ライフサイエンスコーパス: SM

1 SM inhibited the transcription-activating function of DH

2 SM interactions are known to be important for StnII bind

3 SM is a substrate for the lysosome-resident enzyme acid

4 SM(39:1) was around half as abundant in in utero groups

5 SM-164 also inhibited expression of RANKL, which mediate

6 SM-164 did not kill adriamycin-resistant BC cells, while

7 SM-ILF and M-ILF showed distinct patterns of distributio

8 appa(w)] = 0.75 [95% CI: 0.74, 0.76]; site 1 SM: kappa(w) = 0.71 [95% CI: 0.64, 0.78]; site 2 SM: kap

9 ction of PE with five metabolites (SM C28:1, SM C30:1, LysoPC C19:0, LysoPE C20:0, propane-1,3-diol).

10 er 3, 2011, and October 31, 2014, and 34 180 SM/DBT examinations between January 7, 2015, and Februar

11 kappa(w) = 0.71 [95% CI: 0.64, 0.78]; site 2 SM: kappa(w) = 0.72 [95% CI: 0.70, 0.75]).

12 Furthermore, PC(18:0/18:1), PE(18:1/18:2), SM(d18:0/22:0) and several other molecular species were

13 failure and higher levels of Cer-22, SM-20, SM-22, and SM-24 with decreased risk of heart failure.

14 f heart failure and higher levels of Cer-22, SM-20, SM-22, and SM-24 with decreased risk of heart fai

15 rformed DNA and RNA sequencing on 80 STs, 26 SMs, and 22 melanomas with Spitzoid features (MSF).

16 mptomatic patients, 99 underwent ASA and 378 SM.

17 ], 0.72 vs 0.79, P < .001) by using only 500 SM images from that site.

18 anuary 1, 2012, and May 31, 2016, and 31 929 SM/DBT examinations between June 1, 2016, and March 30,

19 tions, the formation of ordered domains in a SM and cholesterol-rich leaflet can be suppressed by an

20 We observe that the resiliency of a SM network depends on both the network structure and par

21 In the liver, smooth muscle alpha-actin (SM alpha-actin) is up-regulated in hepatic stellate cell

22 However, after multivariable adjustment, SM/DBT was associated with a slightly higher recall rate

23 Similarly, after multivariable adjustment, SM/DBT was associated with slightly lower specificity co

24 10% of cases that progress to more advanced SM (advSM), which has a significantly poorer outcome.

25 utated clones are characteristic of advanced SM (advSM), especially SM with an associated hematologic

26 SM and no definitive progression to advanced SM was found.

27 Progressions to advanced SM were observed in 2.9% of these patients.

28 tion of S100A4, RhoA or rhotekin from airway SM tissues using short hairpin RNA or small interfering

29 myosin II undergoes polymerization in airway SM and regulates contraction by catalysing the assembly

30 action of S100A4 with NM myosin II in airway SM at the cell cortex and catalysed NM myosin filament a

31 s to the regulation of contraction in airway SM by regulating a pool of NM myosin II at the cell cort

32 itical role in tension development in airway SM tissue by catalysing NM myosin filament assembly, and

33 In airway SM, ACh (acetylcholine) stimulated the binding of S100A4

34 es the polymerization of NM myosin in airway SM, which is necessary for tension development.

35 ritical function in the regulation of airway SM contraction by catalysing NM myosin filament assembly

36 llular sphingomyelin (SM) levels and altered SM distribution.

37 SIAB linker yielded superior results over an SM(PEG)2 linker but combinations of carriers, conjugatio

38 aphylaxis (n = 8 of 47; [17%; P = .006]) and SM (n = 10 of 82 [12.2%; P = .03]) relative to the contr

39 s in the RBCs outer leaflet with PC 34:1 and SM 34:1 being the most abundant species.

