media/Prevotella nigrescens, Capnocytophaga, Propionibacterium, yeasts, Actinobacillus actinomycetemc