ライフサイエンスコーパス: SPF

1 SPF >/= 15 sunscreen use was associated with significant

2 SPF IL-10-knockout mice had no significant difference in

3 SPF mice colonized with rETBF mimicked WT-ETBF, whereas

4 SPF microbiota did not induce HO-1 in colons of germ-fre

5 SPF optoacoustic imaging was applied to imaging arteries

6 tion between sunscreen use (never, SPF < 15, SPF >/= 15) and melanoma risk by calculating hazard rati

7 neata the proportions were approximately 20% SPF, 5% PMF, and PRF was absent.

8 et ~ UV Index (355 reviews), VisualDx (306), SPF (128), iSore (61), and SpotMole (50).

9 all ESTs) and PRF (15%), with less than 0.5% SPF.

10 C56BL/6J SPF and GF mice were placed on custom diets containing d

11 leading computational prediction algorithms: SPF-Cancer and TransFIC.

12 ng GGQ to reach the PTC while still allowing SPF-stop-codon interaction.

13 Although SPF and co-housed mice were comparably susceptible to ac

14 in/+) ;Il10(-/-) mice conventionalized by an SPF microbiota had significantly more colon tumors compa

15 The tissue specimens were HPV typed using an SPF(10) line probe assay HPV detection system.

16 004), grade (P =.004), ploidy (P =.006), and SPF (P =.05) were associated with time to recurrence; th

17 tor, progesterone receptor, patient age, and SPF.

18 onserved amino acid motifs (NIKS in eRF1 and SPF in RF2) and by the conserved tripeptide (GGQ) intera

19 insofar as being identical in germ-free and SPF mice.

20 sely, colonic macrophages from germ-free and SPF-derived colitis-prone Il10(-/-) mice demonstrated ro

21 is was not significantly different in GF and SPF mice, there was a delay in intestinal epithelial rep

22 We compared the results of HC2 and SPF(10) testing of cervical specimens.

23 valence of carcinogenic HPV types by HC2 and SPF(10)-LiPA among women with normal, atypical squamous

24 observed very good agreement between HC2 and SPF(10)-LiPA for carcinogenic HPV type detection.

25 ong positive correlation between mitosin and SPF (r = 0.57; P = 0.0001), and there were significant n

26 d grade were associated with both ploidy and SPF (P </=.01).

27 the genes of two of these proteins (PRF and SPF) are prone to incessant evolution driven by positive

28 Like PRF and SPF, PMF is produced by a multigene family characterized

29 xtreme in these dimensions than both PRF and SPF.

30 Because SPF mice exaggerated humoral and T cell protection upon

31 iable with broad-spectrum sunscreens because SPF with primarily UVB sunscreens is dependent on time o

32 For RF2, the separation between SPF and GGQ elongates from 32 angstroms in the crystal t

33 as 35% (n=1,962) by HC2 and 35% (n=2,003) by SPF(10)-LiPA.

34 Samples were tested for HPV by SPF(10) PCR/DEIA/LiPA(25), version 1.

35 Cereal-SPF rats displayed increased gut CD3(+) and CD8alpha(+)

36 T1D was highest in cereal-SPF (65%) and cereal-GF rats (53%) but inhibited and del

37 +) was highest in HC-GF and lowest in cereal-SPF rats.

38 Clean SPF mice had attenuated responses to gluten compared to

39 inst gluten-induced immunopathology in clean SPF mice was reversed after supplementation with a membe

40 nscreen, the UVA filter, and the combination SPF 15 sunscreen and UVA filter, resulting in increasing

41 nd the side chain of serine of the conserved SPF motif of RF2 recognize U1 and A2 of the stop codon,

42 n two separate experiments; however, control SPF kittens housed with highly bacteremic kittens in the

43 ared to 73.6g glucose/100g starch in control SPF and 65.9 g in SPS noodles.

44 thogens and Proteobacteria, and conventional SPF mice that harbor a complex microbiota that includes

45 luten compared to germ-free and conventional SPF mice.

46 uten-induced immunopathology in conventional SPF mice.

47 t effect, fecal microbiota transfer from DIO SPF mice into germ-free mice also accelerated graft reje

48 Importantly, hyperglycemia in DIO SPF mice could be reduced by the addition of the gut com

49 nation of Sun Protection Factor for DNA (DNA-SPF), using specific DNA repair enzymes and antibodies,

50 -10-deficient mice were maintained in either SPF conditions or germfree conditions or were populated

51 rom mice housed in pathogen free facilities (SPF).

