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1 gens such as Marburg virus, Nipah virus, and SARS coronavirus.
2 might be unique to the SUD and, thus, to the SARS coronavirus.
3 P-PMO showed low inhibitory activity against SARS coronavirus.
4 ected with H5N1 avian influenza virus or the SARS-coronavirus.
5 xperiments to address the above question for SARS coronaviruses.
6 s found to be conserved in several human non-SARS coronaviruses.
7 efforts to develop polymerase inhibitors for SARS coronaviruses.
8 ory syndrome (SARS) donor and identified 200 SARS coronavirus 2 (SARS-CoV-2) binding antibodies that
11 s disease 2019 (COVID-19) pandemic caused by SARS coronavirus 2 (SARS-CoV-2) has caused significant m
15 pid antigen immunoassay for the detection of SARS coronavirus 2 (SARS-CoV-2) proteins from nasal or n
16 reakthrough cases and infection by different SARS coronavirus 2 (SARS-CoV-2) variants have remained l
17 2 (TMPRSS2) is a human protease required for SARS coronavirus 2 (SARS-CoV-2) viral entry and may repr
21 ss the magnitude, function, and phenotype of SARS coronavirus 2-specific (SARS-CoV-2-specific) CD4+ T
24 ive vaccines, therapies, and diagnostics for SARS- coronavirus 2 (CoV-2), the development of which wi
25 ng impact of the COVID-19 pandemic caused by SARS-coronavirus 2 (SARS-CoV-2) has raised important que
26 nse to the ongoing global pandemic caused by SARS coronavirus-2 (SARS-CoV-2), we use this vaccine pla
27 caused by severe acute respiratory syndrome (SARS)-coronavirus-2 (CoV-2) has not only taken millions
34 specially severe acute respiratory syndrome (SARS) coronavirus, and Middle East respiratory syndrome
35 og in the severe acute respiratory syndrome (SARS) coronavirus appears to be group 1-like in that it
36 luenza A virus (IAV), Zika virus, HIV-1, and SARS coronaviruses by inhibiting virus-cell membrane fus
37 how that the PLpro domains from the MERS and SARS coronaviruses can recognize and process the same su
39 that infection of human airway epithelia by SARS coronavirus correlates with the state of cell diffe
40 zate (PubChem CID 16725315) were tested in a SARS coronavirus (CoV) and Ebola virus-pseudotype infect
43 es of the severe acute respiratory syndrome (SARS) coronavirus (CoV) are sensitive to neutralization
44 lation of severe acute respiratory syndrome (SARS) coronavirus (CoV) from Himalayan palm civets (Pagu
47 oteins of severe acute respiratory syndrome (SARS) coronavirus (CoV) to protective immunity by expres
50 In 2002, severe acute respiratory syndrome (SARS)-coronavirus (CoV) appeared as a novel human virus
51 hat the respiratory tract is a major site of SARS-coronavirus (CoV) infection and disease morbidity.
53 hods by: 1) identifying the sites explaining SARS coronavirus differences between human, bat and palm
56 COVID-19, the K18-hACE2 mouse, expresses the SARS-coronavirus entry receptor, human angiotensin-conve
61 tified hinge regions are highly conserved in SARS coronaviruses, highlighting their functional import
62 acting alone, can prevent replication of the SARS coronavirus in the lung, a promising observation fo
63 3 of the severe acute respiratory syndrome (SARS) coronavirus includes a "SARS-unique domain" (SUD)
66 70, was among the best characterized against SARS coronavirus infection, showing weight loss and othe
68 odels for severe acute respiratory syndrome (SARS) coronavirus infection of humans are needed to eluc
69 o severe acute respiratory syndrome-related (SARS) coronavirus, inhibit the classical nuclear import
71 combinant severe acute respiratory syndrome (SARS) coronavirus lacking the E gene (rSARS-CoV-DeltaE)
73 s) of the severe acute respiratory syndrome (SARS) coronavirus may encode determinants of virus virul
74 me entry site (IRES)-driven mRNAs, including SARS coronavirus mRNAs, hepatitis C virus (HCV), and cri
75 with BHPIV3/SARS-S induced the production of SARS-coronavirus-neutralising serum antibodies, indicati
77 gand that was found to inhibit -1 PRF in the SARS coronavirus on the conformational dynamics of the S
80 rotein of severe acute respiratory syndrome (SARS) coronavirus plays important roles in both viral re
81 )) of the severe acute respiratory syndrome (SARS) coronavirus proteolyzes viral polyproteins and has
83 tion, the severe acute respiratory syndrome (SARS) coronavirus replicated to high titers in the respi
84 al methyltransferases play critical roles in SARS coronavirus replication and are therefore promising
85 COGs) database, Retroviral Genotyping Tools, SARS Coronavirus Resource, SAGEmap, Gene Expression Omni
86 A vectored mucosal vaccine expressing the SARS-coronavirus S protein alone may be highly effective
88 rotein microarray that harbors proteins from SARS coronavirus (SARS-CoV) and five additional coronavi
89 highly contagious disease that is caused by SARS coronavirus (SARS-CoV) and for which there are curr
90 ar receptor for two divergent coronaviruses, SARS coronavirus (SARS-CoV) and human coronavirus NL63 (
97 , for "middle of the SARS-unique domain") in SARS coronavirus (SARS-CoV) nonstructural protein 3 (nsp
98 pidemiologic studies have suggested that the SARS coronavirus (SARS-CoV) originated from animals.
99 tructures of NL63 coronavirus (NL63-CoV) and SARS coronavirus (SARS-CoV) receptor-binding domains (RB
100 ribe the construction of a panel of isogenic SARS coronavirus (SARS-CoV) strains bearing variant spik
101 evere acute respiratory syndrome (SARS), the SARS coronavirus (SARS-CoV), is a member of this large f
102 in-converting enzyme 2 (ACE2), isolated from SARS coronavirus (SARS-CoV)-permissive Vero E6 cells, th
108 rotein of severe acute respiratory syndrome (SARS) coronavirus (SARS-CoV) is not only responsible for
109 cation of severe acute respiratory syndrome (SARS) coronavirus (SARS-CoV) requires proteolytic proces
110 ) against severe acute respiratory syndrome (SARS) coronavirus (SARS-CoV) spike (S) glycoprotein conf
111 rotein of severe acute respiratory syndrome (SARS) coronavirus (SARS-CoV) were mapped by Pepscan anal
112 ks of the severe acute respiratory syndrome (SARS) coronavirus (SARS-CoV), but concerns remain over t
113 TR in the severe acute respiratory syndrome (SARS) coronavirus (SCoV) can each replace the 301-nt 3'
116 he elderly to severe SARS and the ability of SARS coronavirus to replicate in mice led us to examine
117 NL63 and severe acute respiratory syndrome (SARS) coronavirus to determine if DUB activity mediates