ライフサイエンスコーパス: SCR

1 SCR also mitigates DPF-related increases in nitrogen dio

2 SCR and SCL23 are expressed specifically in the BS cells

3 SCR contributes to increased BVR by interspersed prolong

4 SCR drives the Na(+)/Ca(2+) exchange current inducing a

5 SCR systems increase emissions of nitrous oxide (N(2)O)

6 SCR-2-3-4 also inhibited C5a generation (IC(50) ~0.16 mu

7 SCR-2-3-4 inhibited both lectin and alternative pathway

8 SCRs were added to lepirudin plasma or sera from differe

9 Human phospholipid scramblase 1 (SCR) catalyzes phospholipid transmembrane (flip-flop) mo

10 Structural modeling suggested that SCR-2, SCR-3 and SCR-4 are critical for binding to the alternat

11 wed an overall length of 26-29 nm for its 20 SCR domains.

12 cial V(2)O(5)-WO(3)/TiO(2) catalyst in NH(3)-SCR of NO(x) under dry conditions has been analyzed in d

13 his indicates that the low-temperature NH(3)-SCR reaction proceeds via a reaction of these complexes

14 lyst to CO(2) and NO, which enters the NH(3)-SCR reaction.

15 meric complexes in the low-temperature NH(3)-SCR reaction.

16 ediated selective catalytic reduction (NH(3)-SCR) of NOx over Cu-ion-exchanged chabazite (Cu-CHA) cat

17 Under conditions for low-temperature NH(3)-SCR, oxygen only reacts with Cu(I) ions, which are prese

18 M), whereas shorter domains (SCR-3, SCR-2-3, SCR-3-4) were ineffective.

19 ) ~0.6 muM), whereas shorter domains (SCR-3, SCR-2-3, SCR-3-4) were ineffective.

20 ins, and 93 genes exhibiting SCR encoding 94 SCR isoforms.

21 issions from diesel vehicles equipped with a SCR.

22 Accurate SCR predictions can benefit homology modelling and seque

23 The low percentage of activity SCR over the local and near-dock cycles contributed to a

24 lockade led to further AP prolongation after SCR, and this strongly correlated with exaggerated BVR.

25 d during Ca(2+)-induced Ca(2+) release after SCR, and this contributes to AP prolongation.

26 In this study, an ammonia SCR system was examined with respect to its impact on bo

27 ral modeling suggested that SCR-2, SCR-3 and SCR-4 are critical for binding to the alternative pathwa

28 issions due to the rapid adoption of DPF and SCR systems by the California truck fleet are: (1) a 65%

29 2013 model years) equipped with both DPF and SCR were 69 +/- 15%, 92 +/- 32%, and 66 +/- 35% lower, r

30 The plant-specific GAI, RGA and SCR (GRAS) family proteins play critical roles in plant

31 tion of FANCJ helicase in regulating SCR and SCR associated gene amplification/duplications and imply

32 and annotated datasets of selenoprotein and SCR transcript and protein records to serve as annotatio

33 Our model reveals the timing of SHR and SCR dynamics and allows us to understand how protein mov

34 Ectopic activation of SHR and SCR in legumes is sufficient to induce root cortical cel

35 inary differential equation model of SHR and SCR in the QC and CEI which incorporated the stoichiomet

36 ameters into a mathematical model of SHR and SCR, which shows that SHR reaches a steady state in minu

37 stream transcriptional regulation of SHR and SCR.

38 GA in part through the regulation of SHR and SCR.

39 We find that Sox2 and SCR show no evidence of enhanced spatial proximity and t

40 formation experiments using chimeric SRK and SCR genes constructed with SC- and S36-derived sequences

41 24 accession, in which expression of SRK and SCR had been shown to exhibit a robust SI response.

42 clone was found to contain complete SRK and SCR sequences located close by one another in the derive

43 xpectation, however, coexpression of SRK and SCR was found to inhibit SRK-mediated signaling and to d

44 ck between the action potential upstroke and SCR.

