ライフサイエンスコーパス: Pseudomonas

1 a), I-E (Pseudomonas and Serratia), and I-C (Pseudomonas).

2 erstanding the pan metabolic capabilities of Pseudomonas.

3 lysis by the L-glutaminase-asparaginase from Pseudomonas 7A (PGA) was investigated using structural,

4 heir ability to modify the environment, with Pseudomonas able to utilise secondary metabolites produc

5 thogens were Staphylococcus aureus (34%) and Pseudomonas aeruginosa (17%), whereas blood cultures mos

6 ogens were Klebsiella pneumoniae (25.6%) and Pseudomonas aeruginosa (18.9%).

7 Qualifying baseline pathogens: Pseudomonas aeruginosa (77%), Klebsiella spp. (16%), oth

8 athogens in liver transplant recipients, and Pseudomonas aeruginosa (9%) in lung transplant recipient

9 We test our predictions by competing Pseudomonas aeruginosa (a tit-for-tat species) with Vibr

10 enemase producers among carbapenem-resistant Pseudomonas aeruginosa (CRPA) isolates warrants an expan

11 ug/mL), and also exhibited activity against Pseudomonas aeruginosa (MIC 16 ug/mL) and Staphylococcus

12 o investigate the prevalence, antibiogram of Pseudomonas aeruginosa (P. aeruginosa), and the distribu

13 Elimination of pulmonary Pseudomonas aeruginosa (PA) infections is challenging to

14 sensitivity analyses based on lung function, Pseudomonas aeruginosa (PA) status, and follow-up time i

15 A), vancomycin-resistant Enterococcus (VRE), Pseudomonas aeruginosa (PA), and Candida albicans (CA)].

16 r bacteria, including the common CF pathogen Pseudomonas aeruginosa (Pa).

17 thogens, Escherichia coli (Ec, m/z 1797) and Pseudomonas aeruginosa (Pa, m/z 1446) using on-tissue ac

18 purified small RNAs isolated from pathogenic Pseudomonas aeruginosa (PA14) is sufficient to induce pa

19 tudy the biochemical properties of FtsZ from Pseudomonas aeruginosa (PaFtsZ) and the effects of its t

20 Emerging evidence suggests the Pseudomonas aeruginosa accessory genome is enriched with

21 Pathogens such as Pseudomonas aeruginosa advantageously modify animal host

22 development associated with the isolation of Pseudomonas aeruginosa after lung transplantation.

23 reviously characterized in the regulation of Pseudomonas aeruginosa alginate biosynthesis(3,4), as a

24 kingdom QS interactions between a bacterium, Pseudomonas aeruginosa and a yeast, Candida albicans, in

25 n-resistant Staphylococcus aureus (MRSA) and Pseudomonas aeruginosa and accelerates their removal by

26 icant improvement in the inactivation of MDR Pseudomonas aeruginosa and Acinetobacter baumannii (plan

27 undreds of bacteria, including the pathogens Pseudomonas aeruginosa and Acinetobacter baumannii.

28 Pseudomonas aeruginosa and Burkholderia cepacia complex

29 tam antibiotics against carbapenem-resistant Pseudomonas aeruginosa and carbapenem-resistant Enteroba

30 Using microfluidic experiments with Pseudomonas aeruginosa and Escherichia coli, we demonstr

31 dal activity toward both Gram stain-negative Pseudomonas aeruginosa and Gram stain-positive Staphyloc

32 tablished that UQ(9) is the major quinone of Pseudomonas aeruginosa and is required for growth under

33 m progressive and severe pneumonia caused by Pseudomonas aeruginosa and poor wound healing.

34 Pseudomonas aeruginosa and Staphylococcus aureus are opp

35 xperimentally using both model membranes and Pseudomonas aeruginosa and Staphylococcus aureus, repres

36 i, Salmonella enteritidis, Listeria innocua, Pseudomonas aeruginosa and Streptococcus pneumoniae did

37 l activity against Staphylococcus aureus and Pseudomonas aeruginosa and with their geographical origi

38 Chronic infections by Pseudomonas aeruginosa are characterized by biofilm form

39 ials (e.g., -0.2 to 1.0 V vs Ag/AgCl), using Pseudomonas aeruginosa as a model organism.

