ライフサイエンスコーパス: RNA polymerase II

1 the recruitment of chromatin remodelers and RNA polymerase II.

2 A repair, mRNA processing, and regulation of RNA polymerase II.

3 hunt for factors that regulate elongation by RNA polymerase II.

4 y dephosphorylating the C-terminal domain of RNA polymerase II.

5 on of most, if not all, genes transcribed by RNA Polymerase II.

6 RNP interacts with transcriptionally engaged RNA polymerase II.

7 xygenase-1 locus and promotes recruitment of RNA polymerase II.

8 n factors and transcriptional machinery like RNA Polymerase II.

9 n chromatin interaction networks mediated by RNA polymerase II.

10 ly related to the C-terminal domain (CTD) of RNA polymerase II.

11 cription by linking transcription factors to RNA polymerase II.

12 the biochemical analysis of transcription by RNA polymerase II.

13 o the expression of all genes transcribed by RNA polymerase II.

14 regulatory element for genes transcribed by RNA polymerase II.

15 and in the transcription of coding genes by RNA polymerase II.

16 st cancer cells by increasing recruitment of RNA polymerase II.

17 ectly stimulates transcription elongation by RNA polymerase II.

18 1 (DCL1) and Hyponastic Leaves 1 (HYL1) with RNA Polymerase II.

19 and operates by recruiting and/or initiating RNA Polymerase II.

20 s interactions with the C-terminal domain of RNA polymerase II.

21 y modulating the association of Mediator and RNA polymerase II.

22 ex, which regulates transcription pausing of RNA-polymerase II.

23 ple-arise from Bre1 and Rad6 travelling with RNA polymerase II(2), the mechanism of H2B ubiquitinatio

24 co-binding of the tumor suppressor BRCA1 and RNA polymerase II, a well-known transcriptional pair in

25 nhancers and super-enhancers, and leading to RNA polymerase II activation and expression of downstrea

26 ion, and none of them affected host cellular RNA polymerase II activities.

27 ty controls transcription factor binding and RNA polymerase II activity, validating a mechanism propo

28 reversible association and disassociation of RNA polymerase II and associated co-factors from genes a

29 at transcriptionally active regions bound by RNA polymerase II and Brahma, its recruitment to the tra

30 RNA methylation machinery, the NuRD complex, RNA polymerase II and factors involved in the regulation

31 einitiation complex (PIC), which consists of RNA polymerase II and general transcription factors.

32 omain (CTD) of the largest subunit (RPB1) of RNA polymerase II and is essential for the transition fr

33 mentalization of the gene-control machinery, RNA polymerase II and its cofactors, within biomolecular

34 lex critical for transcription initiation by RNA polymerase II and nucleotide excision DNA repair.

35 TFIID binds promoter DNA to recruit RNA polymerase II and other basal factors for transcript

36 osphorylating the carboxy-terminal domain of RNA polymerase II and selectively affects the expression

37 ly the method to subunits Rpb1-Rpb2 of yeast RNA polymerase II and subunits RpoB-RpoC of bacterial RN

38 ntified transcriptome-wide binding sites for RNA polymerase II and the exosome cofactors Mtr4 (TRAMP

39 through chromatin looping and by recruiting RNA polymerase II and the histone-code modifiers p300 an

40 Ubp15 interacts with both RNA polymerase II and the nuclear pore complex, and its

41 demonstrate interaction between MTA and both RNA Polymerase II and TOUGH (TGH), a plant protein neede

42 CUT&Tag by profiling histone modifications, RNA Polymerase II and transcription factors on low cell

43 from HS2 of major transcription factors and RNA Polymerase II, and consequently in loss of factors a

44 art site of genes occupied by high levels of RNA polymerase II, and terminates at their polyadenylati

45 esized at DNA double-strand breaks (DSBs) by RNA polymerase II are necessary for DNA-damage-response

46 ive viral transcription by focal assembly of RNA polymerase II around Kaposi's sarcoma-associated her

47 ive heterochromatin, and the organization of RNA polymerase II around transcription factories.

