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1                                              S. marcescens does not normally colonize human skin, but
2                                              S. marcescens expresses prodigiosin, a bright red and ce
3                                              S. marcescens facilitates arboviral infection through a
4                                              S. marcescens induced neutrophil recruitment to the corn
5                                              S. marcescens induces corneal inflammation by activation
6                                              S. marcescens isolates were compared using restriction-e
7                                              S. marcescens oxyR mutants were severely impaired in bio
8                                              S. marcescens was identified from a pharmacy water fauce
9 isolates, 5 Enterobacter cloacae isolates, 2 S. marcescens isolates, 1 Proteus mirabilis isolate, and
10 ene knockout mice was abraded, and 1 x 10(7) S. marcescens were added in the presence of a silicone h
11    Here, the development and evaluation of a S. marcescens cgMLST scheme is reported with the goal of
12                  It has been reported that a S. marcescens chimera, SM : rS5'ec, in which five diverg
13  example, for growth on N-acetylglucosamine, S. marcescens exhibits high pentose phosphate pathway ac
14         The activity of the fraction against S. marcescens was explained by (R)-(-)-mellein alone, an
15 tic (R)-(-)-mellein and micromolide, against S. marcescens and a Gram-positive bacterium, Staphylococ
16 a (2%), E. coli (<1%), K. oxytoca (<1%), and S. marcescens (<1%).
17 tion of bronchoscopes with P. aeruginosa and S. marcescens and possible infection of patients at a co
18 ducted an investigation of P. aeruginosa and S. marcescens isolates related to bronchoscopy at a comm
19 aerogenes, K. pneumoniae, P. aeruginosa, and S. marcescens) became more susceptible.
20  with Acinetobacter spp., P. aeruginosa, and S. marcescens, 5/6 with Citrobacter spp., 13/14 with Ent
21  seven of these had Enterobacter cloacae and S. marcescens in the same culture.
22 nce birth, but gut colonization with GBS and S. marcescens occurred closer to time of bloodstream inf
23                        All K. pneumoniae and S. marcescens isolates were resistant to ampicillin, and
24  were E. cloacae complex, K. pneumoniae, and S. marcescens.
25                        Cases were defined as S. marcescens BSIs in patients receiving PN from the pha
26  its transporter ShlB resulted in attenuated S. marcescens strains that failed to cause profound weig
27 eement was observed, excluding A. baumannii, S. marcescens, and S. pneumoniae, for which >/=4-fold di
28 n foreskin fibroblasts was also inhibited by S. marcescens secretomes indicating that the effect is n
29                                   Currently, S. marcescens is poorly characterized and studies on int
30 , that is located upstream of NucC-dependent S. marcescens promoters and the late promoters of P2-rel
31 f multiple members of the recently described S. marcescens complex causing hospital- or community-ass
32 gellum mutants fliR and flhD in two distinct S. marcescens strains.
33 P, and prodigiosin concentration changes for S. marcescens during cultivation in batch culture.
34                     Several risk factors for S. marcescens infection were identified, but hospital an
35          Field Aedes mosquitoes positive for S. marcescens were more permissive to dengue virus infec
36 individuals exhibited reduced preference for S. marcescens, and dauers from some strains preferred E.
37                  Both genes are required for S. marcescens to escape the gut lumen into the hemocoel,
38 there is no standardized analytic scheme for S. marcescens core genome multilocus sequence typing (cg
39        In summary, a novel cgMLST scheme for S. marcescens was developed and evaluated.
40                            Purified LPS from S. marcescens, but not from Escherichia coli or S. marce
41 roduce SlpB as a new cytotoxic protease from S. marcescens.
42 haracterise the tripartite SmhABC toxin from S. marcescens and propose a mechanism of pore assembly.
43 ing that serratiochelin is required for full S. marcescens pathogenesis in the bloodstream.
