ライフサイエンスコーパス: SAP

1 SAP activates the high-affinity IgG receptor Fcgamma rec

2 SAP and 1866 may inhibit some, but not all, of the effec

3 SAP consisted of single-shot, intravenous infusion of 1.

4 SAP domains are common among many other SUMO E3s, and ar

5 SAP in the first 14 days was diagnosed by a criteria-bas

6 SAP performed significantly better than FDT in predictin

7 SAP reduced serum levels of IL-23, IFN-beta, MCP-1, and

8 SAP, SLP, and OCT outcomes were compared between the con

9 SAP-deficient mice were highly susceptible to this infec

10 SAPs channel food allergens to lamina propria mucosal ma

11 Interestingly, in the presence of Ca(2+), SAP first inhibited, then significantly accelerated D76N

12 if they had a minimum of 2 NEI VFQ-25 and 5 SAP tests during follow-up.

13 they had a minimum of 2 NEI VFQ-25 and >/=5 SAP during follow-up.

14 Subjects had a mean 4.5 +/- 0.8 SAP and OCT tests for short-term variability assessment.

15 Subjects had an average of 4.8 SAP tests (range, 2-28), 3.6 SD-OCT tests (range, 2-10),

16 s had a median of 3 NEI VFQ-25, 8 FDT, and 8 SAP tests during follow-up.

17 a under the curve, 0.81; 95% CI, 0.72-0.90), SAP mean sensitivity (area under the curve, 0.80; 95% CI

18 eveloped endophthalmitis despite receiving a SAP by cefuroxime at the end of cataract surgery.

19 atients had normal or nonrepeatable abnormal SAP at baseline.

20 d to construct expression vectors to achieve SAP overexpression, and both genetic and functional assa

21 nosis of SAP were assessed using adjudicated SAP as the reference standard.

22 )) than physician diagnosis with adjudicated SAP.

23 e evidence of a difference in SSI risk after SAP administration 60-30 minutes or 30-0 minutes before

24 urve, 0.80; 95% CI, 0.69-0.88; P = .03), and SAP pattern standard deviation (area under the curve, 0.

25 (C-statistic, 0.85; 95% CI, 0.77-0.92), and SAP <1.5 x ULN (C-statistic, 0.65; 95% CI, 0.55-0.73).

26 he sectoral measurements between the BCI and SAP.

27 control T-helper function, such as CD40L and SAP.

28 present on mouse lung epithelial cells, and SAP and the aminothiazole potentiate IL-10 production fr

29 CRP and SAP dose-dependently and substoichiometrically inhibited

30 The serum CRP and SAP increases correlated with tau post-CPB levels signif

31 CRP and SAP interacted with fresh and aggregated Abeta(1-40) and

32 lar visual fields were estimated for FDT and SAP and used to calculate rates of change.

33 er than short-term variability on SD-OCT and SAP.

34 n of radiolabelled peptide p5+14 with p5 and SAP, in amyloid-laden mice, using dual-energy SPECT imag

35 the presence of concentrations of serum and SAP that normally completely inhibit fibrocyte different

36 ls regulate B cell survival in a SLAMF6- and SAP-dependent manner.

37 tral-domain optical coherence tomography and SAP were performed at 6-month intervals.

38 Treatment with CPHPC followed by an anti-SAP antibody safely triggered clearance of amyloid depos

39 pecifically targeted by therapeutic IgG anti-SAP antibodies.

40 fused a fully humanized monoclonal IgG1 anti-SAP antibody.

41 In response to antigen, SAP-deficient mice form extrafollicular B cell responses

42 , 10-20% of which can evolve into severe AP (SAP) causing significant morbidity and mortality.

43 that regulates the level of STERILE APETALA (SAP) protein in the developing petals.

44 gue of Arabidopsis thaliana STERILE APETALA (SAP), that forms part of an SKP1/Cullin/F-box E3 ubiquit

45 te differentiation, and sialidases attenuate SAP function.

46 a key paracrine factor of BMSCs attenuating SAP targeting the NF-kappaB1/p50 gene and suppressing th

47 For binocular SAP mean sensitivity, each 1 dB/year change was associat

48 Each 1-dB change in binocular SAP MS per year was associated with a change of 2.9 unit

49 Each 1 decibel (dB)/year change in binocular SAP MS was associated with a change of 2.0 units in the

50 ty index, each 1 dB/year change in binocular SAP MS was associated with a change of 3.0 units in the

51 severity and the rate of change in binocular SAP sensitivity, each 1-mum-per-year loss of RNFL thickn

52 res during follow-up and change in binocular SAP sensitivity.