40 or risk factors, higher levels of Cer-16 and SM-16 were associated with higher risk of incident heart

41 ations of higher plasma levels of Cer-16 and SM-16 with increased risk of heart failure and higher le

42 s: Cer and SM with palmitic acid (Cer-16 and SM-16), species with arachidic acid (Cer-20 and SM-20),

43 16), species with arachidic acid (Cer-20 and SM-20), species with behenic acid (Cer-22 and SM-22), an

44 M-20), species with behenic acid (Cer-22 and SM-22), and species with lignoceric acid (Cer-24 and SM-

45 d higher levels of Cer-22, SM-20, SM-22, and SM-24 with decreased risk of heart failure.

46 and species with lignoceric acid (Cer-24 and SM-24).

47 obtained from patients with IS (N = 508) and SM (N = 349; defined by absence of a diffusion weighted

48 Guideline-based referral for ASA and SM leads to excellent outcomes with low procedural morta

49 e studied the associations of plasma Cer and SM species with incident heart failure in the Cardiovasc

50 We examined 8 species: Cer and SM with palmitic acid (Cer-16 and SM-16), species with a

51 proach (fluorescent analogs of both Chol and SM) in combination with a series of StnII tryptophan (Tr

52 Norway included women screened with DBT and SM (study group) or DM alone (control group) between Feb

53 , 59 years +/- 6) were screened with DBT and SM and 57 763 women in the control group (mean age, 59 y

54 were observed in women screened with DBT and SM compared with women screened with DM.

55 breast cancer in women screened with DBT and SM versus those screened with DM alone and (b) screen-de

56 r reading modes: DM, DM + CAD, DM + DBT, and SM + DBT.

57 demonstrated strong performance on FFDM and SM images from two institutions without training on SM i

58 .0, PC.ae.C34.2, C3.DC..C4.OH, glutamine and SM.C16.1, being the most significant metabolites.

59 ercross between two inbred strains (LG/J and SM/J); this GWAS identified 23 quantitative trait loci.

60 survival was worse in MSF than in the ST and SM groups (P = 0.0073); and (iv) classification incorpor

61 By contrast, soluble sugars and SM remained relatively constant in aboveground young org

62 The majority of STs and SMs have kinase fusions as primary initiating genomic ev

63 The odds of any SM phenotype were significantly higher in children with

64 etected in cholesterol-containing asymmetric SM+DOPC outside/DOPC inside vesicles, but ordered domain

65 In cholesterol-containing asymmetric SM+PC outside/PC inside vesicles, the PC-containing inne

66 the model using real-world global automotive SM networks.

67 significant inter-model correlation between SM and air temperature biases (R = -0.65), (ii) a wet/co

68 rther improved when we distinguished between SM, GM, and junction genes responsible for reactions sha

69 study, we identified an interaction between SM and DHX9, a DExH-box helicase family member, by mass

70 but also emphasize the relationship between SM alpha-actin and fibrogenesis in hepatic myofibroblast

71 treolysin A (OlyA), which specifically binds SM/cholesterol complexes in membranes, to study the SM-s

72 Bangalore S, Maron DJ, O'Brien SM, et al.

73 and abolish the S(o) phase domains formed by SM:Cer.

74 on genes responsible for reactions shared by SM and GM pathways, indicating that topological consider

75 plays a role in BoHV-1 entry, while cellular SM does not.

76 essed by asymmetry in cholesterol-containing SM+POPC outside/POPC inside vesicles over the entire tem

77 nd expansion of an avirulent gene-containing SM cluster through transposable element-mediated whole-c

78 duplication of an avirulent gene-containing SM cluster, ACE1, in a clonal M. oryzae population (Clad

79 studied, the paracrine signaling controlling SM regionalization and how this is coordinated with epit

80 ecall rate at repeat screening with both DBT+SM (3.71% vs 3.40%, respectively; P = .10) and FFDM (3.7

81 is randomized controlled trial comparing DBT+SM and DM was performed in Bergen as part of BreastScree

82 cancer increased with denser breasts for DBT+SM but not for DM.