48 small nucleolar RNAs that are transcribed by RNA Polymerase II as precursors, and whose 5' and 3' end

49 plex (PAF1C), a transcriptional regulator of RNA polymerase II, as a suppressor of G4C2-associated to

50 One of the transcription factors, the PAF1C (RNA polymerase II associated factor 1 complex) is report

51 In absence of HBV replication, RNA polymerase II associated with SALL4 exon1.

52 PAF1, a RNA polymerase II-associated factor 1 complex (PAF1C) co

53 function of VIP proteins, components of the RNA polymerase II-associated factor 1 complex (Paf1c).

54 F1 in CSC maintenance was independent of its RNA polymerase II-associated factor 1 complex component

55 e same strand by promoting the 3' pausing of RNA polymerase II at the upstream gene.

56 AM1 promoter resulted in graded RelA/p65 and RNA polymerase II binding that gave rise to a distributi

57 ty of the epidermis, HNRNPK is necessary for RNA Polymerase II binding to proliferation/self-renewal

58 Rapid reduction of RNA polymerase II binding was accompanied by reduced bin

59 ediated C-terminal domain phosphorylation of RNA polymerase II both in vitro and in vivo.

60 anscriptional machinery, including NCOA3 and RNA polymerase II, but does not alter AR binding itself.

61 anscription factor II H (TFIIH) it activates RNA polymerase II by hyperphosphorylation of its C-termi

62 In addition, through its interaction with RNA Polymerase II C-terminal domain (CTD) and affecting

63 Post-translational modifications of the RNA polymerase II C-terminal domain (CTD) coordinate the

64 res conversion of an arginine residue in the RNA polymerase II C-terminal domain (CTD) to citrulline,

65 Set1/COMPASS associates with the RNA polymerase II C-terminal domain (CTD) to establish p

66 The RNA polymerase II carboxyl terminal domain (RNAPII CTD)

67 that both enhancer classes are enriched for RNA Polymerase II, CBP, and architectural proteins but t

68 nhancers and other cis-regulatory regions by RNA polymerase II chromatin interaction analysis by pair

69 Although transcribed by RNA polymerase II, cluster transcripts lack splicing sig

70 The RNA polymerase II complex (pol II) is responsible for tr

71 0454) disrupts BRD4 binding to the NF-kappaB-RNA polymerase II complex and inhibits its histone acety

72 ologous end-joining pathway factor, that the RNA polymerase II component ELOF1 modulates the response

73 The C-terminal domain (CTD) of RNA polymerase II contains a repetitive heptad sequence

74 s influenced by the Thr4 phospho-site in the RNA polymerase II CTD and the 3' processing/termination

75 the structure and function of SSU72 homolog, RNA polymerase II CTD phosphatase (Ssu72, from Drosophil

76 Alpha-satellite expression occurs through RNA polymerase II-dependent transcription, but does not

77 complex as a terminator of promoter-proximal RNA polymerase II during piRNA biogenesis.

78 nd thereby reduced the recruitment of active RNA polymerase II during transcription initiation.

79 bly by controlling Integrator recruitment or RNA polymerase II dynamics.

80 effects of THZ1 and other CDK inhibitors on RNA polymerase II dynamics.

81 RNA polymerase II elongation complexes (ECs) were assemb

82 MeCP2 binding to methylated miRNA loci halts RNA polymerase II elongation, leading to enhanced proces

83 this region exhibited basal eRNA production, RNA polymerase II enrichment, and looping to the TSS, pl

84 NA degradation and larger Ser2p CTD-modified RNA polymerase II foci.

85 We hypothesize that waves of slowed-down RNA polymerase II formed behind these sites travel backw

86 Processive elongation of RNA Polymerase II from a proximal promoter paused state

87 lex, which plays key roles in the release of RNA polymerase II from promoter-proximal pausing and in

88 matin marks at the IFNG locus, but displaces RNA polymerase II from the locus.

