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1 gens such as Marburg virus, Nipah virus, and SARS coronavirus.
2 might be unique to the SUD and, thus, to the SARS coronavirus.
3 P-PMO showed low inhibitory activity against SARS coronavirus.
4 ected with H5N1 avian influenza virus or the SARS-coronavirus.
5 xperiments to address the above question for SARS coronaviruses.
6 s found to be conserved in several human non-SARS coronaviruses.
7 efforts to develop polymerase inhibitors for SARS coronaviruses.
8 ory syndrome (SARS) donor and identified 200 SARS coronavirus 2 (SARS-CoV-2) binding antibodies that
9                                              SARS Coronavirus 2 (SARS-CoV-2) emerged in late 2019, le
10                                              SARS coronavirus 2 (SARS-CoV-2) has caused an ongoing gl
11 s disease 2019 (COVID-19) pandemic caused by SARS coronavirus 2 (SARS-CoV-2) has caused significant m
12                                          The SARS coronavirus 2 (SARS-CoV-2) main protease (Mpro) is
13                                  We used the SARS coronavirus 2 (SARS-CoV-2) mouse-adapted, passage 1
14                                          The SARS coronavirus 2 (SARS-CoV-2) pandemic is a global cha
15 pid antigen immunoassay for the detection of SARS coronavirus 2 (SARS-CoV-2) proteins from nasal or n
16 reakthrough cases and infection by different SARS coronavirus 2 (SARS-CoV-2) variants have remained l
17 2 (TMPRSS2) is a human protease required for SARS coronavirus 2 (SARS-CoV-2) viral entry and may repr
18                                        Since SARS coronavirus 2 (SARS-CoV-2) was identified as the ca
19 syndrome, as well as the current pandemic of SARS coronavirus 2 (SARS-CoV-2)(1-4).
20 share extensive sequence homology with human SARS coronavirus 2 (SARS-CoV-2).
21 ss the magnitude, function, and phenotype of SARS coronavirus 2-specific (SARS-CoV-2-specific) CD4+ T
22           Severe acute respiratory syndrome (SARS) coronavirus 2 (SARS-CoV-2) has emerged as the infe
23 the novel severe acute respiratory syndrome (SARS) coronavirus 2, SARS-CoV-2.
24 ive vaccines, therapies, and diagnostics for SARS- coronavirus 2 (CoV-2), the development of which wi
25 ng impact of the COVID-19 pandemic caused by SARS-coronavirus 2 (SARS-CoV-2) has raised important que
26 nse to the ongoing global pandemic caused by SARS coronavirus-2 (SARS-CoV-2), we use this vaccine pla
27 caused by severe acute respiratory syndrome (SARS)-coronavirus-2 (CoV-2) has not only taken millions
28                                              SARS-Coronavirus-2 (SARS-CoV-2) causes Coronavirus disea
29                         After challenge with SARS coronavirus, all monkeys in the control group shed
30                            Here, we identify SARS coronavirus amino-terminal replicase products nsp1,
31 locker of severe acute respiratory syndrome (SARS) coronavirus and Ebola pseudotype virus.
32 dge about severe acute respiratory syndrome (SARS) coronaviruses and their life cycle.
33 sis, influenza, respiratory syncytial virus, SARS-Coronavirus and pneumonia.
34 specially severe acute respiratory syndrome (SARS) coronavirus, and Middle East respiratory syndrome
35 og in the severe acute respiratory syndrome (SARS) coronavirus appears to be group 1-like in that it
36 luenza A virus (IAV), Zika virus, HIV-1, and SARS coronaviruses by inhibiting virus-cell membrane fus
37 how that the PLpro domains from the MERS and SARS coronaviruses can recognize and process the same su
38                                            A SARS-coronavirus challenge was given to all monkeys 28 d
39  that infection of human airway epithelia by SARS coronavirus correlates with the state of cell diffe
40 zate (PubChem CID 16725315) were tested in a SARS coronavirus (CoV) and Ebola virus-pseudotype infect
41 al structure of the N-terminal domain of the SARS coronavirus (CoV) nucleocapsid protein.
