ライフサイエンスコーパス: SSR

1 SSR alleles varied within populations, but recombination

2 SSR analysis allowed the detection of 157 alleles across

3 SSR dose-related suppression of the nickel CHS response

4 SSR growth requires Ral and the exocyst component Sec5 a

5 SSR had the same effect as cis-UCA, but trans-UCA had no

6 SSR loci also exhibit the highest percentage of 'extende

7 SSR loci are least conserved with 12.71 polymorphic base

8 SSR markers and Real Time qPCR analysis showed that diff

9 SSR processes provide a new alternative to understand th

10 SSR-based cluster analysis revealed that varieties with

11 SSRs are found in eukaryotes as well as prokaryotes, and

12 SSRs are subject to slipped-strand mutations and a commo

13 SSRs, on the other hand, are found at low frequency dist

14 The population was genotyped using 2,000 SSR and DArT markers.

15 mer pairs were designed for more than 60,000 SSR-containing sequences based on alignment against asse

16 The accuracies using 135 SSRs were low, with accuracies of the traits around 0.20

17 A total of 15 SSR amplicons unique to P. philippinensis (one of the po

18 2 molecular markers (538 Bin, 258 AFLPs, 175 SSRs, and an EST).

19 The difference was greatest at 3.8 J/cm(2) SSR [mean difference: 11% (95% CI: 0.5%, 21.4%)].

20 agonists such as nicotine, choline, GTS-21, SSR-180711A and PNU-282987 in the presence of the positi

21 enerated in this study was screened using 22 SSR markers located on linkage group 2 which contains a

22 In addition, 23 SSR loci were identified.

23 Among them, a sub set of primers (245 SSR) was validated using polymerase chain reaction (PCR)

24 recting for the multiple tests performed, 25 SSR loci showed evidence of selection.

25 optimal extraction conditions 60% SC and 25% SSR resulted in the highest concentration of anthocyanin

26 We identified and characterized 2749 SSRs of 963 SSR types in the genome of N. crassa.

27 rent study, we simultaneously genotyped 3105 SSRs in eight rice varieties, which were further validat

28 ingerprints of the rice varieties using 3105 SSRs, which offer much greater discriminative power than

29 A set of 400 SSR primer combinations were tested for amplification an

30 uisiana (commercials, wild/exotic) using 423 SSR alleles showed an average gene diversity (h) at 0.20

31 constructed using 2,467 SLAF markers and 43 SSRs, which were distributed on seven linkage groups spa

32 Among 2,431 SSR markers located across the whole peanut genome scree

33 uch greater discriminative power than the 48 SSRs commonly used for rice.

34 a full-sib family, combining 2719 SNP and 84 SSR segregating markers among the parental maps.

35 A genetic linkage map composed of 841 SSR, AFLP, and RAPD markers and phenotypic data from 310

36 We identified 12,956 SSRs, including 2,594 repeats for which primers were des

37 dentified and characterized 2749 SSRs of 963 SSR types in the genome of N. crassa.

38 ntial have been reported, we now show that a SSR also impacts small RNA-mediated posttranscriptional

39 netics and forensics, which require accurate SSRs to calculate intra- and inter-species interactions.

40 tinic acetylcholine receptor (nAChR) agonist SSR 180711 (3.0 mg/kg) eliminated the TTX-induced perfor

41 We present Kmer-SSR, which finds all SSRs faster than most heuristic SSR identification algor

42 Although SSRs are common and highly dispersed in the genome, thei

43 AmpSeq-SSR is able to genotype potentially more than a million

44 l method for SSR genotyping, named as AmpSeq-SSR, which combines multiplexing polymerase chain reacti

45 results showed that the accuracies of AmpSeq-SSR were nearly 100 and 94% with a single base resolutio

46 To demonstrate the power of AmpSeq-SSR, we adopted it in two applications.

47 the work we present focused on rice, AmpSeq-SSR can be readily extended to animals and micro-organis

48 Again, 50% had an LLR, but none had an SSR.

49 sing a full-sib population of 238 plants and SSR and STS markers to access the degree of preferential

50 The consensus SNP and SSR map contains a total of 1201 markers in 23 linkage g

51 h that combines Phenotype MicroArray(TM) and SSR genotyping appeared useful to assess the performance

52 netic map comprising over 600 SFPs, GEMs and SSRs.

