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1 tom characteristic of dcSSc with and without Raynaud's phenomenon).
2 ing significant changes in the management of Raynaud's phenomenon.
3 sure, myopic refractive error (for NTG), and Raynaud's phenomenon.
4 y hypertension are also showing an effect in Raynaud's phenomenon.
5 occupational origin and a form of secondary Raynaud's phenomenon.
6 anoids are being tested for the treatment of Raynaud's phenomenon.
7 disease, including risk of hypertension and Raynaud's phenomenon.
8 itical digital ischemia) in the treatment of Raynaud's phenomenon all determined improvement of sympt
10 er time; features of inflammation as well as Raynaud's phenomenon and esophageal hypomotility diminis
11 gives an update of the current management of Raynaud's phenomenon and its ischaemic complications (di
13 551 and +1922 demonstrated correlations with Raynaud's phenomenon and pulmonary fibrosis, respectivel
14 have been reported into the pathogenesis of Raynaud's phenomenon and the consequences of ischemia.
16 ancy, she developed multiple autoantibodies, Raynaud's phenomenon, and fetal death occurred at 20 wee
17 linically the condition differs from primary Raynaud's phenomenon as persistent pain and paresthesia
18 discrimination between primary and secondary Raynaud's phenomenon as well as a better way to predict
21 l randomized trials of PDE5Is, patients with Raynaud's phenomenon demonstrated improved blood flow, f
24 It is characterized by calcinosis cutis, Raynaud's phenomenon, esophageal involvement, sclerodact
26 y is likely to confer benefit in SSc-related Raynaud's phenomenon, further research is required to co
27 (VAS) to evaluate SSc organ system symptoms, Raynaud's phenomenon, gastrointestinal (GI) tract and lu
28 al studies of large cohorts of patients with Raynaud's Phenomenon have addressed the predictors of de
29 95% CI, 1.1 to 2.4, respectively), developed Raynaud's phenomenon (HR, 1.9; 95% CI, 1.1 to 3.6) or dy
30 uggested that more than one defect may cause Raynaud's phenomenon, including increased alpha-2 sympat
33 p1 were white women with fatigue, arthritis, Raynaud's phenomenon, malar rash, and photosensitivity.
35 fication criteria in less than one year from Raynaud's Phenomenon onset (Early Onset Subset-EOS), and
37 d artery stiffness as well as prevalences of Raynaud's phenomenon, pulmonary hypertension, and athero
39 d dosage, in 210 patients with lcSSc or with Raynaud's phenomenon (RP) and the presence of SSc-specif
44 e or absence of 1) lupus skin disease and 2) Raynaud's phenomenon (RP) was determined for each patien
49 ogressive SSc and a more recent onset of non-Raynaud's phenomenon symptoms had higher numbers of mast