53 res during follow-up and change in binocular SAP sensitivity.

54 HFD causes steatosis, and both SAP and 1866 reduced it.

55 of change by spectral-domain OCT vs 9.1% by SAP (P < .001).

56 Rates of visual field loss were assessed by SAP.

57 thickness change in eyes that progressed by SAP were faster than in those that did not progress (-1.

58 s progressing at moderate or faster rates by SAP vs 26.5% by spectral-domain OCT (P = .055).

59 ectral-domain OCT were classified as slow by SAP and vice versa.

60 CD45 mAbs conjugated with saporin CD45 (CD45-SAP).

61 In contrast to irradiation, CD45-SAP completely avoided neutropenia and anemia, spared bo

62 tively, were conditioned with CD45-SAP, CD45-SAP plus 2 Gy of total body irradiation (TBI), 2 Gy of T

63 Conditioning with CD45-SAP allows multilineage engraftment and robust immune re

64 Conditioning with CD45-SAP enabled high levels of multilineage engraftment in b

65 on, respectively, were conditioned with CD45-SAP, CD45-SAP plus 2 Gy of total body irradiation (TBI),

66 In SLE T cells, SAP protein is also subject to increased degradation by

67 iating the need for increasingly challenging SAP timing recommendations.

68 ter first using CPHPC to deplete circulating SAP, we infused a fully humanized monoclonal IgG1 anti-S

69 (conA), and human serum amyloid P component (SAP) at elevated temperatures prior, during, and after d

70 protein (CRP) and serum amyloid P component (SAP), two major classical pentraxins in humans, are solu

71 saminoglycans and serum amyloid P component (SAP).

72 l plasma protein, serum amyloid P component (SAP).

73 NUSAP1 contains a conserved SAP domain (SAF-A/B, Acinus, and PIAS).

74 scular signals) and was attenuated by DbetaH-SAP treatment (coherence: 0.74 +/- 0.12 vs.

75 0.54 +/- 0.10, HF+Veh vs. HF+DbetaH-SAP rats; P < 0.05).

76 +/- 2.5 events h(-1) ; HF+Veh vs. HF+DbetaH-SAP; P < 0.05) and improved cardiac autonomic control in

77 amine beta-hydroxylase-saporin toxin (DbetaH-SAP) was used to selectively lesion RVLM-C1 neurones.

78 In anti-DBH-SAP injected SHR, the antihypertensive effect of repeate

79 ith anti-DBH conjugated to saporin (anti-DBH-SAP).

80 ockade of IL-13-induced CD38/cADPR-dependent SAP antigen passaging in mice inhibited induction of cli

81 IL-13-CD38-cADPR-dependent SAP sampling of food allergens was conserved in human in

82 carboxylic acid (CPHPC) efficiently depletes SAP from the plasma but leaves some SAP in amyloid depos

83 ate of FDT PSD change in eyes that developed SAP visual field loss was 0.07 dB/year versus 0.02 dB/ye

84 algorithm-defined versus physician-diagnosed SAP in 1088 patients who had dysphagic acute stroke from

85 Physicians diagnosed SAP in 176/1088 (16%) and the algorithm in 123/1088 (11.

86 CI 56% to 73%), respectively) in diagnosing SAP.

87 tic and functional assays confirmed elevated SAP activity in transformed strains.

88 EternaBrain-SAP outperforms previously published RNA design algorith

89 tients examined for glaucoma and to evaluate SAP, OCT and RGC counts capability to discriminate the w

90 Neutrophils were found to express SAP; however, adoptive transfer experiment supported a n

91 Finally, SAP inhibits the heat-induced amorphous aggregation of h

92 For SAP with short infusion time no clear superior timing in

93 61 +/- 0.34 dB, respectively (P < 0.001) for SAP MD and 1.95 +/- 1.86 mum vs. 0.81 +/- 0.56 mum, resp

94 A damage of approximately -6 dB for SAP MD, indicating relatively early visual field loss, m

95 tem, and that FcgammaR are not necessary for SAP function.

96 levels could serve as an early predictor for SAP.

97 The role for SAP was subset-specific, as Vgamma1Vdelta6.3, Vgamma4, V

98 the Guided Progression Analysis software for SAP.

99 5% CI, 0.86-0.96), which was larger than for SAP mean deviation (area under the curve, 0.81; 95% CI,

100 thickness and -0.09 +/- 0.36 dB per year for SAP mean deviation.

