ライフサイエンスコーパス: SEP

1 SEP amplitude but not latency effects recovered after 7

2 SEP indicators at 3 life stages (childhood, adulthood, a

3 SEP inequalities in CRP emerged in 30s, increased up to

4 SEP inequalities in fibrinogen decreased with age.

5 SEP is associated with T2DM risk factors in children but

6 SEP over the life course is related to the risk of incid

7 SEP-1 performance was associated with structural hospita

8 SEP-1 reporting was more common in larger, nonprofit hos

9 SEP-225289 is a novel compound that, based on in vitro p

10 perative mJOA (P = .927), T2 HSI (P = .176), SEP amplitude (P = .154), and latency (P = .260) did not

11 We validated expression of a SEP from its endogenous RNA, and demonstrated the specif

12 95% CI, 1-13) 72 hours after CPR, and absent SEPs during hypothermia (FPR 3; 95% CI, 1-7) and after r

13 Here, we show that a 69-amino acid SEP, MRI-2, physically interacts with the Ku heterodimer

14 ity with respect to both childhood and adult SEP are urgently required.

15 inal data to examine how childhood and adult SEP relates to BMI across adulthood in three national Br

16 The strength of association between adult SEP and BMI did not vary with age in any consistent patt

17 nces hemostatic markers more than does adult SEP.

18 l hygiene) were the mediators, and adulthood SEP (education and income) represented the exposure-indu

19 adulthood that was not mediated by adulthood SEP and behaviors.

20 etween early life, young and later adulthood SEP and the expression of 845 genes involved in human in

21 ly life, young adulthood and later adulthood SEP with each inflammatory markers separately, or by com

22 ustment for young and mid-to-older adulthood SEP and established heart failure risk factors attenuate

23 Of the SEP measures, mid-to-older adulthood SEP showed the strongest association with incident heart

24 s, where participants with a less advantaged SEP have higher levels of inflammation.

25 to 66%), z-severity (-2.4 down to -2.5), and SEP (-195 to -227) (P = 0.0062).

26 easure of cumulative SEP (range, 0 to 8) and SEP mobility.

27 concentrations by LC-MS/MS (rPred > 0.90 and SEP < 100 mug/kg).

28 istics (mother's age and place of birth) and SEP.

29 assessing associations of race/ethnicity and SEP to health outcomes with CIs); illustrate the applica

30 lammation measured as C-reactive protein and SEP across four countries (Britain, Ireland, Portugal an

31 The obtained results (RSQ and SEP, respectively) for the global model of red and white

32 des (SEPs) to validate smORF translation and SEP stability.

33 nd 36 hours and 48 hours after collapse; and SEPs during hypothermia and after rewarming.

34 the anterior hypothalamus (AH), septal area (SEP), and ventromedial nucleus of the posterior division

35 performance on the sepsis measure, known as SEP-1.

36 Harmonized social class-based SEP data (Registrar General's Social Class) was ascertai

37 Because SEPs are shorter and less abundant than average proteins

38 rse SEP-1 performance, hospitals with better SEP-1 performance tended to be smaller, for-profit, nont

39 varied significantly with depth and between SEP pools.

40 e countries reporting an association between SEP (e.g., income, education, occupation, private medica

41 ind a consistent inverse association between SEP and CRP across cohorts, where participants with a le

42 Associations between SEP and BMI were examined using linear regression and mu

43 Conversely, in women, associations between SEP and nonadherence were weak and inconsistent.

44 y the theory, absolute differentials between SEP groups reflect the pattern of societal income inequa

45 s evidence for a complex interaction between SEP and other variables, such as ethnicity, sex, and the

46 onal study assessed the relationship between SEP and human infection within a single community in wes

47 We investigated the relationship between SEP and statin adherence in a country with universal cov

48 equality to measure the relationship between SEP and use of eye care services across the entire distr

49 SEP-1 measure, and the relationship between SEP-1 performance and performance on other quality measu

50 We examined the relationships between SEP and T2DM risk factors in UK children of South Asian,

51 h we cannot rule out residual confounding by SEP.

52 fferences in the use of eye care services by SEP persist among US adults with eye diseases.

53 inflammatory marker examined rather than by SEP measures.

