ライフサイエンスコーパス: SPC

1 SPC activity in ONH lysate was significantly higher than

2 SPC films plasticized with 40-50% glycerol showed a time

3 SPC flow prior to Fontan measured 1.5+/-0.9 L/min/m(2),

4 SPC number and intracellular content of hypoxia-inducibl

5 SPC recruitment and protein changes were inhibited by si

6 SPC was extracted from soybean flour, produced during th

7 SPC-Cre(+) Vcan(-/-) mice demonstrated enhanced recruitm

8 SPC-IAP's high brightness (L* = 79.55), low redness (a*

9 SPC-Tert cKO mice did not develop pulmonary fibrosis spo

10 SPCs exhibit testicular repopulating activity in vivo an

11 SPCs performed only 18% of these operations (15,002).

12 SPCs were diagnosed in 16.8% of penile cancer patients a

13 tisfaction with SPC availability (P < .001), SPC acceptance of patients receiving chemotherapy (P < .

14 hosphoprotein 65 antigen SPCs were both >100 SPCs per 250 000 cells (cutoff 2), and a low CMV-CMI whe

15 ediatric appendectomies were performed in 21 SPCs and 183 DGHs in England.

16 any and 7,560 in Sweden, overall 752 and 349 SPCs were recorded, respectively.

17 sexD3 was recruited for biosynthesis of 3UFA SPCs in M. sexta lineage via gene duplication and neofun

18 (mean age, 60.4 years; 48.8% women), 156 442 SPC cases and 88 818 SPC deaths occurred during 11 197 8

19 ; 48.8% women), 156 442 SPC cases and 88 818 SPC deaths occurred during 11 197 890 person-years of fo

20 8.6 x 10,000 PY), and 22 of them developed a SPC (annual incidence: 3.9 x 10,000 PY).

21 The annual incidence of a SPC in the transplanted patients who developed a first c

22 ype and presence of a fenestration, absolute SPC flow was significantly associated with hospital dura

23 In addition, SPC treatment, but not genistin treatment, significantly

24 or gene Trp53(F/F) mice infected with Adeno5-SPC-Cre and Adeno5-CC10-Cre viruses displayed difference

25 ounting for 31% to 33% of mortality from all SPCs.

26 to both small airway (CCSP(+)) and alveolar (SPC(+)) epithelial cells in three-dimensional (3D) organ

27 sing a bleomycin model of lung injury and an SPC-driven inducible cre to fate-map AECs, we found the

28 greater risk of developing and dying from an SPC, compared with the general population.

29 han that of retinal lysate; however, when an SPC inhibitor was added, activity in ONH decreased more

30 Risk for these genital and anal SPCs are high and a follow-up plan should be agreed at d

31 with SPC service availability (P < .001) and SPC service acceptance of patients on chemotherapy (P <

32 Combined absence of PAK-1 and SPC-1 induced complete axis retraction, owing to defecti

33 of ClayFF, constant-valence force field, and SPC water model.

34 Mice treated with genistin and SPC had reduced final tumor weights by 56% (P < 0.05) an

35 -transformed duration of hospitalization and SPC flow as a proportion of total aortic (rho=0.31, P=0.

36 Da secretory protein (CC10+) airway-like and SPC+ saccular structures within 6 days.

37 athology results (rho = 0.87; P < .001), and SPCs correlated with histopathology results (rho = 0.88;

38 s to observe and characterize Dirac WGMs and SPCs, and speculate that these features can potentially

39 when the IE-1 and phosphoprotein 65 antigen SPCs were both >100 SPCs per 250 000 cells (cutoff 2), a

40 isks were calculated bidirectionally for any SPC after skin cancer and for skin cancer as SPC.

