ライフサイエンスコーパス: Schaffer

1 Schaffer collateral axons form excitatory synapses that

2 Schaffer collateral synapses on CA2 neurons are distinct

3 Schaffer collateral-CA1 tetanic long-term potentiation d

4 Schaffer-collateral long-term potentiation (LTP) was imp

5 y ramifying in the pyramidal cell layer; (2) Schaffer collateral/commissural pathway-associated inter

6 whenever tested (n = 5; 1 basket cell and 4 Schaffer-associated interneurons), were abolished by the

7 we measure this replenishment rate at active Schaffer collateral terminals by determining the maximum

8 ed transcripts in rat hippocampus 3 hr after Schaffer-CA1 LTP induction in vivo, we identified a neur

9 voltage and calcium signals in spines after Schaffer collateral synapse stimulation.

10 ium to block synaptic transmission, allowing Schaffer-collateral axon fiber volleys to be recorded fr

11 path-dentate gyrus granule cell, CA3-CA3 and Schaffer collateral-CA1 synapses without effects at moss

12 of anterior piriform (olfactory) cortex, and Schaffer-commissural synapses were studied in slices of

13 d activation of entorhinal cortical (EC) and Schaffer collateral (SC) inputs to hippocampal CA1 pyram

14 Maynard Smith's infinite population ESS and Schaffer's finite population ESS.

15 imulation in the hippocampal mossy fiber and Schaffer collateral pathways.

16 e dendrites were unlabeled, mossy fibers and Schaffer collaterals in the hippocampal formation, baske

17 l CA1 neurons through the perforant path and Schaffer collateral synapses, respectively.

18 ively direct formation of perforant-path and Schaffer-collateral synapses, respectively, to hippocamp

19 n excitatory inputs, the perforant path, and Schaffer collaterals during theta and non-theta behavior

20 at the CA3 recurrent collateral synapses and Schaffer collateral-CA1 synapses, but not at the mossy f

21 terneuron, the so-called 'basket cells' and 'Schaffer collateral-associated' cells, which innervate s

22 At Schaffer collateral synapses, the induction of long-term

23 At Schaffer collateral-CA1 synapses in the hippocampus, pre

24 red the function of SNAP-25a and SNAP-25b at Schaffer collateral-CA1 synapses in hippocampus using 4-

25 (AMPA)-type glutamate receptors (AMPARs) at Schaffer collateral to CA1 synapses in postischemic hipp

26 nylglycine (DHPG) was markedly attenuated at Schaffer collateral-CA1 synapses of mice lacking caveoli

27 This occurred both at Schaffer collateral-CA1 synapses and at CA3-CA3 synapses

28 ficits and specific synaptic dysfunctions at Schaffer collateral to CA1 synapses.

29 NMDA receptor-dependent LTP was enhanced at Schaffer collateral-CA1 pyramidal cell synapses.

30 s of both AMPAR and NMDAR currents evoked at Schaffer collateral synapses.

31 Although CB1Rs are also expressed at Schaffer-collateral excitatory terminals, long-term plas

32 e of cleft glutamate concentrations found at Schaffer collateral synapses.

33 SR in regulating synaptic NMDAR function at Schaffer collateral (CA3)-CA1 synapses.

34 ly disrupted, revealing hyperexcitability at Schaffer collateral-CA1 synapses and depression of mossy

35 nd chemically induced NMDAR-dependent LTD at Schaffer collateral synapses but did not affect potentia

36 stores each prevent the induction of LTD at Schaffer collateral-CA1 synapses.

37 GluR-dependent long-term depression (LTD) at Schaffer collateral to CA1 pyramidal synapses of the hip

38 erm depression of synaptic strength (LTD) at Schaffer collateral-CA1, commissural/associational-CA3 a

39 ese interneurons in the modulation of LTP at Schaffer collateral synapses onto pyramidal cells.

40 lectively required for those forms of LTP at Schaffer collateral synapses that recruit a presynaptic

41 LTP at Schaffer collateral-CA1 pyramidal cell synapses remained

42 tance of CC2D1A in the maintenance of LTP at Schaffer collateral-CA1 synapses and the formation of hi

43 show normal synaptic transmission and LTP at Schaffer collateral-CA1 synapses, and have no deficits i

44 idylinositol 3-kinase (PI3-kinase) in LTP at Schaffer collateral/commissural fiber-CA1 synapses in ra

45 aCaMKII is essential for induction of LTP at Schaffer commissural-CA1 synapses in vivo, but is not re

46 equent threshold for the induction of LTP at Schaffer-commissural synapses through a reduction in the

47 mpal slices, long-term potentiation (LTP) at Schaffer collateral fiber synapses onto CA1 pyramidal ce

48 to modulate long-term potentiation (LTP) at Schaffer collateral-CA1 hippocampal synapses.

49 we show that long-term potentiation (LTP) at Schaffer collateral-CA1 synapses is greatly enhanced in

50 late-phase long-term potentiation (L-LTP) at Schaffer collateral/commissural fiber-CA1 synapses in ra

51 of synaptic transmission was not observed at Schaffer collateral-CA1 synapses, while the submaximal t

52 rences in short- and long-term plasticity at Schaffer collateral (SC) synapses in the dorsal and vent

53 on activity-dependent synaptic plasticity at Schaffer collateral and perforant path synapses in the h

54 ates the induction of synaptic plasticity at Schaffer collateral synapses and hippocampal-dependent l

55 arget cell-specific short-term plasticity at Schaffer collateral synapses in which the activation of

56 II mGlu receptors to synaptic plasticity at Schaffer collateral-CA1 synapses in acute slices of adul

57 uates activity-driven synaptic plasticity at Schaffer collateral-CA1 synapses.