40 in an untargeted fashion while in contrast, Pseudomonas aeruginosa assembles and fires its T6SS appa

41 The optimal antibiotic regimen for Pseudomonas aeruginosa bacteremia is controversial.

42 Health care-associated infections such as Pseudomonas aeruginosa bacteremia pose a major clinical

43 Pseudomonas aeruginosa belongs to the group of three "cr

44 challenging sample (>90% water) of a mature Pseudomonas aeruginosa biofilm in its native state.

45 As an example, Pseudomonas aeruginosa biofilm is grown from single cell

46 of the in vitro electroceutical treatment of Pseudomonas aeruginosa biofilms is demonstrated both at

47 -cost method was proposed for the imaging of Pseudomonas aeruginosa biofilms on metallic surfaces usi

48 contributes to the structure and function of Pseudomonas aeruginosa biofilms.

49 nteractions with extracellular DNA (eDNA) in Pseudomonas aeruginosa biofilms.

50 vaU act coordinately as global repressors in Pseudomonas aeruginosa by binding to AT-rich regions of

51 iofilms of the pathogens Vibrio cholerae and Pseudomonas aeruginosa can induce large deformations of

52 Pseudomonas aeruginosa causes severe multidrug-resistant

53 atelet activation after intratracheal LPS or Pseudomonas aeruginosa challenge.

54 injury in CF mice after intratracheal LPS or Pseudomonas aeruginosa challenge.

55 (GFP) to construct a novel SDS biosensor in Pseudomonas aeruginosa chassis.

56 lone and during polymicrobial infection with Pseudomonas aeruginosa Colonization, persistence, and vi

57 Pseudomonas aeruginosa colonizing airways is consistentl

58 ke many bacteria, the opportunistic pathogen Pseudomonas aeruginosa contains two ClpP homologs: ClpP1

59 ess this question, we purified a five-member Pseudomonas aeruginosa division complex consisting of Ft

60 terobacterales, Acinetobacter baumannii, and Pseudomonas aeruginosa during susceptibility testing.

61 It has been shown that Pseudomonas aeruginosa elastase (LasB) and Clostridium h

62 fy independent risk factors for mortality in Pseudomonas aeruginosa episodes.

63 Pseudomonas aeruginosa exhibits a high requirement for i

64 Next, a hexasaccharide fragment of the Pseudomonas aeruginosa exopolysaccharide Pel was assembl

65 Instead, Pseudomonas aeruginosa exposed to CSE (CSE-PSA) had incr

66 We demonstrated that their preexposure to Pseudomonas aeruginosa flagellin modify their inflammato

67 t a change in the rates of ESBL, CRE and MDR Pseudomonas aeruginosa following ASP.

68 nical isolates of the opportunistic pathogen Pseudomonas aeruginosa from patients with cystic fibrosi

69 The human pathogen Pseudomonas aeruginosa harbors three paralogous zinc pro

70 In particular, electrogenic Pseudomonas aeruginosa has been studied with the utility

71 IGPS from Pseudomonas aeruginosa has the highest turnover number o

72 so efficacious at killing the model organism Pseudomonas aeruginosa in biofilms and in a murine chron

73 ects the lung from injury upon intratracheal Pseudomonas aeruginosa in mice.

74 We hypothesized that the isolation of Pseudomonas aeruginosa in respiratory specimens would in

75 strate that upon hitting a host cell, motile Pseudomonas aeruginosa induce a specific gene expression

76 Pseudomonas aeruginosa infection elicits the production

77 ng protein (BPI) is strongly associated with Pseudomonas aeruginosa infection in cystic fibrosis (CF)

78 Pseudomonas aeruginosa infections are increasingly multi

79 ed by a prophage in P. aeruginosa IMPORTANCE Pseudomonas aeruginosa is a Gram-negative bacterium freq

80 The opportunistic pathogen Pseudomonas aeruginosa is a leading cause of morbidity a

81 The opportunistic pathogen Pseudomonas aeruginosa is a major cause of antibiotic-to

82 Pseudomonas aeruginosa is a priority pathogen for the de

83 Pseudomonas aeruginosa is an extracellular opportunistic

84 Pseudomonas aeruginosa is an opportunistic bacterium of

85 Pseudomonas aeruginosa is an opportunistic human pathoge

86 Pseudomonas aeruginosa is an opportunistic pathogen that

87 The prevalence of carbapenem-resistant Pseudomonas aeruginosa is increasing.

88 The opportunistic pathogen Pseudomonas aeruginosa is responsible for much of the mo

89 Also, 46.2% of Pseudomonas aeruginosa isolates were carbapenem-resistan

90 terobacterales, Acinetobacter baumannii, and Pseudomonas aeruginosa isolates were evaluated across th