54 nt have on neuronal activity, as measured by SEPs, and on the amplitude of the BOLD fMRI signal in ra

55 Lower childhood SEP was associated with higher adult BMI in both genders

56 provides additional evidence that childhood SEP is associated with a sustainable upregulation of the

57 ogen and C-reactive protein, while childhood SEP influences hemostatic markers more than does adult S

58 rthologs together with AGL6 encode classical SEP floral organ identity and floral termination functio

59 ers for Medicare and Medicaid Services (CMS) SEP-1 BCx component, 30-day readmission, and all-cause i

60 Census tract-level composite SEP index in quintiles was assigned based on residence a

61 An oral compound, SEP-363856, that does not act on dopamine D2 receptors b

62 xamined the impact of changes in life-course SEP on incidence of dementia and cognitive impairment bu

63 authors examined the effects of life-course SEP on inflammatory and hemostatic markers: fibrinogen,

64 IND with every 1-unit increase in cumulative SEP disadvantage across the life course (hazard ratio =

65 were used to derive a measure of cumulative SEP (range, 0 to 8) and SEP mobility.

66 dominantly mediate perisomatic and dendritic SEPs, respectively.

67 , worsening cardiac denervation (global diff-SEP > 9) occurred in 14 of 23 (60.9%) patients over 2 y,

68 cline in lateral and inferior segments (diff-SEP -82 to -99) compared with anterior and septal segmen

69 nervation as the 2-y difference in SEP (diff-SEP).

70 od having a more prominent role than earlier SEP.

71 ylogenetic and expression analysis for eight SEP-like GERBERA REGULATOR OF CAPITULUM DEVELOPMENT (GRC

72 Neutralization of either SEP-196 or Arg-201 leads to a channel with reduced activ

73 We expressed SEP-TAC-7.3 in dissociated rat hippocampal neurons to ex

74 nce interval: -0.04, 0.13) after control for SEP and RPF.

75 takeholder workgroup recognizes the need for SEP-1 but strongly believes that multiple antibiotics li

76 l quality, providing preliminary support for SEP-1 performance as a marker of timely hospital sepsis

77 ibiotics listed in the antibiotic tables for SEP-1 are not appropriate and the use of these antibioti

78 henotypes were not socially patterned or had SEP patterns that were not related to smoke exposure.

79 odels, participants with a continuously high SEP had lower hazard ratios for dementia/CIND compared w

80 val: 0.66, 0.98) than did children with high SEP.

81 evidence.In the general population, a higher SEP appears to be associated with an increased 'hypothet

82 , and skin color (lower versus middle/higher SEP).

83 ce rate ratios for children with the highest SEP were 1.29 (95% confidence interval (CI): 1.15, 1.44)

84 iduals living in households with the highest SEP.

85 tween individual HIV infection and household SEP.

86 The relationships between household SEP and individual infection with Plasmodium falciparum,

87 us relationships can exist between household SEP and infection risk for different pathogens in low in

88 We examined how SEP inequalities in inflammation -assessed using C-react

89 early life to adulthood, we investigate how SEP indicators at different points across the life cours

90 Future research needs to study how SEP impacts the potential donor population from willingn

91 The discovery of additional human SEPs and their conservation indicate the potential impor

92 ve led to the detection of several new human SEPs (novel human genes), improved confidence in the SEP

93 apoptotic suppressor ced-9/Bcl-2 implicating SEP-1 in execution of apoptosis.

94 Importantly, SEPs recorded after InsP3 infusion into distal astrocyte

95 eps in the SEP discovery workflow to improve SEP isolation and identification.

96 In SEP, cortical amplitudes were reduced during self-touch,

97 athetic innervation as the 2-y difference in SEP (diff-SEP).

98 ter understand the observed heterogeneity in SEP differences in health across the lifespan.

99 ked potential (SEP) amplitudes and increased SEP latencies in control genotypes.

100 If the SEP moiety has been internalized, SEP will remain fluorescent even during application of t

101 accompanied by the suppression of only late SEP components (P300) and was stronger for individuals w

102 At 8 months, posture influenced mid-latency SEP components, but by 10 months effects were observed a

103 ive decline and examine how individual-level SEP factors (educational level, annual income, and occup

104 ors examined the relation between early-life SEP and incident, hospitalized heart failure among middl

105 Individuals from the lowest early-life SEP had a higher risk of moderate-to-severe periodontiti

106 Early-life SEP indicators assessed included parental education, occ

107 s the controlled direct effect of early-life SEP on periodontitis.