45 no direct correlation between REM sleep and SCRs, indicating that REM may only modulate fear acquisi

46 genotype, gender, and race were evaluated as SCR risk factors.

47 modern pollution reduction measures such as SCR and fuel with low sulfur content.

48 anted to establish whether the below average SCR performance observed in this study is a systemic iss

49 trast, maltreated children exhibited blunted SCR to the CS+ and failed to exhibit differential SCR to

50 uM for C3 opsonization in sera), followed by SCR-1-2-3-4 (IC(50) ~0.6 muM), whereas shorter domains (

51 enuation of Ca(2+)-induced Ca(2+) release by SCR underlies AP prolongation via increased I(CaL.) Thes

52 tive catalytic reduction (SCR) or a combined SCR+DPF (SCRT) device.

53 uisition, despite no evidence of compromised SCR to unconditioned stimulus or compromised declarative

54 vmPFC lesion failed to produce a conditioned SCR during threat acquisition, despite no evidence of co

55 configurations including engine-out, DOC+CuZ-SCR+AMOX, V-SCR+AMOX, and DOC+DPF+CuZ-SCR+AMOX.

56 OC+CuZ-SCR+AMOX, V-SCR+AMOX, and DOC+DPF+CuZ-SCR+AMOX.

57 Both decreased SCR and the increase in CAG expansions were due to the u

58 NCJ pathological mutants exhibited defective SCR with an increased frequency of LTGC.

59 Defining SCRs requires the comparison of two or more homologous s

60 Forty-eight children (12.1%) demonstrated SCR, 44 of 398 children (11.1%; 95% confidence interval,

61 ive interaction with its pollen coat-derived SCR ligand.

62 o the CS+ and failed to exhibit differential SCR to the CS+ vs CS- during early conditioning.

63 behavior relationships: Neither differential SCR nor fear ratings during fear acquisition or extincti

64 Disrupting SCR function abolished periclinal divisions in this late

65 SCI was used to distinguish SCRs from non-SCRs.

66 (IC(50) ~0.6 muM), whereas shorter domains (SCR-3, SCR-2-3, SCR-3-4) were ineffective.

67 orms, MTCH2x and MTCH2xx (single- and double-SCR products, respectively), in addition to the canonica

68 Thus, double-SCR of MTCH2 regulates its own expression levels contrib

69 erved average N(2)O emission levels for "DPF+SCR" technology were relatively high at 182 mg/kg fuel,

70 een Cu(I) and Cu(II) oxidation states during SCR reaction.

71 threshold that is expected for an efficient SCR system, even when the SCR system was above the optim

72 322 selenoproteins, and 93 genes exhibiting SCR encoding 94 SCR isoforms.

73 We also show that stigma-expressed SCR causes entrapment of its SRK receptor in the endopla

74 se structures readily accommodated the extra SCR-1/2 domain pair present in CFHR5 nephropathy.

75 e substitution effects on US sire fertility (SCR).

76 the substitution effects on sire fertility (SCR, R = 0.6) between the US and IL samples.

77 al specificity in predicting eligibility for SCR; a quarter of eligible patients would receive inadeq

78 ecessary to further explore risk factors for SCR and to identify which individuals with SCD are at ri

79 In conclusion, in KT, screening for SCR more than once during the first year is not economic

80 3' UTR of MTCH2 is the necessary signal for SCR.

81 nding controversy about the active sites for SCR of NO with NH3 by supported V2O5-WO3/TiO2 catalysts.

82 gnificant effects of sleep were observed for SCRs or subjective ratings.

83 en Cu(OH)(2) and terminal Al species forming SCR inactive, Cu-aluminate like species.

84 Children exhibited higher SCR to the CS + Parent and CS + Stranger relative to the

85 rocess of fear conditioning predicted higher SCR for the child to the CS + Parent.

