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1  by complementation with a gene encoding the S-adenosylhomocysteine hydrolase.
2     H19 deficiency increased the activity of S-adenosylhomocysteine hydrolase, a regulator of DNA met
3                                              S-Adenosylhomocysteine hydrolase (AdoHcy hydrolase) crys
4                                              S-Adenosylhomocysteine hydrolase (AdoHcyase) catalyzes t
5       A site-directed mutagenesis, D244E, of S-adenosylhomocysteine hydrolase (AdoHcyase) changes dra
6 ed a causative mutation in the gene encoding S-adenosylhomocysteine hydrolase (Ahcy).
7 es of nine nucleoside analogue inhibitors of S-adenosylhomocysteine hydrolase, an important target fo
8 echanistic findings reveal that TMAO targets S-adenosylhomocysteine hydrolase and disrupts the methio
9 oteomics study reveals that two other genes (S-Adenosylhomocysteine hydrolase and Serine hydroxymethy
10 ylation, S-adenosylmethionine synthetase and S-adenosylhomocysteine hydrolase, are increased in respo
11 y overproduction, activity and expression of S-adenosylhomocysteine hydrolase (converts S-adenosylhom
12 L cells to nucleoside analogue inhibitors of S-adenosylhomocysteine hydrolase correlates directly wit
13 uding accumulation of dATP and inhibition of S-adenosylhomocysteine hydrolase enzyme activity.
14 ties of methionine adenosyltransferase II or S-adenosylhomocysteine hydrolase in the brain tissue of
15 f deoxyadenosine and dATP, and inhibition of S-adenosylhomocysteine hydrolase in the thymus, spleen,
16 hymus and spleen, and a marked inhibition of S-adenosylhomocysteine hydrolase in these organs.
17 adenosine, as well resulting dATP levels and S-adenosylhomocysteine hydrolase inhibition in bone marr
18 ause neither homocysteine thiolactone nor an S-adenosylhomocysteine hydrolase inhibitor (adenosine di
19 tudy, we demonstrate that treatment with the S-adenosylhomocysteine hydrolase inhibitor 3-deazaneplan
20 ibited following treatment with a reversible S-adenosylhomocysteine hydrolase inhibitor, DZ2002.
21                                              S-Adenosylhomocysteine hydrolase is not involved because
22 hanistically, TMAO noncompetitively inhibits S-adenosylhomocysteine hydrolase, leading to accumulatio
23 eptococcus pneumoniae 5'-methylthioadenosine/S-adenosylhomocysteine hydrolase (MTAN) catalyzes the hy
24 s methionine alpha,gamma-lyase (rMETase) and S-adenosylhomocysteine hydrolase (rSAHH) cloned from Pse
25                                              S-adenosylhomocysteine hydrolase (SAH) is a key enzyme i
26 son's disease (WD) through the inhibition of S-adenosylhomocysteine hydrolase (SAHH) by copper (Cu) a
27 r identical or nearly identical to predicted S-adenosylhomocysteine hydrolase (SAHH) from two Nicotia
28                                              S-Adenosylhomocysteine hydrolase (SAHH) is an NAD(+)-dep
29                          This assay utilizes S-adenosylhomocysteine hydrolase (SAHH) to hydrolyze the
30 hed for methylation cycle enzymes, including S-adenosylhomocysteine hydrolase (SAHH), the only known
31 y regulated H19 lncRNA binds to and inhibits S-adenosylhomocysteine hydrolase (SAHH), the only mammal
32  in a Superose column fraction that contains S-adenosylhomocysteine hydrolase (SAHH), which has a hig
33 based molecular beacon (MB) used for probing S-adenosylhomocysteine hydrolase (SAHH)-catalyzed hydrol
34  of adenosine, based on adenosine inhibiting S-adenosylhomocysteine hydrolase (SAHH)-catalyzed hydrol
35 NA methylation by modulating the activity of S-adenosylhomocysteine hydrolase (SAHH).
36 T1A), glycine-N-methyltransferase (GNMT) and S-adenosylhomocysteine hydrolase (SAHH).
37 e basis of the available X-ray structures of S-adenosylhomocysteine hydrolases (SAHHs), free energy s
38 red by expressing the Pseudomonas aeruginosa S-adenosylhomocysteine hydrolase that synthesizes homocy
39 ction, adenosine dialdehyde, an inhibitor of S-adenosylhomocysteine hydrolase, was found to block cyt
40                 Furthermore, introduction of S-adenosylhomocysteine hydrolase, which restores the met