108 We found that early-life SEP was associated with the development of periodontitis

109 proportional hazards regression, early-life SEP was inversely related to incident heart failure afte

110 Early-life SEP was measured at participant birth based on income, h

111 ehaviours, body mass index and later-in-life SEP.

112 iotic- and fluid-focused sepsis bundles like SEP-1 improve survival but do not demonstrate the superi

113 t antibiotic- and fluid-focused bundles like SEP-1 would probably show benefit, but evidence supporti

114 a-analysis of studies of sepsis bundles like SEP-1.

115 Low SEP in childhood is associated with modest increase in a

116 Low SEP was associated with overall and rapidly increasing s

117 Nearly 24% of the sample maintained a low SEP throughout life.

118 odds of asthma alone in children with a low SEP.

119 compared with those with a continuously low SEP at all 3 life stages (hazard ratio = 0.49, 95% confi

120 clinical characteristics, and copayment, low SEP was associated with statin nonadherence (proportion

121 In high-income countries, low SEP is a risk factor for hospital death as well as other

122 dherence trajectories showed that men in low SEP were likely to belong to trajectories presenting a f

123 cognitive performance among these older, low-SEP Latinos.

124 Lower SEP was associated with a reduced likelihood of receivin

125 In a commercial setting, lower SEP motivates people to donate.

126 Across all of the studies, lower SEP groups have higher mean concentrations of CRP and, a

127 Individuals in households with the lowest SEP were at greatest risk of infection with P. falciparu

128 As compared with the lowest SEP, the adjusted incidence rate ratios for children wit

129 ; mean severity z score, -2.47 to -2.0; mean SEP, -167 to -139), with lateral and proximal inferior s

130 ; mean severity z score, -2.8 to -2.62; mean SEP, -271 to -230).

131 everity z score was -2.7+/-1.2, and the mean SEP was -202+/-131 (range, -358 to 0).

132 Here, we measured SEP gating and perceptual sensory attenuation in a singl

133 Neighborhood SEP was derived by linking the participant's individual

134 graphic characteristics, higher neighborhood SEP was significantly associated with cognitive function

135 r individual educational level, neighborhood SEP remained associated with baseline cognition but not

136 ults suggest that the effect of neighborhood SEP on cognitive decline among Latinos is primarily acco

137 netics, and compartmentalization of neuronal SEPs.

138 nced the amplitude and frequency of neuronal SEPs.

139 faster detection and characterization of new SEPs and smORFs.

140 At all ages we observed SEPs over central regions contralateral to the stimulate

141 Co-administration of SEP and L-Cit decreased infarct size and improved corona

142 Co-administration of SEP and L-Cit limits diabetic cardiomyopathy and ischemi

143 litus were abrogated by co-administration of SEP and L-Cit.

144 roximately 24 h after oral administration of SEP-225289, to assess occupancy at trough levels.

145 Assessment of SEP was based on parental occupation defined using NS-SE

146 estimate direct and indirect associations of SEP and RPF, and we estimated average marginal controlle

147 Plasma concentration of SEP-225289 was assessed before ligand injection, and sub

148 biological embedding and the consequences of SEP experiences from early life to adulthood, we investi

149 ferent ethnic groups and the contribution of SEP to ethnic differences in T2DM risk in young people h

150 tudy was conducted during the development of SEP-225289 to investigate its dopamine and serotonin tra

151 may differ according to other dimensions of SEP such as education or income.

152 Different single doses of SEP-225289 were administered to healthy volunteers in 3

153 cupancies increased with increasing doses of SEP-225289.

154 The present study examined the effects of SEP and L-Cit on diabetic cardiomyopathy and ischemia/re

155 y to confirm the effects and side effects of SEP-363856, as well as its efficacy relative to existing

156 use of social class as the sole indicator of SEP-while it was available in each cohort in both childh

157 However, the influence of SEP on emerging T2DM risks in different ethnic groups an

158 Loss of SEP-1 results in extra surviving cells in a weak ced-3(-

159 ld income) and distal (education) markers of SEP were used.