42 st the papain-like protease (PLpro) from the SARS coronavirus (CoV).
43 es of the severe acute respiratory syndrome (SARS) coronavirus (CoV) are sensitive to neutralization
44 lation of severe acute respiratory syndrome (SARS) coronavirus (CoV) from Himalayan palm civets (Pagu
45  model of severe acute respiratory syndrome (SARS) coronavirus (CoV) lung disease.
46  from the severe acute respiratory syndrome (SARS) coronavirus (CoV) that stimulated -1 PRF.
47 oteins of severe acute respiratory syndrome (SARS) coronavirus (CoV) to protective immunity by expres
48 nVs), and severe acute respiratory syndrome (SARS) coronavirus (CoV).
49 s against severe acute respiratory syndrome (SARS) coronavirus (CoV).
50  In 2002, severe acute respiratory syndrome (SARS)-coronavirus (CoV) appeared as a novel human virus
51 hat the respiratory tract is a major site of SARS-coronavirus (CoV) infection and disease morbidity.
52                 Recent studies indicate that SARS-coronavirus (CoV) spike protein (S protein) and its
53 hods by: 1) identifying the sites explaining SARS coronavirus differences between human, bat and palm
54                                          The SARS coronavirus encodes a novel membrane protein 6 that
55       The severe acute respiratory syndrome (SARS) coronavirus encodes several RNA-processing enzymes
56 COVID-19, the K18-hACE2 mouse, expresses the SARS-coronavirus entry receptor, human angiotensin-conve
57                    We expressed the complete SARS coronavirus envelope spike (S) protein from a recom
58              The envelope (E) protein of the SARS coronavirus forms a pathogenic cation-selective cha
59                                         Both SARS coronavirus genomes encode two overlapping large po
60 ot group 2-like, as would be expected by the SARS coronavirus group 2-like 3' UTR structure.
61 tified hinge regions are highly conserved in SARS coronaviruses, highlighting their functional import
62 acting alone, can prevent replication of the SARS coronavirus in the lung, a promising observation fo
63  3 of the severe acute respiratory syndrome (SARS) coronavirus includes a "SARS-unique domain" (SUD)
64  lungs of severe acute respiratory syndrome (SARS)-coronavirus-infected humans.
65                  We identified 32 studies of SARS coronavirus infection and severe influenza.
66 70, was among the best characterized against SARS coronavirus infection, showing weight loss and othe
67           Severe acute respiratory syndrome (SARS) coronavirus infection and growth are dependent on
68 odels for severe acute respiratory syndrome (SARS) coronavirus infection of humans are needed to eluc
69 o severe acute respiratory syndrome-related (SARS) coronavirus, inhibit the classical nuclear import
70            The hepatitis associated with the SARS coronavirus, Isolated Ductular Hyperplasia in patie
71 combinant severe acute respiratory syndrome (SARS) coronavirus lacking the E gene (rSARS-CoV-DeltaE)
72                                          The SARS coronavirus main proteinase (SARS CoV main proteina
73 s) of the severe acute respiratory syndrome (SARS) coronavirus may encode determinants of virus virul
74 me entry site (IRES)-driven mRNAs, including SARS coronavirus mRNAs, hepatitis C virus (HCV), and cri
75 with BHPIV3/SARS-S induced the production of SARS-coronavirus-neutralising serum antibodies, indicati
76           Severe acute respiratory syndrome (SARS) coronavirus nonstructural protein 1 (nsp1) binds t
77 gand that was found to inhibit -1 PRF in the SARS coronavirus on the conformational dynamics of the S
78       The severe acute respiratory syndrome (SARS) coronavirus papain-like protease (PLpro) is a DUB
79 tivirals and vaccines, but also for studying SARS coronavirus pathogenesis.
