ライフサイエンスコーパス: Proteus

1 completely sequenced plasmid from the genus Proteus.

2 multicellular migration, most strikingly in Proteus.

3 alvei, Hafnia paralvei, and Obesumbacterium proteus.

4 ates were formed mainly with Pseudomonas and Proteus.

5 n the Prostate Cancer and Environment Study (PROtEuS), a population-based case-control study conducte

6 We have developed Proteus, a web-based, context-specific tool for building

7 up of bacteria, including species of Vibrio, Proteus and Caulobacter that use the flagellum as a surf

8 letely understood, is far more palpable than Proteus and is (in most cases) much more readily subdued

9 Measurement sensitivity for E.coli, Proteus and Klebsiella was high, with concentrations of

10 ification systems, the flanking sequences in Proteus and Salmonella are completely different.

11 . coli and B. subtilis colonies, swarming by Proteus and Serratia, and spatially organized interspeci

12 on-UTI bacteria, Staphylococcus, Klebsiella, Proteus and Shigella.

13 fied TonB homologs in Shigella, Citrobacter, Proteus, and Kluyvera species.

14 Europe's obligate cave-dwelling amphibian Proteus anguinus inhabits subterranean waters of the nor

15 Toolkits in Proteus are context-independent representations of biolo

16 acteria), present in the xD strain of Amoeba proteus as required cell components, synthesize and expo

17 n range, we established a likely presence of Proteus at seven new sites, extending its range to Monte

18 sium ammonium phosphate), with the crystals, Proteus bacteria, and extracellular DNA forming mixed bi

19 PICE (Serratia, Pseudomonas, indole-positive Proteus, Citrobacter, and Enterobacter) has served as a

20 Proteus combines these choices into a system of ODEs, wh

21 i-HER2 (225)Ac-PRIT (37 kBq/cycle as (225)Ac-Proteus DOTA), separated by a 1-wk interval.

22 n one of these we found both black and white Proteus eDNA together.

23 Proteus FlgN has leucine zipper-like motifs arranged on

24 Proteus flgN is arranged in an operon with the class III

25 obes to discriminate the rare black morph of Proteus from the closely related white morph, we detecte

26 vskite materials through the screening robot PROTEUS has emerged as a powerful tool in the search for

27 Proteus has migrated outwards because of tidal interacti

28 t of these two cytotoxins is critical during Proteus infection.

29 -bp PCR product hybridized strongly with all Proteus isolates (n = 9) and 25% of 355 Escherichia coli

30 The expression of mannose-resistant/Proteus-like (MR/P) fimbria is phase variable because of

31 The mannose-resistant, Proteus-like (MR/P) fimbria, responsible for mannose-res

32 The mannose-resistant, Proteus-like (MR/P) fimbriae and flagellum are among vir

33 ene cluster, which encodes mannose-resistant Proteus-like (MR/P) fimbriae of Proteus mirabilis, indic

34 mrp gene cluster encoding mannose-resistant Proteus-like (MR/P) fimbriae of uropathogenic Proteus mi

35 ract infections, expresses mannose-resistant Proteus-like (MR/P) fimbriae whose expression is phase v

36 lled bacteria or purified mannose-resistant, Proteus-like (MR/P) fimbriae, a surface antigen expresse

37 pregulated in vivo encoded mannose-resistant Proteus-like (MR/P) fimbriae, urease, iron uptake system

38 tract infection, expresses mannose-resistant/Proteus-like (MR/P) fimbriae.

39 e were examined for PTEN mutations, only the Proteus-like patient was found to harbour a germline R33

40 Thus, PTEN may be involved in Proteus-like syndrome with its implications for cancer d

41 ley-Ruvalcaba syndrome, Proteus syndrome and Proteus-like syndrome, collectively classified as PTEN h

42 ey-Ruvalcaba syndrome, Proteus syndrome, and Proteus-like syndrome.

43 Five had Proteus syndrome and one, a Proteus-like syndrome.

44 as broadened to include Proteus syndrome and Proteus-like syndromes.