53 etween asymptomatic sensitization, LLRs, and SSRs.

54 Because SSRs are commonly located in promoters, untranslated reg

55 From 40 BES-SSRs based on long motifs SSRs (>/= trinucleotides) anal

56 In total, 3,789 BES-SSRs were identified, with a distribution of one SSR (si

57 This study identified highly polymorphic BES-SSRs containing tri- to hexanucleotides motifs and bring

58 atistically significant associations between SSRs and gene functional classifications were detected,

59 hereditary neurodegenerative disorders, but SSR alleles can also contribute to normal variation in b

60 h and the percentage of immunosuppression by SSR was calculated.

61 thousands of transgenic insertions carrying SSR recognition sites have been distributed throughout t

62 en used successfully to identify a causative SSR variation that perfectly segregates with a repeat ex

63 We thoroughly characterize SSR density and variability as a function of N-->C posit

64 and identified section-specific chloroplast SSR motifs.

65 In contrast, SSRs are rare in aHC.

66 In contrast, SSRs occur ~350 ms later, with a strong preference for c

67 We propose that seven core SSR primer pairs, namely, SMC31CUQ, SMC336BS, SMC597CS,

68 ile being the most comprehensive and correct SSR detection software available.

69 Public cotton SSR libraries (17,343 markers) were curated for sequence

70 The absorption enhancement of CVDL (SSR = 5 relative to SSR = 1) was higher across mucus-pro

71 rocess that generalises the recently defined SSR processes and is able to produce power laws with arb

72 ttempted to develop, characterize and design SSR (simple sequence repeat) markers using online geneti

73 mmon prefix arrays for efficiently detecting SSRs in large sets of sequences.

74 is the first attempt to extensively develop SSR markers from Peronosclerospora genomic DNA.

75 between GC content and the span of (AG)n-Di-SSR variation.

76 tology (GO) terms with abundance of (AG)3-Di-SSRs shared by the VP strains were associated with purin

77 he numbers of triplexes formed from (AG)n-Di-SSRs between VP and N_VP in Mycoplasma suggested the pot

78 sma suggested the potential role of (AG)n-Di-SSRs in gene regulation.

79 Di-amino acids coded by (AG)n-Di-SSRs included all three six-fold code amino acids (Arg,

80 of (AG)n dimeric simple sequence repeats (Di-SSRs) in the genomes, coding and non-coding regions than

81 d Syndapin colocalize with Past1 in distinct SSR subdomains and collapse into Amphiphysin-dependent m

82 number of different bands detected for each SSR primer pair using a LI-COR- DNA Analyzer ranged from

83 and polymorphism information content of each SSR locus was calculated with an adapted formula taking

84 ng fruiting body development and develop EST-SSR markers assessing the genetic value of breeding mate

85 developed 26 polymorphic and informative EST-SSR markers to assess the genetic diversity in 82 strain

86 variation (no more than two variants per EST-SSR genotype) and recently published chromosomal evidenc

87 sed-sequence tag simple-sequence repeat (EST-SSR)) primers to survey genetic variation in populations

88 These EST-SSRs exhibited high transferability in closely related s

89 100% recall and accurately identifies every SSR of any specified length.

90 determinants of several natural and evolved SSRs including Cre, evolved variants of Cre, and other S

91 Existing SSR detection applications are hampered by one or more l

92 steomyelitis and cystic fibrosis carry fewer SSRs than do isolates from patients with acute infection

93 7-2018 pricing data from the investment firm SSR Health for branded products available before January

94 . trichocarpa genome assembly using flanking SSR markers with known physical positions.

95 c situations and it should be considered for SSR computation.

96 etic marker, we developed a novel method for SSR genotyping, named as AmpSeq-SSR, which combines mult

97 However, the existing methods for SSR genotyping suffer from low sensitivity, low accuracy

98 ysis demonstrates that miR-8 is required for SSR morphogenesis.

99 lated, positively for TEs and negatively for SSRs, with relative nucleosome binding affinities along

100 a 9.5-fold higher risk for LLRs but not for SSRs.

101 ng specificity determinants not evident from SSR:DNA structures.

