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1 d storage (14% survival with CRS vs. 0% with Ringer's solution).
2 els was evoked with long exposure to high K+ Ringer's solution.
3 rch solution); 5% human albumin; or lactated Ringer's solution.
4 ither hypertonic saline, Isosal, or lactated Ringer's solution.
5 HDL or placebo), and then by 1 L of lactated Ringer's solution.
6 CAI-dentate area was perfused with modified Ringer's solution.
7 osm/L of 7.5% hypertonic saline, or lactated Ringer's solution.
8 nd 74%, respectively, compared with lactated Ringer's solution.
9 sive or moderate resuscitation with lactated Ringer's solution.
10 king the resulting block in a PO(4)-enriched Ringer's solution.
11 n ventilators and resuscitated with lactated Ringer's solution.
12 n ventilators and resuscitated with lactated Ringer's solution.
13 ne (5 to 10 microg/kg) or hyperoxic lactated Ringer's solution.
14 th EGTA or by bathing such cells in low-Ca2+ Ringer's solution.
15 um proteins when incubated in buffered chick Ringer's solution.
16 volume was returned in the form of lactated Ringer's solution.
17 uld restore membrane resealing in low Ca(2+) Ringer's solution.
18 volume was returned in the form of lactated Ringer's solution.
19 ith its occurrence in animals given lactated Ringer's solution.
20 and perfused at a rate of 5 microL/min with Ringer's solution.
21 testinal sleeve incubated in a physiological Ringer's solution.
22 volume was returned in the form of lactated Ringer's solution.
23 , (4) anti-bFGF antibody, (5) ngIgG, and (6) Ringer's solution.
24 ients receiving a rapid infusion of lactated Ringer's solution.
25 ravenous strategy that consisted of lactated Ringer's solution (100 ml per kilogram of body weight) a
26 rfusion bath with a low-HCO(3)(-) Cl(-)-free Ringer's solution (2.85 mM; pH 6.5), in the presence or
28 nhibitor) in 50/50 dimethyl sulfoxide (DMSO)/Ringer's solution, 300 KIU aprotinin (a serine protease
32 large body surface area burn, using lactated Ringer's solution, 6% hetastarch, and hypertonic saline
35 ntrols (n = 6) received intravenous lactated Ringer's solution according this dosing schedule: 1.5 mL
38 urned to baseline immediately after lactated Ringer's solution administration, while PAOP remained in
42 ron chelator deferoxamine (DFO), or lactated Ringer's solution alone (LR) on lung injury parameters a
43 in a lactated Ringer's solution or lactated Ringer's solution alone during the first 6 hours of flui
45 ndex admission was 20.3+/-3.5% with lactated Ringer's solution and 21.4+/-3.3% with normal saline (ad
47 CLOVERS trial, 622 (39.8%) received lactated Ringer's solution and 690 (44.1%) received 0.9% saline a
48 to 145 mmol/L) was measured in the lactated Ringer's solution and hetastarch groups (130 to 133 mmol
50 rs with the endothelium bathed in a modified Ringer's solution and the epithelium bathed with silicon
51 s were similarly perfused with cold lactated Ringer's solution and were followed without immunosuppre
52 ts in normal frog Ringer's solution, Ca-free Ringer's solution, and BAPTA AM-pretreated preparations;
53 ellate) morphology by exposure to serum-free Ringer's solution; and (3) swelling cells by exposure to
54 timand was the effect of the use of lactated Ringer's solution as compared with normal saline average
55 tation in all three groups was with lactated Ringer's solution as needed to maintain baseline cardiac
56 All groups received intravenous lactated Ringer's solution at 4 mL.kg-1.%burn(-1).24 hrs-1 for re
58 n, during which either PentaLyte or lactated Ringer's solution-based resuscitation was administered.
60 the conjunctiva with Na(+)-free bicarbonated Ringer's solution (BRS) were used to estimate contributi
62 le; however, after 15 minutes of exposure to Ringer's solution, [Ca2+]i in fibers from the outer cort
63 Initial fluid resuscitation with lactated Ringer's solution, compared with 0.9% saline, might be a
65 nd 58.9 +/- 3.9 minutes, respectively) or in Ringer's solution containing 1 mM [Ca2+]o + 50 microM ve
67 The proteolytic activity of fiber cells in Ringer's solution containing 10(-)(6) M and 2 x 10(-)(3)
68 vely, compared with sucrose-EDTA solution or Ringer's solution containing 10(-)(8) M [Ca(2+)](o).
