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1 ructure present in the lipopolysaccharide of Ralstonia solanacearum.
2 ps4 from Pseudomonas syringae and PopP2 from Ralstonia solanacearum.
3 effective in another P. syringae strain and Ralstonia solanacearum.
4 ound in phytopathogenic Xanthomonas spp. and Ralstonia solanacearum.
5 ella burnetii, as well as the plant pathogen Ralstonia solanacearum.
6 ltiple virulence genes in the plant pathogen Ralstonia solanacearum.
7 m Xanthomonas campestris pv. vesicatoria and Ralstonia solanacearum.
8 e similarity was with Ralstonia eutropha and Ralstonia solanacearum.
9 a TALE-like effector from the plant pathogen Ralstonia solanacearum.
10 t invasion by the plant-pathogenic bacterium Ralstonia solanacearum.
11 dopsis thaliana (Arabidopsis), Resistance to Ralstonia solanacearum 1 (RRS1-R) and Resistance to Pseu
13 ogenetic lineages, to suppress the bacterium Ralstonia solanacearum, a global phytopathogen capable o
15 lation of tomato (Solanum lycopersicum) with Ralstonia solanacearum, a soilborne pathogen that causes
18 vorite pathogens, Phytophthora infestans and Ralstonia solanacearum, among others, are considered.
21 DNase activity in plant-pathogenic bacteria (Ralstonia solanacearum) and fungi (Cochliobolus heterost
22 binding module, a fucose-binding lectin from Ralstonia solanacearum, and human norovirus VA387 P part
23 icum) suffering from wilt disease (caused by Ralstonia solanacearum) as source for potential prebioti
25 sp. and Cercomonas sp.) affect the pathogen Ralstonia solanacearum, both on individual beneficial ba
29 tect tomato plants against the phytopathogen Ralstonia solanacearum in a T4BSS-dependent manner, sugg
30 ct invasion resistance to the plant pathogen Ralstonia solanacearum in microcosms and in tomato plant
31 YopJ effector produced by the plant pathogen Ralstonia solanacearum, in complex with inositol hexapho
40 ropeller formed by oligomerization as in the Ralstonia solanacearum lectin and not by sequential doma
41 environmental variability, the biocontrol of Ralstonia solanacearum, one of the most destructive plan
42 large inhibition zones were produced against Ralstonia solanacearum only when grown in the presence o
44 iens, Pantoea stewartii, Erwinia carotovora, Ralstonia solanacearum, Pseudomonas syringae, Pseudomona
49 ecreted effector from the bacterial pathogen Ralstonia solanacearum, targets the plant E3 ubiquitin l
51 resistance against Pseudomonas syringae and Ralstonia solanacearum through activation of elicitor-me
52 eudomonas syringae and the vascular pathogen Ralstonia solanacearum Thus, the GFP strand system can b
54 tein secreted by the bacterial wilt pathogen Ralstonia solanacearum, undergoes phosphorylation at spe
56 Pseudomonas syringae pv. pisi and PopP2 from Ralstonia solanacearum via an integrated WRKY domain in
57 assessed the growth of a bacterial invader, Ralstonia solanacearum, when introduced into communities
59 winia amylovora, Pectobacterium carotovorum, Ralstonia solanacearum, Xanthomonas campestris, Xanthomo