ライフサイエンスコーパス: Salmonella enterica

1 m impacted by the metabolic stress of 2AA in Salmonella enterica.

2 ilament among B. subtilis, P. aeruginosa and Salmonella enterica.

3 of the essential endoribonuclease RNase E in Salmonella enterica.

4 ISPR based redox conduit in both E. coli and Salmonella enterica.

5 natural and challenged C. jejuni and natural Salmonella enterica.

6 nt-invasive E. coli and the related pathogen Salmonella enterica.

7 ltry to infections with distinct serovars of Salmonella enterica.

8 eractions of B. mallei, Yersinia pestis, and Salmonella enterica.

9 for virulence in Yersinia enterocolitica and Salmonella enterica.

10 sponsible for eptA regulation in E. coli and Salmonella enterica.

11 t Listeria innocua, Pseudomonas fluorescens, Salmonella enterica and Bacillus cereus has not been pre

12 ntibiotic resistance-conferring mutations in Salmonella enterica and E. coli.

13 We show related protein domains from Salmonella enterica and Escherichia coli possess similar

14 s of Vibrio cholerae, Citrobacter rodentium, Salmonella enterica and ETEC were capable of complementi

15 d the PT reaction using purified DndCDE from Salmonella enterica and IscS from Escherichia coli.

16 burnetii, Leptospira spp., Rickettsia spp., Salmonella enterica and Salmonella enterica serovar Typh

17 st Escherichia coli, Listeria monocytogenes, Salmonella enterica and Staphylococcus aureus.

18 -propanediol utilization microcompartment of Salmonella enterica and use it to analyze the function o

19 virulence of the important enteric pathogens Salmonella enterica and Vibrio cholerae by repressing Ar

20 ng and analyzing 720 Listeria monocytogenes, Salmonella enterica, and Escherichia coli short-read dat

21 In Escherichia coli, Salmonella enterica, and many other gamma-proteobacteria

22 Strains of Salmonella enterica, and other organisms lacking RidA, h

23 ella pneumoniae, Mycobacterium tuberculosis, Salmonella enterica, and Staphylococcus aureus, we repor

24 eria, including Corynebacterium diphtheriae, Salmonella enterica, and Vibrio cholerae, are infected w

25 ying host range distribution in a lineage of Salmonella enterica, and we discuss many other potential

26 Salmonella enterica are invasive intracellular pathogens

27 Escherichia coli and Salmonella enterica are models for many experiments in m

28 with cfDNA from Mycobacterium tuberculosis, Salmonella enterica, Aspergillus fumigatus, and Epstein-

29 at viable Francisella tularensis, as well as Salmonella enterica bacteria transferred from infected c

30 Salmonella enterica bloodstream infections are an import

31 ed vitamin B-12, [13C]-cyanocobalamin, using Salmonella enterica by providing [13C2]-ethanolamine as

32 rom Escherichia coli, Shigella flexneri, and Salmonella enterica can all fold to form self-complement

33 -propanediol utilization microcompartment of Salmonella enterica can be controlled using two strategi

34 Collectively, our findings indicate that Salmonella enterica can promote transformation of geneti

35 luding the causative agent of salmonellosis, Salmonella enterica, can occur as a result of eco-evolut

36 Salmonella enterica catabolizes ethanolamine inside a co

37 Serovars of Salmonella enterica cause both gastrointestinal and syst

38 Salmonella enterica causes systemic diseases (typhoid an

39 ericia coli, phosphorothioate epigenetics in Salmonella enterica Cerro 87, and oxidation-induced abas

40 DNA MTases, like those from Vibrio cholerae, Salmonella enterica, Clostridioides difficile, or Strept

41 SpvD is a Salmonella enterica effector protein that we previously

42 The Salmonella enterica effector SteD depletes mature MHC cl

43 vation of MAPK and AKT pathways, mediated by Salmonella enterica effectors secreted during infection,

44 yptosporidium parvum, Entamoeba histolytica, Salmonella enterica, enterotoxigenic Escherichia coli, V