101 After adjusting for the contribution from SAP, 26% (95% CI, 12%-39%) of the variability of change

102 baseline and rate of change information from SAP had stronger ability to predict change in NEI VFQ-25

103 os et al. combining light sensitivities from SAP and retinal thickness from OCT.

104 iferative disease (XLP), who lack functional SAP, were hyperresponsive to PD-1 signaling, confirming

105 Intraseptal GAT1-SAP treatment did not alter baseline or behaviorally sti

106 alculated in 87 eyes with advanced glaucoma (SAP MD </=-12 dB).

107 scriminate eyes with repeatable glaucomatous SAP defects vs eyes with normal fields.

108 ncreased adipocyte size; for mice on an HFD, SAP improved glucose tolerance test results and reduced

109 However, SAP and CRP bind the same Fcgamma receptors (FcgammaR) w

110 Our results identify SAP as an inhibitor of PD-1 function and SHP2 as a poten

111 In SAP patients, PAr had greater predictive strength than d

112 1.30, 1.63) and 1.31 (95% CI: 1.21, 1.41) in SAP and AMI patients, respectively.

113 te the effect of HbA1c on rates of change in SAP mean deviation (MD) and OCT RNFL thickness loss over

114 r subjects with the same amount of change in SAP sensitivity, those with shorter follow-up times had

115 The resulting decrease in SAP activity leads to a shortening of the cell prolifera

116 evaluated the role of pancreatic lipases in SAP-associated visceral fat necrosis, the inflammatory r

117 een cystathionine and all-cause mortality in SAP patients was stronger among nonsmokers and those wit

118 of unreliable responses on the MD and PSD in SAP.

119 GC B cell formation was markedly reduced in SAP-deficient NOD mice, T cells with a GC Tfh phenotype

120 e a blocked anti-insulin vaccine response in SAP-deficient NOD mice.

121 uced similar anti-RABV antibody responses in SAP-deficient and wild-type mice, demonstrating that BAF

122 own to contain anti-CD3/TCR Abs, resulted in SAP downregulation.

123 years; however, its pathogenesis and role in SAP outcomes are poorly understood.

124 tic fat lipolysis plays an important role in SAP, we evaluated the role of pancreatic lipases in SAP-

125 ntours (CSIs) may reduce test variability in SAP by identifying regions of the visual field with stat

126 mum set of items that should be addressed in SAPs for clinical trials, developed with input from stat

127 ems that should be addressed and included in SAPs for clinical trials.

128 o a complete ophthalmic evaluation including SAP and Spectral Domain OCT (SD-OCT) of RNFL and macular

129 was inversely correlated with intracellular SAP levels.

130 of transcription 1 (STAT1) and utilizing its SAP-domain function.

131 ormed in Europe by using iodine-123-labelled SAP; however, this tracer is not available in the US.

132 acceptor site in COP1 and the SUMO E3 ligase SAP and Miz 1 (SIZ1).

133 the day, versus 24 h use of patient-managed SAP only, in free-living conditions.

134 odel adjusting for baseline MoCA score, mean SAP MD, age, sex, race/ethnicity, educational level, inc

135 Mechanistically, SAP opposed PD-1 function by acting as a molecular shiel

136 Conversely, compared with control mice, SAP knockout mice fed on a normal diet had increased whi

137 IGN ligand and anti-DC-SIGN antibodies mimic SAP effects in vitro.

138 isual field was estimated from the monocular SAP tests, and rates of change in mean sensitivity (MS)

139 Moreover, SAP bound principally to hepatosplenic amyloid, whereas

140 teins associated with the silicalemma, named SAPs for Silicalemma Associated Proteins.

141 They also reveal a novel SAP adaptor-independent function for a SLAM receptor.

142 he pro- and anti-amyloidogenic activities of SAP are not mutually exclusive, but reflect two sides of

143 igen through the recruitment and activity of SAP-Fyn and SHP-1, respectively.

144 compared early versus late administration of SAP before surgery.

145 Early administration of SAP did not significantly reduce the risk of SSI compare

146 amily receptor signaling through deletion of SAP resulted in impaired thymic Vgamma1 and Vgamma4 T ce

147 were highly predictive of the development of SAP visual field loss in glaucoma suspects.

148 Mean deviation of SAP was -0.47 +/- 0.9 dB and -1.43 +/- 2.3 dB in the con

149 OR of algorithmic and physician diagnosis of SAP were assessed using adjudicated SAP as the reference

150 Forced expression of SAP in SLE T cells normalized IL-2 production, calcium (

151 port depicting innate protective function of SAP in an acute pulmonary infection.