160 ere the outcome was reported by a measure of SEP.

161 uantify the associations between measures of SEP and use of healthcare in the last year of life.

162 n future studies using different measures of SEP.

163 Virginia Orange that match the properties of SEP.

164 the present findings support the revision of SEP-1 to allow flexibility in treatment according to phy

165 trial to evaluate the efficacy and safety of SEP-363856 in adults with an acute exacerbation of schiz

166 ith a pH sensitivity that approaches that of SEP, making it amenable for detection of single exocytos

167 ectra in absorbance units produced values of SEP that ranged from 0.41 to 2.02 mM and an average of 1

168 In vivo animation of SEPs and somatosensory-induced gamma oscillations both m

169 romoted the expansion and differentiation of SEPs in the spleen.

170 in E2 (PGE2) promotes the differentiation of SEPs.

171 We substantiated the astrocytic origin of SEPs through paired neuron-astrocyte recordings, where w

172 DNA repair and underscores the potential of SEPs to serve important biological functions in mammalia

173 mic stress induce the rapid proliferation of SEPs, but they do not differentiate until serum erythrop

174 P assignments, and enabled quantification of SEPs under different cellular conditions.

175 With repetitive recordings of SEPs, this somatotopic pattern was reliably recorded for

176 meostasis, trauma had exaggerated effects on SEP amplitude and mitochondrial morphology, suggesting a

177 Better hospital performance on SEP-1 was associated with higher rates of timely head CT

178 The dipole on SEPs is consistent with the previously accepted notion t

179 cortex does not significantly reduce the P1 SEP component but strongly reduces the N1 and P2 compone

180 m reflexes 72 hours after CPR and performing SEP.

181 rane chimera, named super ecliptic phluorin (SEP)-TAC-7.3, functions and traffics as a wild-type (WT)

182 The GFP-based superecliptic pHluorin (SEP) enables detection of exocytosis and endocytosis, bu

183 scent protein mutant superecliptic pHluorin (SEP) is particularly well suited to these applications.

184 ent protein, such as superecliptic pHluorin (SEP), which is brightly fluorescent at pH 7.4 but not fl

185 To demonstrate the utility of the pHuji plus SEP pair, we perform simultaneous two-color imaging of c

186 n reading frame (sORF)-encoded polypeptides (SEPs) has raised important questions about the functiona

187 ause they detect smORF-encoded polypeptides (SEPs) to validate smORF translation and SEP stability.

188 Socio-economic position (SEP) and ethnicity influence type 2 diabetes mellitus (T

189 rtance of household socio-economic position (SEP) in shaping individual infectious disease risk is in

190 cal process linking socio-economic position (SEP) to health.

191 ble pathway linking socio-economic position (SEP) to premature ageing and is a possible explanation f

192 haracteristics, and socio-economic position (SEP).

193 relationship between socioeconomic position (SEP) across the life course and inflammation (as measure

194 anges in life-course socioeconomic position (SEP) and cognitive decline or incidence of dementia.

195 preeclampsia, higher socioeconomic position (SEP) and maternal birth in Hong Kong.

196 lating to link lower socioeconomic position (SEP) and poorer health, and the inflammatory system stan

197 associations between socioeconomic position (SEP) and risk of childhood acute lymphoblastic leukemia

198 Children with a low socioeconomic position (SEP) had more asthma alone (adjusted multinomial odds ra

199 Lower socioeconomic position (SEP) has consistently been associated with poorer health

200 Lower socioeconomic position (SEP) in both childhood and adulthood is associated with

201 Socioeconomic position (SEP) is a multi-dimensional construct reflecting (and in

202 Low socioeconomic position (SEP) is recognized as a risk factor for worse health out

203 er (>=60 years), low-socioeconomic position (SEP) Latinos (predominantly of Mexican descent) in Sacra

204 eview focuses on how socioeconomic position (SEP) may influence hypothetical and actual living kidney

205 financial impacts by socioeconomic position (SEP) of a 20% SSB tax for Australia.