45 Recent papers have shown that the Salmonella enterica FinO-domain protein ProQ binds a lar

46 Insertions in the Salmonella enterica fra locus, which encodes the fructos

47 without POTRA domains from Escherichia coli, Salmonella enterica, Haemophilus ducreyi and Neisseria g

48 Neisseria meningitidis, Vibrio cholerae, and Salmonella enterica harbor these prophages.

49 Detection of fluoroquinolone resistance in Salmonella enterica has become increasingly difficult du

50 Multidrug-resistant (MDR) Salmonella enterica has been deemed a high-priority path

51 The intracellular pathogen Salmonella enterica has evolved an array of traits for p

52 enetic and biochemical studies, primarily in Salmonella enterica, have defined a role for RidA in res

53 , we present crystal structures of wild type Salmonella enterica HisA (SeHisA) in its apo-state and o

54 infection with Burkholderia pseudomallei and Salmonella enterica HMBA treatment was also associated w

55 tructures of GusRs from Escherichia coli and Salmonella enterica in complexes with a glucuronide liga

56 luorescence in situ hybridization identified Salmonella enterica in the liver, subsequently confirmed

57 d in the EnvZ-OmpR two-component system from Salmonella enterica in vitro and in vivo, which directly

58 aMN):DMB phosphoribosyltransferases (CobT in Salmonella enterica), in a reaction that is considered t

59 the foodborne pathogens E. coli O157:H7 and Salmonella enterica, in detail a nucleic acid lateral fl

60 Salmonella enterica including Salmonella Typhi and nonty

61 ver natural ClpS substrates in the bacterium Salmonella enterica, including SpoT, the essential synth

62 We report that Salmonella enterica increases the specificity of the bro

63 and Salmonella Typhi infection, we show that Salmonella enterica induces malignant transformation in

64 he most common manifestation of nontyphoidal Salmonella enterica infections, but little is known abou

65 ity and mortality for invasive non-typhoidal Salmonella enterica infections, we excluded cases attrib

66 Salmonella enterica is a leading cause of community-acqu

67 EutT from Salmonella enterica is a member of a class of enzymes te

68 Salmonella enterica is among the most burdensome of food

69 mance of PRAP by analyzing the genomes of 26 Salmonella enterica isolates from Shanghai, China.

70 Two hundred and sixty-four Salmonella enterica isolates recovered over a 16-year pe

71 their ability to detect low-level-resistant Salmonella enterica isolates that are not serotype Typhi

72 isolates of the pathogens Escherichia coli, Salmonella enterica, Klebsiella pneumoniae and Acinetoba

73 ccus pneumoniae, Mycobacterium tuberculosis, Salmonella enterica, Klebsiella pneumoniae, and Escheric

74 in vivo mechanisms underlying the switch in Salmonella enterica lifestyles from the infectious form

75 ted microorganisms such as Escherichia coli, Salmonella enterica, Listeria innocua, Mycobacterium par

76 re formed upon recombinant expression of the Salmonella enterica LT2 ethanolamine utilization bacteri

77 ression experiments, WbaP(Mx) complemented a Salmonella enterica mutant lacking the endogenous WbaP t

78 the total allelic diversity (panallelome) of Salmonella enterica, Mycobacterium tuberculosis, Pseudom

79 Of pathogens, 171 (44.6%) were Salmonella enterica of which 129 (75.4%) were Salmonella

80 Phase variation of the Salmonella enterica opvAB operon generates a bacterial l

81 uencing (Grad-seq) in the bacterial pathogen Salmonella enterica, partitioning its coding and noncodi

82 into nonphagocytic host cells is central to Salmonella enterica pathogenicity and dependent on multi

83 The most common Salmonella enterica pathovariant associated with invasiv

84 We hypothesized that the Salmonella enterica phage SPN3US could be a useful model

85 man bacterial pathogens Escherichia coli and Salmonella enterica produce a biofilm matrix composed pr

86 With hosts such as Salmonella enterica, Pseudomonas aeruginosa, and Erwinia

87 s)RidA-2 complemented the growth defect of a Salmonella enterica ridA mutant, an in vivo model of 2AA

88 ldwide develop foodborne illnesses caused by Salmonella enterica (S. enterica) every year.