89 R 4b (TAF4b), which encodes a subunit of the RNA polymerase II general transcription factor TFIID.

90 n addition to phosphodiester bond formation, RNA polymerase II has an RNA endonuclease activity, stim

91 n cryo-electron microscopy map of a Mediator-RNA polymerase II holoenzyme reveals that changes in the

92 Second, in eight tested cell types, RNA polymerase II IMPACT annotations capture more cis-eQ

93 scernable decrease in the elongating form of RNA polymerase II in either mutant.

94 g signals from transcriptional regulators to RNA polymerase II in eukaryotes.

95 Here, we show that IR associates with RNA polymerase II in the nucleus, with striking enrichme

96 r1 phosphorylation causes an accumulation of RNA polymerase II in the promoter region as detected by

97 viability, development, and the dynamics of RNA polymerase II in vivo.

98 In animals, RNA polymerase II initiates transcription bidirectionall

99 etylated regions are formed after inhibiting RNA polymerase II initiation.

100 Transcribing RNA Polymerase II interacts with multiple factors that o

101 RNA polymerase II interacts with various other complexes

102 eport that zinc finger protein ZPR1 binds to RNA polymerase II, interacts in vivo with SMN locus and

103 The molecular process of transcription by RNA Polymerase II is highly conserved among eukaryotes (

104 matin occupancy of serine 2-unphosphorylated RNA polymerase II is increased, and that of topoisomeras

105 Gene transcription by RNA polymerase II is regulated by activator proteins tha

106 ent recruitment of transcription factors and RNA polymerase II leads to conventional patterns of dive

107 hment at HIF target gene promoters increased RNA polymerase II loading through p300.

108 We propose that such binding may block RNA polymerase II-mediated transcription.

109 itical for the fidelity and effectiveness of RNA polymerase II-mediated transcription.

110 igned with its capacity to primarily augment RNA polymerase II-mediated transcriptional elongation, b

111 ependent kinase (Cdk8) is a component of the RNA polymerase II Mediator complex that predominantly re

112 d kinetics of post-translational histone and RNA polymerase II modifications.

113 trong divergent transcription, together with RNA Polymerase II occupancy and an increase in DNA acces

114 heat shock they display increased HSF-1 and RNA polymerase II occupancy and up-regulation of nearby

115 We further show that KDM2 proteins shape RNA Polymerase II occupancy but not chromatin accessibil

116 demonstrate that PTEN modulates genome-wide RNA Polymerase II occupancy in cells undergoing glucose

117 was coordinate with increased P300, BRG1 and RNA polymerase II occupancy.

118 co-activator, DDX5-mediated stabilization of RNA polymerase II on chromatin.

119 f NF-kB, P-TEFb, and serine 2 phosphorylated RNA Polymerase II on the HEXIM1 gene.

120 vators CRTC2 and BRD4, leading to release of RNA polymerase II over the target gene body.

121 , we show that CDK8 kinase activity promotes RNA polymerase II pause release in response to interfero

122 we report that widespread promoter-proximal RNA polymerase II pausing in resting macrophages is mark

123 We found that integrator and NELF, an RNA polymerase II pausing protein, were associated with

124 depends on sequence signals associated with RNA polymerase II pausing.

125 tiotemporal relationship between R-loops and RNA polymerase II pausing/pause release, as well as link

126 eads to CDK7 phosphorylation, which promotes RNA Polymerase II phosphorylation and transcription.

127 ib, THZ1, and LDC4297 lead to a reduction in RNA Polymerase II phosphorylation on the SLC2A1 promoter

128 Surprisingly, NXF1 downregulation results in RNA polymerase II (Pol II) accumulation at the 3' end of

129 d by reduced remodeler binding and increased RNA polymerase II (Pol II) activity.

130 RNA polymerase II (Pol II) and its general transcription

131 any promoters in mouse sperm are occupied by RNA polymerase II (Pol II) and Mediator.