52 actor (PRF), Sodefrin Precursor-Like Factor (SPF), and Plethodontid Modulating Factor (PMF).

53 cterization of Schistosome Paralysis Factor (SPF), a novel tetracyclic alkaloid produced by the rotif

54 y use sunscreens with sun protection factor (SPF) 15.

55 en (UVA superior, UVB sun protection factor (SPF) 50) delayed the onset of UVR-driven melanoma, but o

56 tudies have not taken sun protection factor (SPF) into consideration and used nonusers of sunscreen a

57 benzophenone-3 with a sun protection factor (SPF) of 15, the UVA filter butyl methoxydibenzoylmethane

58 Furthermore, in vitro sun protection factor (SPF) of berry waxes depicted good UV-B absorbing capacit

59 of wild ferns and the sun protection factor (SPF) of the extracts.

60 ompare high- with low-sun protection factor (SPF) sunscreens in relation to sunbathing habits in a la

61 n and is expressed as Sun Protection Factor (SPF).

62 ology [AAD] criteria, sun protection factor [SPF], or vehicle) could be used to predict price per oun

63 eens with comparable sun protection factors (SPFs), but with different levels of UVA protection, espe

64 rats remained diabetes-free, whereas HC-fed SPF rats were less protected (7 vs. 29%).

65 ignment by 1.3-fold for soy protein fibrils (SPF) and 1.2-fold for pea protein fibrils (PPF).

66 cattery cats transmitted B. henselae to five SPF kittens in two separate experiments; however, contro

67 6 at 10%, 15% and 20% in sweet potato flour (SPF) and 5% and 10% in sweet potato starch (SPS) in redu

68 samples such as simulated peritoneal fluid (SPF).

69 sma cells in subcapsular proliferative foci (SPF).

70 with elongation at break reaching 221 % for SPF and 101.62 % for PPF films.

71 nd HPV73 (which is not targeted by HC2), for SPF(10)-LiPA, we defined the carcinogenic HPV types as t

72 : tensile strength increased to 4.88 MPa for SPF and 3.83 MPa for PPF films, with elongation at break

73 54.96% for NUTRIOSE (15%)+GG (1%) fortified SPF noodles and 53.3% for NUTRIOSE (5%)+GG (0.5%) fortif

74 Four SPF H. felis-uninfected cats served as controls.

75 om the SSF into the soymilk pellet fraction (SPF) following incubation with 0.9% PGA for 1h.

76 ctor in breast cancer, and S-phase fraction (SPF), as measured by flow cytometry, is the most clinica

77 low cytometric (ploidy and S-phase fraction [SPF]) and histopathologic analyses (Nottingham Combined

78 They applied a single-pulse-frame (SPF) sequence, which enabled motion insensitive optoacou

79 abnormally hygienic specific pathogen free (SPF) barrier facilities.

80 -free (GF) mice with specific-pathogen free (SPF) mice at weaning (exGF) results in altered intestina

81 ucted between GF and Specific-Pathogen Free (SPF) mouse models of autoimmunity.

82 ricted flora (RF) vs specific pathogen free (SPF).

83 nd the microbiota in specific pathogen-free (SPF) and germ-free (GF) mice given more than 40 unique d

84 Specific pathogen-free (SPF) C57BL/6J or germfree 129S6/SvEv mice were orally in

85 ed, 20 feral, and 30 specific pathogen-free (SPF) cats for pre-existing AAV-binding antibodies agains

86 feces collected from specific-pathogen-free (SPF) chickens experimentally infected with avian HEV wer

87 characterization in specific-pathogen-free (SPF) chickens.

88 ry mice housed under specific pathogen-free (SPF) conditions are the standard model in biomedical res

89 ry mice housed under specific pathogen-free (SPF) conditions are the standard model in biomedical res

90 s animals died under specific pathogen-free (SPF) conditions between 6 and 7 months of age.

91 o, WT mice raised in specific pathogen-free (SPF) conditions fared better against I/R-induced injury

92 C/Tnd mice housed in specific pathogen-free (SPF) conditions induced KLK5 and activated the protease-

93 models are raised in specific pathogen-free (SPF) conditions with relatively uniform microbial commun

94 se when reared under specific-pathogen-free (SPF) conditions, suggesting the involvement of a microbi

95 causes colitis under specific pathogen-free (SPF) conditions.