206 ence of neighborhood socioeconomic position (SEP) on cognitive decline and examine how individual-lev

207 luence of early-life socioeconomic position (SEP) on incident heart failure in blacks and whites is u

208 effect of early-life socioeconomic position (SEP) on periodontitis at age 31 years, controlling for a

209 l and mental health, socioeconomic position (SEP), and longevity.

210 g cancer varies with socioeconomic position (SEP), it is not known whether socioeconomic inequalities

211 a single measure of socioeconomic position (SEP).

212 earch shows that low socioeconomic position (SEP; especially low income) is associated with statin no

213 ignificantly reduced sound-evoked potential (SEP) amplitudes and increased SEP latencies in control g

214 (N140, P300) somatosensory-evoked potential (SEP) amplitudes.

215 nents of the somatosensory evoked potential (SEP) before and during movement.

216 re regarding somatosensory evoked potential (SEP) gating is commonly cited as a potential mechanism u

217 y (HSI), and somatosensory evoked potential (SEP) were analyzed by using a logistic regression model

218 omponent of somatosensory evoked potentials (SEP) and the area of high-frequency oscillations (HFO).

219 decomposed somatosensory evoked potentials (SEP) into three major components: P1, N1, and P2, where

220 ys recorded somatosensory evoked potentials (SEP) with an almost twice SNR (signal-to-noise ratio) wh

221 dent spontaneous slow excitatory potentials (SEP) originate at dendritic locations and are significan

222 esting, and somatosensory-evoked potentials (SEPs) at spinal and cortical levels.

223 we recorded somatosensory evoked potentials (SEPs) from 6.5-, 8-, and 10-month-old infants while pres

224 g the extracellular sound-evoked potentials (SEPs) from the antennal nerve while monitoring the likel

225 ecording of somatosensory evoked potentials (SEPs) in an ovine model.

226 edian nerve somatosensory-evoked potentials (SEPs) to predict poor outcome in patients treated with m

227 signal and somatosensory evoked potentials (SEPs) using short, typically 300 micros, square stimulat

228 tion 0.982 and standard error of prediction (SEP) between 0.03% and 0.06%.

229 values of the standard error of prediction (SEP) for glucose concentration are maintained to a range

230 mug kg(-1) and standard error of prediction (SEP) of 14.8 mug kg(-1).

231 Pred)>0.91 and standard error of prediction (SEP) of 24mg/100g phenolics, 7mg/100g monomeric anthocya

232 (PLSR) showed standard error of prediction (SEP) ~2.33, 0.06, 0.41, 0.30 and 0.014 g/L for estimatio

233 tion (RSQ) and standard error of prediction (SEP), respectively) for the developed models were: 0.82

234 bination of sequential extraction procedure (SEP) and chemical composition analysis of impoundment sl

235 elivered median nerve stimulation to produce SEPs during a force-matching paradigm (used to quantify

236 score severity and severity-extent product (SEP) on 9-segment bullseye maps and decline in cardiac s

237 score severity, and severity-extent product (SEP).

238 Immature stress erythroid progenitors (SEPs) are derived from short-term hematopoietic stem cel

239 e expansion of stress erythroid progenitors (SEPs) in the spleen.

240 diatoms, are the main cause of the prominent SEP.

241 ter and less abundant than average proteins, SEP detection using proteomics faces unique challenges.

242 a four-step sequential extraction protocol (SEP) and pyrolytic thermodesorption.

243 ulate matter during the summer export pulse (SEP) average 408, 283, 24.1, 1.1, and 67.5 mumol.m(-2).d

244 The recurrent SEP is enigmatic because it is focused in time despite t

245 ng health promotion policies may help reduce SEP inequalities in health.

246 Adjustment for lifestyle indicators reduces SEP differentials by between 45% and 52% but cannot acco

247 of En and Inv in postmitotic neurons reduces SEP amplitude but also reduces synaptic strength at the

248 we show that rather than directly regulating SEPs, Epo promotes this transition from proliferation to

249 he analysis, of which 2,851 (86.8%) reported SEP-1 performance data.