89 sed investigation of a recurrent blood-borne Salmonella enterica serotype Enteritidis (S.

90 or comparing molecular subtyping methods for Salmonella enterica serotype Enteritidis and survey the

91 vestigated a large multi-country outbreak of Salmonella enterica serotype Enteritidis in the EU and E

92 Between March 1, 2010, and Jan 31, 2014, 135 Salmonella enterica serotype Typhi (S Typhi) and 94 iNTS

93 We reviewed Salmonella enterica serotype Typhi infections reported t

94 cal failure (ie, blood cultures positive for Salmonella enterica serotype Typhi, or Paratyphi A, B, o

95 Salmonella enterica serotype Typhimurium (S. Typhimurium

96 mportant for colonization and persistence of Salmonella enterica serotype Typhimurium in chickens.

97 Salmonella enterica serotype Typhimurium is a food-borne

98 teric fever, a bacterial infection caused by Salmonella enterica serotypes Typhi and Paratyphi A, fre

99 detects typhoid-causing infectious bacteria Salmonella enterica serovar (Salmonella typhi) in 10 muL

100 nterica serovar Enteritidis, and 3% (3) were Salmonella enterica serovar Arizonae.

101 Analysis of the distribution of Salmonella enterica serovar Derby (S.

102 4 protected mice against the group D serovar Salmonella enterica serovar Dublin (85% vaccine efficacy

103 aluated the capacities of S. Typhimurium and Salmonella enterica serovar Enteritidis DeltaguaBA Delta

104 Salmonella enterica serovar Enteritidis is a common caus

105 Salmonella enterica serovar Enteritidis is a major cause

106 e show that neonatal chick colonization with Salmonella enterica serovar Enteritidis requires a virul

107 enterica serovar Typhimurium, 10% (10) were Salmonella enterica serovar Enteritidis, and 3% (3) were

108 An epidemiological paradox surrounds Salmonella enterica serovar Enteritidis.

109 Salmonella enterica serovar Infantis (S.

110 We also show that CBA120 infects Salmonella enterica serovar Minnesota, and this host ran

111 from the pathogenic, Gram-negative bacteria Salmonella enterica serovar Montevideo.

112 Here, we present a focused minireview on Salmonella enterica serovar Panama, a serovar responsibl

113 o develop the first human challenge model of Salmonella enterica serovar Paratyphi A infection.

114 Salmonella enterica serovar Paratyphi A is a human-speci

115 r with Salmonella enterica serovar Typhi and Salmonella enterica serovar Sendai, causes enteric fever

116 1 protected mice against the group B serovar Salmonella enterica serovar Stanleyville (91% vaccine ef

117 In areas of Asia, multidrug-resistant Salmonella enterica serovar Typhi (S Typhi) has been the

118 ting protein, to influence susceptibility to Salmonella enterica serovar Typhi (S Typhi) infection.

119 Salmonella enterica serovar Typhi (S Typhi) is responsib

120 Salmonella enterica serovar Typhi (S. Typhi) causes subs

121 ing the ingestion and subsequent invasion of Salmonella enterica serovar Typhi (S. Typhi), a human-re

122 The population of Salmonella enterica serovar Typhi (S. Typhi), the causat

123 imicrobial resistance (AMR) in the bacterium Salmonella enterica serovar Typhi (S. Typhi).

124 a human-specific serovar that, together with Salmonella enterica serovar Typhi and Salmonella enteric

125 Salmonella enterica serovar Typhi causes the systemic di

126 Salmonella enterica serovar Typhi causes typhoid fever o

127 Multiyear epidemics of Salmonella enterica serovar Typhi have been reported fro

128 man populations, with the causative pathogen Salmonella enterica serovar Typhi implicated in many out

129 Salmonella enterica serovar Typhi is a human-restricted

130 Salmonella enterica serovar Typhi is the etiological age

131 We report a typhoid fever case with a Salmonella enterica serovar Typhi isolate showing extend

132 Salmonella enterica serovar Typhi isolates from the 2 ho

133 n, whereas the secretion of Typhoid toxin in Salmonella enterica serovar Typhi relies on a muramidase

