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1 cus, Finegoldia, Micrococcus, Prevotella and Propionibacterium).
2 ptures skin commensal microbiota, especially Propionibacterium.
3 scherichia coli (3), Candida tropicalis (1), Propionibacterium (1), and Rothia (1).
4 Mobiluncus (8), Peptostreptococcus (16), and Propionibacterium (24).
5 (10) CFU/g), Bifidobacterium (1 x 10(10)/g), Propionibacterium (3 x 10(10)/g), Acetobacter (1 x 10(6)
6 acteria Staphylococcus, Corynebacterium, and Propionibacterium (6.5, 5.7, 5.4 log10 copies/100 mL, re
7 10.5%), Olsenella (9.4%), Prevotella (8.8%), Propionibacterium (7.2%), Streptococcus (3.9%), Selenomo
8 investigated the acid tolerance mechanism of Propionibacterium acidipropionici at microenvironmental
9 46% vs 80%; 3) the most frequent isolate was Propionibacterium acnes (11/26) vs coagulase-negative St
10  1), Propionibacterium species (n = 15), and Propionibacterium acnes (n = 19) isolates; all of these
11 S (20 microg/mouse) 7 days after heat-killed Propionibacterium acnes (P. acnes) injection into wild-t
12 monstrate that fermentation of glycerol with Propionibacterium acnes (P. acnes), a skin commensal bac
13 ne treatment as a natural antibiotic against Propionibacterium acnes (P. acnes), which promotes folli
14 e investigated the roles of TLR2 and TLR4 in Propionibacterium acnes (P. acnes)-primed, LPS-induced l
15 nd cDC2s during bacterial infection, notably Propionibacterium acnes (P. acnes).
16 nas and Peptoniphilus species (type IV), and Propionibacterium acnes (type V).
17 In this issue, Kistowska et al. confirm that Propionibacterium acnes activates inflammasomes leading
18 asts, 6 nonfermenters, and 1 isolate each of Propionibacterium acnes and coagulase-negative staphyloc
19                                              Propionibacterium acnes and coagulase-negative Staphyloc
20 olleagues present strain-based resolution of Propionibacterium acnes and its association with the com
21  the presence of the Gram-positive bacterium Propionibacterium acnes and its potential association wi
22                      In vivo Th1 response to Propionibacterium acnes and lipopolysaccharide in IFN-ga
23 nished in the sera of ICE-/- mice exposed to Propionibacterium acnes and lipopolysaccharide.
24 s associated with formation of resistance in Propionibacterium acnes and other bacteria, with clinica
25 ndependent growth, requiring helpers such as Propionibacterium acnes and Prevotella intermedia for st
26 on in common skin commensal bacteria such as Propionibacterium acnes and Staphylococcus epidermitis.
27              Low virulence organisms such as Propionibacterium acnes are the most common culprit orga
28                     We report a patient with Propionibacterium acnes bacteremia and late prosthetic v
29 ites from the abundant skin-resident microbe Propionibacterium acnes can influence cytokine expressio
30 n are compounded by increasing resistance of Propionibacterium acnes clinical isolates.
31 resulting from the immune response targeting Propionibacterium acnes has a significant role in its pa
32                                              Propionibacterium acnes has been identified as a signifi
33                 The microaerophylic organism Propionibacterium acnes has shown consistent association
34 ase-negative staphylococci in 6.0% (27/448), Propionibacterium acnes in 4.7% (21/448), and Pseudomona
35                      Agak et al. report that Propionibacterium acnes induces IL-17 expression in peri
36                                              Propionibacterium acnes induction of IL-1 cytokines thro
37                                              Propionibacterium acnes induction of inflammatory respon
38  have been sensitized to endotoxin damage by Propionibacterium acnes infection.
39                                              Propionibacterium acnes is a critical component in the p
40                                              Propionibacterium acnes is a key pathogen involved in th
41                                              Propionibacterium acnes is a key therapeutic target in a
42                                              Propionibacterium acnes is a known cause of postneurosur
43                                              Propionibacterium acnes is a major etiological factor of
44                  The Gram-positive bacterium Propionibacterium acnes is a member of the normal human
45                                              Propionibacterium acnes is a skin commensal bacterium th
46                                              Propionibacterium acnes is a well-known cause of delayed
47                                              Propionibacterium acnes is also associated with inflamma
48             The opportunistic human pathogen Propionibacterium acnes is composed of a number of disti
49                                              Propionibacterium acnes is increasingly recognized as an
50                                              Propionibacterium acnes isolates usually have relatively
51 stigated the in vitro susceptibilities of 23 Propionibacterium acnes ophthalmic isolates to ertapenem
52     Mycothiol production was not detected in Propionibacterium acnes or in representative species of
53  relative abundances of propionibacteria and Propionibacterium acnes phage in healthy skin.