132 nd found that H2A.Z eviction is dependent on RNA Polymerase II (Pol II) and the Kin28/Cdk7 kinase, wh

133 RNA Polymerase II (Pol II) and transcription factors for

134 The journey of RNA polymerase II (Pol II) as it transcribes a gene is a

135 gene expression or transcription factor and RNA polymerase II (Pol II) association with viral DNA pr

136 al mammary cells form upon release of paused RNA polymerase II (Pol II) at promoters, 5' splice sites

137 ription of eukaryotic mRNA-encoding genes by RNA polymerase II (Pol II) begins with assembly of the p

138 tacc-seq), we investigated the landscapes of RNA polymerase II (Pol II) binding in mouse embryos.

139 The vRNAP core enzyme resembles eukaryotic RNA polymerase II (Pol II) but also reveals many virus-s

140 ylates the carboxyl-terminal domain (CTD) of RNA polymerase II (pol II) but its roles in transcriptio

141 te the degradation of Rpb1, a subunit of the RNA polymerase II (Pol II) complex, and therefore hamper

142 The RNA polymerase II (Pol II) core promoter is the strategi

143 The former is regulated by RNA polymerase II (pol II) de novo recruitment or loss;

144 After synthesis of a short nascent RNA, RNA polymerase II (pol II) dissociates general transcrip

145 romatin organization correlates with altered RNA polymerase II (Pol II) distribution.

146 Pausing of RNA polymerase II (Pol II) during early transcription, m

147 S-PP1 phosphatase is a negative regulator of RNA polymerase II (Pol II) elongation rate.

148 nsation, KAS-seq uncovers a rapid release of RNA polymerase II (Pol II) from a group of promoters.

149 The transition of RNA polymerase II (Pol II) from initiation to productive

150 inal domain (CTD) of the RPB1 subunit of the RNA polymerase II (Pol II) has been revived in recent ye

151 either T7 RNA polymerase (T7 RNAP) or human RNA polymerase II (pol II) have inhibitory and mutagenic

152 ps in gene regulation involve the pausing of RNA polymerase II (Pol II) in early elongation, and the

153 mere regions retain transcriptionally active RNA polymerase II (Pol II) in mitosis.

154 Here we show, however, that RNA polymerase II (Pol II) inside human nucleoli operate

155 The synthesis of pre-mRNA by RNA polymerase II (Pol II) involves the formation of a t

156 nscription, and promoter-proximal pausing of RNA polymerase II (Pol II) is a critical step in transcr

157 Transcription by RNA polymerase II (Pol II) is carried out by an elongati

158 The C-terminal domain (CTD) of RNA polymerase II (Pol II) is composed of repeats of the

159 Transcription by RNA polymerase II (Pol II) is dictated in part by core p

160 cates within the nucleus coopting the host's RNA Polymerase II (Pol II) machinery for production of v

161 Condensates containing RNA polymerase II (Pol II) materialize at sites of activ

162 esent mechanical and energetic barriers that RNA Polymerase II (Pol II) must overcome during transcri

163 t its disruption manifests as a reduction of RNA polymerase II (Pol II) occupancy downstream of trans

164 minal domain (CTD) of the largest subunit of RNA polymerase II (Pol II) orchestrates dynamic recruitm

165 istone H2B ubiquitination (H2Bub) facilitate RNA polymerase II (Pol II) passage through chromatin, ye

166 al regions wherein transcriptionally engaged RNA polymerase II (Pol II) pauses before proceeding towa

167 RNA polymerase II (Pol II) pauses downstream of the tran

168 The phenomenon of RNA polymerase II (Pol II) pausing at transcription star

169 RNA polymerase II (Pol II) pausing is a general regulato

170 RNA polymerase II (Pol II) pausing is a key regulatory s

171 To better understand human RNA polymerase II (Pol II) promoters in the context of p

172 at stimuli predominantly affect the rates of RNA polymerase II (Pol II) recruitment and polymerase re

173 Active NL genes with higher RNA polymerase II (Pol II) recruitment levels tend to di

174 iption is regulated at many steps, including RNA polymerase II (Pol II) recruitment, transcription in