83 The outcomes were compared for DBT+SM versus DM by VDG in descriptive analyses.

84 For DBT+SM, adjusted relative risk of recall (VDG 2: 1.8; P < .0

85 s synthetic mammography (SM) (hereafter, DBT+SM) and the repeat screening round with DBT with SM (her

86 ening round with DBT with SM (hereafter, DBT+SM) or full-field digital mammography (FFDM) with FFDM s

87 l synthetic mammography (SM) (hereafter, DBT+SM) versus DM.

88 th synthetic two-dimensional mammograms (DBT+SM) versus digital mammography (DM) yielded lower recall

89 when repeat screening was performed with DBT+SM (23.8% vs 12.0%, P < .01).

90 women, 14 380 of whom were screened with DBT+SM and 14 369 of whom were screened with DM (both groups

91 ) underwent repeat screening-16 198 with DBT+SM and 16 672 with FFDM.

92 igher was 14.5% (19 of 131 cancers) with DBT+SM and 8.5% (five of 59 cancers) with FFDM, both of whic

93 2015/1238) included women screened with DBT+SM between April 2015 and March 2017 and rescreened with

94 2015 and March 2017 and rescreened with DBT+SM or FFDM between April 2017 and March 2019.

95 CDR was higher for repeat screening with DBT+SM than for the control group with FFDM (8.1 per 1000 wo

96 l rate was lower for women screened with DBT+SM versus those screened with DM for VDG 1 (2.1% [81 of

97 , the Adolescent Brain Cognitive Development(SM) (ABCD) study is the largest in the U.S. assessing br

98 ment delay in patients with UM and different SM phenotypes using age-adjusted mixed-effects regressio

99 ignaling interactions, we generate different SM subtypes from human pluripotent stem cells (hPSCs), w

100 both infection and severe malarial disease (SM).

101 nary bladder, or megabladder, with disrupted SM in its wall.

102 To generate diversified SMs that enable different functions, SM-coding regions r

103 ocks EBV virion production by inhibiting EBV SM function.

104 ctivity is particularly dependent on the EBV SM protein, a nuclear protein expressed early during lyt

105 While EBV SM has been thought to act posttranscriptionally, we pro

106 interaction between Epstein-Barr virus (EBV) SM protein and cellular helicase DHX9, exploring the rol

107 and symmetric lipid vesicles composed of egg SM, cholesterol, and either unsaturated dioleoyl PC (DOP

108 cteristic of advanced SM (advSM), especially SM with an associated hematologic neoplasm (AHN).

109 midopropionamido)-hexaethyleneglycol] ester (SM(PEG)(6)) spacer.

110 membrane fusion requires Sec1/Munc18-family (SM) proteins, SNARE chaperones that can function as temp

111 ining on SM images and improved by using few SM images.Supplemental material is available for this ar

112 Based on these findings, SM-OCT represents a novel approach for intraoperative tu

113 e found remarkably inhibited in Asah1(fl/fl)/SM(Cre) CASMCs as shown by GCaMP3 Ca(2+) imaging and Por

114 Asah1(fl/fl)/SM(Cre) mice were found to have more severe AMC in both

115 ooth muscle cells (CASMCs) from Asah1(fl/fl)/SM(Cre) mice, high dose of P(i) led to a significantly i

116 midase (Ac) gene knockout mice (Asah1(fl/fl)/SM(Cre)) were used to demonstrate the role of lysosomal

117 compared to their littermates (Asah1(fl/fl)/SM(wt) and WT/WT mice) after receiving a high dose vitam

118 sal and mucosal isolated lymphoid follicles (SM-ILFs and M-ILFs, respectively) as well as in GALT-fre

119 alue (P = .78), or sensitivity (P = .33) for SM/DBT versus DM/DBT overall or within either institutio

120 /DBT (7.0% [4630 of 66 109 examinations] for SM/DBT vs 7.9% [6742 of 85 254 examinations] for DM/DBT;

121 aternal antibodies that protect infants from SM and suggest that vaccination of pregnant women with P

122 etabolic changes for differentiating IS from SM.