96 mouse models and in specific pathogen-free (SPF) control mice.

97 colitis when kept in specific-pathogen-free (SPF) environments.

98 ce with conventional specific pathogen-free (SPF) gut microbiota increases both bone formation and re

99 Five specific-pathogen-free (SPF) Helicobacter-free cats were studied before and for

100 otentiate colitis in specific-pathogen-free (SPF) IL-10(-/-) mice.

101 lar to those seen in specific pathogen-free (SPF) IL-2(-/-) mice.

102 biota composition of specific pathogen-free (SPF) INS-GAS mice was quantified by pyrosequencing.

103 rring these fleas to specific-pathogen-free (SPF) kittens housed in a controlled, arthropod-free Univ

104 ings generated using specific pathogen-free (SPF) laboratory mice to humans.

105 Specific Pathogen-Free (SPF) layer chickens were infected with ALV-J or maintain

106 sma and intestine of specific pathogen-free (SPF) Leghorn chickens challenged with Eimeria maxima, wi

107 were not detected in specific pathogen-free (SPF) macaques free of RhCMV and six other viruses; howev

108 ve CD4(+) T cells in specific pathogen-free (SPF) mice are characterized by transcriptional heterogen

109 Specific-pathogen-free (SPF) mice are widely used in biomedical research to mode

110 erm-free mice, clean specific-pathogen-free (SPF) mice colonized with a microbiota devoid of opportun

111 erminal centres from specific-pathogen-free (SPF) mice contain highly dominant 'winner' B cell clones

112 free (GF) and normal specific-pathogen-free (SPF) mice have revealed the impact of host immunosurveil

113 robial experience to specific pathogen-free (SPF) mice induces durable immunological changes that bet

114 onventionally raised specific pathogen-free (SPF) mice treated with azoxymethane (AOM) and dextran su

115 , antibiotic-treated specific-pathogen-free (SPF) mice were given jejunal, cecal, or fecal microbiota

116 ops spontaneously in specific pathogen-free (SPF) mice with a targeted disruption in the IL-10 gene (

117 n antibiotic-treated specific pathogen-free (SPF) mice, and prevented or treated an anaphylactic resp

118 in sharp contrast to specific pathogen-free (SPF) mice, germ-free (GF) mice are resistant to Concanav

119 ared with disease in specific pathogen-free (SPF) mice, ileitis in GF mice is significantly attenuate

120 h-fat diet (HFD)-fed specific-pathogen-free (SPF) mice, supporting a role for the microbiota in promo

121 on alveolar bone in specific-pathogen-free (SPF) mice.

122 were colonized with specific pathogen-free (SPF) microbiota.

123 H pylori-specific pathogen-free (SPF) monkeys were experimentally challenged with wild-ty

124 and exposed them to specific-pathogen-free (SPF) or colorectal cancer-associated bacteria.

125 rats were housed in specific pathogen-free (SPF) or germ-free (GF) conditions and weaned onto diabet

126 , the development of specific-pathogen-free (SPF) oysters has enabled assessment of the infection pro

127 nts, we utilized the specific-pathogen-free (SPF) pig model for HEV and a unique inoculation procedur

128 nstrated in mice and specific pathogen-free (SPF) piglets.

129 ifferent conditions: specific-pathogen-free (SPF) pigs and domestic pigs from the same bred, and indi

130 tious DNA clones, 40 specific-pathogen-free (SPF) pigs were randomly assigned into five groups of eig

131 ely transferred into specific pathogen-free (SPF) RAG-/- mice, but not in germfree RAG-/- mice.

132 ere transferred from specific pathogen-free (SPF) to conventional (non-SPF) animal rooms, and evaluat

133 espiratory tracts of specific-pathogen-free (SPF) young turkeys.

134 iate in conventional specific pathogen-free (SPF), and highest in wild-type mice, even those infected

135 uces stool output in specific pathogen-free (SPF), but not GF mice.

136 g antibiotic-treated specific pathogen-free (SPF), germ-free (GF), and wildling mice.

137 we show in multiple specific pathogen-free (SPF), gnotobiotic, and germ-free murine models of GI GVH

138 s from germ-free and specific pathogen-free (SPF)-derived mice produce IL-10, but not IL-12 p40, when

139 wo generations under specific pathogen-free (SPF, n = 69) or non-SPF conditions (n = 48).