91 nited States, where 309 Enterobacterales and Pseudomonas aeruginosa isolates were evaluated by NG-Tes

92 We previously identified Pseudomonas aeruginosa isolates with characteristics typ

93 rs (126 isolates of the Enterobacterales, 50 Pseudomonas aeruginosa isolates, and 50 Acinetobacter sp

94 isolates (775 Enterobacterales isolates, 119 Pseudomonas aeruginosa isolates, and 83 Acinetobacter ba

95 rtain enteroaggregative Escherichia coli and Pseudomonas aeruginosa isolates, whereas to other bacter

96 f-5 as a model to elucidate PelX function as Pseudomonas aeruginosa lacks a pelX homologue in its pel

97 ologous to the historic phage B3 that infect Pseudomonas aeruginosa Like other phage groups, the B3-l

98 cocrystal structures of inhibitors bound to Pseudomonas aeruginosa LpxC as guides, resulted in the d

99 used whole genome sequencing to characterise Pseudomonas aeruginosa MDR clinical isolates from a hosp

100 Citrobacter rodentium, Escherichia coli, or Pseudomonas aeruginosa mutant strain DeltapopB Moreover,

101 highest for Enterobacterales and lowest for Pseudomonas aeruginosa Nevertheless, even for Enterobact

102 gation-ready trisaccharide repeating unit of Pseudomonas aeruginosa O11 via a highly stereoselective

103 epidermidis but did not inhibit biofilms by Pseudomonas aeruginosa or Bacillus subtilis, and inhibit

104 During infection of a host, Pseudomonas aeruginosa orchestrates global gene expressi

105 xoelectrogens, Shewanella oneidensis MR1 and Pseudomonas aeruginosa PA01, and many other mutants of t

106 Pseudomonas aeruginosa PAO1, an opportunistic human path

107 Pseudomonas aeruginosa pneumonia elicits endothelial cel

108 al characterization of the CopG protein from Pseudomonas aeruginosa Results from biochemical analyses

109 Using two Pseudomonas aeruginosa strains carrying resistance plasm

110 tized, and tested against colistin-resistant Pseudomonas aeruginosa strains including clinical isolat

111 Pseudomonas aeruginosa strains with loss-of-function mut

112 terial lawns including Bacillus subtilis and Pseudomonas aeruginosa strongly alter the collective dyn

113 Here, we show that the ClpXP protease of Pseudomonas aeruginosa suppresses its antimicrobial acti

114 terobacterales, Acinetobacter baumannii, and Pseudomonas aeruginosa tested.

115 ng ventilator-associated pneumonia caused by Pseudomonas aeruginosa that acquired increasing levels o

116 rbapenem susceptible" at breakpoint; and (4) Pseudomonas aeruginosa that merely lack porin OprD?

117 reagents for ticks, we also show that adding Pseudomonas aeruginosa to drinking water quickly leads t

118 By fusing transcription activation domain to Pseudomonas aeruginosa type I-F Cas proteins, we activat

119 Pseudomonas aeruginosa uses a type III secretion system

120 d by an extensively drug-resistant strain of Pseudomonas aeruginosa was detected in a hospital in Mad

121 Pseudomonas aeruginosa was the most common CRGNB each ye

122 produced in planktonic- and biofilm-cultured Pseudomonas aeruginosa We identified a core assembly of

123 al activities against Enterobacteriaceae and Pseudomonas aeruginosa were identified.

124 Staphylococcus aureus and Pseudomonas aeruginosa were isolated in 10.5 and 6.4% of

125 as well as bacterial killing curves against Pseudomonas aeruginosa were utilized.

126 A common Gram-negative pathogenic bacterium Pseudomonas aeruginosa wild-type PAO1 and first-line ant

127 nerated large deletions (7-424 kilobases) in Pseudomonas aeruginosa with near-100% efficiency, while

128 erpretive categories to other organisms like Pseudomonas aeruginosa without supporting evidence.