86 replication fork stalling and chromosomal HR/SCR in mouse cells.

87 n gene products, BRCA1 and BRCA2, control HR/SCR at stalled replication forks.

88 mpleted an image recognition task where HRD, SCRs and affective ratings were recorded again.

89 worst predictions pinpoints difficulties in SCR definitions.

90 less coherent fibers and less myelination in SCR and PCR only in male infants, but these abnormalitie

91 of the Fe centers (reduced in NH3, partly in SCR mixture, slight reduction in NO) strongly changed.

92 conditions including low-temperature (473 K) SCR catalysis and are rationalized through first-princip

93 CFHR4 lacks SCRs homologous to the complement inhibitory domains of

94 kdown of hCtr1 (536 +/- 191 mm(3) for Lenti- SCR-shRNA-PC-3 or 208 +/- 104 mm(3) for Lenti-SCR-shRNA-

95 CR-shRNA-PC-3 or 208 +/- 104 mm(3) for Lenti-SCR-shRNA-DU-145, P < 0.01).

96 -shRNA-PC-3 and 5.57 +/- 1.20 %ID/g in Lenti-SCR-shRNA-DU-145, P < 0.001) by PET quantification.

97 kdown of hCtr1 (7.21 +/- 1.48 %ID/g in Lenti-SCR-shRNA-PC-3 and 5.57 +/- 1.20 %ID/g in Lenti-SCR-shRN

98 fter learning, the THC group exhibited lower SCRs to the extinguished cue with no significant extinct

99 naling, we coexpressed an Arabidopsis lyrata SCR variant with its cognate SRK receptor in the stigma

100 e third analogue is a scrambled peptide (Mel-SCR) that contains the amino acid composition of melitti

101 ear-dock driving cycles, resulted in minimal SCR activity.

102 reshold, which are used in some of the newer SCRs, have the potential to control NOx emissions during

103 tor the Cu-CHA catalyst in action during NH3-SCR in the 150-400 degrees C range, targeting Cu oxidati

104 ective catalytic reduction of NO by NH3 (NH3-SCR) over a Fe-ZSM-5 zeolite catalyst.

105 ion kinetics on low-temperature standard NH3-SCR, supplemented by DFT calculations, as strong evidenc

106 and the isostructural Cu-SAPO-34) in the NH3-SCR of NOx.

107 SCI was used to distinguish SCRs from non-SCRs.

108 In addition to SPIO NWs, SCR-2-3-4 effectively inhibited C3 opsonisation and C5a

109 e where a diverse mix of trucks is observed, SCR systems on 2010 and newer engines reduce emitted nit

110 The ability of SCR to utilize unstructured data and produce spatially e

111 ntaneous activation of SRK in the absence of SCR ligand, these thioredoxins are thought to be essenti

112 ated to the previously described affinity of SCR for cholesterol-rich domains in membranes.

113 n of randomness by a statistical analysis of SCR events, which do not follow a Poisson process observ

114 0 physically realistic trial arrangements of SCR domains for scattering curve fits.

115 otential risk factors for the development of SCR.

116 ry elements drive the cortical expression of SCR, and stele-expressed SHR protein accumulates in cort

117 ence context all influence the likelihood of SCR.

118 In this study, the performance of SCR was studied by utilizing NOx, NH3, and particle meas

119 veness from the health sector perspective of SCR screening in the first year after KT using a Markov

120 floaters were associated with progression of SCR and may be criteria for referral for retinal examina

121 s and reveals a proportional relationship of SCR reaction rate to [surface VO(x) concentration](2) ,

122 emographic associations with the severity of SCR in 296 patients seen at both our SCD specialty clini

123 tween each clinical variable and severity of SCR.

124 action potential upstroke, waiting times of SCR events after the upstroke are narrowly distributed,

125 s were prevalence, age at onset, and type of SCR, based on examination by an ophthalmologist.

