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1                                              S. suis bacteria express capsular polysaccharides (CPS)
2                                              S. suis can induce disease and also quietly reside on mu
3                                              S. suis infection resulted in a rapid depletion of mitoc
4                                              S. suis interaction with human and pig IEC correlates wi
5                                              S. suis is also a very successful colonizer of mucosal s
6                                              S. suis type II was recovered from their brains and join
7 hree genetic markers that differentiated 115 S. suis isolates into disease-associated and non-disease
8               Streptococcus suis serotype 2 (S. suis 2) is a highly invasive pathogen in pigs and hum
9 udy was to characterize the diversity of 208 S. suis isolates collected between 2014 and 2017 across
10                             We identified 29 S. suis serotypes and 4 reclassified serotypes as putati
11 housekeeping gene fragments from each of 294 S. suis isolates obtained from various S. suis diseases
12                   This study evaluated 2,379 S. suis central nervous system (CNS) isolates from diagn
13 c potential, we compared whole genomes of 98 S. suis isolates from human patients and pigs with invas
14 subtype, thus increasing the knowledge about S. suis strains circulating in the United States.
15 and administered with emulsifying adjuvants, S. suis type 2 CPS is able to induce potent IgM and isot
16 (TNF), and IL-10, was observed in mice after S. suis type 2 infection.
17 hat Ssads could impair PMN's defense against S. suis 2 with decreasing of oxidative activity and degr
18 ortant roles in developing therapies against S. suis 2 infection.
19  confirm extensive taxonomic diversity among S. suis-like organisms, support the recognition of a bro
20               According to genomic analysis, S. suis is divided into asymptomatic carriage, respirato
21 (11.5743) compared to S. pyogenes (1.03) and S. suis (0.57).
22 in Streptococcus pyogenes, S. agalactiae and S. suis.
23 al role in the activation of neutrophils and S. suis clearance, which further reduced severe inflamma
24 minimizing disease associated with PRRSV and S. suis coinfection.
25  reduced mortality associated with PRRSV and S. suis coinfection.
26  and three from cows: all were classified as S. suis by MALDI-TOF MS but tested negative by the recN
27 ased swine and three from cows identified as S. suis by MALDI-TOF MS, but which tested negative by a
28 atories still report such isolates simply as S. suis, due to the limited resolution of current diagno
29 oor indicator of genetic relatedness between S. suis isolates, these findings suggest that capsular g
30 functions (Nudix hydrolase and DNA binding), S. suis 2 retains a single regulatory role in the modula
31 anisms, support the recognition of a broader S. suis complex, demonstrate the need for genome-based m
32 infection and disease following challenge by S. suis type II.
33 h to be a competitive method to characterize S. suis isolates recovered from pigs on UK farms and one
34 njugate prototypes were prepared by coupling S. suis type 2 CPS to tetanus toxoid, and the immunologi
35  little geographical clustering of different S. suis subpopulations, and the bacterium undergoes high
36                         The virulence factor S. suis adhesin P (SadP) recognizes the galabiose Galalp
37 jection for three consecutive days following S. suis challenge was the most effective regimen for min
38 multilocus sequence typing (MLST) scheme for S. suis developed in order to begin to address these iss
39            The cleavage patterns of DNA from S. suis isolated from the sows matched the cleavage patt
40 ws matched the cleavage patterns of DNA from S. suis isolated from their respective pigs.
41  undercooked pork is a risk factor for human S. suis serotype 2 (SS2) infection.
42 of a structured S. suis complex and identify S. suis sensu stricto-specific markers that may inform i
43  insufficient for defining a pathotype, (ii) S. suis serotypes and STs associated with CNS infection
44 o understand the genetic basis of disease in S. suis, we study the genomes of 375 isolates with detai
45                Furthermore, nrtR knockout in S. suis serotype 2 reduces its capacity to form biofilms
46 robustness of the core metabolic pathways in S. suis during the infection process.
47 cose starvation induced adaptive response in S. suis makes a great contribution to understanding bett
48 A/(p)ppGpp in glucose starvation response in S. suis, the growth curves and transcriptional profiles
49 ndicate that Ssads play an important role in S. suis 2 escaping human innate immunity in the context
50  intramuscular doses of an autogenous killed S. suis vaccine (group 6) prior to S. suis challenge or
51 ion of MALDI-TOF MS results, can misidentify S. suis-like isolates.
52                                    Moreover, S. suis infection increased cleavage of caspase-3, caspa
53 uencing revealed that most isolates were not S. suis sensu stricto but instead belonged to other reco
54 llenged intranasally with strain MN 87555 of S. suis type II.
55 we identified 38 genes conserved in >=95% of S. suis sensu stricto genomes that may support future ma
56 ting isotopologue patterns in amino acids of S. suis grown under in vitro and ex vivo conditions.
57 To dissect the central metabolic activity of S. suis under different conditions of nutrient availabil
58 present, to our knowledge, the first case of S. suis arthroplasty infection and streptococcal toxic s
59                          Characterization of S. suis strains originating from pigs with histologic co
60 which correlated well with host clearance of S. suis.