41 mong men, the overall risk of developing any SPCs was statistically significantly higher for 18 of th

42 ng women, the overall risk of developing any SPCs was statistically significantly higher for 21 of th

43 the 30 FPC types, and risk of dying from any SPCs was statistically significantly higher for 27 of 30

44 the 31 FPC types, and risk of dying from any SPCs was statistically significantly higher for 28 of 31

45 SPC after skin cancer and for skin cancer as SPC.

46 rranted to apply soy phytochemicals, such as SPC, as a potent prevention regimen for bladder cancer p

47 thetic templates, an approach we refer to as SPC-sequencing.

48 genital cancers might run in families and as SPCs are associated with human papilloma virus and smoki

49 were for invasive and in situ skin cancer as SPCs (14.59 and 16.71, respectively).

50 participate in an anonymous survey assessing SPC referral practices.

51 function may contribute to SPCs by assessing SPCs associated with known immune responsive skin cancer

52 ractor muscles and in the spicule-associated SPC and PCB cholinergic neurons.

53 r, only a few smoking- or obesity-associated SPCs, such as lung, urinary bladder, oral cavity/pharynx

54 ency included comprehensiveness of available SPC services (P = .004), satisfaction with SPC availabil

55 ck from mTORC1 to the GDNF receptor balances SPC growth with self-renewal.

56 plications in gas separation/storage because SPCs do not inherently suffer from the recyclability pro

57 e sought to evaluate the association between SPC flow and acute post-Fontan clinical outcomes using a

58 BM SPC recruitment is a repair response to dimethylnitrosam

59 uitment of SPCs was analyzed by examining BM SPC proliferation, mobilization to the circulation, engr

60 soidal endothelial cell progenitor cells (BM SPCs) repopulate the sinusoid as liver sinusoidal endoth

61 ays, 40% of all LSECs came from engrafted BM SPCs.

62 After partial hepatectomy, BM SPCs provide hepatocyte growth factor, promote hepatocyt

63 t, bone marrow progenitor cells of LSECs (BM SPCs), which are rich in HGF, are recruited to the liver

64 th factor (VEGF) regulates recruitment of BM SPCs and their effects on liver injury.

65 ethylnitrosamine-induced proliferation of BM SPCs and their mobilization to the circulation, reduced

66 Hepatic VEGF regulates recruitment of BM SPCs to liver and reduces this form of liver injury.

67 s in the bone marrow, and mobilization of BM SPCs to the circulation increased 2- to 4-fold by 24 hou

68 Proliferation of BM SPCs, the number of SPCs in the bone marrow, and mobiliz

69 er regeneration from the perspective that BM SPCs that have been recruited to the liver, rather than

70 mong week-to-week assessments of blood-borne SPC HIF factors.

71 demonstrated that the number of blood-borne SPCs and the cellular content of HIFs at study entry and

72 We conclude that both SPC-expressing alveolar type 2 cells and CC10-expressing

73 , migration, and tube formation through both SPC-dependent and -independent pathways.

74 Furthermore, both SPCs and MASCs express GPR125, an orphan adhesion-type G

75 s and in them 45.9% of deaths were caused by SPC (other than penile cancer).

76 ce reduces processing time for genotyping by SPC-SBE and allows direct spotting of sample for rapid a

77 Oxidative stress from lactate metabolism by SPCs accelerated further SPC recruitment and differentia

78 -RasG12D expression in Surfactant Protein C (SPC)(+) alveolar type 2 cells and in Clara cell antigen

79 atic cancer, and sporadic pancreatic cancer (SPC) kindreds as families without such an affected pair.

80 ed the incidence of a second primary cancer (SPC) in 7,636 patients who underwent a kidney, liver, lu

81 a on the risk of subsequent primary cancers (SPCs) among survivors of adult-onset cancers are limited

82 Second primary cancers (SPCs) are becoming a common cancer entity, which may int

83 ginal cancers and in second primary cancers (SPCs) following these cancers are limited.