126 ovide a better understanding of the usage of SCR in combination with a higher sulfur level fuel and a

127 A database of SCRs was compiled from 386 SCOP superfamilies containing

128 ened arousal and increased generalization of SCRs and explicit measures of shock expectancy.

129 ial conditioning to the CS+ vs CS-, based on SCR and self-reported fear.

130 Based on SCR, both groups generalized the shock contingency to th

131 hus, the proposed state of the art review on SCR will create a renewed interest at all levels includi

132 Optimized SCR and SCRT retrofit technology reduced real-world NO (

133 H4(+),ads intermediates dominate the overall SCR reaction, especially for hydrothermally aged catalys

134 Results favored 1-time screening at peak SCR incidence rather than repeated screening.

135 protein gene products and failure to predict SCR.

136 it is still possible to effectively predict SCRs for a protein.

137 ogous structures, it is necessary to predict SCRs of a protein using information from only a set of h

138 neural networks were then trained to predict SCRs with various features deduced from a single structu

139 ) current (I(CaL)) with and without previous SCR indicated that I(CaL) was increased during Ca(2+)-in

140 ust temperature being high enough for proper SCR function.

141 oat-localized S-locus cysteine-rich protein (SCR).

142 For AMA-1, the seroconversion rates (SCRs) ranged from 0.121 (Ngodhe) to 0.202 (Ungoye), and

143 Stop codon readthrough (SCR) occurs when the ribosome miscodes at a stop codon.

144 n a process known as stop codon readthrough (SCR), producing extended protein isoforms with potential

145 ed a photographic spatial capture-recapture (SCR) methodology incorporating animal movement to estima

146 rubbing sites and spatial capture-recapture (SCR) modeling to estimate local density and population g

147 vity of 16 synthetic carbohydrate receptors (SCRs) that inhibit ZIKV infection in Vero and HeLa cells

148 combination, sister chromatid recombination (SCR), and break-induced replication whereas H2A.2 does n

149 processed by sister chromatid recombination (SCR), generating error-free or error-prone homologous re

150 hesis during sister chromatid recombination (SCR).

151 troduction of selective catalytic reduction (SCR) aftertreatment to meet stringent diesel NOx emissio

152 e presence of selective catalytic reduction (SCR) and selective noncatalytic reduction (SNCR) technol

153 in Cu/SSZ-13 selective catalytic reduction (SCR) catalysts have been recently identified as isolated

154 al Cu/SAPO-34 selective catalytic reduction (SCR) catalysts have experienced unexpected and quite per

155 anadium-based selective catalytic reduction (SCR) catalysts, and ammonia oxidation (AMOX) catalysts a

156 ith EGR and a selective catalytic reduction (SCR) device were measured on two different routes with t

157 s compared to selective catalytic reduction (SCR) equipped diesel vehicles.

158 rs (DPFs) and selective catalytic reduction (SCR) increased rapidly.

159 ssion limits, selective catalytic reduction (SCR) is increasingly utilized in ships, likely also in c

160 NH3-assisted selective catalytic reduction (SCR) of harmful nitrogen oxides and to unveil the SCR me

161 ctive for the selective catalytic reduction (SCR) of nitrogen oxides (NO x ) with ammonia (NH3), but

162 mistry in the selective catalytic reduction (SCR) of NO over Cu-exchanged SSZ-13.

163 The selective catalytic reduction (SCR) of NO(x) with NH(3) to N(2) with supported V(2) O(5

164 technologies, selective catalytic reduction (SCR) of NOx by NH3 over Cu- and Fe-ion exchanged zeolite

165 ivity for the selective catalytic reduction (SCR) of NOx with NH3 are established through experimenta

166 , and ammonia selective catalytic reduction (SCR) of NOx.

167 ers (DPF) and selective catalytic reduction (SCR) on heavy-duty diesel truck emissions were studied a

168 ers (DPF) and selective catalytic reduction (SCR) or a combined SCR+DPF (SCRT) device.