61       Oligosaccharides resembling the CPS of S. suis serotypes 2, 3, 9, and 14 have been synthesized,
62  diagnoses, alongside the complex ecology of S. suis, poses significant challenges in comprehending t
63              Although the virulence genes of S. suis have been extensively studied, the mechanisms by
64 cial role of PEP carboxylation for growth of S. suis in the host was supported by experiments with a
65 isolates from farms with no known history of S. suis-associated disease.
66          We compared in vitro interaction of S. suis with human and porcine intestinal epithelial cel
67  distribution of disease-causing isolates of S. suis, most isolates previously characterized as of hi
68  we find that several pathogenic lineages of S. suis emerged in the 19th and 20th centuries, during a
69 e the most common clinical manifestations of S. suis 2 infection.
70                   However, the mechanisms of S. suis 2 surviving in human blood remains unclear, so t
71 e details about the impact and mechanisms of S. suis on specific populations of thymic and immune cel
72 d us to examine its effect on the outcome of S. suis infection.
73  the pathogenicity and zoonotic potential of S. suis is yet to be fully realized.
74  results indicate that zoonotic potential of S. suis results from gene loss, recombination and horizo
75              The recombinant BgaC protein of S. suis was purified to homogeneity.
76 sistently emerging from one subpopulation of S. suis and acquiring genes through horizontal transfer
77 signated as Ssads (the adenosine synthase of S. suis 2).
78 sponse, and the outcome of highly pathogenic S. suis infection in a mouse model.
79  Because the strategies to target pathogenic S. suis are limited, new therapeutic approaches are need
80 alactiae, S. equi, S. mutans, S. pneumoniae, S. suis and S. uberis, as well as representative enteroc
81 with additional diagnostic tools for precise S. suis identification, and (iv) VAGs remain an unreliab
82             Most of the piglets in the PRRSV-S. suis dual-infection group developed suppurative menin
83   Overall, 20 out of 22 piglets in the PRRSV-S. suis dual-infection group died within 1 week after ch
84 ence supporting the proposal of a structured S. suis complex and identify S. suis sensu stricto-speci
85 ing methods used to characterize and subtype S. suis isolates.
86 tigens for the development of semi-synthetic S. suis serotypes 2 and 9 glycoconjugate veterinary vacc
87 g to Gb4 could be used to selectively target S. suis in systemic disease without interfering with com
88                  Therefore we concluded that S. suis BgaC is an atypical surface-exposed protein with
89 immune electron microscopy demonstrated that S. suis BgaC is an atypical surface-anchored protein in
90                          Here, we found that S. suis 2 can escape phagocytic clearance by adenosine s
91  classical growth experiments, we found that S. suis is auxotrophic for Arg, Gln/Glu, His, Leu, and T
92        Bioinformatics analyses revealed that S. suis BgaC shared the conserved active sites (W240, W2
93 ck end labeling (TUNEL) assays revealed that S. suis infection induced apoptosis in CD3(+), CD14(+),
94   Taken together, these results suggest that S. suis infection can cause atrophy of the thymus and in
95           However, little is known about the S. suis population structure, the clonal relationships b
96  to SSU05_1971 on the reverse strand) in the S. suis 05ZYH33 that causes streptococcal toxin shock-li
97 ection-only group and 5 of 23 piglets in the S. suis-challenge-only group (1 of 12 in trial 1 and 4 o
98                            Intriguingly, the S. suis NrtR naturally contains a single amino acid subs
99 pulations worldwide, making detection of the S. suis species in asymptomatic carrier herds of little
100 genes may be moving horizontally through the S. suis population.
101                                        Thus, S. suis induced thymocyte apoptosis through a p53- and c
102 control of NAD(+) homeostasis contributes to S. suis virulence.
103 us killed S. suis vaccine (group 6) prior to S. suis challenge or a single 2-ml intramuscular dose of
104 ed a more severe proinflammatory response to S. suis infection and increased the mortality rate, whil
105 gulated in swine spleen cells in response to S. suis infection.
106  collection of 50 previously uncharacterized S. suis isolates, in comparison to existing methods used
107 they form a broader, previously unrecognized S. suis complex.
108 f 294 S. suis isolates obtained from various S. suis diseases and from asymptomatic carriage represen
109 encing revealed that only four isolates were S. suis sensu stricto, while most others belonged to Str
110 led that fewer than half of cases from which S. suis was isolated from the brain had histologic evide
111 roup died within 1 week after challenge with S. suis (10 of 11 in each of two trials).
112 ets showed clinical symptoms compatible with S. suis infection 24-48 hours after ingestion of SS2.
113 ction with human and pig IEC correlates with S. suis serotype and genotype, which can explain the zoo
114                    Labeling experiments with S. suis grown ex vivo in blood or cerebrospinal fluid re
115                     We identified a zoonotic S. suis clone that diverged from a non-zoonotic clone by
116 th food-borne transmission in Asia, zoonotic S. suis infections are mainly occupational hazards elsew

 
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