152 supported a neutrophil-extrinsic function of SAP in neutrophil extracellular trap formation.

153 y unrecognized innate protective function of SAP, an adaptor protein primarily reported in T cells, N

154 show that for mice on an HFD, injections of SAP and a synthetic CD209 ligand (1866) reduced HFD-incr

155 C B cell development were blocked by loss of SAP in K/BxN mice.

156 s of NOD mice, which was enhanced by loss of SAP NOD T cells override SAP requirement to undergo acti

157 necrosis volume of 112.5 ml was a marker of SAP and 433.0 ml cut-off value could be used to predict

158 473.4 pg/ml) were reliable early markers of SAP.

159 n hepatic amyloid load, as shown by means of SAP scintigraphy and measurement of extracellular volume

160 us SAP use during the day versus 2 months of SAP use only under free-living conditions.

161 ained largely intact, increased mortality of SAP-deficient mice correlated with increased accumulatio

162 ty, educational level, income, and number of SAP tests, each 5-point decline in MoCA score was associ

163 Mean rate of SAP MD change was -0.09 +/- 0.20 decibel/year (median -0

164 Ordinary least squares linear regression of SAP mean deviation (MD) and SD-OCT global retinal nerve

165 ordinary least squares linear regressions of SAP mean deviation (MD) values over time.

166 ncrease of 0.23 dB in the SD of residuals of SAP MD (95% CI, 0.11-0.35; P < .001).

167 ncrease of 0.18 dB in the SD of residuals of SAP MD (R2 = 4.3%; 95% CI, 0.06-0.30; P = .003).

168 Restoration of SAP levels in SLE T cells corrects the overexcitable lup

169 The risk of SAP increased in a dose-response manner with delays in S

170 SALT) were associated with patients' risk of SAP.

171 r stroke, are associated with higher risk of SAP.

172 OR 2.01, 1.76 to 2.30) had a higher risk of SAP.

173 mmune processes, we investigated the role of SAP in T1D and autoantibody-mediated arthritis.

174 Silencing of SAP augmented and overexpression blocked PD-1 function.

175 The association between timing of SAP and SSI was assessed using multivariable logistic re

176 The importance of appropriate timing of SAP before surgery has long been recognized.

177 the longitudinal measurement variability of SAP mean deviation (MD) using linear regressions.

178 gest that the physiological contributions of SAPs to vaginal immunopathology require hypha formation,

179 Whereas spontaneous arthritis depended on SAP in the autoantibody-mediated K/BxN model, organized

180 hms for detection of glaucoma progression on SAP and SD-OCT have relied on short-term variability dat

181 Forty-six eyes (8.4%) showed progression on SAP during follow-up.

182 nts) was conducted to establish consensus on SAPs.

183 enhanced by loss of SAP NOD T cells override SAP requirement to undergo activation and proliferation

184 rombin may be an initial trigger to override SAP inhibition of fibrocyte differentiation to initiate

185 lated human serum proteins, serum amyloid P (SAP) and C-reactive protein (CRP), strongly affect fibro

186 lylated glycoprotein called serum amyloid P (SAP) inhibits fibrocyte differentiation, and sialidases

187 The systemic pentraxin serum amyloid P (SAP) inhibits inflammation.

188 f C-reactive protein (CRP), serum amyloid P (SAP), and alpha-2-macroglobulin (A2M).

189 bited by the plasma protein serum amyloid P (SAP), and healthy tissues contain very few fibrocytes.

190 ith the fibrocyte inhibitor serum amyloid P (SAP; pentraxin-2) significantly prolonged survival and s

191 n associated with severe acute pancreatitis (SAP) for over 100 years; however, its pathogenesis and r

192 ry cells (termed secretory antigen passages [SAPs]) that are localized to the SI villous and crypt re

193 linked lymphoproliferative disease patients, SAP deficiency reduces CD74 expression, resulting in the

194 ients with suspected stable angina pectoris (SAP) (3033 patients; median 10.7 y follow-up; 648 deaths

195 graphy for suspected stable angina pectoris (SAP) (n = 4131) and an independent cohort of patients wh

196 r angiography due to stable angina pectoris (SAP) or acute coronary syndrome (ACS).

197 6N beta2-m fibril was coated with pentameric SAP.