84 ribution and risk of second primary cancers (SPCs) in multiple myeloma (MM) survivors in Germany and

85 uencing method using solid-phase capturable (SPC) dideoxynucleotides and MALDI-TOF mass spectrometry

86 CD34(+) SPCs from lactate-supplemented Matrigel exhibited signif

87 There were over one million CD34(+) SPCs per Matrigel plug 18 h after Matrigel implantation,

88 ab-on-a-screen-printed electrochemical cell (SPC) based on iron-sparked graphite working electrode mo

89 Using spermatogonial progenitor cells (SPCs) as a model system, we show that mTORC1 impairs ste

90 ative adult spermatogonial progenitor cells (SPCs) can be efficiently obtained by cultivation on mito

91 iation of circulating stem/progenitor cells (SPCs) in subcutaneous Matrigel in mice was assessed.

92 sized that studies of stem/progenitor cells (SPCs) in the early weeks of standard wound management co

93 We observed that CML stem/progenitor cells (SPCs) produce tumor necrosis factor-alpha (TNF-alpha) in

94 rvival signals to CML stem/progenitor cells (SPCs) with contradictory results.

95 f postnatal spermatogonial progenitor cells (SPCs).

96 models [i.e., extended simple point charge (SPC/E) vs. four-site transferrable intermolecular potent

97 ding yeast, the spindle position checkpoint (SPC) delays mitotic exit until the mitotic spindle moves

98 The spindle position checkpoint (SPC) ensures correct mitotic spindle position before all

99 ay known as the spindle position checkpoint (SPC) to ensure the arrival of one end of the mitotic spi

100 ircuit components, including the cholinergic SPC and PCB and the glutamatergic PCA sensory-motor neur

101 d on the concept of split proximity circuit (SPC) to achieve both high sequence selectivity and assay

102 The tunable binary citrem/SPC nanoplatform holds promise for future development of

103 hway as a relevant therapeutic target in CML SPCs and endorse the current use of nilotinib in combina

104 Furthermore, we demonstrate that in CML SPCs, inhibition of autocrine TNF-alpha signaling via a

105 highlight a novel survival mechanism of CML SPCs and suggest a new putative therapeutic target for t

106 a and found that it supports survival of CML SPCs by promoting nuclear factor kappaB/p65 pathway acti

107 s correlated with increased apoptosis of CML SPCs in vitro and a reduction in primitive quiescent CML

108 1A1+ airway cell population that coexpresses SPC, a marker for type II alveolar cells that promotes a

109 f systemic-to-pulmonary arterial collateral (SPC) vessels in single ventricle patients are poorly und

110 Systemic-pulmonary collateral (SPC) flow occurs commonly in single ventricle patients a

111 bjected to a supervised principal component (SPC) analysis to predict progression-free survival (PFS)

112 RCC-specific supervised principal component (SPC) risk score prognostic gene signature was constructe

113 y the molecular basis of novel SP component (SPC) acquisition, we used the tobacco hornworm (Manduca

114 with individual sparse principal components (SPCs) are also reported, showing that both high- and low

115 Availability of comprehensive SPC, especially for patients receiving chemotherapy, and

116 oflavone-rich soy phytochemical concentrate (SPC) on the growth and metastasis of 253J B-V tumors in

117 coacervation of soybean protein concentrate (SPC) by using calcium salts and spray-drying.

118 l properties of soybean protein concentrate (SPC) films, plasticized with varying levels of glycerol

119 sing that the isolated protein concentrates (SPC-IAP) show high protein content (69.08% d.b) as well

120 (ML) for monitoring soluble pectin content (SPC) changes in orange juice.

121 d inhibited spontaneous phasic contractions (SPCs) of colonic muscle through activation of a SK condu

122 proach based on statistical process control (SPC), which is able to monitor the response to a treatme

123 luated by using statistical process control (SPC).

124 by a subtilisin-like proprotein convertase (SPC).

125 Subtilisin-like proprotein convertases (SPCs) are a family of calcium-dependent cleavage enzymes

126 y of subtilisin-like proprotein convertases (SPCs).