175 C-terminal heptapeptide repeat domain of the RNA polymerase II (Pol II) subunit RPB1, which is an imp

176 depends on many factors that together direct RNA polymerase II (pol II) through the different stages

177 nd that prp5 alleles decrease recruitment of RNA polymerase II (Pol II) to an intron-containing gene,

178 tory step in gene expression, which requires RNA polymerase II (pol II) to escape promoter proximal p

179 In eukaryotes, RNA polymerase II (pol II) transcribes all protein-codin

180 RNA polymerase II (Pol II) transcribes all protein-codin

181 RNA polymerase II (Pol II) transcribes hundreds of thous

182 NA-binding motif protein 7 (RBM7) stimulates RNA polymerase II (Pol II) transcription and promotes ce

183 factor Paf1C regulates several stages of the RNA polymerase II (Pol II) transcription cycle, although

184 The RNA polymerase II (Pol II) transcription elongation fact

185 scription factor that stimulates the rate of RNA polymerase II (Pol II) transcription elongation in v

186 RNA polymerase II (Pol II) transcription is tightly regu

187 The pervasive nature of RNA polymerase II (Pol II) transcription requires effici

188 to a lesser extent, exon-targeted ASOs cause RNA polymerase II (Pol II) transcription termination in

189 ption involves the complex interplay between RNA polymerase II (Pol II), regulatory factors (RFs), an

190 ribed from > 15,000 discrete genomic loci by RNA polymerase II (Pol II), resulting in 28 nt short-cap

191 ociation between influenza RdRP and cellular RNA polymerase II (Pol II), which is the source of nasce

192 we show that hepatocyte-specific ablation of RNA polymerase II (Pol II)-associated Gdown1 leads to do

193 on, resulting in degradation of the residual RNA polymerase II (Pol II)-associated RNA by XRN2 and di

194 binding and recruitment of coactivators and RNA polymerase II (Pol II).

195 ipt invades the DNA duplex behind elongating RNA polymerase II (Pol II).

196 SHM requires IgV transcription by RNA polymerase II (Pol II).

197 template strand that block translocation of RNA polymerase II (Pol II).

198 1 (HSV-1) genes are transcribed by cellular RNA polymerase II (Pol II).

199 ls from DNA-binding transcription factors to RNA polymerase II (Pol II).

200 f serine-2 in the C-terminal domain (CTD) of RNA-polymerase II (Pol II), and reduces the expression o

201 RNA polymerase II (Pol2) movement through chromatin and

202 functional promoters that include a complete RNA polymerase II preinitiation complex, MED1 and CDK9.

203 modifications and RNA methylation, regulate RNA polymerase II processivity, co-transcriptional splic

204 acterium ND2006 (Lb) in plants, using a dual RNA polymerase II promoter expression system.

205 the best-studied cis-regulatory elements in RNA polymerase II promoters and enhancers have variable

206 e guide RNA (sgRNA) expression strategy with RNA polymerase II promoters.

207 sses such as transcription factor occupancy, RNA polymerase II recruitment and initiation, nascent tr

208 ve historically focused on events leading to RNA polymerase II recruitment and transcription initiati

209 romatin marks were associated with increased RNA polymerase II recruitment to the ATF3 promoter, a sy

210 Tat-TAR inhibition results in loss of RNA polymerase II recruitment to the SIV promoter.

211 duction of TNF and LTA mRNA synthesis and of RNA polymerase II recruitment to their promoters.

212 nd 13 phosphorylate the C-terminal domain of RNA polymerase II, regulating transcription and co-trans

213 ar multisubunit RNA polymerases IV and V are RNA Polymerase II-related enzymes that synthesize non-co

214 ing of tissues to the mechanistic control of RNA polymerase II remains a long-term goal of developmen

215 Fusing Set1 to RNA polymerase II results in H3K4me2 throughout transcri

216 RNA polymerase II (RNA Pol II) contains a disordered C-t

217 Precise control of the RNA polymerase II (RNA Pol II) cycle, including pausing

218 Hyperacetylation removes RNA polymerase II (RNA Pol II) from core regulatory gene

219 RNA polymerase II (RNA Pol II) is generally paused at pr

220 ore increases Mediator-driven recruitment of RNA polymerase II (RNA Pol II) to promoters and enhancer