134 Here we used a live attenuated Salmonella enterica serovar Typhi strain to create a biv

135 ded-spectrum-beta-lactamase (ESBL)-producing Salmonella enterica serovar Typhi was identified, whole-

136 p., Rickettsia spp., Salmonella enterica and Salmonella enterica serovar Typhi, and Yersinia pestis),

137 e that the causative agent of typhoid fever, Salmonella enterica serovar Typhi, can partially subvert

138 ince the 1970s, this threat has increased in Salmonella enterica serovar Typhi, driven in part by the

139 Enteric fever, caused by Salmonella enterica serovar Typhi, is an important publi

140 Typhoid toxin is a virulence factor of Salmonella enterica serovar Typhi, the causative agent o

141 Salmonella enterica serovar Typhi, the causative agent o

142 the serotyped salmonellae, 14% (21/152) were Salmonella enterica serovar Typhi, whereas 86% (131/152)

143 n the development of antimicrobial-resistant Salmonella enterica serovar Typhi.

144 childhood bacteremia, with a predominance of Salmonella enterica serovar Typhi.

145 osa, nontypeable Haemophilus influenzae, and Salmonella enterica serovar Typhi/Typhimurium.

146 ss-reactivity to bacterial pathogens such as Salmonella enterica serovar Typhimurium (7.8%), Listeria

147 FrmR from Salmonella enterica serovar typhimurium (a CsoR/RcnR-lik

148 scherichia coli, Yersinia enterocolitica and Salmonella enterica serovar Typhimurium (all gram-negati

149 Salmonella enterica serovar Typhimurium (S Typhimurium)

150 ly showed that l-asparaginase II produced by Salmonella enterica serovar Typhimurium (S Typhimurium)

151 Salmonella enterica serovar Typhimurium (S Typhimurium)

152 The human pathogen Salmonella enterica serovar Typhimurium (S Typhimurium)

153 BA would be more resistant to infection with Salmonella enterica serovar Typhimurium (S Typhimurium).

154 Salmonella enterica serovar Typhimurium (S.

155 wo intracellular [Listeria monocytogenes and Salmonella enterica serovar Typhimurium (S.

156 the ancestral regulatory system PhoP/PhoQ of Salmonella enterica serovar Typhimurium (S. Typhimurium)

157 Infection of human macrophages with Salmonella enterica serovar Typhimurium (S. Typhimurium)

158 udy, we used a mouse model of infection with Salmonella enterica serovar Typhimurium (STM) to identif

159 host defense against the bacterial pathogen Salmonella enterica serovar Typhimurium (STm).

160 uick and accurate detection of low levels of Salmonella enterica serovar typhimurium and enteritidis

161 ial amyloids using curli fibers, produced by Salmonella enterica serovar Typhimurium and Escherichia

162 ependent bacteria (Escherichia coli O157:H7, Salmonella enterica serovar Typhimurium and S.

163 rolling infection by two bacterial pathogens-Salmonella enterica serovar Typhimurium and Shigella fle

164 flexneri and other pathogens that use T3SS, Salmonella enterica serovar Typhimurium and Yersinia pse

165 Using two species, Escherichia coli and Salmonella enterica serovar Typhimurium as model microbe

166 Shigella flexneri and the vT3SS and fT3SS of Salmonella enterica serovar Typhimurium at ~5 and ~4 nm

167 The food-borne pathogen Salmonella enterica serovar Typhimurium benefits from ac

168 Salmonella enterica serovar Typhimurium can inject effec

169 Conversely, Salmonella enterica serovar Typhimurium causes gastroent

170 The Gram-negative intracellular bacterium Salmonella enterica serovar Typhimurium causes persisten

171 l compartments and a reduced ability to kill Salmonella enterica serovar Typhimurium compared to that

172 Bloodstream infections by Salmonella enterica serovar Typhimurium constitute a maj

173 The ST313 pathovar of Salmonella enterica serovar Typhimurium contributes to a

174 We also demonstrate that the Salmonella enterica serovar Typhimurium core promoter is

175 that the facultative intracellular pathogen Salmonella enterica serovar Typhimurium decreases H-NS a