127 d by subtilisin-like proprotein convertases (SPCs).

128 y of subtilisin-like proprotein convertases (SPCs).

129 opsy samples using stereologic point counts (SPCs).

130 rmediate-early 1 (IE-1) antigen spot counts (SPCs) were >100 (cutoff 1) or when the IE-1 and phosphop

131 Soft porous crystals (SPCs) that exhibit stimuli-responsive dynamic sorption b

132 e an emerging class of soft porous crystals (SPCs) with potential for high working capacity for gas s

133 ), also referred to as soft porous crystals (SPCs), show reversible structural transitions dependent

134 The sneak path current (SPC) problem is one of the main difficulties in crossbar

135 enceforth the term superpersistent currents (SPCs).

136 p-angle cycled (SFC), rf pulse phase cycled (SPC), and pulsed field gradient (PFG) strength cycled (S

137 thesized that embolization acutely decreases SPC flow and increases systemic blood flow (Q(S)).

138 In differentiating SPCs, Sall4 levels transiently increase and Sall4 physic

139 substrate-based assay was used to elucidate SPC enzyme activity within human retina and optic nerve

140 cle embolization of angiographically evident SPC vessels.

141 1b+ cells give rise to adenomas that express SPC and TTF1.

142 e show that surfactant protein C-expressing (SPC-expressing) alveolar epithelial type II cells (AECII

143 d solid phase capture-single base extension (SPC-SBE) method.

144 vitro assays indicated that MMP8 facilitated SPC migration across endothelial cells and through Matri

145 arrows of ApoE(-/-)/MMP8(-/-) mice had fewer SPCs in atheromas and smaller lesions than ApoE(-/-)/MMP

146 In addition, a fluorogenic SPC substrate-based assay was used to elucidate SPC enzy

147 zed with GLY had the highest EE (74.54%) for SPC-GLY-0.25%HC (P < 0.05).

148 groups were different at 95% confidence for SPC average values.

149 sponse to GDNF, a growth factor critical for SPC self-renewal, via negative feedback at the level of

150 g Plzf, a transcription factor essential for SPC maintenance, have enhanced mTORC1 activity.

151 Similar associations exist for SPC flow as a percentage of total aortic (OR=1.09, P=0.0

152 ointestinal track model system was found for SPC-CHO-0.5%HC.

153 an intrachain disulfide bond is required for SPC-mediated cleavage and that SPC-mediated cleavage is

154 cer in family members; additionally risk for SPCs was calculated.

155 The biological effects resulting from SPC/GPR4 interactions involve the activation of both pho

156 of such cells with culture supernatant from SPCs without MMP8 knockdown, and this compensatory effec

157 ctate metabolism by SPCs accelerated further SPC recruitment and differentiation through Trx1-mediate

158 Long-term cultures of GPR125+ SPCs (GSPCs) also converted into GPR125+ MASC colonies.

159 All patients who had SPC flow quantified by CMR imaging before Fontan were re

160 An important question is how SPC activity is coordinated with mother-daughter polarit

161 These results identify SPC and its receptor, GPR4, as critical regulators of th

162 , showing that both high- and low-importance SPCs were associated with cell death pathways, though th

163 ion, demonstrating a significant decrease in SPC flow and Q(P):Q(S) and increase in Q(SVC) and Q(S).

164 lization, we found a significant decrease in SPC flow of 0.9 (range, 0.6-1.3) L/(min.m(2)) (P=0.03);

165 t encapsulation efficiency (EE) was found in SPC-CHO-0.5%HC (P < 0.05) (85.42%), while liposome stabi

166 he spontaneous lung inflammation observed in SPC-TSLP mice reflects a TSLP-driven predisposition towa

167 metalloproteinase-8 (MMP8) played a role in SPC migration and their recruitment into atheromas.