102 We have used the FLP/FRT SSR system to provide a proof of concept, though any of

103 These findings provide new genes, SSR markers, and germplasm to enhance the breeding of co

104 as a dataset of the recently published genic SSRs (eSSRs) generated on the same germplasm.

105 Genic-SSR's coupled with the functional annotations provided b

106 Of the 80 genic-SSR primer sets, 62 were amplified in C. ensifolium succ

107 The 7,936 genic-SSR markers were identified.

108 were analyzed based on 55 polymorphic genic-SSR data among 59 accessions.

109 nce repeats derived from gene regions (genic-SSR).

110 The genic-SSR identified in this study constitute a set of markers

111 Unigenes containing the 62 genic-SSRs were searched against Non-redundant (Nr), Gene Onto

112 A total of 80 genic-SSRs were selected, and primers were designed according

113 Only 5 (5.3%) patients had SSRs, but 41 (43.6%) had large local reactions (LLRs) af

114 l homozygosity underlying fixed heterozygote SSR genotypes, which in turn reflect a selfing mating sy

115 ch finds all SSRs faster than most heuristic SSR identification algorithms in a parallelized, easy-to

116 We discuss several applications showing how SSR processes can be used to understand Zipf's law in wo

117 tions, accurately and intuitively identifies SSRs quickly and in a more user-friendly manner than any

118 we show that imposing energy conservation in SSR cascades allows us to recover Fermi's classic result

119 olerance were performed with 267 informative SSR markers in this study.

120 Kmer-SSR, coupled with the filter options, accurately and int

121 The exhaustive Kmer-SSR option has 100% precision and 100% recall and accura

122 We present Kmer-SSR, which finds all SSRs faster than most heuristic SSR

123 eta11 (imp-beta11) and dfz2 mutants had less SSR, and some boutons lacked the postsynaptic marker Dis

124 Long SSRs in Mycobacterium leprae are mostly associated with

125 erally rare in prokaryotic genomes, and long SSRs composed of iterations of mono-, di-, tri-, and tet

126 ons were detected, suggesting that most long SSRs are not related to a particular cellular function o

127 Significantly long SSRs are generally rare in prokaryotic genomes, and long

128 A lower SSR using CE was defined as the best scenario for using

129 th greater effect than the RE leads to lower SSR using the CE regardless of the AE proportion and the

130 or their placement with independently mapped SSRs, demonstrating the previously un-utilized power tha

131 nomic- and EST-based microsatellite markers (SSRs) from previous de novo assemblies (Staton et al., P

132 integration of SNP data and microsatellite (SSR) data resulted in a final map comprising 17 LGs with

133 genotypes and an established microsatellite (SSR) framework map, we produced a high-density genetic m

134 s of nuclear and organellar microsatellites (SSRs) for most Juglans genomes have not been previously

135 to genotype potentially more than a million SSRs at once using the current sequencing techniques.

136 SSR is 100% accurate and detected >1000 more SSRs than the second best algorithm, while offering grea

137 From 40 BES-SSRs based on long motifs SSRs (>/= trinucleotides) analyzed in high-resolution ge

138 sporter 1 (GlyT1) inhibitors including NFPS, SSR 504734, Lu AA21279, Org 25935, SB-710622, GSK931145,

139 Noisy SSR processes further allow us to explain a wide range o

140 We compared the frequency of nuclear SSR motifs and their lengths across Juglans, and identif

141 The characterization of SSR length by standard sequencing methods is complicated

142 in a higher proportion than other classes of SSR-motifs.

143 Moreover, comparison of SSR-based data with phenylpropanoid molecules exhibited

144 The density of SSR in the walnut genome analyzed was also slightly high

145 Given the high density of SSR recognition-site insertions in Drosophila, our metho

146 nt immunity, revealing the complex nature of SSR resistance.

147 The peak of SSR density is centered around the -35 bp position where

148 to account for miR-8-mediated regulation of SSR architecture, consistent with its localization in th

149 ective of this study was to develop a set of SSR markers useful for genetic studies in sainfoin and t

150 Specificity of SSR markers was verified using controls consisting of: i

151 ence repeats (SSRs) constitutes the basis of SSRs as an effective genetic marker with various applica

152 d 2,000 were suitable for the development of SSRs, of which 194 were evaluated in low-resolution scre

153 tive variability and density distribution of SSRs in coding regions.

