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1 STEMI and the lack of coronary revascularization determi
2 STEMI-SCAD represents an important STEMI subset, particu
3 bility, NSTEMI: 19.7% versus 11.4%, P<0.001; STEMI: 14.8% versus 6.4%, P<0.001; impaired functional m
4 irment, NSTEMI: 20.6% versus 14.3%, P<0.001; STEMI: 20.6% versus 12.4%, P=0.001; activities of daily
6 than men (NSTEMI: 82.1 versus 81.3, P<0.001; STEMI: 82.2 versus 80.6, P<0.001) and had lower rates of
8 structive coronary disease (NSTEMI: P<0.001; STEMI: P=0.02), driven by lower rates of 3-vessel or lef
12 ospital mortality, achievement of at least 2 STEMI care metrics was associated with significantly red
16 ased strategies at our hospital to improve 3 STEMI care metrics: (1) prompt guideline-directed medica
17 centage with achievement of zero, 1, 2, or 3 STEMI care metrics was 7.1%, 24.1%, 43.8%, and 25.1%; an
18 Methods and Results We identified 169 505 STEMI patients in the Chest Pain-Myocardial Infarction R
21 tients with acute myocardial infarction (59% STEMI) admitted to cardiac intensive care units in metro
22 disease than men (STEMI: 38.8% versus 58.7%; STEMI: 24.3% versus 32.1%), and underwent revascularizat
24 del used to combine the gene expression in a STEMI vs healthy donors score showed an AUC of 0.95.
26 pt study, we defined two groups (1) an acute STEMI group (n = 6, 83% male, age 54 +/- 12 years) compl
29 o presented between 2009 and 2014 with acute STEMI were combined with population data to generate inc
30 aterials and Methods Participants with acute STEMI were prospectively enrolled from May 11, 2011, to
31 rst overall survival (log-rank P=0.04) after STEMI during a median follow-up of 5.2 (3.6, 6.9) years
33 eling and more impaired LV deformation after STEMI compared with those with normal BMI, amid similar
35 scharge, women developed heart failure after STEMI (women, 22.5%, versus men, 14.9%) as well as after
36 risk to develop de novo heart failure after STEMI and women with de novo heart failure have worse su
40 te heart failure, and related outcomes after STEMI in patients with no prior history of heart failure
43 e nonculprit artery in the early phase after STEMI and determine the real prevalence of microvascular
46 eart failure in the subsequent 5 years after STEMI or NSTEMI, even after accounting for differences i
47 ective multicenter cardiac MRI studies (AIDA STEMI [NCT00712101] and TATORT NSTEMI [NCT01612312]) inc
48 des infarctus du myocarde) that enrolls all STEMI managed by EMS in the Greater Paris Area, includin
53 eated within target time windows for AIS and STEMI (median DTN time <60 minutes: 21% [interquartile r
57 trial, we assigned 50 patients with anterior STEMI to LV unloading by using the Impella CP followed b
60 s of acute coronary syndrome, including both STEMI and NSTEMI, but relative and absolute reductions w
61 ber of PCI procedures for patients with both STEMI (438 PCI procedures per week in 2019 vs 346 by the
62 iated cytokines were largely not affected by STEMI, except for pro-inflammatory cytokines IL-6, IL-18
63 of cytokine clusters affected differently by STEMI now permits investigation of their differential co
64 isson distribution were calculated comparing STEMI rates between smokers and nonsmokers stratified by
65 consecutive patients admitted with confirmed STEMI treated with primary percutaneous coronary interve
67 model may serve as an example for developing STEMI systems of care in other low- to middle-income cou
76 r SCA were age, heart failure, and extensive STEMI, while male sex and cardiovascular risk factors we
77 actors, symptoms of heart failure, extensive STEMI, and short pain onset-to-call and call-to-EMS arri
82 ional Inpatient Sample from 2012 to 2015 for STEMI hospitalizations with Medicaid or private insuranc
85 a historical cohort of patients admitted for STEMI during the analogous time period (February 21 to A