169 tathesis, and selective catalytic reduction (SCR) reactions.

170 el and have a Selective Catalytic Reduction (SCR) system for NO(X) abatement.

171 uipped with a Selective Catalytic Reduction (SCR) system in California.

172 d with a urea selective catalytic reduction (SCR) system.

173 er (DPF), and selective catalytic reduction (SCR) were tested on a chassis dynamometer.

174 equipped with selective catalytic reduction (SCR), two LNG's equipped with three-way catalyst (TWC),

175 Organization recommends shortcourse regimen (SCR) to treat multidrug resistant tuberculosis for patie

176 its essential enhancer Sox2 Control Region (SCR) in living embryonic stem cells (ESCs).

177 dentification of a stability control region (SCR), residues 97-118, in the Tm sequence that controls

178 revalence of structurally conserved regions (SCRs) even in highly divergent protein families.

179 to the BS cells, where it directly regulates SCR and SCL23 expression.

180 vel function of FANCJ helicase in regulating SCR and SCR associated gene amplification/duplications a

181 ly contains nine short complement regulator (SCR) domains, but a FHR5 mutant has been identified with

182 We believe that subclinical rejection (SCR) may be 1 of the factors that contribute to graft lo

183 Subclinical rejection (SCR) screening in kidney transplantation (KT) using prot

184 due to spontaneous calcium (Ca(2+)) release (SCR) from intracellular stores after the end of a preced

185 alternans, and spontaneous calcium release (SCR) incidence were determined.

186 lymorphism occurs in short consensus repeat (SCR) 7 of FH and results in decreased binding affinity o

187 ene encodes a unique short consensus repeat (SCR) domain protein.

188 n of the N-terminal short consensus repeats (SCRs) that are conserved in FHR2 and FHR5.

189 e effect of various short consensus repeats (SCRs, "sushi" domains) of human CD55 on nanoparticle-med

190 4-fold) in a schedule-controlled responding (SCR) behavioral assay.

191 ning by assessing skin conductance response (SCR) and declarative memory of stimulus-outcome continge

192 extinction while skin conductance response (SCR) and fear ratings were recorded.

193 ck expectancy and skin conductance response (SCR).

194 Skin conductance responses (SCR) and self-reported fear were acquired.

195 delivered) while skin conductance responses (SCR) were recorded.

196 ficant impact on skin conductance responses (SCR).

197 ration (HRD) and skin conductance responses (SCRs) were monitored.

198 Additionally, skin conductance responses (SCRs) were weakly correlated to the activity in the amyg

199 c arousal [i.e., skin conductance responses (SCRs)] and explicit measures of shock expectancy served

200 ors associated with sickle cell retinopathy (SCR) to inform development of screening guidelines for a

201 kinase (SRK) and the S-locus cysteine-rich (SCR) genes, as well as unlinked modifier loci required f

202 localized ligand, the S-locus cysteine-rich (SCR) protein.

203 ological synchronization between the child's SCR during observational learning and the parent's SCR d

204 ring observational learning and the parent's SCR during the actual process of fear conditioning predi

205 ct with the AtSHR binding protein, Scarecow (SCR).

206 tion factors SHORT-ROOT (SHR) and SCARECROW (SCR) are key regulators of root growth and of the asymme

207 AS proteins, SHORT-ROOT (SHR) and SCARECROW (SCR), cooperatively direct asymmetric cell division and

208 OOT (SHR) and its binding partner SCARECROW (SCR).

209 iption factors, SHORT-ROOT (SHR), SCARECROW (SCR) and SCARECROW-LIKE 23 (SCL23), affect BS cell fate

210 Arabidopsis, the SHORT-ROOT (SHR)/SCARECROW (SCR) transcription factor dimer activates CYCLIND6;1 (CY

211 action with its downstream target SCARECROW (SCR) control root patterning and cell fate specification

212 root primordia, within which the SCARECROW (SCR) transcription factor was specifically expressed.