154 enic bacteria, we observe over enrichment of SSRs near protein N-termini significantly beyond expecta

155 Although several examples of SSRs that affect transcription or coding potential have

156 which is expected if the primary function of SSRs is their contribution to antigenic variation.

157 o study evolution and biological function of SSRs.

158 gnaling in eradication of S. aureus, loss of SSRs may represent an advantageous mechanism to adapt to

159 an inverse correlation between the number of SSRs and the length of disease course.

160 and immune cells, depending on the number of SSRs, which leads to differences in neutrophil recruitme

161 In order to fully exploit the potential of SSRs as genetic marker, we developed a novel method for

162 o Hymenoptera venoms was common, the risk of SSRs in sensitized subjects was low in our study.

163 that allow users to easily view a subset of SSRs based on user input.

164 nants and potential off-target substrates of SSRs in a comprehensive and unbiased manner.

165 was to adapt a genotyping approach based on SSR (Simple Sequence Repeat) marker to a discriminating

166 bly amplify the aerosol extinction effect on SSR.

167 n and wind stilling amplification effects on SSR variations at a decadal scale.

168 equence, we assess evolutionary pressures on SSRs.

169 were identified, with a distribution of one SSR (simple sequence repeat) per 8.36 kbp and 2,000 were

170 ploys 63 de novo developed microsatellite or SSR (Single Sequence Repeat) markers in different datase

171 mprehensive report of nuclear and organellar SSRs in Juglans and the generation of validated SSR prim

172 ding Cre, evolved variants of Cre, and other SSR family members.

173 n a more user-friendly manner than any other SSR identification algorithm.

174 roof of concept, though any of several other SSR systems could be used in the same way.

175 To identify more biologically pertinent SSRs, we also developed several filters that allow users

176 distinctive physiological properties of pHC-SSR enable an alternative mechanism for hippocampal enga

177 ngly phase-locked with parietal-SSs, and pHC-SSRs were phase-coupled with pHC-SSs and parietal-SSs.

178 of fluorescence-labeled highly poloymorphic SSR primers and a capillary electrophoresis (CE) detecti

179 A total of 167 polymorphic SSR alleles were identified by CE with a mean value of p

180 genetic diversity analysis using polymorphic SSRs and found to separate into seven groups with 0.73 t

181 Using polymorphic SSRs, we have built linkage maps from three line-cross p

182 of primer designs including generic primers, SSR primers together with SSR detection, and SNP genotyp

183 In prokaryotes, SSR loci can cause phase variation, a microbial survival

184 also a module available for finding putative SSRs in the analysed datasets.

185 or 7.6 J/cm(2) of solar-simulated radiation (SSR) and to 3 unexposed control sites.

186 It was found that surface solar radiation (SSR) decreased considerably with wind stilling in heavil

187 (8-13 Hz) is one such instance: alpha-range SSRs have typically been found to increase in power when

188 The survival rate (SSR) and success rate (SR) were calculated using life-ta

189 concentration (SC) and solid solvent ratio (SSR) had a significant effect on the anthocyanin content

190 vent-shift method at supersaturation ratios (SSR) of 5 and 6, respectively.

191 at risk for severe systemic sting reactions (SSR), and conversely, patients with weakly positive or e

192 tera venom without systemic sting reactions (SSRs) is commonly observed in the general population.

193 15-crown-5 ether, a selective shift reagent (SSR), was added after the last chromatographic dimension

194 Site-specific recombinases (SSRs) are valuable tools for manipulating genomes.

195 e development of site-specific recombinases (SSRs) as genome editing agents is limited by the difficu

196 scribe a simple site-specific recombination (SSR) strategy that simultaneously removes pre-defined DN

197 Sample Space Reducing (SSR) processes are simple stochastic processes that offe

198 emonstrate that these sample-space-reducing (SSR) processes necessarily lead to Zipf's law in the ran

199 A total of 3607 non-redundant SSR primer pairs was designed based on the sequences of

200 (referred as to single surface referencing (SSR)) uses a single functionalized surface split into th

201 sin gene (RH1) and 9 microsatellite regions (SSRs) were amplified and analysed.