86 A total of 26 patients were admitted for STEMI during the study period, and 7 (26.9%) of these pa
88 primary PCI remains the standard of care for STEMI patients at PCI capable hospitals when it can be p
92 eat Multi-vessel Disease After Early PCI for STEMI) trial demonstrated that staged nonculprit lesion
93 eat Multi-vessel Disease After Early PCI for STEMI) trial, angiography-guided percutaneous coronary i
94 eat Multi-Vessel Disease After Early PCI for STEMI), we performed optical coherence tomography of at
95 tients who underwent successful stenting for STEMI and had left ventricular dysfunction (ejection fra
96 the largest study of cell-based therapy for STEMI completed in the United States and provides eviden
97 erformance on door-to-balloon (D2B) time for STEMI and door-to-needle (DTN) time for AIS, with and wi
98 OVID-19 and low likelihood of mortality from STEMI and use of preventive strategies such as preproced
100 ofile highlights five genes able to identify STEMI patients and to discriminate them in the backgroun
106 risk of death during hospitalization but in STEMI patients a lower HDL-C was paradoxically associate
108 d organizations involved with improvement in STEMI care in LMICs, it also provides some specific targ
109 ted with a significantly greater increase in STEMI rate for women than men (IRR: 6.62; 95% confidence
112 ent, this difference remained significant in STEMI (adjusted odds ratio, 1.42 [95% CI, 1.24-1.64]) bu
115 pproved indication of reperfusion therapy in STEMI (streptokinase, tenecteplase, alteplase, and retep
116 The DTU-STEMI pilot trial (Door-To-Unload in STEMI Pilot Trial) represents the first exploratory stud
122 ST-segment elevation myocardial infarction (STEMI) and multivessel coronary artery disease (CAD).
123 ST-segment elevation myocardial infarction (STEMI) and non-ST-segment elevation myocardial infarctio
124 ST-segment elevation myocardial infarction (STEMI) care are formidable in low- to middle-income coun
125 ST-segment-elevation myocardial infarction (STEMI) care have traditionally focused on improving door
126 ST-segment elevation myocardial infarction (STEMI) caused by type 1 myocardial infarction in patient
128 ST-segment elevation myocardial infarction (STEMI) complicated by symptoms of acute de novo heart fa
129 ST-segment-elevation myocardial infarction (STEMI) decreased drastically, mainly through reduction i
130 ST-segment elevation myocardial infarction (STEMI) from accessing the emergency system, with subsequ
132 ST-segment-elevation myocardial infarction (STEMI) has been widely used; however, recent trials have
135 ST-segment-elevation myocardial infarction (STEMI) in context of the coronavirus disease 2019 (COVID
136 ST-segment-elevation myocardial infarction (STEMI) in particular is increasing at an unprecedented r
137 ST-segment elevation myocardial infarction (STEMI) in settings where health-care resources are scarc
138 ST-segment elevation myocardial infarction (STEMI) is the most acute manifestation of coronary arter
139 ST-segment elevation myocardial infarction (STEMI) may not be uniform over time, which may affect th
140 ST-segment elevation myocardial infarction (STEMI) or non-STEMI (NSTEMI) who were undergoing PCI and
141 ST-segment elevation myocardial infarction (STEMI) patients treated by primary percutaneous coronary
142 ating an ST-elevation myocardial infarction (STEMI) presentation, stress cardiomyopathy, non-ischemic
143 ST-segment elevation myocardial infarction (STEMI) relates to smaller infarct size and preserved lon
146 -ST-segment-elevation myocardial infarction (STEMI) type 2 (31.9%), non-STEMI type 1 (31.5%), unstabl
148 nts with ST-elevation myocardial infarction (STEMI) undergoing primary percutaneous coronary interven
149 ST-segment-elevation myocardial infarction (STEMI) victims remain at risk for infarct expansion, hea