213 two-base mismatches (1MM, 2MM) or scrambled (SCR) sequences was highlighted and the applicability for

214 wards one base mismatch (1-MM) or scrambled (SCR) sequences was obtained.

215 SHR, SCR and SCL23 homologs are present in many plant species

216 a pathway that partially overlaps with SHR, SCR, PLETHORA1 and PLETHORA2 (PLT1 and PLT2).

217 io-temporal dynamics of SHR movement and SHR-SCR interaction is currently unavailable.

218 uggests that acquisition of the cortical SHR-SCR module enabled cell division coupled to rhizobial in

219 The cortical SHR-SCR network is conserved across legume species, responds

220 e SHR-SCR binary and JACKDAW (JKD)/IDD10-SHR-SCR ternary complexes.

221 Here we show that a SHORTROOT-SCARECROW (SHR-SCR) stem cell program in cortical cells of the legume M

222 Thus, our results support that SHR-SCR protein complex stoichiometry and regulation of SHR

223 we present the crystal structures of the SHR-SCR binary and JACKDAW (JKD)/IDD10-SHR-SCR ternary compl

224 lidation, showed that high levels of the SHR-SCR complex are associated with more CEI division but le

225 ch incorporated the stoichiometry of the SHR-SCR complex as well as upstream transcriptional regulati

226 eady state in minutes, while SCR and the SHR-SCR complex reach a steady-state between 18 and 24 hr.

227 ,ads intermediates exhibit a higher specific SCR activity (TOF).

228 e canonical Brassicaceae S locus genes (SRK, SCR), and is situated in a genomic position that differs

229 ocation or recombination event involving SRK/SCR and Lal2/SCRL likely occurred, together with neofunc

230 ctively, and crossing data show that the SRK/SCR haplotype is functional in self-incompatibility.

231 Significantly, the low-temperature standard SCR mechanism proposed here provides full consistency wi

232 ce both Cu(II) and Cu(I) ions under standard SCR conditions at 473 K.

233 To study SCR in vivo in a genome-wide manner, we treated mammalia

234 = .02) and higher diffusivities in superior (SCR) and posterior corona radiatae (PCR) (group x age x

235 Suppressing SCR via inhibition of ryanodine receptors, Ca(2+)/calmod

236 enting the active sites for high-temperature SCR.

237 ovides full consistency with low-temperature SCR kinetics.

238 Low-temperature SCR, up to approximately 200 degrees C, is characterized

239 been identified with a duplicated N-terminal SCR-1/2 domain pair that causes CFHR5 nephropathy.

240 on oxide nanoworms (SPIO NWs), we found that SCR-2-3-4 was the most effective inhibitor (IC(50) ~0.24

241 Our findings indicate that SCR contains an amino acid segment at the C-terminal reg

242 p experiments and sugar assays, we show that SCR is primarily involved in sugar transport whereas SCL

243 The study shows that SCR retrofit programs can be effective for NO (x) reduct

244 believe that there are data to suggest that SCR leads to progressive fibrosis and loss of graft func

245 Structural modeling suggested that SCR-2, SCR-3 and SCR-4 are critical for binding to the a

246 The SCR revaluation effect was not dependent on explicit mem

247 The particles were measured after the SCR with an engine exhaust particle sizer spectrometer (

248 energies and apparent reaction orders at the SCR conditions, even on zeolite frameworks other than SS

249 for typical highway driving conditions, the SCR technology is proving to be effective in controlling

250 ing that promoter incorporation enhances the SCR reaction by a structural effect generating adjacent

251 NO (x) emissions in real-time, evaluate the SCR system performance, and identify vehicle operating m

252 eveal both the dual-site requirement for the SCR reaction and the important structural promotional ef

253 al effect that tungsten oxide offers for the SCR reaction by V(2) O(5) /TiO(2) catalysts.