198 ity indices in standard automated perimetry (SAP) affect the global indices of visual field (VF) resu

199 Standard automated perimetry (SAP) and eye tracking perimetry (saccadic vector optokin

200 monitored with standard automated perimetry (SAP) and had longitudinal assessment of cognitive abilit

201 re tested with standard automated perimetry (SAP) at 6-month intervals, and evaluation of rates of vi

202 d annually and standard automated perimetry (SAP) at 6-month intervals.

203 d annually and standard automated perimetry (SAP) at 6-month intervals.

204 eld defects on standard automated perimetry (SAP) at baseline.

205 relations with standard automated perimetry (SAP) global indices were compared between the human grad

206 Differences in standard automated perimetry (SAP) mean deviation (MD) and integrated binocular mean s

207 regression of standard automated perimetry (SAP) mean deviation (MD) values over time.

208 re assessed by standard automated perimetry (SAP) mean deviation (MD).

209 t one reliable standard automated perimetry (SAP) test, while RNFL measurements were obtained using t

210 CT) and 19,812 standard automated perimetry (SAP) tests from 6138 eyes of 3669 patients with >=6 mont

211 ast 2 reliable standard automated perimetry (SAP) tests, 2 spectral domain OCT (SD-OCT) tests, and 2

212 resentation of standard automated perimetry (SAP) visual fields using 29,161 fields from 3,832 patien

213 eld loss using standard automated perimetry (SAP) when considering different frequencies of testing u

214 BCI device and standard automated perimetry (SAP) within 3 months.

215 Standard automated perimetry (SAP), the most common form of perimetry used in clinical

216 be observed by Standard Automated Perimetry (SAP), the second by Optic Coherence Tomography (OCT) tha

217 try to current standard automated perimetry (SAP).

218 asurements and standard automated perimetry (SAP).

219 -25), FDT, and standard automated perimetry (SAP).

220 irubin, albumin, serum alkaline phosphatase (SAP) times the upper limit of normal (ULN), platelets, a

221 atio (C/N ratio), soil available phosphorus (SAP), soil NH4(+)-N, soil NO3(-)N, aboveground biomass (

222 e report that the SAF-A/B, Acinus, and PIAS (SAP) domain of MANF selectively associates with the nucl

223 uired content of statistical analysis plans (SAPs) to support transparency and reproducibility.

224 move predictions with single-action-playout (SAP) of six strategies compiled by human players solves

225 nths of AP use from dinner to waking up plus SAP use during the day versus 2 months of SAP use only u

226 Diagnosing stroke-associated pneumonia (SAP) is challenging and may result in inappropriate anti

227 ces the risk of stroke-associated pneumonia (SAP), or of how quickly it should be done after admissio

228 tion method based on superabsorbent polymer (SAP) beads.

229 ncluding 510 single amino acid polymorphism (SAP) peptides, 2 INDEL peptides, 49 splice junction pept

230 Stretch-attend posture (SAP) occurs during risk assessment and is prevalent in c

231 act of three different set anode potentials (SAPs; -0.25, 0, and 0.25 V vs. standard hydrogen electro

232 arning method named Simpler Angle Predictor (SAP) to train simpler DNN models that enhance protein ba

233 em, and the symptom association probability (SAP) test might distinguish extraesophageal manifestatio

234 In 87% of the procedures, SAP was administered within 60 minutes before incision.

235 ation of surgical antimicrobial prophylaxis (SAP) for the prevention of surgical site infection (SSI)

236 isplay reduced levels of the adaptor protein SAP, probably as a result of continuous T cell activatio

237 ytic activation molecule-associated protein (SAP) adaptor are important in the development of several

238 cyte activation molecule-associated protein (SAP) adaptors.

239 ytic activation molecule-associated protein (SAP) are highly susceptible to one specific viral pathog

240 es, mediated by the SLAM-associated protein (SAP) family of adaptors.

241 ytic activation molecule-associated protein (SAP) was functionally and mechanistically analyzed for i

242 ytic activation molecule-associated protein (SAP), a critical intracellular signaling molecule for T-

243 tivation molecule (SLAM)-associated protein (SAP)-deficient mouse model.

244 -95, PSD-93, and synapse-associated protein (SAP)102 and combining electrophysiology and transmission

245 The T. pseudonana SAPs (TpSAPs) are characterized by their motif organizat

246 (UEV) domain with a pocket that can bind PT/SAP motifs and another pocket that can bind Ub.

247 0 patients were randomly assigned to receive SAP early (2798 patients) or late (2782 patients).

248 ter-generated list in a 1:1 ratio to receive SAP early in the anaesthesia room or late in the operati

249 Data regarding SAP via intracameral injection were also retrieved.

250 inal nerve fiber layer scans, and 2 reliable SAP tests were included.