71 ither background illumination or exposure to Ringer's solution containing low Ca2+ and/or cyclic GMP-
73 elongated fibers, which, in the presence of Ringer's solution (containing 2 mM Ca2+), underwent disi
74 nutes, the [Ca2+]i of fiber cells exposed to Ringer's solution, containing 2 mM Ni2+ (574.7+/-29 nM;
77 0.9% saline (n = 6), 5% dextrose in lactated Ringer's solution (D5RL) (n = 6), or 5% dextrose in wate
78 en cells were wounded twice in normal Ca(2+) Ringer's solution, decreases in tension at the second wo
80 ate-buffered saline, normal saline, lactated Ringer's solution, dextran, hespan, 5% human albumin, 25
81 emoglobin) or a control infusion of lactated Ringer's solution (each infusion given over a total of 4
82 = 9) solution made up exactly like lactated Ringer's solution except for the substitution of either
85 ed rats were then resuscitated with lactated Ringer's solution, four times the maximum shed blood vol
86 was superfused with glutathione bicarbonate Ringer's solution (GBR); with GBR and 10 nM, 100 nM, or
90 Test solutions (10 mL/kg of either lactated Ringer's solution, hetastarch, or hypertonic saline dext
91 sterior surface is continually bathed with a Ringer's solution in equilibrium with a CO2-gas air mixt
93 obules generated from fiber cells exposed to Ringer's solution; in addition, no high molecular weight
94 est the hypothesis that intravenous lactated Ringer's solution, infused at a rate used in resuscitati
96 were resuscitated by administering lactated Ringer's solution intravenously to achieve and maintain
100 on of the isolated rat lens fiber cells with Ringer's solution led to their globulization in 30 +/- 3
102 ed in lactated Ringer's solution or lactated Ringer's solution (LR) alone were given by intravenous i
103 ovolemic shock, HSD (250 mL) versus lactated Ringer's solution (LR) as the initial resuscitation flui
104 CLP-induced sepsis and treated with lactated Ringer's solution (LR, n = 13) survived longer than thos
107 d bathing solutions were iso-osmotic Cl-free Ringer's solutions modified using N-methyl-D-glucamine a
109 o receive a 1-hr infusion of either lactated Ringer's solution (n = 6), 0.9% saline (n = 6), 5% dextr
110 ed infusion pumps to deliver blood, lactated Ringer's solution, norepinephrine, and in ReFit1 dobutam
111 plantation, kidneys were flushed with either Ringer's solution or CRS at 35-37 degrees C or were not
112 ontrol mice overnight in distilled water, in Ringer's solution or in Ringer's solution with added 1 M
113 Three doses of EP dissolved in lactated Ringer's solution or lactated Ringer's solution (LR) alo
114 eceive either bolus of albumin in a lactated Ringer's solution or lactated Ringer's solution alone du
116 s in Ontario, Canada, to use either lactated Ringer's solution or normal saline hospital-wide for a p
118 after CRS was significantly higher than with Ringer's solution or without flushing (80% vs. 25% and 1
119 .086 min[-1]) and in K+-free (0.062 min[-1]) Ringer's solution, or when the fibers were suspended in
122 de of the epithelium, was enhanced in simple Ringer's solution over that in tissue culture medium, an
126 s to investigate the effects of ATP added to Ringer's solution perfusing the retinal-facing (apical)
129 e than 50%, while administration of lactated Ringer's solution provoked an approximately 2.5 times gr
130 hospital-wide policy to administer lactated Ringer's solution rather than normal saline did not resu
132 scitation with red blood cells plus lactated Ringer's solution (RL) is more effective than RL alone i
134 lood + 0.12, 0.24, or 0.36 g/kg) or lactated Ringer's solution (shed blood + 2 x volume of shed blood
135 exposed to 1 mM [Ca2+]o in Na+ - or K+ -free Ringer's solution (tg = 66.7 +/- 5.3 and 58.9 +/- 3.9 mi
136 ter (p < .001) in animals receiving lactated Ringer's solution than in rabbits receiving either collo
137 crease if cells were wounded in a low Ca(2+) Ringer's solution that inhibited both membrane resealing
138 esence of phosphatidylcholine (0.025 g/ml in Ringer's solution), the injured cells initially responde
140 at, for fibroblasts wounded in normal Ca(2+) Ringer's solution, the membrane tension decreased dramat
142 When these cells were placed back in normal Ringer's solution, they remained viable and active for s
143 bsequently either resuscitated with lactated Ringer's solution (three times shed blood volume, n = 18
144 on, awakened, and resuscitated with lactated Ringer's solution titrated to maintain hematocrit +/- 3%
145 blood was then returned along with lactated Ringer's solution (two times the shed blood volume) to p
146 compared between subjects receiving lactated Ringer's solution vs. subjects receiving normal saline.
147 studied in those subjects receiving lactated Ringer's solution vs. those persons receiving normal sal
148 0, or 100 mg/kg) or equal volume of lactated Ringer's solution was infused on day 1; the alternate so
149 mL/kg test solution dose was given, lactated Ringer's solution was infused to achieve the same end po
150 ncrease of [Ca2+]i in fiber cells exposed to Ringer's solution was measured, and the effects on the i
152 he currents observed in divalent cation-free Ringer's solution were due to Cx46 hemichannel opening,
153 After recovery, Intralipid 20 or Lactated Ringer's Solution were infused according to a clinical g
154 ly discovered that small amounts of lactated Ringer's solution, which are inadequately cleared from a
156 Currents were recorded in chloride-free Ringer's solution with low or high concentrations of ext