54 overed, including Escherichia coli phage T3, Propionibacterium acnes phage PA6, and Streptococcus mit
55 on of early childhood acne, the emergence of Propionibacterium acnes resistance, and the rare but ser
56  observed the decline of an early-colonizing Propionibacterium acnes strain similar to SK137 and the
57 ny countries reporting that more than 50% of Propionibacterium acnes strains are resistant to topical
58 es of the human skin microbiome suggest that Propionibacterium acnes strains may contribute different
59  Prevotella oris, Staphylococcus aureus, and Propionibacterium acnes strains.
60 ddress the emergence of antibiotic-resistant Propionibacterium acnes strains.
61 s-wide comparative analysis of 90 genomes of Propionibacterium acnes that represent the known diversi
62 nflammation is due in part to the ability of Propionibacterium acnes to activate TLR2.
63                    C57BL/6 mice treated with Propionibacterium acnes to elicit high levels of macroph
64 ntial evidence that links the skin bacterium Propionibacterium acnes to the condition.
65       The culture supernatant of a strain of Propionibacterium acnes was investigated for its phospho
66                                              Propionibacterium acnes was the commonest species detect
67 cular hyperkeratinization, and the action of Propionibacterium acnes within the follicle.
68                Cutibacterium acnes (formerly Propionibacterium acnes) is recognized as a pathogen in
69 iome at the strain level and genome level of Propionibacterium acnes, a dominant skin commensal, betw
70  mechanism by which S. aureus may commandeer Propionibacterium acnes, a key member of the human skin
71                                              Propionibacterium acnes, causative agent of chronic pros
72 Bradyrhizobium, Anaerococcus, Peptoniphilus, Propionibacterium acnes, Dorea, and Ruminococcus and red
73 rial pathogens including Bacillus anthracis, Propionibacterium acnes, Enterococcus faecalis, and both
74           Among the airborne microorganisms, Propionibacterium acnes, Escherichia coli, Acinetobacter
75 e primed 12 days in advance with heat-killed Propionibacterium acnes, IL-18BP-Fc prevents LPS-induced
76      In patients with AgP, Actinomyces oris, Propionibacterium acnes, P. aeruginosa, Staphylococcus a
77 teen of the species or phylotypes, including Propionibacterium acnes, Staphylococcus spp., and the op
78 rect antimicrobial activity in vitro against Propionibacterium acnes, the bacterium linked to the pat
79 ct to sequence multiple clinical isolates of Propionibacterium acnes, we have produced a draft genome
80 teria such as Staphylococcus epidermidis and Propionibacterium acnes, were identified in bacteriologi
81 ulture showed antimicrobial activity against Propionibacterium acnes, whereas the enhanced antimicrob
82 ica charantia Linn. var. abbreviata Ser.) on Propionibacterium acnes-induced inflammation and to iden
83 us studies showed that TLR9 and TLR2 mediate Propionibacterium acnes-induced sensitization to lipopol
84 ion of 2.0 micrograms per milliliter against Propionibacterium acnes.
85 against Escherichia coli., and 12 mm against Propionibacterium acnes.
86 licles on the face, neck, chest, and back by Propionibacterium acnes.
87 e immune response to the commensal bacterium Propionibacterium acnes.
88 antimicrobial activity against S. aureus and Propionibacterium acnes.
89 es, is caused by multiple factors, including Propionibacterium acnes.