176 toxified outer membrane vesicles (OMVs) from Salmonella enterica serovar Typhimurium displaying the v

177 provide an important colonization niche for Salmonella enterica serovar Typhimurium during gastroint

178 A recent study showed that Salmonella enterica serovar Typhimurium exhibits sliding

179 Salmonella enterica serovar Typhimurium exploits the hos

180 ite sequencing (TraDIS) to screen mutants of Salmonella enterica serovar Typhimurium for their abilit

181 ur genome-wide analysis of core genes within Salmonella enterica serovar Typhimurium genomes reveals

182 g different susceptibilities to infection by Salmonella enterica serovar Typhimurium has just been pu

183 e human chitotriosidase and a chitinase from Salmonella enterica serovar Typhimurium hydrolyze LacNAc

184 Slc11a1 is proposed to restrict growth of Salmonella enterica serovar Typhimurium in host tissues

185 ity (mAb 3H3) can disrupt biofilms formed by Salmonella enterica serovar Typhimurium in vitro and in

186 We found that intracellular Salmonella enterica serovar Typhimurium induced the binu

187 n simultaneously in pathogen and host during Salmonella enterica serovar Typhimurium infection and re

188 induction renders cells more susceptible to Salmonella enterica serovar Typhimurium infection in a N

189 Salmonella enterica serovar Typhimurium infection of imm

190 CARD9 is suppressed during Salmonella enterica serovar Typhimurium infection, facil

191 During Salmonella enterica serovar Typhimurium infection, host

192 In addition, the intracellular pathogen Salmonella enterica serovar Typhimurium initiates an ant

193 Salmonella enterica serovar Typhimurium is a common caus

194 Salmonella enterica serovar Typhimurium is a facultative

195 Salmonella enterica serovar Typhimurium is an intracellu

196 h of the in vivo innate immune resistance of Salmonella enterica serovar Typhimurium is attributed to

197 tedly, the allosteric mechanism of FrmR from Salmonella enterica serovar Typhimurium is triggered by

198 a non-thermal intervention for inactivating Salmonella enterica serovar Typhimurium LT2 (ST2) in ten

199 utilization bacterial microcompartment from Salmonella enterica serovar Typhimurium LT2, one of the

200 alis, Escherichia coli K12, E. coli O157:H7, Salmonella enterica serovar Typhimurium LT2, Staphylococ

201 ed a metabolically competent, but avirulent, Salmonella enterica serovar Typhimurium mutant for its a

202 we present the structure of the prototypical Salmonella enterica serovar Typhimurium pathogenicity is

203 ntroduction of the tviA gene in nontyphoidal Salmonella enterica serovar Typhimurium reduced flagelli

204 in the gut by the enteropathogenic bacterium Salmonella enterica serovar Typhimurium requires a T6SS

205 r ring-forming proteins PrgH and PrgK in the Salmonella enterica serovar Typhimurium Salmonella patho

206 We genetically engineered a Salmonella enterica serovar Typhimurium strain of multil

207 antigen from Mycobacterium tuberculosis, in Salmonella enterica serovar Typhimurium strain SL3261.

208 sine harbors bacteriostatic activity against Salmonella enterica serovar Typhimurium that is not shar

209 egulatory system coordinates the response of Salmonella enterica serovar Typhimurium to diverse envir

210 he current study, we examined the ability of Salmonella enterica serovar Typhimurium to infect the ce

211 bound in vivo by the CspA family members of Salmonella enterica serovar Typhimurium to link the cons

212 Here we show that the intestinal pathogen Salmonella enterica serovar Typhimurium uses specialized

213 Salmonella enterica serovar Typhimurium utilizes molecul

214 instance, pigs experimentally infected with Salmonella enterica serovar Typhimurium was investigated

215 C3, D1, E1, G, I, K, N, O, and Q); however, Salmonella enterica serovar Typhimurium was the most pre

216 Listeria monocytogenes V7 and Salmonella enterica serovar Typhimurium were used as mod

217 ive real-time PCR detection of low levels of Salmonella enterica serovar Typhimurium without culture

