ライフサイエンスコーパス: SA-beta-gal

コーパス検索結果 (left1)

通し番号をクリックするとPubMedの該当ページを表示します

1 ence-associated beta-galactosidase activity (SA beta-gal) and cell cycle inhibitors such as p21 or p1

2 rkers of cellular senescence, p16(Ink4a) and SA-beta-gal.

3 IL6, TNF, DPP4, and IL8, as well as elevated SA-beta-gal activity.

4 nated bone marrow progenitor cells expressed SA-beta-gal and senescence-associated proteins p53 and p

5 tosis and senescence, tested as staining for SA-beta-gal, also expressed p16(INK4A).

6 Senescence induction, tested as staining for SA-beta-gal, in reoxygenated progenitor cells was closel

7 h arrest and senescence-associated beta-gal (SA-beta-gal) activity.

8 Senescence-associated beta-galactosidase (SA beta-gal) activity was observed in lymphomas from Emu

9 of senescence-associated beta-galactosidase (SA beta-gal) activity, apparently irreparable genomic DN

10 diameter) anti-PECAM/SA-beta galactosidase (SA-beta-gal) conjugates bound selectively to PECAM-expre

11 n, senescence-associated beta-galactosidase (SA-beta-gal) activation, and increased mRNA expression o

12 of senescence-associated beta-galactosidase (SA-beta-gal) activity [2,3,13-15].

13 ed senescence-associated beta-galactosidase (SA-beta-gal) activity and production of intracellular re

14 nd senescence-associated beta-galactosidase (SA-beta-gal) activity but an increase in adenosine triph

15 xygen species (iROS), SA-beta-galactosidase (SA-beta-gal) activity, and autofluorescence (AF) was ass

16 of senescence-associated beta-galactosidase (SA-beta-gal) positive cells.

17 on senescence-associated beta-galactosidase (SA-beta-gal), have been developed to selectively elimina

18 ested as staining for SA-beta-galactosidase (SA-beta-gal), of bone marrow progenitor cells.

19 by senescence-associated beta-galactosidase (SA-beta-gal), p16Ink4a, and p53 in lamin A/C-deficient m

20 ss senescence-associated beta-galactosidase (SA-beta-gal).

21 With age, NSCs exhibited increased SA-beta-gal activity and decreased proliferation and poo

22 conditions and significantly decreased iROS, SA-beta-gal, and AF normally induced by hyperoxic condit

23 ith enhanced expression of senescent markers SA-beta-gal, PML, and p16(INK4a).

24 oreover, inhibition of ATM signaling reduced SA-beta-gal positivity but increased apoptosis of reoxyg

25 ells that utilizes a prodrug composed of the SA-beta-gal substrate galactose (galacto) and the proteo