129 ibit antibiotic-resistant bacteria (MRSA and Pseudomonas aeruginosa), the most common cause of biomat

130 PapRecT enables efficient recombineering in Pseudomonas aeruginosa, a concerning human pathogen.

131 Pseudomonas aeruginosa, a Gram-negative bacterium that c

132 sis is the key mechanism for host control of Pseudomonas aeruginosa, a motile Gram-negative, opportun

133 Multiple clinical isolates of Pseudomonas aeruginosa, an important human pathogen, hav

134 encompassing 288 Staphylococcus aureus, 456 Pseudomonas aeruginosa, and 1588 Escherichia coli genome

135 d to interpret results for Enterobacterales, Pseudomonas aeruginosa, and Acinetobacter baumannii comp

136 emophilus influenzae, Staphylococcus aureus, Pseudomonas aeruginosa, and Aspergillus infections were

137 5 to 2 mg/L for MDR Gram-negative, excluding Pseudomonas aeruginosa, and between 0.03 and 1 mg/L for

138 bsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, and Enterobacter spp.) directly

139 tory activity against Staphylococcus aureus, Pseudomonas aeruginosa, and Enterococcus faecalis althou

140 uation in PDI against Staphylococcus aureus, Pseudomonas aeruginosa, and Escherichia coli A significa

141 ms of uropathogenic Escherichia coli (UPEC), Pseudomonas aeruginosa, and Staphylococcus aureus, with

142 ebsiella pneumoniae, Acinetobacter baumanii, Pseudomonas aeruginosa, and Staphylococcus aureus.

143 onella enterica, Mycobacterium tuberculosis, Pseudomonas aeruginosa, and Staphylococcus aureus.

144 rally required for growth arrest survival of Pseudomonas aeruginosa, and that this requirement is ind

145 luding the human pathogens Escherichia coli, Pseudomonas aeruginosa, and Vibrio cholerae, and the pla

146 e of a SctK protein family member, PscK from Pseudomonas aeruginosa, as well as the structure of its

147 its DNA from immunity nucleases of its host, Pseudomonas aeruginosa, by constructing a proteinaceous

148 piric coverage (e.g., Staphylococcus aureus, Pseudomonas aeruginosa, Clostridium difficile, and funga

149 ng members of the order Enterobacterales and Pseudomonas aeruginosa, enriched for drug resistance.

150 In Pseudomonas aeruginosa, expression of the T3SS is regula

151 of the environmental opportunistic pathogen Pseudomonas aeruginosa, it has been shown that overexpre

152 Pseudomonas aeruginosa, like many bacilliforms, are not

153 All FARs showed bactericidal effect against Pseudomonas aeruginosa, making PA the most susceptible o

154 using culture-dependent methods to quantify Pseudomonas aeruginosa, opportunistic pathogens capable

155 ne complexes from Salmonella Typhimurium and Pseudomonas aeruginosa, respectively, reveals that Eag c

156 In the opportunistic pathogen Pseudomonas aeruginosa, RsmA is an RNA-binding protein t

157 type III secreted phospholipase effector of Pseudomonas aeruginosa, serves as a prototype to model l

158 tral forms still present in Vibrio cholerae, Pseudomonas aeruginosa, Shewanella oneidensis and Methyl

159 l activity against many pathogens, including Pseudomonas aeruginosa, Staphylococcus aureus, and Esche

160 nd effectively inhibits biofilm formation in Pseudomonas aeruginosa, the most widely used model for s

161 In Pseudomonas aeruginosa, the transition between planktoni

162 xt of the modal MIC for Enterobacterales and Pseudomonas aeruginosa, the variability of MIC tests, an

163 HE domain of chemoreceptors PctA and TlpQ of Pseudomonas aeruginosa, thus inducing chemotaxis and bio

164 tam (TOL-TAZ) affords broad coverage against Pseudomonas aeruginosa.

165 airway epithelial cells and cocultures with Pseudomonas aeruginosa.

166 traits in opportunistic pathogens including Pseudomonas aeruginosa.

167 hat often retains activity against resistant Pseudomonas aeruginosa.

168 evelopment of effective strategies to target Pseudomonas aeruginosa.

169 oad array of bacteria, including E. coli and Pseudomonas aeruginosa.

170 Escherichia coli, Citrobacter rodentium, and Pseudomonas aeruginosa.

171 it from three Podoviridae phages that infect Pseudomonas aeruginosa.

172 nas gingivalis, Fusobacterium nucleatum, and Pseudomonas aeruginosa.

173 S) production and anthranilate metabolism in Pseudomonas aeruginosa.

174 obacter baumannii, Klebsiella pneumoniae and Pseudomonas aeruginosa.