45 (4.4%), Stenotrophomonas maltophilia (4.3%), Proteus mirabilis (4.0%), Klebsiella oxytoca (2.7%), and

46 lla oxytoca (3), Klebsiella pneumoniae (29), Proteus mirabilis (9), Pseudomonas aeruginosa (10), and

47 ae (n = 4), Pseudomonas aeruginosa (n = 14), Proteus mirabilis (n = 3), Serratia spp. (n = 10), Steno

48 ifferentiation of Klebsiella pneumoniae from Proteus mirabilis 16S rRNA target sequences differing by

49 Proteus mirabilis alternates between motile and adherent

50 have shown previously using hyperflagellated Proteus mirabilis and a motile but non-swarming flgN tra

51 significant, especially against the bacteria Proteus mirabilis and Antibiotic resistant Escherichia c

52 treptococcus pyogenes, Streptococcus mutans, Proteus mirabilis and Candida albicans.

53 of Gram-negative bacterial cells, including Proteus mirabilis and Caulobacter crescentus, initiates

54 We focused on Lrp from Proteus mirabilis and E. coli, orthologs with 98% identi

55 The closely related Proteus mirabilis and Enterobacterlaceae plasmid-encoded

56 nodes in these mice isolated K. pneumoniae, Proteus mirabilis and Enterococcus gallinarum, which wer

57 of a urease-negative mutant of uropathogenic Proteus mirabilis and its wild-type parent strain was as

58 during UTI caused by the major uropathogens Proteus mirabilis and Klebsiella pneumoniae, in addition

59 operon as a major assimilatory checkpoint in Proteus mirabilis and other Gram-negative bacteria and e

60 The Proteus mirabilis and plasmid-encoded urease loci contai

61 The urease-positive species Proteus mirabilis and Providencia stuartii are two of th

62 zation by urease-positive organisms, such as Proteus mirabilis and Providencia stuartii, commonly occ

63 lebsiella oxytoca, Klebsiella pneumoniae, or Proteus mirabilis and receiving PDAT on blood culture co

64 s II promoter sequences of Escherichia coli, Proteus mirabilis and Salmonella typhimurium allowed det

65 cherichia coli, Pseudomonas aeruginosa PAO1, Proteus mirabilis and Serratia marcescens, possibly by i

66 segregate from other human pathogens such as Proteus mirabilis and Staphylococcus aureus that outcomp

67 ted urinary tract infections (UTI) caused by Proteus mirabilis are associated with severe pathology i

68 Fimbriae of the human uropathogen Proteus mirabilis are the only characterized surface pro

69 Using Proteus mirabilis as a model, we investigate the role of

70 ol for application of the mini-Tn7 system in Proteus mirabilis as an example of a bacterium with a se

71 lebsiella pneumoniae, Klebsiella oxytoca, or Proteus mirabilis at >=50 000 colony-forming units/mL; (

72 coli, Klebsiella pneumoniae, K. oxytoca, or Proteus mirabilis at >=50,000 CFU/mL, (2) identification

73 One of the six predicted Proteus mirabilis autotransporters (ATs), ORF c2341, is

74 onstructed from the 50 %-identical TEM-1 and Proteus mirabilis beta-lactamases.

75 become encrusted and blocked by crystalline Proteus mirabilis biofilms.

76 lebsiella pneumoniae, Klebsiella oxytoca, or Proteus mirabilis BSI from a urinary source from May 201

77 Klebsiella pneumoniae, Escherichia coli, and Proteus mirabilis by acidifying the proximal colon and t

78 Proteus mirabilis can rapidly utilize choline to enhance

79 Proteus mirabilis causes complicated urinary tract infec

80 fundamental behaviors of motile, rod-shaped Proteus mirabilis cells (3 mum in length) adsorbed to th

81 unds on the inhibition of ureolysis in whole Proteus mirabilis cells showed a very good potency (IC(5