140 ntionally colonized (specific pathogen-free [SPF]) animals and SCFA supplementation to SPF mice even

141 hat stimulation with purified fecal Ags from SPF, but not GF mice leads to the generation of IL-4-sec

142 -10(-/-) mice were colonized with stool from SPF mice that harbored or did not harbor endogenous H. h

143 PF conditions from birth or transferred from SPF conditions at weaning have predominantly ileal tumor

144 is study, these structure-property-function (SPF) and structure-property-hazard (SPH) relationships a

145 of establishing structure-property-function (SPF) and structure-property-hazard (SPH) relationships t

146 rse and richer gut microbiota than did group SPF.

147 e abundant in groups XZ and JD than in group SPF, whereas Firmicutes showed the inverse pattern.

148 ntly lower in groups XZ and JD than in group SPF.

149 Furthermore, transfer of MLN cells from BM-->SPF Tg(epsilon26) mice into SPF Tg(epsilon26) recipients

150 beit in lower frequency than in control BM-->SPF Tg(epsilon26) mice.

151 Also, most samples result in higher SPF than P. aureum's rhizome.

152 How SPF housing conditions alter early-life immune developme

153 In SPF and germfree mice, ETBF caused colitis but was letha

154 says revealed higher beta-sheet alignment in SPF compared to PPF.

155 estored by anti-CD8beta Ab, and augmented in SPF mice bearing the TAP(-/-) genotype.

156 ast size and interface with alveolar bone in SPF mice.

157 ive immunity against intranasal challenge in SPF chickens.

158 TBF induced acute then persistent colitis in SPF mice and rapidly lethal colitis in WT germfree mice.

159 al microbiota and the severity of colitis in SPF mice, but not in GF mice or mice given antibiotics.

160 ose fiber reduced the severity of colitis in SPF mice, whereas methylcellulose increased severity.

161 inant microbiota antigen, induced colitis in SPF RAG-/- mice.

162 bacter hepaticus, induces chronic colitis in SPF-reared IL-10(-/-) mice and that the disease is accom

163 e the SI-Ep under steady-state conditions in SPF mice.

164 MAPK pathway, and neurocognitive deficits in SPF aGVHD mice.

165 ined by rapid and selective pDC depletion in SPF mice transferred with RF CD8(+) T cells.

166 -1 (Erdr1), is induced during development in SPF but not GF or exGF mice and localizes to Lgr5(+) ste

167 umerous impairments in immune development in SPF conditions relative to natural immune development.

168 is article, we compare immune development in SPF-raised mice with mice born from immunologically expe

169 Bacterial communities differed in SPF rats fed cereal compared with HC.

170 alysis showed flexible, worm-like fibrils in SPF and PPF.

171 itis was induced in wild-type mice housed in SPF conditions by infection with Salmonella typhimurium.

172 latform induced robust antitumor immunity in SPF mice, it failed to do so in microbially experienced

173 ignificantly correlated with inflammation in SPF-housed Apc(Min/+) ;Il10(-/-) , but not in Apc(Min/+)

174 IL-10-deficient mice kept in SPF conditions developed colitis in all segments of the

175 eeks, and smaller hypofluorescent lesions in SPF rats at ages 10-16 weeks, especially in the inferior

176 osteoclastogenesis and alveolar bone loss in SPF mice compared with GF mice.

177 ed naturally occurring alveolar bone loss in SPF mice.

178 %) significantly reduced the cooking loss in SPF noodles, this was enhanced in SPS noodles and guar g

179 ut microflora compared to mice maintained in SPF facilities.

180 -binding antibodies were highly prevalent in SPF cats (83%), but this was primarily due to cross-reac

181 as against 33.8% and 40.68%, respectively in SPF and SPS controls.

182 s, Actinobacteria, and Bacteroidetes seen in SPF conditions.

183 indings demonstrate that the colitis seen in SPF IL-2(-/-) mice depends upon the presence of intestin

184 ic cells lining the alveolar bone surface in SPF compared with GF mice.

185 tres is markedly higher in germ-free than in SPF mice, and winner B cells in germ-free germinal centr

186 more and larger tumors compared with that in SPF mice after AOM and DSS treatment despite the lack of

187 rging evidence indicates that variability in SPF microbiota plays a significant role in data inconsis

188 aralleling detection of IAV in H9N2-infected SPF chickens and chickens from LBM showed that pan-IAV F

189 Stomachs of H pylori-infected SPF male mice had significant reductions in Bacteroidete

190 is was inoculated by gastric intubation into SPF C57BL/6 wild-type and p53 hemizygous mice that were

191 cells from BM-->SPF Tg(epsilon26) mice into SPF Tg(epsilon26) recipients induced active colitis, but

192 s from these mice were then transferred into SPF or GF recipients.