90 t contributes to the pathogenesis of acne is Propionibacterium acnes; yet, the molecular mechanism by
91 and four different Cutibacterium (previously Propionibacterium) acnes strains, and compare outcomes w
92 ilar to SK137 and the proliferation of novel Propionibacterium and Peptoniphilus species late in colo
93                  Increases in Streptococcus, Propionibacterium, and Corynebacterium species were obse
94 e of the genera Enterococcus, Lactobacillus, Propionibacterium, and Streptococcus and two clones were
95 r proportions of Shinella, Terribacillus and Propionibacterium, and these genera are known to have to
96 cus, Finegoldia, Micrococcus, Prevotella and Propionibacterium at all time points, suggesting their p
97 loss of skin commensals (Corynebacterium and Propionibacterium) at the surgical site, which were repl
98 loss of skin commensals (Corynebacterium and Propionibacterium) at the surgical site, which were repl
99           Regardless, 12 genera-Pseudomonas, Propionibacterium, Bradyrhizobium, Corynebacterium, Acin
100 rder Corynebacterineae; revisions within the Propionibacterium, Clostridium, Borrelia, and Enterobact
101  well as bacterial genus-specific probes for Propionibacterium, Corynebacterium, Streptococcus, and S
102  with aeroallergen sensitization compared to Propionibacterium dominance (OR: 4.48, FDR = 0.03).
103                                              Propionibacterium FMA5 was significantly associated with
104  identity to PP(i)-PFKs from two eubacteria, Propionibacterium freudenreichii and Sinorhizobium melil
105          Co-expression of the cobA gene from Propionibacterium freudenreichii and the cbiA, -C, -D, -
106                  The well-studied PPi-PFK of Propionibacterium freudenreichii is highly divergent and
107 acillus casei, Levilactobacillus brevis, and Propionibacterium freudenreichii resulted in the product
108 human active vitamin B12 in cell extracts of Propionibacterium freudenreichii subsp. shermanii and af
109 through solid-state fermentation (SSF) using Propionibacterium freudenreichii.
110   We have tentatively named this new species Propionibacterium humerusii.
111 dobacterium animalis subsp. lactis BB-12 and Propionibacterium jensenii 702.
112 s developed as a useful tool for engineering Propionibacterium jensenii, and two key enzymes-glycerol
113 healthy controls vs HS skin, indicating that Propionibacterium may be part of the pathogenesis in HS.
114 reported that the newly discovered commensal Propionibacterium, P.
115 gibacterium, Peptostreptococcus, Prevotella, Propionibacterium, Selenomonas, Solobacterium, Streptoco
116                Both activities purified from Propionibacterium shermanii are the functional propertie
117 tetradehydrocorrin, factor IV, isolated from Propionibacterium shermanii has been established by mult
118                        Transcarboxylase from Propionibacterium shermanii is a 1.2 MDa multienzyme com
119 million Dalton (Da) multienzyme complex from Propionibacterium shermanii that couples two carboxylati
120                   A 75-residue sequence from Propionibacterium shermanii that is biotinylated in mamm
121 in-accepting domain from the 1.3S subunit of Propionibacterium shermanii transcarboxylase (PSBT) is t
122 hift by fusing a compact protein domain, the Propionibacterium shermanii transcarboxylase domain (PST
123                   Transcarboxylase (TC) from Propionibacterium shermanii, a biotin-dependent enzyme,
124 .7.1.90) which was purified from extracts of Propionibacterium shermanii.
125 by the action of the biosynthetic enzymes of Propionibacterium shermanii.
126                      Several taxa, including Propionibacterium, showed a significantly higher relativ
127                             The isolation of Propionibacterium sp. from broth only usually represente
128                          To detect anaerobic Propionibacterium sp., a well-described cause of these i
129 cluded Bacteroides thetaiotaomicron (n = 1), Propionibacterium species (n = 15), and Propionibacteriu
130                                          The Propionibacterium species differed in their ability to m
131 uded multiple Actinomyces species in wounds, Propionibacterium species in joints and cerebrospinal fl
132  produced a draft genome sequence of a novel Propionibacterium species that is closely related to, ye
133 ative Staphylococcus, Candida, Fusarium, and Propionibacterium species).
134 phaga species, six Actinomyces species, four Propionibacterium species, and eight Streptococcus speci
135                                              Propionibacterium species, coagulase-negative staphyloco
136 e gut microbiota, particularly enrichment of Propionibacterium species.
137 9 (51%) of these bottles grew Cutibacterium (Propionibacterium) species.
138 ertinent to former designations of or within Propionibacterium spp., Corynebacterium spp., Clostridiu
139 m infants or a newly identified and cultured Propionibacterium strain, P.
140 ptostreptococcus, Porphyromonas, Provetella, Propionibacterium, Tisierella, and Veillonella and 36 of
141 media/Prevotella nigrescens, Capnocytophaga, Propionibacterium, yeasts, Actinobacillus actinomycetemc

 
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