82 A total of 63 clinical isolates of Proteus mirabilis collected over a 19-month period were

83 Proteus mirabilis colonies exhibit striking geometric re

84 Proteus mirabilis commonly infects the complicated urina

85 Proteus mirabilis compromises the care of many patients

86 Proteus mirabilis forms extensive crystalline biofilms o

87 Proteus mirabilis forms extensive crystalline biofilms o

88 Klebsiella pneumoniae, Escherichia coli, and Proteus mirabilis from the intestinal lumen and represen

89 In this study, we describe wosA, a Proteus mirabilis gene identified by its ability to incr

90 In a search for Proteus mirabilis genes that were regulated by cell-to-c

91 rming motility by the urinary tract pathogen Proteus mirabilis has been a long-studied but little und

92 -ray crystal structure of a Crl homolog from Proteus mirabilis in conjunction with in vivo and in vit

93 , Seo et al. (2015) show that the pathobiont Proteus mirabilis induces NLRP3 inflammasome-dependent i

94 hat provides a clear visual early warning of Proteus mirabilis infection and subsequent blockage.

95 Proteus mirabilis is a biofilm-forming, multidrug-resist

96 The enteric bacterium Proteus mirabilis is a common cause of complicated urina

97 Proteus mirabilis is a common cause of urinary tract inf

98 Proteus mirabilis is a common uropathogen in patients wi

99 Proteus mirabilis is a dimorphic motile bacterium well k

100 Proteus mirabilis is a dimorphic, motile bacterium often

101 The gram-negative enteric bacterium Proteus mirabilis is a frequent cause of urinary tract i

102 Proteus mirabilis is a Gram-negative bacterium that exis

103 Proteus mirabilis is a Gram-negative bacterium that unde

104 Proteus mirabilis is a Gram-negative uropathogen and fre

105 Proteus mirabilis is a model organism for urease-produci

106 Proteus mirabilis is a predominant cause of catheter ass

107 Proteus mirabilis is a urinary tract pathogen and well k

108 Proteus mirabilis is a urinary tract pathogen that diffe

109 Proteus mirabilis is an opportunistic pathogen that is f

110 The enteric bacterium Proteus mirabilis is associated with a significant numbe

111 The bacterium Proteus mirabilis is capable of movement on solid surfac

112 Swarming by Proteus mirabilis is characterized by cycles of rapid an

113 rotease, ZapA, of the urinary tract pathogen Proteus mirabilis is co-ordinately expressed along with

114 Proteus mirabilis is one of the most common causes of mo

115 loacae isolates, 2 S. marcescens isolates, 1 Proteus mirabilis isolate, and 2 A. baumannii isolates)

116 Swarming colonies of independent Proteus mirabilis isolates recognize each other as forei

117 6 (3%) Klebsiella sp. isolates, and 7 (100%) Proteus mirabilis isolates tested were CTX-M positive, w

118 ebsiella pneumoniae, Klebsiella oxytoca, and Proteus mirabilis isolates, including phenotypically ESB

119 gh level of similarity to sequences encoding Proteus mirabilis mannose-resistant fimbriae.

120 lular migration in a non-swarming but motile Proteus mirabilis mutant lacking the FIgN facilitator of

121 Molecular analyses have revealed that Proteus mirabilis possesses two genes, flaA and flaB, th

122 Uropathogenic Proteus mirabilis produces at least four types of fimbri

123 An isolate of Proteus mirabilis recovered from blood cultures of a dia

124 Escherichia coli, Klebsiella pneumoniae, and Proteus mirabilis remains unknown.

125 structures of CaiT from Escherichia coli and Proteus mirabilis revealed an inverted five-transmembran

126 atment enriched the relative proportion of a Proteus mirabilis strain in the ceca of animals not pre-

127 y used to assess the relatedness of swarming Proteus mirabilis strains, was used to study 15 P. aerug

128 Proteus mirabilis swarming behavior is characterized by

129 Here, we identified a gene from Proteus mirabilis that encodes a 135-amino acid residue

130 MR/P fimbriae of uropathogenic Proteus mirabilis undergo invertible element-mediated ph

131 Proteus mirabilis urease catalyzes the hydrolysis of ure

132 Expression of Proteus mirabilis urease is governed by UreR, an AraC-li

133 We tested the hypothesis that experimental Proteus mirabilis urinary tract infection in mice would

134 truncated form of hemolysin A (HpmA265) from Proteus mirabilis using a series of functional and struc