123 wing the highest value in separating IS from SMs were asymmetrical and symmetrical dimethylarginine,

124 bolites differentiated patients with IS from SMs with an area under the curve (AUC) of 0.90 in the re

125 rsified SMs that enable different functions, SM-coding regions rapidly evolve in fungal genomes.

126 riving force and genetic mechanism of fungal SM diversification in the context of host-pathogen inter

127 Higher SM% at midlife in 1985/1986 was associated (P < 0.05) wi

128 Being in the highest SM% quartile at midlife increased (adjusted OR: 2.32; 95

129 A HOPS SM-family subunit binds the R- and Qa-SNAREs.

130 In SM patients, GAG-DMMB and GAG-MS were increased in those

131 ew York Heart Association class I/II (96% in SM and 90% in ASA).

132 t Chol is able to displace ceramide (Cer) in SM bilayers and abolish the S(o) phase domains formed by

133 GAG-MS (g/mol creatinine) were increased in SM (mean, 95% confidence interval: 3.98, 2.44-5.53 and 6

134 was found between age and telomere length in SM.

135 copresence of long and asymmetric chains in SMs and diphytanoyl-phosphatidylcholine in the lipid mix

136 rther, we quantified sphingolipids including SM and Ceramide (Cer) using Multiple Reaction Monitoring

137 sistant BC cells, while adriamycin inhibited SM-164-resistant BC cell growth, similar to parental cel

138 treatment or by siRNA transfection, inhibits SM-dependent late lytic gene transcription but not trans

139 Last, SMs also increased IL-9 and IL-13 production and, under

140 uestionnaire responses based on a non-linear SM and validate our method compared to other methods.

141 esterol-enriched domains with 16 carbon-long SMs were most thermally stabilized by cholesterol.

142 djusted OR: 2.32; 95% CI: 1.53, 3.53; lowest SM% quartile as reference) and being in the highest BF%

143 telet-rich plasma or whole blood whereas LPS(SM) and LPS(RS) inhibited platelet activation under spec

144 t of lymphoid organ sinus lining macrophage (SMs) that provide a cell-cell contact portal, which faci

145 pping results were compared with sole maize (SM) and sole soybean (SS).

146 rease the risk of developing severe malaria (SM), but access to treatment remains low in most high-bu

147 g round with DBT plus synthetic mammography (SM) (hereafter, DBT+SM) and the repeat screening round w

148 with two-dimensional synthetic mammography (SM) (hereafter, DBT+SM) versus DM.

149 two-dimensional (2D) synthetic mammography (SM) or standard 2D digital mammography (DM) results in d

150 utcomes incorporating synthetic mammography (SM) with DBT have suggested that SM is an acceptable non

151 nsional embedding of a statistical manifold (SM).

152 Current Supplier-Manufacturer (SM) networks are highly complex and susceptible to local

153 mple of a low-grade glioma resection margin, SM-OCT is able to resolve the brain tumor margin.

154 s of body fat (BF) and skeletal muscle mass (SM) were calculated using validated formulas (including

155 ell disorders (cMCD), systemic mastocytosis (SM) and monoclonal mast cell activation syndrome (MMAS),

156 Cohorts with systemic mastocytosis (SM) and venom as well as idiopathic anaphylaxis from ref

157 Systemic mastocytosis (SM) has greatly benefited from the broad application of

158 Mechanistically, SM-164-induced BC cell death is TNFalpha-dependent, with

159 ies of Spitz tumors (ST) and Spitz melanoma (SM).

160 across a plant-derived symbiosome membrane (SM), which encloses rhizobia to form a symbiosome.

161 Water Maze (MWM) and reduced social memory (SM).

162 DE) and the surrounding splanchnic mesoderm (SM).

163 the prediction of PE with five metabolites (SM C28:1, SM C30:1, LysoPC C19:0, LysoPE C20:0, propane-

164 e production of these secondary metabolites (SMs) (also called natural products) is often encoded by

165 Secondary metabolites (SMs) are crucial for fungi and vary in function from ben

166 isms produce numerous secondary metabolites (SMs) with various biological activities.