221 s with transcription factor GATA2 to promote RNA polymerase II (RNA-POL-II) recruitment and activate

222 sphorylation within the C-terminal domain of RNA polymerase II (RNAP II) and in the recruitment of th

223 Here, we report that ATXN3 associates with RNA polymerase II (RNAP II) and the classical nonhomolog

224 of key residues at the C-terminal domain of RNA polymerase II (RNAP2-CTD) coordinates transcription,

225 t by positioning and stabilizing stalling of RNA polymerase II (RNAPII) at DNA lesions.

226 The RNA polymerase II (RNAPII) C-terminal domain kinase, CDK

227 The rate of RNA polymerase II (RNAPII) elongation has an important r

228 le 1-beta-D-ribofuranoside (DRB), to measure RNA polymerase II (RNAPII) elongation rates in vivo, a t

229 rosine kinase c-Abl/ABL1 causes formation of RNA polymerase II (RNAPII) foci, predominantly phosphory

230 nd phosphorylating the C-terminal domain for RNA polymerase II (RNAPII) for activation.

231 nitiation and regulation of transcription by RNA polymerase II (RNAPII) in eukaryotes rely on the tra

232 ption of transcription termination (DoTT) of RNA polymerase II (RNAPII) in host genes.

233 transcript elongation of subsets of genes by RNA polymerase II (RNAPII) in the chromatin context.

234 Transcription by RNA polymerase II (RNAPII) is a dynamic process with fre

235 RNA polymerase II (RNAPII) passes through the nucleosome

236 Given that the elongating RNA polymerase II (RNAPII) stalls at this well positione

237 elongation factors associate with elongating RNA polymerase II (RNAPII) to control the efficiency of

238 In eukaryotes, RNA polymerase II (RNApII) transcribes messenger RNA fro

239 e that scaling relies on the coordination of RNA polymerase II (RNAPII) transcription initiation rate

240 RNA polymerase II (RNAPII) transcription is governed by

241 Although correlations between RNA polymerase II (RNAPII) transcription stress, R-loops

242 sphorylated carboxy-terminal domain (CTD) of RNA polymerase II (RNAPII) using RGG motifs in its low-c

243 tant ulp2 cells show impaired association of RNA polymerase II (RNAPII) with, and diminished expressi

244 inase 9 (Cdk9) occur during transcription by RNA polymerase II (RNAPII), and are mutually dependent i

245 scription-coupled DNA repair, degradation of RNA polymerase II (RNAPII), and genome-wide transcriptio

246 e 2 (Ser2) of the carboxy-terminal domain of RNA polymerase II (RNAPII), which is initiated when RNAP

247 Ccr4-Not associates with elongating RNA polymerase II (RNAPII), which raises the possibility

248 Elongin is an RNA polymerase II (RNAPII)-associated factor that has be

249 recruited to DNA damage sites in a PARP- and RNA polymerase II (RNAPII)-dependent manner.

250 In vitro, TRIM28, together with the RNA polymerase II (RNAPII)-interacting protein RECQ5, pr

251 Regulation of RNA polymerase II (RNAPII)-mediated transcription contro

252 inding complex (CBC) binds to the 5' caps of RNA polymerase II (RNAPII)-synthesized transcripts and s

253 osphorylating the C-terminal domain (CTD) of RNA polymerase II (RNAPII).

254 ex(3), concordant with increased stalling of RNA polymerase II (RNAPII).

255 t of PRC-bound genes actively transcribed by RNA polymerase II (RNAPII).

256 feration and migration, and to interact with RNA Polymerase II (RNAPII).

257 the fate of many nascent RNAs transcribed by RNA polymerase II (RNAPII).