135 Patients with Proteus mirabilis UTIs were more likely to have a foreig

136 higher than the averages were observed with Proteus mirabilis versus imipenem and with Klebsiella pn

137 ty in Escherichia coli, Klebsiella spp., and Proteus mirabilis was evaluated, using incidence rates o

138 to virulence in other pathogens, its role in Proteus mirabilis was investigated by constructing a str

139 A TnphoA mutant of Proteus mirabilis was isolated, which had lost the abili

140 his group of compounds was also confirmed in Proteus mirabilis whole-cell-based inhibition assays.

141 ebsiella pneumoniae, Klebsiella oxytoca, and Proteus mirabilis with an ertapenem-susceptible extended

142 plementation studies with hemolysin-negative Proteus mirabilis WPM111 (a HpmA(-) mutant of BA6163) tr

143 ram-negative pathogens (Escherichia coli and Proteus mirabilis) and variable antifungal activity agai

144 Here we characterize PmDsbA from Proteus mirabilis, a bacterial pathogen increasingly ass

145 Proteus mirabilis, a cause of complicated urinary tract

146 Proteus mirabilis, a cause of complicated urinary tract

147 Recently, we identified a genomic island of Proteus mirabilis, a common agent of catheter-associated

148 Proteus mirabilis, a common cause of nosocomial and cath

149 Proteus mirabilis, a common cause of urinary tract infec

150 Proteus mirabilis, a gram-negative bacterium associated

151 Proteus mirabilis, a gram-negative bacterium, is a frequ

152 Proteus mirabilis, a Gram-negative bacterium, represents

153 Proteus mirabilis, a leading cause of catheter-associate

154 Proteus mirabilis, an etiologic agent of complicated uri

155 ier studies, lrp genes from Vibrio cholerae, Proteus mirabilis, and E. coli were introduced into the

156 al species including Listeria monocytogenes, Proteus mirabilis, and Escherichia coli in various host

157 ct on the numbers of Salmonella typhimurium, Proteus mirabilis, and Escherichia coli internalized by

158 ization of wild-type Salmonella typhimurium, Proteus mirabilis, and Escherichia coli.

159 i, Salmonella muenchen, Serratia marcescens, Proteus mirabilis, and Proteus vulgaris).

160 as Klebsiella pneumoniae, Escherichia coli, Proteus mirabilis, and Salmonella enterica serovar Typhi

161 olates of Escherichia coli, Klebsiella spp., Proteus mirabilis, and Salmonella spp. and are associate

162 by susceptible isolates of Escherichia coli, Proteus mirabilis, and Staphylococcus saprophyticus.

163 Proteus mirabilis, associated with complicated urinary t

164 re also found in cell-free supernatants from Proteus mirabilis, Citrobacter freundii and Enterobacter

165 bacterial pathogens: Pseudomonas aeruginosa, Proteus mirabilis, Enterococcus faecalis, Klebsiella pne

166 ns), Listeria monocytogenes (three strains), Proteus mirabilis, Escherichia coli (three strains), and

167 ted EA rates of >=80%, with the exception of Proteus mirabilis, for which clinical and challenge isol

168 se-resistant Proteus-like (MR/P) fimbriae of Proteus mirabilis, indicate that MrpB functions as the t

169 acteriaceae and in particular the pathobiont Proteus mirabilis, induced robust IL-1beta release that

170 ssified as Corynebacterium frankenforstense, Proteus mirabilis, Lactobacillus johnsonii, and Bacteroi

171 Intestinal colonization with K. pneumoniae, Proteus mirabilis, or Enterobacter cloacae promoted grea

172 kb PAI, designated ICEPm1, that is common to Proteus mirabilis, Providencia stuartii, and Morganella