254 g information along the coiled-coil from the SCR.

255 In the case of the LSD and ULSD fuels, the SCR system also significantly reduced emissions of compo

256 However, for all tested fuels, the SCR system produced significantly (p < 0.05) higher emis

257 its desired role as a reducing agent in the SCR and diminishes NO conversion and N(2) selectivity.

258 Greater FA in the SCR and precentral gyrus white matter were associated wi

259 the hydrophobic core is destabilized in the SCR by Ala residues at three consecutive d positions.

260 rtly in the first amino acid residues in the SCR C-terminal extracellular coil.

261 rity Fe sites was synthesized, tested in the SCR reaction and characterized by UV-vis, X-ray absorpti

262 oth species were found to participate in the SCR reaction, their relative population depends on the c

263 ammonia level was induced downstream of the SCR catalyst.

264 We demonstrate the importance of the SCR in controlling and transmitting the stability signal

265 verall NO (x) conversion efficiencies of the SCR system on many vehicles were well below the 90% thre

266 For all studied fuels, the use of the SCR system yielded statistically significant (p < 0.05)

267 None of the SCR systems were found to lead to a substantial increase

268 , we explore the possible interaction of the SCR TMD with cholesterol by using a variety of experimen

269 loading, the low temperature behavior of the SCR was enhanced.

270 The single mutation A109L prevents the SCR from transmitting stabilizing information and separa

271 of harmful nitrogen oxides and to unveil the SCR mechanism.

272 d for an efficient SCR system, even when the SCR system was above the optimum operating temperature t

273 However, under operations where the SCR's do not reach minimum operating temperature, like c

274 with the proposed mode of action, where the SCRs likely inhibit binding between the virus and cell-s

275 infection assay, our data demonstrates these SCRs are highly potent with IC(50)s as low as 0.16 muM a

276 Time-of-addition studies showed that these SCRs inhibit the early stages of the virus infection, wh

277 Stop codon read-through (SCR) is a process of continuation of translation beyond

278 diated Cu(I)-->Cu(II) redox step integral to SCR.

279 ense beta-adrenergic stimulation, leading to SCR.

280 catalyzing the hydrolysis reaction prior to SCR.

281 hasize the need for model inputs relative to SCR performance as a function of driving cycle and engin

282 cies are solvated and mobilized by NH3 under SCR conditions.

283 ns including engine-out, DOC+CuZ-SCR+AMOX, V-SCR+AMOX, and DOC+DPF+CuZ-SCR+AMOX.

284 hment in lateral root primordia operates via SCR-mediated formative cell division and coincides with

285 lternative pathway C3bBb convertase, whereas SCR-1 is dispensable.

286 e upstroke are narrowly distributed, whereas SCR amplitudes follow a broad normal distribution with a

287 s (in 25-30 nm size), the formation of which SCR either increased or decreased.

288 SHR reaches a steady state in minutes, while SCR and the SHR-SCR complex reach a steady-state between

289 H severity assessed were not associated with SCR and are not necessary for screening guidelines.

290 This could potentially be associated with SCR catalyst deterioration on some engines.

291 ng therapies most frequently associated with SCR in SCD patients.

292 exposure and were negatively associated with SCR to the CS+ during early conditioning in the total sa

293 lity, oligomeric state, and association with SCR using a combination of Fluorescent Correlation Spect

294 tic detection sampling approach coupled with SCR modeling allowed us to estimate spatially variable g

295 nd one hydraulic hybrid diesel equipped with SCR, were measured using a portable emissions measuremen

296 k factors were associated significantly with SCR.

297 tients would receive inadequate therapy with SCR.

298 omposition of NO(x) emitted with and without SCRs and SNCRs; further the isotopic composition of powe

299 allow active and inactive species in deNO(x)-SCR to be identified.

300 ic reduction of NO(x) using ammonia (deNO(x)-SCR) and characterizing the underlying distribution of c