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1                                              R. solanacearum RpoS (RpoS(Rso)) was demonstrated to fun
2                                              R. solanacearum synthesized putrescine via a SpeC ornith
3 ogy (IVET), we screened a library of 133 200 R. solanacearum strain K60 promoter fusions and isolated
4 r its efficacy and biosafety profile against R. solanacearum.
5  and L-phenylalaninamide were tested against R. solanacearum, E. coli, Staphylococcus sp. and B. subt
6                                           An R. solanacearum mutant lacking the pathogen's two extrac
7 has in recent years led to the concept of an R. solanacearum species complex.
8 ion accelerated wilt symptom development and R. solanacearum growth and systemic spread.
9 hich includes R. eutropha, R. pickettii, and R. solanacearum.
10                                       Cloned R. solanacearum aer1 and aer2 genes restored aerotaxis t
11 lcaligenes eutrophus) that fully complements R. solanacearum phcA mutants.
12 uce an extracellular factor that complements R. solanacearum mutants deficient in production of the 3
13                            Two site-directed R. solanacearum mutants lacking either CheA or CheW, whi
14 psI operon is the major virulence factor for R. solanacearum.
15 rom 80 mug/ml for X. citri to 600 mug/ml for R. solanacearum and X. euvesicatoria.
16  is not a sole carbon or nitrogen source for R. solanacearum, was enriched 76-fold to 37 microM in R.
17 serve as sole carbon or nitrogen sources for R. solanacearum.
18 gesting that the main location in tomato for R. solanacearum during pathogenesis is iron replete.
19 ng one of the exo-PGs, pehB, was cloned from R. solanacearum K60.
20  effectors, named RipI, is required for full R. solanacearum pathogenicity.
21     Our findings suggest that Brg11 may give R. solanacearum a competitive advantage and uncover a ro
22 file of ipx genes suggests that in its host, R. solanacearum confronts and overcomes a stressful and
23 lomics identified 22 metabolites enriched in R. solanacearum-infected sap.
24                The chromosomal pehB genes in R. solanacearum wild-type strain K60 and in an endo-PG P
25 ided no clues as to the role of acyl-HSLs in R. solanacearum gene regulation.
26 o investigate the role of these acyl-HSLs in R. solanacearum virulence gene expression, transposon mu
27 cearum, was enriched 76-fold to 37 microM in R. solanacearum-infected sap.
28 regulation of flagellum-mediated motility in R. solanacearum.
29 SS2(bp) genes, as well as their orthologs in R. solanacearum.
30  production, whereas inactivation of phcA in R. solanacearum increases siderophore production.
31                     Mutation of rpoS(Rso) in R. solanacearum reduced survival during starvation and l
32 e acyl-HSL-dependent autoinduction system in R. solanacearum is part of a more complex autoregulatory
33                                Therefore, in R. solanacearum the acyl-HSL-dependent autoinduction sys
34 quired for a wild-type level of virulence in R. solanacearum although its individual role in wilt dis
35 ildtype rescued DeltaspeC growth, indicating R. solanacearum produced and exported putrescine to xyle
36 scopy revealed that during tomato infection, R. solanacearum forms biofilm-like masses in xylem vesse
37 ssion of many ipx genes was subject to known R. solanacearum virulence regulators.
38                 While Phc is present in most R. solanacearum strains, it is apparently absent from ot
39 inhibits bacterial niche competitors but not R. solanacearum.
40  contributes significantly to the ability of R. solanacearum to locate and effectively interact with
41        plantarum ZPZ inhibited the growth of R. solanacearum by 72.46 +/- 14.42% based on OD(600) mea
42 and a naturally acyl-HSL-defective strain of R. solanacearum to produce acyl-HSLs.
43 actor (VEF) produced by wild-type strains of R. solanacearum.
44 s the gene encoding the catalytic subunit of R. solanacearum's sole assimilatory nitrate reductase, d
45 erophores present in culture supernatants of R. solanacearum, R. metallidurans, and Bacillus megateri
46 ontributes substantially to the virulence of R. solanacearum.
47                                The predicted R. solanacearum FliM closely resembled motor switch prot
48 y suggest that nitrate assimilation promotes R. solanacearum virulence by enhancing root attachment,
49                      K60 generally resembles R. solanacearum CFBP2957, a Caribbean tomato isolate, bu
50        To locate and infect host plant roots R. solanacearum needs taxis, the ability to move toward
51 wledge, this is the first demonstration that R. solanacearum forms biofilms in plant xylem vessels, a
52 em vessels of infected plants, we found that R. solanacearum is essentially nonmotile in planta, alth
53                                We found that R. solanacearum manipulates its host to increase nutrien
54 graphy, and mass spectroscopy indicated that R. solanacearum produces staphyloferrin B rather than sc
55                                          The R. solanacearum genome encodes two putative aerotaxis tr
56  characterized, and mutated two genes in the R. solanacearum flagellar biosynthetic pathway.
57                    The identification of the R. solanacearum enzyme enables us to propose that the an
58 rtholog of HrpB, the master regulator of the R. solanacearum T3SS (T3SS(rso)) and its secreted effect
59 ositively activates motility, in contrast to R. solanacearum where it represses motility.
60  of unknown function, and 13% were unique to R. solanacearum.
61 terial species that were antagonistic toward R. solanacearum.
62 lates virulence gene expression in wild-type R. solanacearum.
63 sal bacteria (not positively correlated with R. solanacearum) but not efficiently used by the pathoge