173 scherichia coli 1021, Klebsiella pneumoniae, Proteus mirabilis, Providencia stuartii, and Pseudomonas

174 ibrary included probes for Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Enterocococcu

175 gram negative bacteria are Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Klebsiella pn

176 prevent colonization by common uropathogens Proteus mirabilis, Staphylococcus aureus and Escherichia

177 ncluded Gram-negative ( Pseudomonas species, Proteus mirabilis, Stenotrophomonas maltophilia ) and Gr

178 by Escherichia coli, Klebsiella pneumoniae, Proteus mirabilis, Streptococcus pyogenes, Bacillus subt

179 een to identify rhomboid-encoding genes from Proteus mirabilis, tatA was identified as a multicopy su

180 In laboratory models of colonization by Proteus mirabilis, the sensor signaled encrustation at a

181 e arfA hairpins from Haemophilus influenzae, Proteus mirabilis, Vibrio fischeri, and Pasteurella mult

182 genesis of urinary tract infection caused by Proteus mirabilis, we constructed a nonmotile, nonswarmi

183 riminating strains of Myxococcus xanthus and Proteus mirabilis, we found the rates of killing between

184 tract pathogens, Pseudomonas aeruginosa and Proteus mirabilis, were made bioluminescent by stable in

185 Here we engineer Proteus mirabilis, which natively forms centimeter-scale

186 inducing drugs also triggered DNA release in Proteus mirabilis, with ssDNA again being more abundant

187 ity to a putrescine-deficient speA mutant of Proteus mirabilis.

188 es in Klebsiella spp., Escherichia coli, and Proteus mirabilis.

189 important virulence factor of uropathogenic Proteus mirabilis.

190 toxin to prevent urinary tract infection by Proteus mirabilis.

191 roteus-like (MR/P) fimbriae of uropathogenic Proteus mirabilis.

192 philus influenzae, Bacteroides fragilis, and Proteus mirabilis.

193 een of the isolates were E. coli and one was Proteus mirabilis.

194 ion, is a virulence factor for uropathogenic Proteus mirabilis.

195 he presence of collagenolytic E faecalis and Proteus mirabilis.

196 acid decarboxylase that inhibits swarming in Proteus mirabilis.

197 d for swarming in the urinary tract pathogen Proteus mirabilis.

198 n CFT073, is a functional homolog of MrpJ of Proteus mirabilis; ectopic expression of papX in P. mira

199 l-Amino acid deaminase from Proteus myxofaciens (PmaLAAD) is a membrane flavoenzyme

200 +)CD11c(+)CD163(low) subset, whereas OTU-10: Proteus, OTU-15: Collinsella tended to concentrate more

201 cetes sharing 96% nucleotide similarity with Proteus phage PM 85 and has a T7-like phage genomic orga

202 nd FSL_SP-069 and the more distantly related Proteus phage PmiS-Isfahan, have similarly sized genomes

203 membrane-bound LAADs mainly express in genus Proteus, Providencia and Morganella.

204 Hippocamp is probably an ancient fragment of Proteus, providing further support for the hypothesis th

205 region of rtn is identical to the published Proteus sequence, with the exception of a single G inser

206 mophilus ducreyi is the newest member of the Proteus/Serratia family of pore-forming toxins.

207 nt aerobes were Escherichia coli (n = 8) and Proteus sp. (n = 7).

208 ccus areus, Pseudomonas aureginosa, E. coli, Proteus sp. and streptococcus sp.) at concentrations up

209 Most pathogenic Proteus species are primarily associated with urinary tr

210 cherichia coli, 21 Klebsiella species, and 6 Proteus species that were resistant to at least one ESC

211 ereas Escherichia coli , Klebsiella species, Proteus species, and Streptococcus species were associat

212 toca, 16/17 with K. pneumoniae, and 0/1 with Proteus spp.

213 Escherichia coli, Klebsiella pneumoniae, and Proteus spp.

214 mograms and recovery of Escherichia coli and Proteus spp. from the livers of infected mice.