175 nem-resistant acinetobacter species, and MDR Pseudomonas aeruginosa.

176 nge in the treatment of infections caused by Pseudomonas aeruginosa.

177 iocontrol agents to the major human pathogen Pseudomonas aeruginosa.

178 of the bacterial endosymbionts Burkholderia, Pseudomonas and Azospirillum on photosynthesis and the a

179 such as carbapenem-resistant Acinetobacter, Pseudomonas and Enterobacterales.

180 rowing plants, while sequences identified as Pseudomonas and Pantoea were abundant in poorly growing

181 domonas, Pectobacterium, and Serratia), I-E (Pseudomonas and Serratia), and I-C (Pseudomonas).

182 rial species from genera such as Paracoccus, Pseudomonas, and Alcaligenes have evolved to use DMF as

183 romosomal MGEs within Enterobacteriaceae and Pseudomonas, and an additional Aca (aca9).

184 In this study, Burkholderia, Pseudomonas, and Staphylococcus species were isolated fr

185 evel: Escherichia, Klebsiella, Enterobacter, Pseudomonas, and Stenotrophomonas.

186 by Gammaproteobacteria at class level and by Pseudomonas at genera level.

187 utants exhibited increased susceptibility to Pseudomonas bacteria and Mucorales fungi, which could be

188 (CCSSD) of PupR, the sigma regulator in the Pseudomonas capeferrum pseudobactin BN7/8 transport syst

189 ells of the plant health-promoting bacterium Pseudomonas chlororaphis O6 (PcO6) are examined.

190 inst non-host strains was demonstrated using Pseudomonas Chlororaphis.

191 o-occurrence of the bacterial spot pathogens Pseudomonas cichorii and Xanthomonas spp. is common in A

192 t association between the number of positive Pseudomonas cultures and the risk of DSA development.

193 The conserved Pseudomonas effector AvrPtoB acts as an E3 ubiquitin lig

194 , Staphylococcus, Klebsiella, Acinetobacter, Pseudomonas, Enterobacter (ESKAPE), and other enteric pa

195 Lower increases in mesophile, psychrophile, Pseudomonas, Enterobacteriaceae and H(2)S producing bact

196 that were either susceptible or resistant to Pseudomonas entomophila infection reveals nutcracker as

197 38 DGRP lines that were orally infected with Pseudomonas entomophila.

198 immunotoxins to contain various deimmunized Pseudomonas exotoxin (PE) domains.

199 (TaAA9A) is compared with that of CopC from Pseudomonas fluorescens (PfCopC) and with the LPMO-like

200 indole enhanced the antibiotic tolerance of Pseudomonas fluorescens 2P24, a PGPR well known for its

201 Over the last two decades, the mechanisms of Pseudomonas fluorescens biofilm formation and regulation

202 Here, we identify a homolog of Pseudomonas fluorescens LapG as a dispersal factor that

203 terial strains Pseudomonas putida KT2440 and Pseudomonas fluorescens LP6a at varying electrolyte conc

204 g adhesion studies of bacterial cells (i.e., Pseudomonas fluorescens).

205 vity of GO and MGO against Listeria innocua, Pseudomonas fluorescens, Salmonella enterica and Bacillu

206 othrix thermosphacta, Enterococcus faecalis, Pseudomonas fluorescens, Salmonella typhimurium, Staphyl

207 ependent analyses revealed that isolation of Pseudomonas from respiratory specimens, acute cellular r

208 g family present in different members of the Pseudomonas genus, and associated with multiple sources

209 , the heme assimilation system (Has) and the Pseudomonas heme utilization (Phu) system.

210 n sequence variants of the genera Ralstonia, Pseudomonas, Hyphomicrobium, and Novosphingobium, which

211 llin-resistant S. aureus, fungal infections, Pseudomonas infections, and C. difficile.

212 ar rejection, lymphocytic bronchiolitis, and Pseudomonas isolation after transplantation are associat

213 In multivariable analyses, Pseudomonas isolation, acute cellular rejection, and lym

214 Collectively, we propose that Pseudomonas jumbo phages evade a broad spectrum of DNA-t

215 aque-forming units (PFUs) per milliliter for Pseudomonas, Klebsiella, and Serratia phages tested.

216 cluding those from the genera Acinetobacter, Pseudomonas, Klebsiella, Enterobacter, Vibrio, Shigella,

217 Consistent with the Pseudomonas Lap system model, our data support a role fo

218 HS samples using the denitrifying bacterium Pseudomonas nitroreducens.

219 lar permeability is a severe complication of Pseudomonas (P.) aeruginosa-induced acute lung injury.