202 ive model (SGAM), Spike and slab regression (SSR), Stochastic gradient boosting (SGB), support vector

203 Simple sequence repeat (SSR) fingerprinting revealed that increased frequencies

204 n many forest trees (simple sequence repeat (SSR) FST=0.21), with major genetic groups corresponding

205 DNA, genomic DNA and simple sequence repeat (SSR) loci were re-sequenced in Gossypium herbaceum (A1 g

206 were genotyped at 25 simple-sequence repeat (SSR) loci.

207 andraces assessed by simple sequence repeat (SSR) markers and allelic variation at the GmTfl1 locus s

208 er analysis with 151 simple sequence repeat (SSR) markers in 90 individuals of the population indicat

209 iously characterised Simple Sequence Repeat (SSR) markers to estimate genetic differentiation across

210 and 6.05 between the simple sequence repeat (SSR) markers umc1165-bnlg1017, umc1065-umc1637, and nc01

211 639033]), developing simple sequence repeat (SSR) markers, and identifying single nucleotide polymorp

212 but also captured a simple sequence repeat (SSR) variation associated with Machado-Joseph disease.

213 ed on a whole-genome simple sequence repeat (SSR)-based genetic map and on mapping sex determination

214 surface antigens by simple sequence repeat (SSR)-mediated phase variation.

215 Microsatellite (simple sequence repeat - SSR) and single nucleotide polymorphism (SNP) markers ar

216 Simple sequence repeats (SSR) from BESs were analyzed and found to be more abunda

217 ait loci (QTLs) and simple sequence repeats (SSR) markers linked to three QTLs for PM resistance.

218 orphisms (AFLP) and simple sequence repeats (SSR)] we have generated genetic linkage maps for Physcom

219 rosatellite loci or simple sequence repeats (SSRs) and single nucleotide polymorphisms (SNPs).

220 tems evaluated were simple sequence repeats (SSRs) and single nucleotide polymorphisms (SNPs).

221 Simple sequence repeats (SSRs) are indel mutational hotspots in genomes.

222 Simple Sequence Repeats (SSRs) are used to address a variety of research question

223 cient genotyping of simple sequence repeats (SSRs) constitutes the basis of SSRs as an effective gene

224 ion of hypermutable simple sequence repeats (SSRs) is a widespread and stochastic mechanism to genera

225 ct, by carrying out simple sequence repeats (SSRs) markers analysis, we characterise the genetic prof

226 hether the number of short sequence repeats (SSRs) present in the polymorphic region modulates the in

227 ts of genome called simple sequence repeats (SSRs) that consist of multiple copies of a short sequenc

228 The simple sequence repeats (SSRs) they bind can also adopt alternative DNA structure

229 n instances such as Simple Sequence Repeats (SSRs), a 'good enough' solution may not accurately portr

230 informative marker simple sequence repeats (SSRs), as long as a sufficient number of loci are employ

231 .5% of BESs contain simple sequence repeats (SSRs), most of which are AT/TA dimer repeats.

232 etrotransposons and simple sequence repeats (SSRs).

233 elements (TEs) and simple sequence repeats (SSRs).

234 I) were scanned for Simple Sequence Repeats (SSRs); unique SSRs were then tested for amplification an

235 33 microsatellites (simple sequence repeats, SSRs) per Mbp in the enset genome, representing 0.28% of

236 a binary CE on the sample size requirement (SSR) has not been addressed.

237 ss, referred to as the Sumo stress response (SSR).

238 [classically termed steady-state responses (SSRs)] that add to the ongoing brain activity.

239 f the subsynaptic muscle membrane reticulum (SSR) at the Drosophila melanogaster larval neuromuscular

240 functions within the subsynaptic reticulum (SSR) surrounding presynaptic terminals.

241 ic development of the subsynaptic reticulum (SSR), an elaboration of the postsynaptic plasma membrane

242 plasma membrane (the subsynaptic reticulum, SSR).

243 rates approximately as many spindle ripples (SSR: ripples phase-locked to local spindles).

244 issect the genetics of Sclerotinia stem rot (SSR) [caused by Sclerotinia sclerotiorum (Lib.) de Bary]

245 SA-SSR is 100% accurate and detected >1000 more SSRs than t

246 Here, we present an innovative algorithm, SA-SSR, based on suffix and longest common prefix arrays fo

247 Our data on large scale SSR based genetic diversity and population structure ana

248 or even negative tests can experience severe SSR.