168 MMP8 plays an important role in SPC migration and their recruitment into atherosclerotic

169 Postoperative length of stay was double in SPCs compared with DGHs (median, 4 vs 2 days).

170 ve appendectomy rates than those operated in SPCs.

171 ons, and 11% more readmissions than those in SPCs.

172 Increasing SPC flow before Fontan, as measured by CMR imaging, is a

173 by crossing floxed Tert mice with inducible SPC-driven Cre mice.

174 own-regulation of GPR4 specifically inhibits SPC-, but not sphingosine-1-phosphate-, or vascular endo

175 forms a noncovalent latent complex with its SPC-cleaved prodomain and that this latent complex is ac

176 (AECII)-specific TERT conditional knockout (SPC-Tert cKO) mice by crossing floxed Tert mice with ind

177 Primary GPR125-LacZ SPC lines retained GPR125 expression, underwent clonal e

178 on of mouse oocytes with the GPR3/12 ligands SPC and S1P delayed spontaneous oocyte maturation.

179 mbedded human posterior sections to localize SPC family members.

180 More detailed analysis of Adora2b(loxP/loxP) SPC Cre(+) mice confirmed elevated lung inflammation and

181 disease susceptibility in Adora2b(loxP/loxP) SPC Cre(+) mice.

182 lveolar epithelial cells (Adora2b(loxP/loxP) SPC Cre(+)) revealed a selective increase in disease sus

183 After lyophilization, SPC-CHO-0.5%HC had higher EE than SPC-GLY-0.25%HC (P < 0

184 Lyophilized SPC-CHO-0.5%HC exhibited higher stability than lyophiliz

185 exhibited higher stability than lyophilized SPC-GLY-0.25%HC during storage at 25 degrees C for 28 da

186 epithelial cell-specific Cre transgenic mice SPC-rtTA/TetO-Cre and floxed-Alk3 mice.

187 epithelial-specific versican-deficient mice [SPC-Cre(+) Vcan(-/-)] demonstrated that RSV infection le

188 More SPCs entered the bloodstream in the first 2 weeks of car

189 type MMTV mice and a triple transgenic mouse SPC-rtTA(+/-)TetoCre(+/-)LoxP-VEGF-A(+/+) to conditional

190 A degree of toxicity toward normal SPCs was observed with the combination treatment, althou

191 of a Sca1 promoter yielded CC10(+), but not SPC(+), hyperplasias, and adenomas.

192 njugation of low-molecular-mass O-SP-core (O-SPC) fragments.

193 ced in young outbred mice by the S. sonnei O-SPC conjugates were significantly higher then those elic

194 The O-SPC conjugates used oxime linkages between the terminal

195 The O-SPC fragments were bound by their reducing ends similar

196 ECT 475) in order to evaluate the ability of SPC to encapsulate and protect bacteria from stress cond

197 ction of GPR4 fully restores the activity of SPC.

198 hesis of the first photoaffinity analogue of SPC.

199 Here, we report the application of SPC-sequencing in characterizing frameshift mutations by

200 uitable strategy for the quick assessment of SPC trends in orange juices.

201 e amino group at C2 of the sphingoid base of SPC analogue 2.

202 Moreover, the effects of SPC on EC require SPC induced trans-phosphorylation and

203 R4 is required for the biological effects of SPC on endothelial cells (EC).

204 The acute efficacy of SPC embolization has not been demonstrated in a quantifi

205 We sought to assess the acute efficacy of SPC embolization on blood flow as quantified by phase co

206 We report on the acute efficacy of SPC embolization, demonstrating a significant decrease i

207 and adenomas, but not for the generation of SPC(+) ATII lesions.

208 Current reference methods of SPC in orange juice are laborious, requiring several ext

209 he simple rapid screening of a wide range of SPC mediated organic reactions.