44 ication methods are insufficient to identify S. marcescens complex and laboratory reporting should be
45 nd interleukin-1 receptor type 1 (IL-1R1) in S. marcescens-induced corneal inflammation and infection
46  and T6SS-mediated antibacterial activity in S. marcescens.
47 behind secondary metabolites biosynthesis in S. marcescens remains limited.
48 quent inactivating mutations, exclusively in S. marcescens isolates of clinical origin.
49 expectedly, fliR but not flhD is involved in S. marcescens-mediated damages of the intestinal epithel
50 itro cytotoxic activity commonly observed in S. marcescens culture filtrates.
51 nergy-dependent and Sec-dependent pathway in S. marcescens.
52  extracellular polysaccharides production in S. marcescens and provides important clues for future st
53  RND efflux systems in biocide resistance in S. marcescens.
54 echanism for the second step of secretion in S. marcescens.
55 sitively controlled prodigiosin synthesis in S. marcescens.
56 characterize the iron acquisition systems in S. marcescens isolate UMH9, which was recovered from a c
57 etermined that the addition of a chi-induced S. marcescens cell lysate to an uninfected culture cause
58 tic screen for loss of virulence of ingested S. marcescens and identified FliR, a structural componen
59                            Tobramycin-killed S. marcescens induced corneal inflammation in C57BL/6 mi
60 encia burhodogranaria, is gram negative like S. marcescens.
61       R-flagellins from Serratia marcescens (S. marcescens) and Salmonella muenchen (S. muenchen) do
62 malized secretomes from wild-type and mutant S. marcescens derivatives.
63 less, the temperature response of the native S. marcescens ATCase suggests a strong entropic effect t
64 fection and in infections with Gram-negative S. marcescens or Gram-positive E. faecalis bacteria, whi
65 4 metabolites based on genomic annotation of S. marcescens Db11.
66                In addition, the clearance of S. marcescens was genetically correlated with the resist
67 opic bacteriophage chi (Chi) to a culture of S. marcescens stimulates a greater than fivefold overpro
68 ase treatments abrogated the cytotoxicity of S. marcescens culture filtrates towards HeLa cells, sugg
69 29 was refined by integrating RNAseq data of S. marcescens growth on three different carbon sources (
70                             LPS depletion of S. marcescens secretomes with polymyxin B agarose render
71    Despite multiple clinical descriptions of S. marcescens nosocomial pneumonia, little is known rega
72 ective means to control the dissemination of S. marcescens, an in-depth analysis of the population st
73 to dengue virus infection than those free of S. marcescens.
74 x strains, 89% of K. pneumoniae, and half of S. marcescens had an extended-spectrum B-lactamase pheno
75 ential therapeutic targets for inhibition of S. marcescens-induced corneal inflammation.
76                         Oral introduction of S. marcescens into field mosquitoes that lack this bacte
77  applicable to emerging clinical isolates of S. marcescens causing bacteremia.
78           We included first-time isolates of S. marcescens identified via culture and confirmed by ma
79              This study describes a model of S. marcescens pneumonia that mimics known clinical featu
80      A case was defined as the occurrence of S. marcescens bacteremia in any patient in the surgical
81                               An outbreak of S. marcescens and E. cloacae bacteremia in a surgical in
82 ding standards contributed to an outbreak of S. marcescens BSIs.
83 was to characterize the metabolic profile of S. marcescens to provide insight for metabolic engineeri
84 tify any environmental or staff reservoir of S. marcescens.
85 in which OxyR contributes to early stages of S. marcescens biofilm formation by influencing fimbrial
86  first reports applying PFGE to the study of S. marcescens, and this method was a useful marker of st
87              Between-patient transmission of S. marcescens complex outside of outbreaks is likely rar
88 istant clones suggests that the treatment of S. marcescens infections will become increasingly diffic
89 estigation, allowing better understanding of S. marcescens genomic epidemiology and facilitating inte
90 Prodigiosin has implications in virulence of S. marcescens and promising clinical applications.
91 marcescens, but not from Escherichia coli or S. marcescens strains with mutations in the waaG and waa
92 colonized with provisionally matching GBS or S. marcescens.