250 The majority of eligible hospitals reported SEP-1 data, and overall bundle compliance was highly var

251 Among hospitals reporting SEP-1 performance data, overall bundle compliance was ge

252 e hospital factors associated with reporting SEP-1 data, the hospital factors associated with perform

253 Mutants in five rice (Oryza sativa) SEP genes suggest both redundant and unique functions in

254 with an acute exacerbation of schizophrenia, SEP-363856, a non-D2-receptor-binding antipsychotic drug

255 SEPALLATA (SEP) MADS box transcription factors mediate floral devel

256 te functional diversification for SEPALLATA (SEP)-like MADS box genes that often function redundantly

257 Members of SEPALLATA (SEP) and APETALA1 (AP1)/SQUAMOSA (SQUA) MADS-box transcr

258 OF OVEREXPRESSION OF CO 1 (SOC1), SEPALLATA (SEP) and SHORT VEGETATIVE PHASE (SVP) Arabidopsis thalia

259 es, such as AN and members of the SEPALLATA (SEP) family, to activate precociously, while the express

260 We report a role for the C. elegans separase SEP-1, primarily known for its essential activity in cel

261 Sepiapterin (SEP) is a tetrahydrobiopterin precursor, and L-citrullin

262 ndent on an interaction between phospho-Ser (SEP)-196 and Arg-201, reducing Arg-201 interaction with

263 Use of eye care services; SEP was measured by the PIR and educational attainment.

264 t a functional characterization of the seven SEP/AGL6 and four AP1/SQUA genes in the distant asterid

265 nagement Bundle, Severe Sepsis/Septic Shock (SEP-1) performance measure to the Hospital Inpatient Qua

266 ng acute and chronic recordings, significant SEP responses in the triangle between the ansate, diagon

267 ity of our labeling approach using synthetic SEP.

268 We found that SEP inequalities in inflammation followed heterogeneous

269 The SEP is approximately threefold greater than mean wintert

270 The SEP-1 performance measure requires, among other critical

271 If the SEP moiety has been internalized, SEP will remain fluore

272 If the SEP moiety is at the surface, its fluorescence will decr

273 vel human genes), improved confidence in the SEP assignments, and enabled quantification of SEPs unde

274 Here, we optimize several steps in the SEP discovery workflow to improve SEP isolation and iden

275 , there is no evidence of discordance in the SEP of donors and recipients that would suggest undisclo

276 e of these antibiotics, as called for in the SEP-1 measure, is not in alignment with prudent antimicr

277 re on the PANSS at baseline was 101.4 in the SEP-363856 group and 99.7 in the placebo group, and the

278 ms; one sudden cardiac death occurred in the SEP-363856 group.

279 thy diet explained part, but not all, of the SEP inequalities in inflammation; in general, BMI exerte

280 Of the SEP measures, mid-to-older adulthood SEP showed the stro

281 lthough it reduced the N120 component of the SEP.

282 ces in the functional diversification of the SEP/AGL6 and AP1/SQUA MADS-box subfamilies during angios

283 l factors associated with performance on the SEP-1 measure, and the relationship between SEP-1 perfor

284 ator of PKC inhibition and suggests that the SEP-196/Arg-201 interaction is critical for maintaining

285 A total of 120 patients were assigned to the SEP-363856 group and 125 to the placebo group.

286 Although the SEPs are capable of self-renewal, they are erythroid res

287 characterization of MRI-2 suggests that this SEP may participate in DNA repair and underscores the po

288 Risk exposure related to SEP accumulates across the life course and contributes t

289 ice at 6 to 8 weeks of age received vehicle, SEP, or L-Cit orally alone or in combination for 8 weeks

290 Acculturation may benefit cognition when SEP is low.

291 Whether SEP and L-Cit are effective at reducing diabetic heart d

292 healthy human subjects to determine whether SEP gating correlated with the behavior.

293 childhood ALL was negatively associated with SEP among Hispanics but was positively associated among

294 and 95% confidence intervals associated with SEP and race/ethnicity, adjusted for sex, age, and year

295 of statin adherence and its association with SEP.

296 Adverse events with SEP-363856 included somnolence and gastrointestinal symp

297 to the risk of incident heart failure, with SEP later in adulthood having a more prominent role than

298 Pharmacological intervention with SEP-363856, a novel psychotropic agent with agonism at t

299 1 ratio to receive once-daily treatment with SEP-363856 (50 mg or 75 mg) or placebo for 4 weeks.

300 Compared with hospitals with worse SEP-1 performance, hospitals with better SEP-1 performan