193 IELs are reduced, and in lethally irradiated SPF IL-2(+/+) mice, reconstituted with IL-2(-/-) bone ma

194 mulative sunburn with two sunscreens labeled SPF 6, but with different UVR-absorbing properties, one

195 They also show that labeled SPF is much more reliable with broad-spectrum sunscreens

196 Sun Protection Factor for Lethal Damage (LD-SPF), by measuring cell viability and apoptosis induced

197 investigated the effect of high- versus low-SPF sunscreens on cSCC, appropriately adjusting for time

198 Eighty 1-day-old male SPF chickens were randomly allocated to one of four grou

199 o develop in germfree INS-GAS mice than male SPF INS-GAS mice.

200 In colons of germ-free, wild-type mice, SPF microbiota induced production of HO-1 via activation

201 Moreover, SPF WT mice i.p. administered 10 mg/kg MDP were protecte

202 he association between sunscreen use (never, SPF < 15, SPF >/= 15) and melanoma risk by calculating h

203 up of mdx mice and controls (housed in a non-SPF facility) using MRI at 1, 3, 6, 9 and 12 months afte

204 fic pathogen-free (SPF) to conventional (non-SPF) animal rooms, and evaluated clinically and histolog

205 ter species [non-specific-pathogen-free (non-SPF) conditions].

206 In non-SPF macaques, coinfection by RhCMV with other common vir

207 es of memory-phenotype CD4(+) T cells in non-SPF parabionts were similar, generalizing these results

208 specific pathogen-free (SPF, n = 69) or non-SPF conditions (n = 48).

209 The non-SPF rats had significantly higher microbiota alpha diver

210 one-fourth of GPX-DKO mice raised under non-SPF conditions from birth or transferred from SPF condit

211 bone height is significantly less in normal SPF mice compared with their age- and strain-matched GF

212 nsfer of CD4-positive cells from GF, but not SPF mice induces severe colitis in SCID recipients.

213 Application of SPF-15 sunscreens to mouse skin before each UV irradiati

214 We compared the severity and sex bias of SPF, GF, and ex-GF mice and found variability in the sev

215 riations in the composition and diversity of SPF microbiota.

216 helper cells in the junctional epithelium of SPF mice compared with GF mice suggest that the adaptive

217 viruses; however, experimental infection of SPF animals with RhCMV strain UCD59, but not RhCMV strai

218 cells and IL17+ cells in the periodontium of SPF mice demonstrate possible molecular mechanisms media

219 ight promote GIN in achlorhydric stomachs of SPF mice.

220 Conclusion Use of SPF >/= 15 rather than SPF < 15 sunscreens reduces melan

221 Moreover, use of SPF >/= 15 sunscreen by all women age 40 to 75 years cou

222 n attributable fraction) with general use of SPF >/= 15 sunscreens by women age 40 to 75 years was 18

223 Cancer Study, we investigated whether use of SPF >=15 versus SPF <15 sunscreens reduces cSCC risk.

224 the potential limitation of exclusive use of SPF mice when testing vaccine efficacy.

225 ock was determined by inoculating 1-week-old SPF chickens intravenously with 200 microl of each of se

226 In 4-week-old SPF chickens, NDV-Belize-3/08 behaved as a typical velog

227 Neonatal germ-free or SPF mice orally gavaged with live B. infantis exhibited

228 Germ-free or SPF-raised wild-type and Il10(-/-) mice were given intra

229 mice was transplanted into germfree (GF) or SPF Tg(epsilon26) mice.

230 nsal microflora without any known pathogens (SPF), <9% of GPX-DKO mice develop tumors in the ileum or

231 determine the fraction of cells in S phase (SPF).

232 epared by solid-phase pressure forming (PVDF-SPF) show a giant ECE of up to 12.8 K at 25 degrees C an

233 ctions with multiple residues beyond the PXT/SPF motifs.