258 Thus, it is perplexing how RNA-polymerase II (RNAPII) can successfully transcribe t

259 n assays (examining histones, accessibility, RNA polymerase II [RNAPII], and transcription factor bin

260 The response to DNA damage-stalled RNA polymerase II (RNAPIIo) involves the assembly of the

261 e, we integrate mRNA sequencing, genome-wide RNA polymerase II (RNPII), chromatin immunoprecipitation

262 horibosyltransferase 1 (HPRT1), DNA-directed RNA polymerase II (RPB2), 18S ribosomal RNA (18S), 28S r

263 increased binding of total and phospho-Ser2 RNA polymerase II specifically at the intron retained un

264 iption-coupled DNA repair (TCR) complex with RNA polymerase II subunit A (POLR2A), ataxin-3, the DNA

265 ogy was found across the range of eukaryotic RNA polymerase II subunits and their associated basal tr

266 How the core subunits of Pol IV, homologs of RNA polymerase II subunits, diverged to support siRNA bi

267 Upon inhibiting RNA polymerase II termination via depletion of the cleav

268 horylation states of Ser(2)/Ser(5) of CTD in RNA polymerase II that occur at different stages of tran

269 Initially devoid of RNA polymerase II, the accessible chromatin domains late

270 tes the binding of the m(6)A MTC to adjacent RNA polymerase II, thereby delivering the m(6)A MTC to a

271 y decreased recruitment of NF-kappaB p65 and RNA polymerase II to COX-2 and IL-8 promoters.

272 5 condensates, which in turn further recruit RNA polymerase II to drive transcriptional output.

273 lex, which bridges transcription factors and RNA polymerase II to facilitate transcription initiation

274 Recruitment of RNA polymerase II to hypoxia-inducible factor (HIF) targ

275 14 (H3K14ac) facilitates the processivity of RNA polymerase II to maintain the high expression of key

276 us possesses an upstream motif that recruits RNA polymerase II to produce an ~28 nt primary transcrip

277 d link the SnRK2.6-mediated ABA signaling to RNA polymerase II to promote immediate transcriptional r

278 s, regulating their expression by recruiting RNA polymerase II to promoters for productive transcript

279 sing H3K4me3, transcription start sites, and RNA polymerase II to represent the mass value in the mod

280 robably through promoting the recruitment of RNA polymerase II to their promoters.

281 that TOE1 promotes maturation of all regular RNA polymerase II transcribed snRNAs of the major and mi

282 guanosine cap is a quintessential feature of RNA polymerase II-transcribed RNAs, and a textbook aspec

283 and Drosophila cells, splicing occurs after RNA polymerase II transcribes several kilobases of pre-m

284 xpressed from an intron that is generated by RNA polymerase II transcribing the circular viral genome

285 accessible chromatin domains later acquired RNA polymerase II, transcribing RNA.

286 Compared to other stages in the RNA polymerase II transcription cycle, the role of chrom

287 The human Mediator complex regulates RNA polymerase II transcription genome-wide.

288 e specifically associated with initiation of RNA Polymerase II transcription of highly expressed gene

289 d Myc mRNA in AGO1-depleted animals requires RNA polymerase II transcription.

290 o conserved epigenetic marks associated with RNA polymerase II transcription.

291 lin-dependent kinases play multiple roles in RNA polymerase II transcription.

292 TBPL2 (TBP2 or TRF3), which is essential for RNA polymerase II transcription.

293 is provided by physical interaction with the RNA polymerase II transcriptional machinery (chromatin r

294 NCBP2), associates with the nascent 5'cap of RNA polymerase II transcripts and impacts RNA fate decis

295 ates distinct isoforms of mRNAs and/or other RNA polymerase II transcripts with different 3'UTR lengt

296 osine cap structure on the 5' end of nascent RNA polymerase II transcripts.

297 of the length and sequence complexity of the RNA polymerase II unstructured C-terminal domain in anim

298 rucial to the regulation of transcription by RNA-polymerase II, via its interaction with the positive

299 mixed-lineage leukemia protein 4 (MLL4), and RNA polymerase II were recruited to the GREB1 enhancer a

300 The elongating form of RNA polymerase II, which is phosphorylated at Ser2 in he