150 ST-segment elevation myocardial infarction (STEMI), a gene-by-gene analysis of the platelet gene exp
151 ST-segment-elevation myocardial infarction (STEMI), infarct size correlates directly with heart fail
152 ified as ST-elevation myocardial infarction (STEMI), non-STEMI (NSTEMI), myocardial infarction of unk
153 ST-segment elevation myocardial infarction (STEMI), percutaneous coronary intervention (PCI) of the
154 ST-segment elevation myocardial infarction (STEMI), the use of percutaneous coronary intervention (P
155 ST-segment-elevation myocardial infarction (STEMI), though use in recent practice is not well descri
156 ST segment elevation myocardial infarction (STEMI), with positive cardiac biomarkers and either isch
168 ST-segment elevation myocardial infarction (STEMI); however, once STEMI occurs, smoking has been ass
169 (ST-segment-elevation myocardial infarction [STEMI] and non-STEMI [NSTEMI]) and subsequently evaluate
170 (ST-segment elevation myocardial infarction [STEMI], n = 399; non-ST-segment elevation acute coronary
171 perintensity was associated with the initial STEMI severity, adverse remodeling, and long-term health
175 boration of investigators interested in LMIC STEMI care have tried to create a consensus document tha
176 the pharmaco-invasive strategy for managing STEMI on 30-day patient mortality and individual provide
177 s of 3-vessel or left main disease than men (STEMI: 38.8% versus 58.7%; STEMI: 24.3% versus 32.1%), a
178 tes and outcomes of ST-segment elevation MI (STEMI) in renal transplant recipients vs the stage 5D CK
181 ime to a comprehensive focus on multifaceted STEMI care offers an opportunity to further improve STEM
182 rdial infarction (STEMI) type 2 (31.9%), non-STEMI type 1 (31.5%), unstable angina (28.5%), and STEMI
183 vation myocardial infarction [STEMI] and non-STEMI [NSTEMI]) and subsequently evaluated for sex-based
184 evation myocardial infarction (STEMI) or non-STEMI (NSTEMI) who were undergoing PCI and receiving tre
185 elevation myocardial infarction (STEMI), non-STEMI (NSTEMI), myocardial infarction of unknown type, o
186 s with presumptive COVID-19 with nonanterior STEMI without cardiogenic shock, PPCI offered a 0.4% abs
189 ncremental prognostic value of each facet of STEMI care on clinical outcomes within a STEMI system of
191 tals in the United Kingdom within 6 hours of STEMI due to a proximal-mid-vessel occlusion of a major
193 CI cannot be performed within 120 minutes of STEMI diagnosis, fibrinolysis therapy should be administ
196 been gained regarding the pathophysiology of STEMI and feed into the development of new treatment str
197 r adjusting for known clinical predictors of STEMI in-hospital mortality, achievement of at least 2 S
202 ents who had cardiac arrest without signs of STEMI to undergo immediate coronary angiography or coron
203 -hospital cardiac arrest and had no signs of STEMI, a strategy of immediate angiography was not found
204 tors showed that serum-PC EPA at the time of STEMI was inversely associated with both incident MACE a
205 e omega-3 consumption) levels at the time of STEMI were associated with a lower incidence of major ad
206 d serum-PC EPA and ALA levels at the time of STEMI were associated with a lower risk of clinical adve
209 (AngeLmed Early Recognition and Treatment of STEMI) pivotal study, subjects at high risk for recurren
210 elMed for Early Recognition and Treatment of STEMI), a multicenter, randomized trial of an implantabl
211 s the persistently dramatic impact of SCA on STEMI and the major importance of PCI in this setting.
212 myocardial infarction (STEMI); however, once STEMI occurs, smoking has been associated with favorable
214 ients with left ventricular dysfunction post STEMI who are at risk for death and major morbidity.
218 oline administration in the early phase post-STEMI in patients with multivessel disease is safe.