215 spp., 99.3%; Pseudomonas aeruginosa, 98.9%; Proteus spp., 100%; Acinetobacter spp., 98.4%; and Citro

216 e most frequently used codons in the AT-rich Proteus spp., AAA (lysine).

217 pital admission) coliform (Escherichia coli, Proteus spp., or Klebsiella spp.) bacteraemia among adul

218 , Klebsiella oxytoca, Klebsiella pneumoniae, Proteus spp., Pseudomonas aeruginosa, and Serratia marce

219 In Proteus swarm cell extracts, all the FlhC was assembled

220 Each Proteus swarm colony terrace corresponds to one swarming

221 opsy samples obtained from patients with the Proteus syndrome and compared the resultant DNA sequence

222 Five had Proteus syndrome and one, a Proteus-like syndrome.

223 syndrome, Bannayan-Riley-Ruvalcaba syndrome, Proteus syndrome and Proteus-like syndrome, collectively

224 r syndrome spectrum has broadened to include Proteus syndrome and Proteus-like syndromes.

225 Interestingly, 27 of 31 Proteus syndrome animals had non-zero blood VAF that is

226 53 individuals with a confirmed diagnosis of Proteus syndrome at our institution from 10/2001 to 10/2

227 issues and cell lines from patients with the Proteus syndrome harbored admixtures of mutant alleles t

228 clinical diagnosis of neurofibromatosis and Proteus syndrome has allowed advancements in the Elephan

229 alence of cardiothoracic imaging findings of Proteus syndrome in a large cohort at our institution.

230 Complications of Proteus syndrome include, among others, progressive skel

231 Proteus syndrome is a life-threatening segmental overgro

232 Proteus syndrome is a mosaic, progressive overgrowth dis

233 Proteus syndrome is a progressive overgrowth disorder wi

234 Proteus syndrome is a rare and sporadic disorder that ca

235 Proteus syndrome is an extremely rare disorder of mosaic

236 The Proteus syndrome is caused by a somatic activating mutat

237 The Proteus syndrome is characterized by the overgrowth of s

238 uorescent protein (GFP) staining in chimeric Proteus syndrome mice showed that in some lesions, hyper

239 ary hyperplasia were seen in 10 of 16 female Proteus syndrome mice with other localized regions of hy

240 from 0% to 50% across numerous tissues in 44 Proteus syndrome mice.

241 a, frequently biliary, were seen in 22 of 44 Proteus syndrome mice.

242 finding with a total of 69 found in 29 of 44 Proteus syndrome mice.

243 These findings extend the spectrum of Proteus syndrome pathological characteristics and sugges

244 Care of patients with Proteus syndrome presents significant challenges to both

245 y of miransertib in adults and children with Proteus syndrome to identify an appropriate dosage start

246 ogenously-regulated mosaic expression of the Proteus syndrome variant.

247 Of 29 patients with the Proteus syndrome, 26 had a somatic activating mutation (

248 hs that covered his body: neurofibromatosis, Proteus syndrome, and a combination of childhood injury,

249 syndrome, Bannayan-Riley-Ruvalcaba syndrome, Proteus syndrome, and Proteus-like syndrome.

250 ent did not meet the diagnostic criteria for Proteus syndrome, he was found to have the c.49G>A, p.Gl

251 is the mildest molecularly confirmed case of Proteus syndrome, occurring in the absence of the charac

252 DNA in 158 samples from 29 patients with the Proteus syndrome.

253 There are no effective treatments for Proteus syndrome.

254 drome, Bannayan-Riley-Ruvalcaba syndrome and Proteus syndrome.

255 nt as a therapeutic option for patients with Proteus syndrome.

256 ling in cells and tissues from patients with Proteus syndrome.

257 onsible for the mosaic overgrowth condition, Proteus syndrome.

258 Merrick was in all likelihood suffering from Proteus syndrome.

259 ise of therapeutic efficacy in children with Proteus syndrome.