218 Salmonella (Salmonella enterica serovar Typhimurium) secrete numerou

219 Gram negative bacteria (Escherichia coli and Salmonella enterica serovar Typhimurium).

220 Of the 102 typed NTS isolates, 40% (41) were Salmonella enterica serovar Typhimurium, 10% (10) were S

221 Salmonella enterica serovar Typhimurium, a Gram-negative

222 Nontyphoidal salmonellae, particularly Salmonella enterica serovar Typhimurium, are a major cau

223 For the human and animal pathogen Salmonella enterica serovar Typhimurium, biofilm formati

224 In Salmonella enterica serovar Typhimurium, DSFs repress th

225 ic bacteria, either Klebsiella pneumoniae or Salmonella enterica serovar Typhimurium, enhanced transl

226 In Salmonella enterica serovar Typhimurium, flagella-mediat

227 We show that both flagellins of Salmonella enterica serovar Typhimurium, FliC and FljB,

228 In Salmonella enterica serovar Typhimurium, Mg(2+) limitati

229 nterohemorrhagic Escherichia coli (EHEC) and Salmonella enterica serovar Typhimurium, or the surrogat

230 create FLIM-phasor maps of Escherichia coli, Salmonella enterica serovar Typhimurium, Pseudomonas aer

231 udy the infection initiation of phage P22 in Salmonella enterica serovar Typhimurium, revealing how a

232 In Salmonella enterica serovar Typhimurium, siroheme is pro

233 Unlike the nontyphoidal Salmonella enterica serovar Typhimurium, the genomes of

234 To examine individual functions, strains of Salmonella enterica serovar Typhimurium, the murine mode

235 hput assay for type III protein secretion in Salmonella enterica serovar Typhimurium, we discovered t

236 d a striking defect in their ability to kill Salmonella enterica serovar Typhimurium, which was rescu

237 that TcpB protein can efficiently attenuate Salmonella enterica serovar Typhimurium-induced pyroptos

238 Testing the methodology on Salmonella enterica serovar Typhimurium-infected murine

239 en Vibrio cholerae and the invasive pathogen Salmonella enterica serovar Typhimurium.

240 t intracellular states of the model pathogen Salmonella enterica serovar Typhimurium.

241 helial oxygenation, and aerobic expansion of Salmonella enterica serovar Typhimurium.

242 oQ in the facultative intracellular pathogen Salmonella enterica serovar Typhimurium.

243 charide stimulation or upon coinfection with Salmonella enterica serovar Typhimurium.

244 ) responses to bacterial infections, such as Salmonella enterica serovar Typhimurium.

245 ls and cell lines, typically challenged with Salmonella enterica serovar Typhimurium.

246 al infection with enteric pathogens, such as Salmonella enterica serovar Typhimurium.

247 acteria, such as Clostridium perfringens and Salmonella enterica serovar Typhimurium.

248 Salmonella enterica serovars Enteritidis and Kentucky di

249 The burden of enteric fever caused by Salmonella enterica serovars Typhi and Paratyphi is subs

250 ivalent to fever (39 degrees C-42 degrees C) Salmonella enterica serovars Typhi, Paratyphi A, and Sen

251 Nontyphoidal Salmonella (NTS), particularly Salmonella enterica serovars Typhimurium and Enteritidis

252 Exposure to the dominant NTS serovars, Salmonella enterica serovars Typhimurium and Enteritidis

253 sess the ability of PCR for the detection of Salmonella enterica shedding and to compare that ability

254 Studies in Escherichia coli and Salmonella enterica showed that such sRNAs are natural p

255 estis), PscF (Pseudomonas aeruginosa), PrgI (Salmonella enterica SPI-1), SsaG (Salmonella enterica SP

256 sa), PrgI (Salmonella enterica SPI-1), SsaG (Salmonella enterica SPI-2), or MxiH (Shigella flexneri).