234 However, in the X-ray structure of RF2, SPF and GGQ are only 23 A apart, indicating that they ca

235 Another conserved motif of RFs (SPF in RF2) has been proposed to interact directly with

236 tions: a specific pathogen-free animal room (SPF), a general animal room (XZ) and a farmhouse (JD).

237 246 and 246X lines had similar SPFs that were approximately twofold greater than contro

238 ot adhere to AAD guidelines (broad spectrum, SPF >/=30, and water resistant) for sunscreens.

239 fared better against I/R-induced injury than SPF Nod2(-/-) mice.

240 y, HC2 was more likely to test positive than SPF(10)-LiPA for the carcinogenic HPV types (87% and 79%

241 Conclusion Use of SPF >/= 15 rather than SPF < 15 sunscreens reduces melanoma risk.

242 The SPF rats had significantly less photoreceptor apoptosis

243 was assessed by eye and objectively, and the SPF of each sunscreen was modeled with changes in solar

244 ys, the Hybrid Capture 2 assay (HC2) and the SPF(10) assay, for the detection of carcinogenic HPV.

245 TSLP also induced scratching behavior in the SPF NC/Tnd mice.

246 itated with the 7S and 11S proteins into the SPF by 0.9% PGA.

247 VA filter butyl methoxydibenzoylmethane, the SPF 15 sunscreen and the UVA filter together, and the lo

248 ripeptide anticodon PVT motif instead of the SPF motif of RF2, which confers the specificity towards

249 ed the highest microbial diversity while the SPF pigs the lowest.

250 henicol acetyl transferase activity with the SPF 15 sunscreen, the UVA filter, and the combination SP

251 he factor-binding site of the ribosome, the 'SPF' loop of the protein is situated close to the mRNA,

252 d development of eczematous lesions in these SPF NC/Tnd mice, which normally do not suffer from AD.

253 g niche competitor (i.e., wildtype B. theta, SPF microbiota) rapidly excludes the acapsular strain du

254 In striking contrast to SPF IL-2(-/-) mice, germfree IL-2(-/-) mice do not devel

255 mice without colitis transferred disease to SPF Tg(epsilon26) recipients within 2-4 weeks.

256 /-) mice before transition from germ-free to SPF conditions reduced their development of colitis.

257 itosin as assessed by IHC may be superior to SPF as a prognostic factor in node-negative breast cance

258 e [SPF]) animals and SCFA supplementation to SPF mice even further exacerbated plaque load.

259 itions, and we subjected minocycline-treated SPF mice to chlorhexidine oral antiseptic rinses.

260 ed alveolar bone loss in minocycline-treated SPF mice, validating that antimicrobial-induced oral dys

261 cervical lymph nodes of minocycline-treated SPF mice.

262 lar bone loss was greater in vehicle-treated SPF versus germ-free mice, demonstrating that the commen

263 or vehicle-control to male mice reared under SPF and germ-free conditions, and we subjected minocycli

264 ection in Men Study were HPV genotyped using SPF(10) PCR-DEIA-LiPA(25).

265 Genotyping for 25 HPVs was performed using SPF(10)/LiPA(25).

266 investigated whether use of SPF >=15 versus SPF <15 sunscreens reduces cSCC risk.

267 cSCC risk of sunscreens with SPF >=15 versus SPF <15 was close to the null when used at any latitudes

268 In addition, we show that GF versus SPF mice have reduced intestinal absorption and increase

269 After 1 week, SPF and GF mice were given dextran sulfate sodium (DSS)

270 At the age of four weeks, SPF rats had significantly shorter dark-adapted a-wave a

271 , we characterize a transmission model where SPF mice are exposed to natural mouse pathogens at physi

272 study, we used a "dirty" mouse model, where SPF laboratory mice were cohoused (CoH) with pet store m

273 han under the laboratory conditions in which SPF is tested and assigned.

274 cantly decreased melanoma risk compared with SPF < 15 use (hazard ratio, 0.67; 95% CI, 0.53 to 0.83).

275 tigens in ConA-treated GF mice compared with SPF counterparts.

276 ice were significantly reduced compared with SPF mice after vaccination.

277 enteric lymph nodes of GF mice compared with SPF mice, as well as lower relative gene expression of F

278 we found no indication that sunscreens with SPF >=15 reduced Norwegian women's cSCC risk more than s

279 The effect on cSCC risk of sunscreens with SPF >=15 versus SPF <15 was close to the null when used

280 women's cSCC risk more than sunscreens with SPF <15, suggesting that either there is no difference i