249 ommended in patients with moderate to severe SSR and in patients with SSR confined to generalized ski

250 rich repeats near termini, yielding U-shaped SSR density curves.

251 ndicate that each stress triggers a specific SSR signature centered on proteins involved in transcrip

252 he integrated linkage map with gene-specific SSR markers coupled with computational prediction of AFL

253 lar type of stress induces a stress-specific SSR that targets a unique, largely nonoverlapping set of

254 constitutes the core of all stress-specific SSRs in yeast.

255 ikingly, whereas the various stress-specific SSRs were largely nonoverlapping, all types of stress te

256 Within each stratum, SSR was computed for multiple scenarios by varying the e

257 In the case of the studied SSRs, the results allowed the initial characterisation a

258 s on average, whereas ripples on successive SSR peaks are separated by only ~80 ms.

259 ped to linkage group (LG) 3 of the sunflower SSR map.

260 Like SWRs, SSRs often co-occur with neocortical theta bursts (TBs),

261 recting the choice of the most appropriate T-SSR system for genetic engineering include that, wheneve

262 Tyrosine-type site-specific recombinases (T-SSRs) have opened new avenues for the predictable modifi

263 in our mechanistic understanding of simple T-SSRs and their application in developmental and syntheti

264 ength variants at individual tetranucleotide SSR loci.

265 This finding supports a hypothesis that SSR polymorphism has functional implications.

266 The SSR markers characterized and tested in this study provi

267 the relative overlap, can help to define the SSR in specific situations and it should be considered f

268 hods were successfully applied to design the SSR and to support the experimental data.

269 mphiphysin-dependent membrane nodules in the SSR of past1 mutants.

270 Across species, sequence identity in the SSR-flanking regions was generally low.

271 Integrating the SSR- and AFLP-based maps generated 31 linkage groups com

272 the elaboration of increasing layers of the SSR and uncover new roles for an EHD protein at synapses

273 ectly corresponds to the mixing ratio of the SSR process and noise.

274 Based on the SSR data, the 115 accessions were classified into seven

275 However, it remains unclear whether the SSR involves a common set of core proteins regardless of

276 The SSRs are robust (with basic PCR methods and agarose gel

277 during persistent carriage by analyzing the SSRs of eight loci in multiple isolates from 21 carriers

278 We used the GMATo program for the SSRs search, and the Bowtie2 and UGENE programs for the

279 From this SSR dependence on wind speed, we further derived proxies

280 Those SSRs capable of switching conformation under physiologic

281 to determine their genetic diversity through SSR markers.

282 We show that by adding noise to SSR processes the corresponding rank distributions remai

283 ion enhancement of CVDL (SSR = 5 relative to SSR = 1) was higher across mucus-producing than non-mucu

284 -mediated site-specific recombination" (tRNA-SSR) for this mechanism.

285 g that these species may evolutionarily tune SSR positions in coding regions to facilitate phase vari

286 large genomic segment that lies between two SSR recognition-site insertions can be "captured" as a t

287 d to the distal part of chr.3 flanked by two SSR markers within an interval of 1.8 cM corresponding t

288 arated by approximately 6 cM, along with two SSR markers.

289 hmic visual stimulation, we find the typical SSR gain effect when emphasizing stimulus-locked neural

290 thereof revealed previously uncharacterized SSR interactions, including specificity determinants not

291 d for Simple Sequence Repeats (SSRs); unique SSRs were then tested for amplification and polymorphism

292 Second, using SSRs and putatively neutral sequenced loci, coalescent s

293 s in Juglans and the generation of validated SSR primers will be a useful resource for future genetic

294 upstream of the TSS at the same point where SSR frequency is at its highest.

295 icant epistatic interactions associated with SSR resistance, with candidate genes involved in a wide

296 moderate to severe SSR and in patients with SSR confined to generalized skin symptoms if quality of

297 g generic primers, SSR primers together with SSR detection, and SNP genotyping primers (including sin

298 , at least some of the genes associated with SSRs encode potential antigens, which is expected if the

299 ulation might also apply to other genes with SSRs, which could be targeting sites of cis- or trans-en

300 rresponding SR of 93.9%, whereas the 16-year SSR and SR for the R implants were 97.2% and 95.2%, resp

301 The M implants had a 16-year SSR of 95.7% and a corresponding SR of 93.9%, whereas th