257 i, S. Paratyphi A, and genes conserved among Salmonella enterica spp. and utilized strongly magnetize

258 hlortetracycline on the temporal dynamics of Salmonella enterica spp. enterica in feedlot cattle.

259 ter rodentium NleB effectors, as well as the Salmonella enterica SseK effectors are glycosyltransfera

260 Enterococcus spp., Escherichia coli, Salmonella enterica, Staphylococcus aureus and Streptoco

261 We report that typhoidal and nontyphoidal Salmonella enterica strains activate MAIT cells.

262 (EPEC), and Citrobacter rodentium Moreover, Salmonella enterica strains encode up to three NleB orth

263 The NGS data set of the Salmonella enterica strains were used as a case study to

264 tic activity of various Escherichia coli and Salmonella enterica strains.

265 S. bongori, S. enterica subsp. salamae, and Salmonella enterica subsp. arizonae The beta-lactamase T

266 aced this domain with a nuclease domain from Salmonella enterica subsp. arizonae This modified V. cho

267 Here, we recovered eight Salmonella enterica subsp. enterica genomes from human s

268 equence the entire chromosome and plasmid of Salmonella enterica subsp. enterica serovar Bareilly and

269 system may contribute to the persistence of Salmonella enterica subsp. enterica serovar Typhimurium

270 nosus R0011 secretome (LrS) on TNF-alpha and Salmonella enterica subsp. enterica serovar Typhimurium

271 Salmonella enterica subsp. enterica serovars Typhimurium

272 tudy, we compared a draft genome assembly of Salmonella enterica subsp. salamae strain 3588/07 agains

273 nterica subspecies enterica serovar Typhi or Salmonella enterica subspecies enterica serovar Paratyph

274 Human infections with Salmonella enterica subspecies enterica serovar Senftenb

275 Typhoid fever is caused by Salmonella enterica subspecies enterica serovar Typhi (S

276 Salmonella enterica subspecies enterica serovar Typhi (S

277 Salmonella enterica subspecies enterica serovar Typhi or

278 We report a traveler who acquired a Salmonella enterica subspecies enterica serovar Typhi st

279 Here we show that Salmonella enterica subspecies enterica serovar Typhimur

280 0 of 620 (74.2%) NTS isolates serotyped were Salmonella enterica subspecies enterica serovar Typhimur

281 We present the genome sequences of Salmonella enterica tailed phages Sasha, Sergei, and Sol

282 The common human pathogen Salmonella enterica takes up citrate as a nutrient via t

283 thogens or prolonged exposure to heat-killed Salmonella enterica, the Gram-positive bacterium Bacillu

284 Here, we report that, in Salmonella enterica, the sirtuin deacylase CobB long iso

285 III secretion system (T3SS) of intracellular Salmonella enterica translocates effector proteins into

286 Within host cells such as macrophages, Salmonella enterica translocates virulence (effector) pr

287 Two major serovars of Salmonella enterica, Typhi and Typhimurium, have evolved

288 onlethal gastric infections of Gram-negative Salmonella enterica Typhimurium (ST), a major source of

289 Using Toxoplasma gondii and Salmonella enterica Typhimurium we demonstrate HRMAn's c

290 in bacterial cells, particularly E. coli and Salmonella enterica Typhimurium.

291 on with the intracellular bacterial pathogen Salmonella enterica Typhimurium.

292 leic acid, a major lipid in E. coli Last, in Salmonella enterica, ubiK was required for proliferation

293 Unlike ScThi5, LpThi5 functioned in vivo in Salmonella enterica under multiple growth conditions.

294 Salmonella enterica variants exhibit diverse host adapta

295 Dap accumulation in Salmonella enterica was previously shown to inhibit grow

296 es in an investigation of host adaptation in Salmonella enterica We highlight the value of the method

297 a on two key organisms, Escherichia coli and Salmonella enterica, we show that copper resistance requ

298 res of R-LPS from either Escherichia coli or Salmonella enterica were directly infused into the mass

299 bserved PPI, including Bacillus subtilis and Salmonella enterica which are predicted to have up to 18

300 rimers specific for E. coli eaeA (151bp) and Salmonella enterica yfiR (375bp) genes.