167 and the production of secondary metabolites (SMs).

168 ces (VHA ONS) issued a Staffing Methodology (SM) Directive, standardizing the method of determining a

169 erived activator of caspase (SMAC) mimetics (SMs) are selective antagonists of the inhibitor of apopt

170 ith ischemic stroke (IS) from stroke mimics (SMs) poses a diagnostic challenge.

171 delivered by a lay counsellor (SEHER Mitra [SM]) or a teacher (Teacher as SEHER Mitra [TSM]), respec

172 a higher concentration of a probe (0.56 mM), SM sensing can be visualized with the naked eye, leading

173 FM pre-amplified receivers and single-mode (SM) pre-amplified receivers with or without adaptive opt

174 hotopic murine glioblastoma xenograft model, SM-OCT readily identifies brain tumor margins with resol

175 Even though surface soil moisture (SM) is often cited as a key control variable in these pr

176 solution transcriptome-wide, single molecule SM-PAT-seq method.

177 Single molecule (SM) detection represents the ultimate limit of chemical

178 ates, which was measured by single-molecule (SM) Forster Resonance Energy Transfer imaging.

179 ions of conventional scanning monochromator (SM) CPL spectrometers, which are costly to acquire and m

180 Squalene monooxygenase (SM) is a rate-limiting enzyme in cholesterol synthesis.

181 The true ecological function of most SMs remains obscure, but understanding of both the activ

182 rce transmission pathways in a multicellular SM ensemble.

183 ty of accessory factors such as Sec1/Munc18 (SM), double C2-domain proteins (DOC2), and additional in

184 ns, and SNARE chaperones of the Sec1/Munc18 (SM), Sec17/alpha-SNAP, and Sec18/NSF families.

185 pin1 played a novel role in skeletal muscle (SM) regeneration.

186 sed in many tissues including smooth muscle (SM), although its role in the regulation of their physio

187 ed in many tissues, including smooth muscle (SM), but its physiologic function is unknown.

188 on fluorescence detection of sulfur mustard (SM) at "room temperature".

189 In the I131C stability mutant (SM), we observe an open-active channel in the presence o

190 ification of BGCs and the discovery of novel SMs.

191 e, maize and soybean produced 98% and 77% of SM and SS yield, respectively, in medium-strips.

192 thout recognition elements and is capable of SM detection.

193 A-1 antibody levels had 51.4% fewer cases of SM compared to individuals with lower anti-PfSEA-1 level

194 identified distinguishing characteristics of SM and GM (general metabolism, traditionally referred to

195 Definitions of SM were standardised across studies to compare treatment

196 in water exhibits fluorescence detection of SM within seconds.

197 echnique for chromo-fluorogenic detection of SM.

198 shows that lipin1 is a major determinant of SM development.

199 This identifies a diversity of SM cell types that develop in close register with the or

200 force transmission in a two-cell ensemble of SM cells.

201 ecological systems, catastrophic failures of SM networks are difficult to predict due to high dimensi

202 in structure of SMs affects the formation of SM- and cholesterol-enriched domains.

203 TF protein by ASMase-mediated hydrolysis of SM.

204 ) panels to profile the genetic landscape of SM variants have been critical adjuncts to the diagnosis

205 Because mixtures of SM, POPC, and cholesterol form domains in symmetric vesi

206 In this work we investigate resiliency of SM networks using the quantitative methods employed to s

207 PfSEA-1 antibodies predict decreased risk of SM in infants and vaccination of mice with PbSEA-1 prior

208 nd 8 other observational clinical studies of SM and UM cases.