93 for Fis are 100% identical in K. pneumoniae, S. marcescens, E. coli, and S. typhimurium and 96 to 98%
94         Four hundred ninety-one high-quality S. marcescens WGS data sets were extracted from public d
95 ematic collection of antimicrobial-resistant S. marcescens associated with bloodstream infections in
96  the clinically isolated multidrug-resistant S. marcescens strain and found that the sdeXY deletion m
97 what is observed in other bacterial species, S. marcescens OxyR is required for oxidative stress resi
98 he genomic sequence of NCBI reference strain S. marcescens Db11 (NZ_HG326223.1) as a starting point-a
99 eal aspiration model of lethal and sublethal S. marcescens pneumonia in BALB/c mice and extensively c
100 ability of the tigecycline Etest for testing S. marcescens, Acinetobacter spp., and S. pneumoniae is
101                          We hypothesize that S. marcescens detects the threat of phage-mediated cell
102 or in vitro cytotoxic activity revealed that S. marcescens mutant strains that are deficient in produ
103                        Our results show that S. marcescens is a diverse species with a high level of
104                        Our results show that S. marcescens model iSR929 can provide reasonable predic
105             Together these data suggest that S. marcescens LPS is sufficient for inhibition of epithe
106                      When hasF, encoding the S. marcescens TolC ortholog, was expressed in KAM43, all
107 ight putative RND efflux system genes in the S. marcescens Db10 genome that included the previously c
108       Finally, recombinant expression of the S. marcescens 56-kDa metalloprotease conferred a cytotox
109 CTP stimulates the catalytic activity of the S. marcescens ATCase and CTP/UTP inhibitory synergism ha
110  measured differences in processivity of the S. marcescens chitinases.
111                Bioinformatic analysis of the S. marcescens Db11 genome revealed three additional open
112                      One crystal form of the S. marcescens enzyme displays a bound pyruvate as well a
113 93-r97) of the regulatory polypeptide of the S. marcescens enzyme have been replaced with their E. co
114 ) retained 455 out of 460 amino acids of the S. marcescens enzyme, it possessed characteristics simil
115 ajority of the K. pneumoniae and half of the S. marcescens isolates were resistant to both cefotaxime
116 n E. coli for extracellular secretion of the S. marcescens nuclease.
117  proteins are the original substrates of the S. marcescens T6SS, before horizontal acquisition of oth
118 yperthermophile Sulfolobus solfataricus, the S. marcescens structure shows similar subunit structures
119                          NucC binding to the S. marcescens nuclease promoter P(nucA) and to the seque
120 ues of E. coli have been replaced with their S. marcescens counterpart, lost both heterotrophic and h
121 auers from some strains preferred E. coli to S. marcescens.
122                      Literature pertinent to S. marcescens-mediated necrotizing fasciitis is also rev
123 and phenoloxidase activity and resistance to S. marcescens in both sexes, but these relationships wer
124 N-acetylneuraminic acid, and n-butanol using S. marcescens.
125                      The mechanisms by which S. marcescens attacks enterocytes and damages the intest
126 (-/-), and TLR4/5(-/-) corneas infected with S. marcescens had significantly increased CFU, indicatin
127 itis of the chest wall due to infection with S. marcescens that initially manifested as bilateral bre
128 ed insect survival after oral infection with S. marcescens.
129 ording to univariate analysis, patients with S. marcescens bacteremia stayed in the surgical intensiv
130                     Twenty-six patients with S. marcescens bacteremia were identified; eight (31 perc
131 tify risk factors, we compared patients with S. marcescens bacteremia with randomly selected controls
132 were positive for P. aeruginosa also yielded S. marcescens.
133 nyl infusions from two case patients yielded S. marcescens and E. cloacae.

 
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