210 study does not indicate an increased risk of SPC in transplanted subjects who already suffered a firs

211 Herein, we report a new type of SPC based on a [2 + 3] imide-based organic cage (NKPOC-1

212 pecific types of FPCs with specific types of SPC risk; however, only a few smoking- or obesity-associ

213 lveolar hyperplasias, exclusively made up of SPC(+) ATII cells, progressed to yield malignant adenoca

214 hat MMP8 deficiency inhibited the ability of SPCs to migrate from the arterial lumen and the adventit

215 The decrease in migratory ability of SPCs with MMP8 knockdown was reduced by incubation of su

216 he expression, localization, and activity of SPCs in the human retina and optic nerve head.

217 We conclude that assays of SPCs during the first weeks of care in patients with DFU

218 NKPOC-1 supports the further development of SPCs for applications in gas separation/storage because

219 Incidence and mortality of SPCs per 10 000 person-years; standardized incidence rat

220 actate concentration increased the number of SPCs by 3.6-fold.

221 Proliferation of BM SPCs, the number of SPCs in the bone marrow, and mobilization of BM SPCs to

222 uman testis could enrich for a population of SPCs for derivation of GPR125+ MASCs, which may be emplo

223 Recruitment of SPCs was analyzed by examining BM SPC proliferation, mob

224 chanism contributing to an increased risk of SPCs.

225 However, the design and synthesis of SPCs is challenging.

226 eden to provide etiological understanding of SPCs and insight into their incidence rates and recordin

227 sintegrin-and-metalloproteinase-domain-10 on SPCs.

228 r GI254023X decreased E-cadherin shedding on SPCs.

229 However, only SPC(+) alveolar type II (ATII) cells were able to form h

230 However, in adult mice, only SPC(+) ATII cells were able to yield malignant adenocarc

231 comprised substantial proportions of overall SPC incidence and mortality among all survivors and high

232 xture of citrem and soy phosphatidylcholine (SPC) at different weight ratios, we describe a library o

233 somes prepared from soy phosphatidylcholine (SPC) with various stabilizers (cholesterol (CHO) or glyc

234 The semiconductor photocatalyzed (SPC) oxidation of toluene is performed inside an NMR spe

235 Aberrant mTORC1 activation in Plzf(-/-) SPCs inhibits their response to GDNF, a growth factor cr

236 with self-rectifying behavior are potential SPC solutions.

237 ed recombined, GFP+ cells were predominantly SPC+.

238 eta4, but little or no pro-surfactant C (pro-SPC), is endowed with regenerative potential.

239 ession of the type II epithelial marker, pro-SPC.

240 LP transgene (surfactant protein C promoter (SPC)-TSLP) develop a spontaneous and progressive asthma-

241 outcomes using a novel method of quantifying SPC flow by cardiac magnetic resonance (CMR) imaging.

242 and that MMP8 knockout significantly reduced SPC numbers in atherosclerotic lesions in apolipoprotein

243 Moreover, the effects of SPC on EC require SPC induced trans-phosphorylation and activation of the

244 Allogeneic resident SPCs, infused 24 hours after injection of dimethylnitros

245 Results SPC analysis allowed stratification based on 11 features

246 ed free-running single photon counting (SEFR-SPC), excitation pulses from inexpensive laser diodes (p

247 We also show that SEFR-SPC provides state-of-the-art sensitivity in the SWIR sp

248 The SEFR-SPC method will be a valuable tool for studies of weak,

249 SIRs) were used to assess risk of a specific SPC compared to risk of the same first cancer in the cor

250 A SOMA for the CCRCC-specific SPC prognostic gene signature that is predictive of dise

251 nder the control of lung epithelial-specific SPC promoter was produced.

252 Significantly elevated SIRs of specific SPCs were observed for acute myeloid leukemia (AML; SIR

253 hway, thereby identifying the alpha-spectrin SPC-1.

254 that the lipid sphingosylphosphorylcholine (SPC) induces angiogenesis in vivo and GPR4 is required f

255 lipid mediator sphingosylphosphorylcholine (SPC) has not yet been identified.