209 ts to the diagnosis and subclassification of SM, and development of clinical-molecular prognostic sco

210 PB to specifically activate transcription of SM target promoters.

211 holesterol accelerates the ubiquitination of SM by membrane-associated ring-CH type finger 6 (MARCH6)

212 ies, displayed a significant upregulation of SM genes and Notch3 target genes, and genetic reduction

213 Of SMs, 77% had fusions and/or truncations with eight invol

214 investigated how the acyl chain structure of SMs affects the formation of SM- and cholesterol-enriche

215 DHX9 was not mediated through its effects on SM.

216 es from two institutions without training on SM images and improved by using few SM images.Supplement

217 on that separates ISM from CM and from other SM variants.

218 t delay was moderately associated with other SM phenotypes (OR [95% CI] >3 to <=4 days versus <=24 ho

219 A total of 129 patients (SM = 43, MSM = 57, HC=29) were recruited.

220 imilated C into future survival by producing SM.

221 he EBV posttranscriptional regulator protein SM, whose expression is limited to the replicative cycle

222 r leaflet of the plasma membrane and reduced SM levels in monocytes.

223 ts with diverse AHNs and relapsed/refractory SM.

224 into the signalling circuitry that regulates SM development, and have important implications for deve

225 nsfer technique also suggested that the same SMs also form the largest L(o) domains.

226 Here we present the structure of a second SM-Qa-SNARE complex, Vps45-Tlg2.

227 e, we use recently developed remotely sensed SM products to analyse the link between spatial patterns

228 ed as static features, single-molecule SERS (SM-SERS) is marked by characteristic time-dependent fluc

229 ne marrow mastocytosis (BMM), and smoldering SM (SSM), 4.1% of disease progressions have been observe

230 Therefore, the specific SM species present in the membranes, together with unsat

231 Under some conditions, a sphingomyelin (SM) and cholesterol-rich ordered domain in one leaflet i

232 The second is a sphingomyelin (SM)-sequestered pool inaccessible to PFO and ALOD4 but t

233 , phosphatidic acid (PA), and sphingomyelin (SM) in a safe manner by removing any need for direct han

234 turated phospholipids such as sphingomyelin (SM), may form.

235 esulting in elevated cellular sphingomyelin (SM) levels and altered SM distribution.

236 bind to membranes containing sphingomyelin (SM) and oligomerize to form pores.

237 terol plus different types of sphingomyelin (SM) are prone to produce bilayer regions with more than

238 In particular, sphingomyelin (SM) species were significantly up-regulated in 5-FU-resi

239 hosphatidylethanolamine (PE), sphingomyelin (SM), phosphatidylcholine (PC) and their molecular specie

240 a favorable interaction with sphingomyelin (SM), together forming domains in the liquid-ordered phas

241 hosphatidylcholines (PC) and sphingomyelins (SM) were the most abundant phospholipids in the RBCs out

242 00A4 depletion did not affect ACh-stimulated SM myosin regulatory light chain phosphorylation.

243 link between spatial patterns of summertime SM, precipitation and air temperature biases over CONUS

244 tability in cholesterol-containing symmetric SM/PC vesicles.

245 e of brainstem compression or syringomyelia (SM).

246 The yeast vacuolar tethering/SM complex HOPS (homotypic fusion and vacuole protein so

247 t DHX9 possesses antiviral activity and that SM may suppress the antiviral functions of DHX9 through

248 We conclude that SM-164 is a promising therapeutic agent for early stage

249 We demonstrate that SM binds and recruits XPB to EBV promoters during lytic

250 Our findings demonstrate that SM proteins can engage Qa-SNAREs using at least two diff

251 ttranscriptionally, we provide evidence that SM also facilitates EBV gene transcription.

