戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (left1)

通し番号をクリックするとPubMedの該当ページを表示します
1                                              RNA pol III activity is unchanged in CycD2(-/-) myocardi
2 RNA (TMER) genes that are transcribed from a RNA pol III type 2-like promoter containing triplicated
3 se (pol) III mafr-1 has been shown to affect RNA pol III transcript abundance, lipid biosynthesis and
4 romoters also show enrichment of alternative RNA pol III transcription termination sequences and are
5           Our results show that all analysed RNA pol III expression cassettes (tRNA, U6, Ad VA1), reg
6                  Induction of TBP, Brf1, and RNA pol III-dependent gene expression is driven by enhan
7  study may inform similar roles for Maf1 and RNA pol III in mammalian male fertility.
8  and point to an important role for MAF1 and RNA pol III-mediated transcription in chemosensitivity a
9 ese results reveal a novel role for MAF1 and RNA pol III-mediated transcription in osteoblast fate de
10 role for SUMOylation in controlling Maf1 and RNA pol III-mediated transcription.
11 Given the emerging roles of SENP1, Maf1, and RNA pol III transcription in oncogenesis, our studies su
12 f1 can inhibit the recruitment of TFIIIB and RNA pol III to immobilized templates.
13 sociation of Brf1 complexes with TFIIIC2 and RNA pol III.
14  by chemical inhibition or knockdown of BRF1 RNA pol III transcription initiation factor subunit (BRF
15                         Proper initiation by RNA pol III requires the transcription factor TFIIIB.
16  structure enriched with RNAs transcribed by RNA pol III and RNA binding proteins.
17 at many polytene chromosome sites containing RNA pol III genes.
18 doxorubicin resistance in HCC by controlling RNA pol III-dependent transcription.
19                          However, decreasing RNA pol III transcription through these perturbations en
20 miting or stress conditions are encountered, RNA pol III transcription is rapidly repressed through t
21 pressors and oncogenic proteins and enhanced RNA pol III transcription is essential for cellular tran
22 TA-binding protein or c-Myc display enhanced RNA pol III transcription.
23 her, these results demonstrate that enhanced RNA pol III transcription is essential for anchorage-ind
24                                     However, RNA pol III bound to preinitiation complexes or in elong
25 ression and facilitated recycling in a human RNA pol III in vitro system.
26                          RNA polymerase III (RNA pol III) transcribes structural RNAs involved in RNA
27                This load-induced increase in RNA pol III activity is augmented in Rb-deficient hearts
28 ption factor Brf1 prevented this increase in RNA pol III transcription.
29                    Furthermore, increases in RNA pol III-dependent gene activity and TBP levels could
30  complex with BRF that plays a major role in RNA pol III transcription.
31                                    Increased RNA pol III-dependent transcription, observed in transfo
32 uitment to chromatin, resulting in increased RNA pol III occupancy and tRNA expression in cancers.
33                              Ethanol induces RNA pol III-dependent transcription in both HepG2 cells
34  regulator of the load-induced activation of RNA pol III.
35 ts ability to facilitate the dissociation of RNA pol III from these promoters.
36 nscription factor, TBP, and the induction of RNA pol III-dependent gene activity.
37 demonstrate that the X-mediated induction of RNA pol III-dependent genes and increase in TBP are both
38 t chimeric RNAs transcribed from a number of RNA pol III promoters.
39 ults show that transcriptional regulation of RNA pol III and the coordinate control of ribosomal prot
40 C1 and FASN, but with impaired repression of RNA pol III targets.
41 ssary for rapid Maf1-dependent repression of RNA pol III transcription.
42                                Repression of RNA pol III-dependent transcription by chemical inhibiti
43 suggest that BRCA1 is a general repressor of RNA pol III transcription.
44 his report is the first comparative study of RNA pol III-driven promoters expressing short chimeric t
45  TATA box binding protein (TBP), subunits of RNA pol III-specific transcription factor B, in adult my
46  the ability to inhibit the transcription of RNA pol III targets, reduce lipid biogenesis, and lower
47 ilization; a phenotype that was dependent on RNA pol III activity.
48     The different modalities used to perturb RNA pol III transcription resulted in distinct gene expr
49                     Deletion of Sch9 reduces RNA pol III transcription in a Maf1-dependent manner, ye
50 provide a molecular mechanism for regulating RNA pol III transcription through the coordinate control
51 n is of functional importance for regulating RNA pol III-transcribed genes.
52 s, the roles of these proteins in regulating RNA pol III transcription were examined.
53 However, other perturbations used to repress RNA pol III transcription, inhibited osteoblast differen
54 ss or reduction in JNK1 expression represses RNA pol III transcription.
55  results support the idea that p53 represses RNA pol III transcription through direct interactions wi
56 rtant new role for SUMOylation in repressing RNA pol III-dependent transcription.
57  BRCA1 inhibits both VAI (tRNA) and U6 snRNA RNA pol III transcription; (2) the AD1 of BRCA1 is respo
58 h overexpression of Brf1 modestly stimulated RNA pol III transcription, expression of a phosphomimic,
59 on and controls its stability and subsequent RNA pol III-dependent transcription.
60         Although it is well established that RNA pol III-dependent transcription is deregulated in tr
61                   Hence, we hypothesize that RNA pol III may be regulated by BRCA1 via the TFIIB fami
62 NK1 positively regulates TBP expression, the RNA pol III-specific factors, Brf1 and Bdp1, JNK2 negati
63 sion levels can be achieved by inserting the RNA pol III expression cassette into the U3 region of th
64 f TARE-6 proceeded in the orientation of the RNA pol III promoter of the Alu dimer and opposite to th
65                  Decreased expression of the RNA pol III-specific transcription factor Brf1 prevented
66                                 In yeast the RNA pol III transcription machinery bound to tRNA genes
67                  We also identify that these RNA pol III type 2-like promoters are conserved in eukar
68 s defective in its ability to associate with RNA pol III.
69      In contrast, gene delivery systems with RNA pol III-based expression cassettes have not been stu
70 protein, the second largest subunit of yeast RNA pol III.