220 against a panel of six type I systems: I-F (Pseudomonas, Pectobacterium, and Serratia), I-E (Pseudom

221 The atomic structure of Pseudomonas phage PaP3 TerS, the first complete structur

222 Pseudomonas phages and plasmids have taken advantage of

223 , less is known about the packaging motor of Pseudomonas-phages that have increasing biomedical relev

224 ted control, six rhizobacterial monoculture (Pseudomonas poae, Pseudomonas sp., Bacillus pumilus., Pa

225 potential role of the putative oxygen sensor Pseudomonas prolyl hydroxylase (PPHD) in the control of

226 he rare gut bacteria Serratia marcescens and Pseudomonas protegens contributed to atrazine metabolism

227 Herein, we have used Pseudomonas protegens Pf-5 as a model to elucidate PelX

228 4) and the well-characterized plant epiphyte Pseudomonas protegens Pf-5, a maize seed inoculant.

229 ies investigated (Pseudomonas putida KT2440, Pseudomonas protegens Pf-5, and Pseudomonas putida S12),

230 eria (Escherichia coli and a cheese-isolated Pseudomonas psychrophila).

231 r these reactions, alkane monooxygenase from Pseudomonas putida (alkB) is able to catalyze the diffic

232 monoaromatic hydrocarbons such as toluene in Pseudomonas putida F1 (PpF1) occurs via lateral diffusio

233 a missing step in the D-lysine catabolism of Pseudomonas putida in which 2OA is converted to D-2-hydr

234 temperature" X-ray crystallography data for Pseudomonas putida ketosteroid isomerase (KSI), and we o

235 fects on the deposition of bacterial strains Pseudomonas putida KT2440 and Pseudomonas fluorescens LP

236 The two As resistance arsRBC operons of Pseudomonas putida KT2440 are followed by a downstream g

237 of the biotechnologically relevant bacterium Pseudomonas putida KT2440 that greatly expands computabl

238 oduct indigoidine, a sustainable pigment, in Pseudomonas putida KT2440, an emerging industrial microb

239 GMP levels in the plant-beneficial bacterium Pseudomonas putida KT2440, identifying L-arginine as the

240 Across the three species investigated (Pseudomonas putida KT2440, Pseudomonas protegens Pf-5, a

241 oteome of three aromatic-catabolic bacteria: Pseudomonas putida KT2440, Rhodoccocus jostii RHA1, and

242 Immobilized dye-decolorizing peroxidase from Pseudomonas putida MET94 (PpDyP) and three variants gene

243 tida KT2440, Pseudomonas protegens Pf-5, and Pseudomonas putida S12), siderophore secretion is higher

244 a acetivorans, Sulfolobus acidocaldarius and Pseudomonas putida) enriched in (13) C, (15) N, (18) O,

245 s) has been generated from Escherichia coli, Pseudomonas putida, and Ralstonia eutropha.

246 nella enteritidis, Enterobacter agglomerans, Pseudomonas putida, Staphylococcus aureus, and Bacillus

247 is are implemented in the platform bacterium Pseudomonas putida.

248 nthranilate regulator) for its regulation of Pseudomonas quinolone signal (PQS) production and anthra

249 Stimulation by denatonium or with Pseudomonas quinolone signaling molecules led to an incr

250 ntegrative and conjugative element ICEclc in Pseudomonas requires development of a transfer competenc

251 ns, Rhodococcus fascians, Xanthomonas citri, Pseudomonas savastanoi, Pantoea agglomerans, 'Candidatus

252 rains from four genera, including Aeromonas, Pseudomonas, Shewanella, and Sphingopyxis.

253 re of chlorothalonil dehalogenase (Chd) from Pseudomonas sp. CTN-3, with 15 of its N-terminal residue

254 Amyloid formed by Pseudomonas sp. protein FapC provides an excellent model

255 hizobacterial monoculture (Pseudomonas poae, Pseudomonas sp., Bacillus pumilus., Pantoea agglomerance

256 e defined the pan-regulon of ErfA in several Pseudomonas species and found ergAB as the sole conserve

257 sm of indole-induced antibiotic tolerance in Pseudomonas species and had important implications on ho

258 te and Fe-limited cells, we uncover how soil Pseudomonas species reprogram their metabolic pathways t

259 s instrumental in the virulence of different Pseudomonas species, ranging from soil- and plant-dwelli

260 llin treatment, when the gut is dominated by Pseudomonas species.