252 s and epithelial cells, we hypothesized that SM utilizes XPB to specifically activate transcription o

253 These data indicate that SM acts as a transcriptional activator that has co-opted

254 Thus, from our study, we propose that SM/Cer and SMPD1 are new potential target molecules for

255 We show that SM binds to and colocalizes DHX9 and may counteract the

256 Here, we show that SM-164, a small molecule inhibitor, which degrades inhib

257 ammography (SM) with DBT have suggested that SM is an acceptable non-radiation dose alternative to DM

258 MP response elements (CREs), suggesting that SM may also act to counteract DHX9's antiviral functions

259 These results demonstrate that SMs shape the gene expression and protein profiles of T(

260 Moreover, proteomics analysis revealed that SMs altered the abundance of proteins associated with ce

261 The SM has deep connections with parametric statistical mode

262 responsible for transport of iron across the SM to bacteroids and plays a crucial role in the nitroge

263 ot only suggest a molecular link between the SM alpha-actin cytoskeleton and classic fibrogenic signa

264 se that catalyses nitrogen fixation, but the SM iron transporter has not been identified.

265 To further characterize the SM-DHX9 interaction, we performed immunofluorescence mic

266 gical considerations may further improve the SM prediction model.

267 h the control group, the participants in the SM intervention group reported improvements in school cl

268 hibition of MEF2c and MyoD expression in the SM of lipin1(Myf5cKO) mice.

269 destabilize ordered domain formation in the SM+PC-containing outer leaflet relative to ordered domai

270 MS in the ion extracted chromatograms of the SM((ox))IRPPY peptide (monoisotopic [M + 2 H](+2) mass =

271 s between membranes; SNARE chaperones of the SM, Sec17/alphaSNAP, and Sec18/NSF families; Rab-GTPases

272 obe the spatial and temporal features of the SM-SERS fluctuations from single silver nanoshells.

273 e investigated to improve performance on the SM datasets, and the effect of dataset size on each adap

274 Paradoxically, the SM protein Munc18-1 traps the Qa-SNARE protein syntaxin-

275 esterol complexes in membranes, to study the SM-sequestered cholesterol pool.

276 Previous studies have revealed that the SM protein Munc18-1 plays a critical role for precise SN

277 gy transfer techniques, we observed that the SM- and cholesterol-enriched domains with 16 carbon-long

278 cells and both proteins are localized to the SM.

279 acilitated lateral segregation most with the SMs that had 16 carbon-long acyl chains.

280 an 'template' SNARE complex assembly through SM recognition of R- and Qa-SNAREs.

281 BV lytic gene expression was not confined to SM-dependent genes, indicating that the antiviral effect

282 The FFDM model was evaluated by using SM datasets from two institutions (site 1: 3842 patients

283 entration quantification down to 10 fM using SM detection events.

284 nes, the saturated phospholipids are usually SMs with different acyl chains, the abundance of which v

285 bly requires catalysis by HOPS, the vacuolar SM and tethering complex.

286 M + DBT, DM versus DM + CAD, DM + DBT versus SM + DBT, and DM versus DM + DBT at double reading.

287 viral entry activity, suggesting that viral SM plays a role in BoHV-1 entry, while cellular SM does

288 Whether SM alpha-actin has specific functional effects on cellul

289 had experienced severe HVA, 32 cMCD (22 with SM and 10 with MMAS) and 14 controls.

290 ctal cancer cells was mainly associated with SM increase and Cer decrease, which are controlled by ac

291 -PS and anti-DNA antibodies in children with SM compared to CC (p < 0.0001 for both).

292 d found that DHX9 partially colocalized with SM in nuclear foci during EBV lytic replication.

293 DA-MB-231 cell apoptosis in combination with SM-164.

294 and the repeat screening round with DBT with SM (hereafter, DBT+SM) or full-field digital mammography

295 The unadjusted recall rate was lower with SM/DBT than with DM/DBT (7.0% [4630 of 66 109 examinatio

296 Among patients with SM, concomitant HalphaT was associated with increased ri

297 between treatment delay and presenting with SM.

298 r outcomes from breast cancer screening with SM/DBT versus DM/DBT.Materials and MethodsThis was a ret

299 ut that becomes accessible by treatment with SM-degrading sphingomyelinase (SMase).

300 m 13 studies of 3,989 (94.1% aged <15 years) SM patients and 5,780 (79.6% aged <15 years) UM cases in