80 rotein of severe acute respiratory syndrome (SARS) coronavirus plays important roles in both viral re
81 )) of the severe acute respiratory syndrome (SARS) coronavirus proteolyzes viral polyproteins and has
82                                We found that SARS coronavirus replicase products can be detected by 4
83 tion, the severe acute respiratory syndrome (SARS) coronavirus replicated to high titers in the respi
84 al methyltransferases play critical roles in SARS coronavirus replication and are therefore promising
85 COGs) database, Retroviral Genotyping Tools, SARS Coronavirus Resource, SAGEmap, Gene Expression Omni
86    A vectored mucosal vaccine expressing the SARS-coronavirus S protein alone may be highly effective
87        We previously showed that recombinant SARS coronavirus (SARS-CoV) (Urbani strain based) lackin
88 rotein microarray that harbors proteins from SARS coronavirus (SARS-CoV) and five additional coronavi
89  highly contagious disease that is caused by SARS coronavirus (SARS-CoV) and for which there are curr
90 ar receptor for two divergent coronaviruses, SARS coronavirus (SARS-CoV) and human coronavirus NL63 (
91                Despite the identification of SARS coronavirus (SARS-CoV) as the etiologic agent of SA
92                     The spike protein (S) of SARS coronavirus (SARS-CoV) attaches the virus to its ce
93                                              SARS coronavirus (SARS-CoV) causes severe acute respirat
94                                              SARS coronavirus (SARS-CoV) encodes several unique group
95                                          The SARS coronavirus (SARS-CoV) has been identified as the e
96 rude fatality rate (around 50% vs. 10%) than SARS coronavirus (SARS-CoV) infection.
97 , for "middle of the SARS-unique domain") in SARS coronavirus (SARS-CoV) nonstructural protein 3 (nsp
98 pidemiologic studies have suggested that the SARS coronavirus (SARS-CoV) originated from animals.
99 tructures of NL63 coronavirus (NL63-CoV) and SARS coronavirus (SARS-CoV) receptor-binding domains (RB
100 ribe the construction of a panel of isogenic SARS coronavirus (SARS-CoV) strains bearing variant spik
101 evere acute respiratory syndrome (SARS), the SARS coronavirus (SARS-CoV), is a member of this large f
102 in-converting enzyme 2 (ACE2), isolated from SARS coronavirus (SARS-CoV)-permissive Vero E6 cells, th
103 ause of its genetic similarities to the 2003 SARS coronavirus (SARS-CoV).
104  Incidentally, ACE2 is also used by group II SARS coronavirus (SARS-CoV).
105 enzyme 2 (ACE2) is a functional receptor for SARS coronavirus (SARS-CoV).
106 s caused by a previously unknown coronavirus-SARS coronavirus (SARS-CoV).
107 ten fatal severe acute respiratory syndrome (SARS) coronavirus (SARS-CoV) infection.
108 rotein of severe acute respiratory syndrome (SARS) coronavirus (SARS-CoV) is not only responsible for
109 cation of severe acute respiratory syndrome (SARS) coronavirus (SARS-CoV) requires proteolytic proces
110 ) against severe acute respiratory syndrome (SARS) coronavirus (SARS-CoV) spike (S) glycoprotein conf
111 rotein of severe acute respiratory syndrome (SARS) coronavirus (SARS-CoV) were mapped by Pepscan anal
112 ks of the severe acute respiratory syndrome (SARS) coronavirus (SARS-CoV), but concerns remain over t
113 TR in the severe acute respiratory syndrome (SARS) coronavirus (SCoV) can each replace the 301-nt 3'
114           Severe acute respiratory syndrome (SARS) coronavirus (SCoV) causes a recently emerged human
115                                           In SARS coronaviruses, the non-structural protein 16 (nsp16
116 he elderly to severe SARS and the ability of SARS coronavirus to replicate in mice led us to examine
117  NL63 and severe acute respiratory syndrome (SARS) coronavirus to determine if DUB activity mediates
118  of the respiratory tract, the major site of SARS- coronavirus transmission and disease.
119       The severe acute respiratory syndrome (SARS) coronavirus virus non-structural protein 15 is a M
120 virus, all monkeys in the control group shed SARS coronavirus, with shedding lasting 5-8 days.

 
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