261 ic tolerance is likely to be conserved among Pseudomonas species.

262 ces reshaped within-host fitness ranks among Pseudomonas spp. field isolates and amplified a subset o

263 ignificant growth boost when compared with a Pseudomonas strain incapable of solubilising Ca(3)(PO(4)

264 2)), application of a phosphate solubilising Pseudomonas strain to plant roots provides a significant

265 Driveline smears revealed Staphylococcus or Pseudomonas strains as the underlying pathogen in most c

266 5, the transfer of an inducible cluster from Pseudomonas stutzeri and Azotobacter vinelandii yields a

267 In the Cu(A) site of Pseudomonas stutzeri N(2)OR, a histidine ligand was foun

268 ors diazotrophic (N(2)-fixing) endobacteria (Pseudomonas stutzeri) that allow JGTA-S1 to fix N(2) and

269 ' vector was functional in Escherichia coli, Pseudomonas syringae and Klebsiella pneumoniae, and endo

270 nt resistance to the hemibiotrophic pathogen Pseudomonas syringae and the necrotrophic pathogen Botry

271 for the activity of INPs from the bacterium Pseudomonas syringae by combining a high-throughput ice

272 Here, we show that the bacterial pathogen Pseudomonas syringae deploys an effector protein, HopO1-

273 Interestingly, infection with Pseudomonas syringae in wild-type (WT) plants downregula

274 ic in nature, isolates such as the Antarctic Pseudomonas syringae Lz4W exhibit considerable psychroto

275 gainst the hemibiotrophic bacterial pathogen Pseudomonas syringae oxr2 mutant plants are more suscept

276 d that TARK1 CRISPR plants were resistant to Pseudomonas syringae pathovar tomato strain DC3000-induc

277 ble to virulent bacterial pathogens, such as Pseudomonas syringae pv maculicola (Psm) and P. syringae

278 Notably, Pseudomonas syringae pv tomato (Pto) bacterial effectors

279 aling and pattern-triggered immunity against Pseudomonas syringae pv tomato DC3000.

280 observed in silenced plants infiltrated with Pseudomonas syringae pv. tabaci expressing AvrPto or Hop

281 utant of Arabidopsis is hyper-susceptible to Pseudomonas syringae pv. tomato (Pst) DC3000, while Arab

282 t basal and effector-triggered resistance to Pseudomonas syringae pv. tomato (Pst) DC3000.

283 Pseudomonas syringae pv. tomato (Pst) delivers effector

284 Flagellin, from the bacterial pathogen Pseudomonas syringae pv. tomato (Pst), contains two MAMP

285 t various pathogens, including the bacterium Pseudomonas syringae pv. tomato (Pst).

286 sed resistance toward the virulent bacterium Pseudomonas syringae pv. tomato DC3000 and the necrotrop

287 betaCA3) is induced by the virulent pathogen Pseudomonas syringae pv. tomato DC3000.

288 persicoides confers resistance to strains of Pseudomonas syringae pv. tomato expressing AvrRpt2 and R

289 nthamiana, compromised nonhost resistance to Pseudomonas syringae pv. tomato T1.

290 For example, the plant pathogen Pseudomonas syringae strain PtoDC3000 uses an indole-3-a

291 s a toxin produced by the bacterial pathogen Pseudomonas syringae that is known to counteract Arabido

292 It was further revealed the EmhR ortholog in Pseudomonas syringae was also responsible for indole-ind

293 e, Xanthomonas oryzae, Erwinia chrysanthemi, Pseudomonas syringae, and Acidovorax avenae, naringenin

294 wed increased susceptibility to the pathogen Pseudomonas syringae, with the double mutant showing a s

295 in the machine shop area were enriched with Pseudomonas, the dominant taxa in MWF.

296 The bacterial pathogen Pseudomonas tolaasii severely damages white button mushr

297 The Ptr1 (Pseudomonas tomato race 1) locus in Solanum lycopersicoi

298 the characterization of a bacterial strain, Pseudomonas veronii JW3-6, which was isolated from a mal

299 The Pseudomonas virulence factor (pvf) operon is essential f

300 Here we report that the soil bacteria Pseudomonas vranovensis is a natural pathogen of the nem