256 ed for their ability to effectively suppress SPC in RRAM arrays.

257 ilization, SPC-CHO-0.5%HC had higher EE than SPC-GLY-0.25%HC (P < 0.05).

258 required for SPC-mediated cleavage and that SPC-mediated cleavage is essential to protein function.

259 preexisting type II AECs, demonstrating that SPC- progenitor cells replenished type II AECs during re

260 ally HIF-1 null myeloid cells indicated that SPC recruitment and lactate-mediated effects were depend

261 Ninety-four percent reported that SPC was available to them, but only 37% reported that th

262 Results show that SPC is a feasible material for the development of probio

263 like growth factor-I levels, suggesting that SPC may contain other bioactive ingredients that have an

264 We find that SPCs lacking Plzf, a transcription factor essential for

265 We found that SPCs in atheromas expressed MMP8 and that MMP8 knockout

266 med the relationship between the RRS and the SPC gene signature (R = 0.45, P < .001, classification a

267 we identified bud2Delta as deficient for the SPC.

268 based on the X -S charts generated from the SPC charts.

269 d-type males, GAR-3(mAChR) expression in the SPC and PCB neurons is required for the male to sustain

270 ings reveal a novel function for Elm1 in the SPC and suggest how checkpoint activity may be linked to

271 nce interval (CI), 4.5-16.1], but not in the SPC kindreds (1.8; 95% CI., 0.22-6.4).

272 The performance of the SPC analysis (OS: IBS, 0.149; C index, 0.654; PFS: IBS,

273 The performance of the SPC analysis model was further improved when combined wi

274 mRNA expression of the SPC family in the human retina and optic nerve head tiss

275 We discussed the design aspects of the SPC recognition regions and demonstrated its plug-and-pl

276 This result shows that the incidence of the SPC was the same as the incidence of a first cancer.

277 kinase Elm1 is an obligate regulator of the SPC, and this function requires localization of Elm1 to

278 nstringent assay condition requirements, the SPC retained good analytical performance to detect subna

279 t dataset to confirm its relationship to the SPC gene signature (n = 70) and determination of patient

280 of the human p53 gene in one tube using the SPC-SBE method.

281 te IR and isotropic Raman spectra, using the SPC/E simulation model, and the results are in good agre

282 of a Cox proportional hazards model with the SPC analysis predictor was assessed with C index and int

283 , Spn4.1 forms SDS-stable complexes with the SPC furin and directly inhibits it.

284 The RRS scaled with the SPC gene signature (R = 0.57, P < .001, classification a

285 These results indicate that the SPCs are expressed in distinct patterns throughout the h

286 A striking finding is that the SPCs can be attributed to a robust type of relativistic

287 orm noncovalent, latent complexes with their SPC-cleaved prodomains.

288 Thus, SPC-CHO liposome could be used as a promising carrier of

289 tients with SPC, 36.4% of deaths were due to SPC.

290 Oncologists referred patients frequently to SPC, but generally late in the disease course for patien

291 One third would refer to SPC earlier if it was renamed supportive care.

292 is that immune dysfunction may contribute to SPCs by assessing SPCs associated with known immune resp

293 cavopulmonary connection patients underwent SPC flow quantification by phase contrast magnetic reson

294 000 cells (cutoff 2), and a low CMV-CMI when SPCs were below these thresholds.

295 reased neutrophils in the lung compared with SPC-Cre(-) RSV-infected littermates.

296 In vulvar/vaginal cancer patients with SPC, 36.4% of deaths were due to SPC.

297 e SPC services (P = .004), satisfaction with SPC availability (P < .001), SPC acceptance of patients

298 ather than later, included satisfaction with SPC service availability (P < .001) and SPC service acce

299 d to interact directly and specifically with SPC and in the definition of the ligand-binding sites.

300 e correlated the (1)H-MRS PDFF findings with SPCs (r = 0.92; P < .001).