260 r discovery of disease-causing alleles using Proteus syndrome; phakomatosis pigmentokeratotica; linea

261 eria including Y. pestis, H. influenzae, and Proteus that cause plague, meningitis, and severe wound

262 Hippocamp orbits close to Proteus, the outermost and largest of these moons, and t

263 sequence identities of 92% (Vibrio) and 98% (Proteus) to E. coli Lrp, including complete conservation

264 Proteus toxic agglutinin (Pta) represents a novel autotr

265 he HpmA hemolysin, a secreted cytotoxin, and proteus toxic agglutinin (Pta), a surface-associated cyt

266 the urinary tract, including a known toxin (Proteus toxic agglutinin) and the high pathogenicity isl

267 NA (eDNA) approach to detect the presence of Proteus using water samples collected from karst springs

268 with serum antibodies cross-reactive against Proteus vulgaris (Weil-Felix reaction).

269 Proteus vulgaris accounted for 81% (13/16) of the sample

270 udomonas aeruginosa, Enterococcus aerogenes, Proteus vulgaris and Enterobacter sakazakii) bacteria, w

271 The rtn gene, identified as coming from Proteus vulgaris ATCC 13315, is present in Escherichia c

272 Serratia marcescens, Erwinia carotovora, and Proteus vulgaris but not in several nonenteric bacteria.

273 We placed 43 isolates belonging to the Proteus vulgaris complex into proposed DNA groups (genom

274 gA antitoxin regulates the expression of the Proteus vulgaris higBA toxin-antitoxin operon from the R

275 of the tna operon of Escherichia coli and of Proteus vulgaris is induced by L-tryptophan.

276 species-specific class A beta-lactamase from Proteus vulgaris K1 was crystallized at pH 6.25 and its

277 oaceticus BD413, Vibrio cholerae El Tor, and Proteus vulgaris K80, were members of a previously descr

278 Proteus vulgaris L-amino acid deaminase (pvLAAD) belongs

279 ound in the O-polysaccharide of the LPS from Proteus vulgaris OX19 used in the Weil-Felix test, sugge

280 ith deletion constructs of the tna operon of Proteus vulgaris supported this interpretation.

281 e solved two x-ray crystal structures of the Proteus vulgaris tetrameric HigB-(HigA)2-HigB TA complex

282 ng of substrates and inhibitors to wild-type Proteus vulgaris tryptophan indole-lyase and to wild typ

283 Urinary tract infections caused by Proteus vulgaris typically form biofilms and are resista

284 Two commercial enzymes, chondroitinase ABC (Proteus vulgaris) and chondroitinase ACII (Arthrobacter

285 scherichia coli, Salmonella typhimurium, and Proteus vulgaris) We also isolated transposition events

286 Serratia marcescens, Proteus mirabilis, and Proteus vulgaris).

287 eas MICs for E. coli, Klebsiella pneumoniae, Proteus vulgaris, and Pseudomonas aeruginosa were > 100

288 we solve the crystal structure of CodB from Proteus vulgaris, at 2.4 angstrom resolution in complex

289 of these enzymes, chondroitinase ABC I from Proteus vulgaris, has the broadest substrate specificity

290 of the PvuII plasmid pPvu1, originally from Proteus vulgaris, making this the first completely seque

291 erived from either Salmonella typhimurium or Proteus vulgaris, microorganisms that have diverged from

292 ression of the tryptophanase (tna) operon of Proteus vulgaris, short deletions were introduced in the

293 st Listeria monocytogenes, Escherichia coli, Proteus vulgaris, Staphylococcus aureus, and Candida alb

294 Serratia marcescens, Erwinia carotovora, and Proteus vulgaris, strongly suggesting that the physiolog

295 nzymes, cABCI and cABCII, were identified in Proteus vulgaris.

296 perative infection with antibiotic-resistant Proteus vulgaris.

297 by expressing an L-amino acid deaminase from Proteus vulgaris.

298 le of the nucleus in the migration of Amoeba proteus we have analyzed the movement trajectories of en

299 nifestations, a reference to the ancient god Proteus, who could assume many forms and thus elude his

300 at the 5-HT3AB receptor (after the Greek god Proteus, who was able to change his shape and appearance