219 f patients presenting with late presentation STEMI was observed in 2020 compared with the historical
220 Compared with controls, patients with recent STEMI exhibited increased LV wall active tension when no
222 tively analyzed (2003 to 2017) at 2 regional STEMI programs (Minneapolis Heart Institute and Cedars-S
224 specific risk scores to stratify reperfused STEMI patients by their risk level for targeted interven
225 Patients (age >18 years) with suspected STEMI and who were eligible for PPCI were randomly alloc
227 elated functional impairments and, among the STEMI subgroup, a higher incidence of overall bleeding e
228 n on infarct size, we analyzed data from the STEMI-Door to Unload (STEMI-DTU) trial and then tested t
230 analyzed data from the STEMI-Door to Unload (STEMI-DTU) trial and then tested the effect of LV unload
231 morbidities, including drug and alcohol use, STEMI acuity (cardiac arrest and cardiogenic shock), and
232 , 0.54 [95% CI, 0.36-0.81] P=0.003), whereas STEMI increased the risk of all-cause death (hazard rati
235 that utilization of PCI in older adults with STEMI and cardiogenic shock is increasing and paralleled
236 nary intervention (PCI) in older adults with STEMI and shock and its influence on in-hospital mortali
238 justed analyses, Medicaid beneficiaries with STEMI had lower rates of coronary revascularization (88.
242 older with symptoms or signs consistent with STEMI at primary care clinics, small hospitals, and PCI
244 tality among the renal transplant group with STEMI was markedly lower compared with the stage 5D CKD
247 ively included 944 consecutive patients with STEMI (mean age 61 years, 209 women) undergoing primary
248 or in 30-day mortality of all patients with STEMI (patients who did and did not undergo PCI, 10.86%
249 mortality than did men in both patients with STEMI (women, 9.4%, versus men, 4.5%) and patients with
250 Milan included all consecutive patients with STEMI admitted to our institute from February 21 to Apri
252 wing culprit-lesion PCI, 4,041 patients with STEMI and multivessel CAD were randomized to staged nonc
257 esults were consistent between patients with STEMI and those with NSTEMI and across other major subgr
258 ated that soluble cytokines in patients with STEMI are upregulated in a coordinated fashion in contra
261 et to hospital admission among patients with STEMI during COVID-19 pandemic compared with the same ti
266 n LD of ticagrelor, 180 mg, in patients with STEMI is feasible and facilitates better early platelet
270 n the treatment and outcome of patients with STEMI treated by primary percutaneous coronary intervent
272 lume PPCI centers and assessed patients with STEMI treated with PPPCI in March/April 2019 and 2020.
275 d from 10 randomized trials of patients with STEMI undergoing primary percutaneous coronary intervent
277 -invasive strategy in selected patients with STEMI with presumptive COVID-19 and low likelihood of mo
278 er mortality gain was found in patients with STEMI with reperfusion therapy or in patients with NSTEM
279 ing and delayed reperfusion in patients with STEMI without cardiogenic shock is safe and feasible.
280 still decreased after 2010 in patients with STEMI without reperfusion therapy, whereas no further mo
282 Participants included 3602 patients with STEMI, aged 18 years or older, who were undergoing prima
283 volunteers without CVD and 62 patients with STEMI, separated soluble and EV fractions, and analyzed
285 a reperfusion therapy in adult patients with STEMI, whether given alone or in combination with adjunc
286 ant impact on the treatment of patients with STEMI, with a 19% reduction in PPCI procedures, especial
292 ferred reperfusion strategy in patients with STEMI; if PCI cannot be performed within 120 minutes of
293 om 66+/-14 to 63+/-14 years in patients with STEMI; it remained stable (68+/-14 years) in patients wi
294 y With PCI Versus PCI Alone in Patients With STEMI]) enrolled 19 047 patients, of whom 18 306 underwe
299 tes of bleeding were higher among women with STEMI (26.2% versus 15.6%, P<0.001) but not NSTEMI (17.8
300 r all patients with PCI and patients without STEMI, there were no significant differences in in-hospi