251 ells (BMSCs) have the potential of repairing SAP, but the detailed mechanism remains unknown.

252 binding of these antibodies to the residual SAP in amyloid deposits activates complement and trigger

253 Cells are permeabilized using saponin (SAP), digitonin (DIG) or recombinant perfringolysin O (r

254 ingle dose of the immunotoxin, CD45-saporin (SAP), enabled efficient (>90%) engraftment of donor cell

255 eyes were compared with global and sectoral SAP parameters.

256 Since SAP is one of the main causes of mortality after acute s

257 Together, these data suggest that the SLAM/SAP signaling pathway plays a larger role in gammadelta

258 we investigated the extent to which the SLAM/SAP signaling pathway regulates the functional programmi

259 depletes SAP from the plasma but leaves some SAP in amyloid deposits that can be specifically targete

260 Algorithm-based approaches can standardise SAP diagnosis for clinical practice and research.

261 described the discovery of sulfonylpyridine (SAP), benzothiazole (BZT), indazole (IND), and tetrahydr

262 binding domain of p65 through its C-terminal SAP-like domain in the nuclei under the condition of inf

263 degradation of propionate in all the tested SAPs was facilitated by syntrophic interactions between

264 r relative abundance varied among the tested SAPs.

265 lactically with cefuroxime demonstrated that SAP alone does not prevent endophthalmitis.

266 We present evidence that SAP protein levels are decreased in T cells and in their

267 We find that SAP is essential when autoantibody-driven immune complex

268 Here, we report that SAP but not CRP binds the receptor DC-SIGN (SIGN-R1) to

269 We then empirically show that SAP can significantly outperform existing state-of-the-a

270 Our data suggest that SAP activates DC-SIGN to regulate the innate immune syst

271 These data suggest that SAP first exhibits anti-amyloidogenic activity possibly

272 We identify that SAPs are a mechanism by which food allergens are channel

273 to bridging of the MEF2C-bound sites by the SAP domain-containing co-activator protein myocardin, an

274 In contrast, the SAP of -0.25 V had the lowest electron flux to current (

275 The nsp1beta L126A mutant in the SAP domain did not bind to Nup62, and in L126A-expressin

276 We demonstrate that antibody blocking of the SAP-dependent 2B4 receptor is sufficient to induce XLP-l

277 cgammaRI with an IgG-blocking Ab reduces the SAP effect on fibrocyte differentiation, and ligating Fc

278 In crystal structures the SAP domain engages the cleft between NBD subdomains Ia a

279 by piloting of the guidance document in the SAPs of 5 trials.

280 tient-managed sensor-augmented pump therapy (SAP) during the day, versus 24 h use of patient-managed

281 nt genera detected on the anode of all three SAPs based on 16S rRNA gene sequencing were Geobacter, S

282 Among the three SAPs tested, 0 V showed the highest electron flux to ele

283 fat by pancreatic lipases convert mild AP to SAP independent of pancreatic necrosis and the inflammat

284 noted obesity to convert mild cerulein AP to SAP with greater cytokines, unsaturated fatty acids (UFA

285 dictions were generated, with comparisons to SAP mean deviation (MD) rates and point-wise (PW) regres

286 and in Phase 1 and Phase 2 clinical trials, SAP affects several aspects of the innate immune system

287 Clinicians now use SAP for the diagnosis and management of glaucoma through

288 In vitro, SAP, but not 1866, treated cells isolated from white fat

289 amma4, Vgamma5, but not Vgamma6 subsets were SAP-dependent.

290 When SAP beads swell with water, small molecules can be sorbe

291 (FcgammaR) with similar affinities, and why SAP and CRP have opposing effects is unknown.

292 ificantly stronger absolute correlation with SAP mean deviation (rho=0.54) than the probability of GO

293 nd inhibiting fibrocyte differentiation with SAP may interfere with this process.

294 For 2 years, consecutive patients with SAP indication undergoing general, orthopedic, or gyneco

295 Among patients with SAP, there was a positive association between plasma cys

296 ) patients were adjudicated as patients with SAP.

297 IOP(cc) had the strongest relationship with SAP MD loss over time (R(2) = 24.5%) and was significant

298 he target range was higher with AP than with SAP use: 66.7% versus 58.1% (paired difference 8.6% [95%

299 the NOD model retains pathogenic Tfh without SAP.

300 saccades became less accurate with worsening SAP sensitivity.