58 f excitatory postsynaptic field potential at Schaffer collateral/commissural fiber-CA1 synapses showe

59 d the induction of long term potentiation at Schaffer collateral synapses.

60 anced induction of long term potentiation at Schaffer-collateral synapses in area CA1 of the hippocam

61 y visualize presynaptic vesicular release at Schaffer collateral-CA1 excitatory synapses in hippocamp

62 r a potential aspartate-mediated response at Schaffer collateral synapses in organotypic hippocampal

63 AMPA-receptor-mediated synaptic responses at Schaffer-collateral synapses monitored in juvenile mice,

64 the rapidly recycling vesicle pool (RRP) at Schaffer collateral terminals in field CA1 of rat hippoc

65 ansmission and spine density specifically at Schaffer collateral synapses in the stratum radiatum (SR

66 (LTP) induced by theta burst stimulation at Schaffer collateral synapses in the hippocampus.

67 m potentiation (LTP) of synaptic strength at Schaffer collateral synapses has largely been attributed

68 erm depression (LTD) of synaptic strength at Schaffer collateral-CA1 synapses by simultaneously eleva

69 Physiological studies at Schaffer collateral-CA1 synapses uncovered a synaptic fu

70 at perforant path synapses is lower than at Schaffer collateral inputs.

71 eurons, deficits in synaptic transmission at Schaffer collateral synapses, and blunted plasticity and

72 n the strength of excitatory transmission at Schaffer collateral-CA1 cell synapses of the hippocampus

73 -term depression of synaptic transmission at Schaffer collateral-CA1 synapses.

74 tors induces LTD of synaptic transmission at Schaffer collateral-CA1 synapses.

75 ansmission and plasticity were unaffected at Schaffer CA1 synapses in mutant mice.

76 Glutamate spillover is observed between Schaffer collateral fiber synapses onto CA1 pyramidal ce

77 ase across a range of Pr at synapses between Schaffer collaterals (SCs) and CA1 pyramidal cells in ac

78 amatergic or GABA-ergic transmission between Schaffer collaterals and CA1 neurons in rat hippocampal

79 cilitated glutamatergic transmission between Schaffer collaterals and CA1 neurons indicated that in a

80 c currents, evoked in CA1 principal cells by Schaffer collateral stimulation, were detected in hippoc

81 amate receptor (mGluR) stimulation either by Schaffer collateral input to CA1 neurones in brain slice

82 duced the amplitude of responses elicited by Schaffer collateral stimulation, without influencing mem

83 inhibits the population spike (PS) evoked by Schaffer collateral stimulation in hippocampal slices.

84 mory and long-term potentiation (LTP) at CA1 Schaffer collateral (SC) synapses.

85 release properties of zinc-positive CA3-CA1 Schaffer collateral/commissural synapses in the stratum

86 dendritic atrophy and LTP attenuation in CA1 Schaffer collateral synapses.

87 issural and mossy fiber synapses but not CA1 Schaffer collateral synapses.

88 esponding to the gamma-paced CA3 output (CA1 Schaffer potentials) and CA3 somatic inhibition within a

89 e dentate perforant path, but not in the CA1 Schaffer collateral pathway.

90 The projections from CA3 to CA1 (Schaffer collaterals), which terminate in the strata rad

91 We provide the first evidence of novel CA3 Schaffer collateral projection to the entorhinal cortex.

92 cture of a single presynaptic input, the CA3 Schaffer pathway, and its contribution to the spontaneou

93 nerated mainly by the entorhinal input, CA3 (Schaffer) collaterals, and voltage-dependent Ca(2+) curr

94 amidal cells and interneurons, the so-called Schaffer-driven spikes, revealing the presynaptic origin

95 ket cells and dendritically projecting, CCK+ Schaffer collateral-associated interneurons.

96 ds preceding the seizure without bursts (cut Schaffer collateral tract) and in comparison with bursts

97 in CA3 pyramidal neurons and its efferents - Schaffer collateral, which causes the depolarization, ac

98 induction of inflammation revealed enhanced Schaffer collateral-induced excitatory field potentials

99 Bilateral excitatory Schaffer gamma waves are generally larger and lead from

100 denosine receptors at neighboring excitatory Schaffer collateral terminals, which could counteract ef

101 LTP is not elicited in CCK-expressing Schaffer collateral-associated cells, which lack CP-AMPA

102 To counteract this amplitude filtering, Schaffer collateral synapses onto CA1 pyramidal cells co

103 h axons ramifying in the SO and SR; all five Schaffer-associated cells whose axons ramified extensive

104 ncreases in the frequency of sEPSC following Schaffer collateral stimulation.

105 amidal neurones revealed that high frequency Schaffer collateral stimulation resulted in a prolonged

106 approximately 80% of the synaptic input from Schaffer collaterals.

107 idal cells co-aligned with the glutamatergic Schaffer collateral/commissural input.

108 torhinal perforant path (PP) and hippocampal Schaffer collateral (SC) inputs to CA1 pyramidal cells s

109 has been studied extensively at hippocampal Schaffer collateral (SC) synapses.

110 nt long-term depression (LTD) at hippocampal Schaffer collateral (SC)-CA1 synapses.

111 Our findings show that at hippocampal Schaffer collateral CA3-CA1 synapses, theta-burst LTP re

112 and neurotransmitter release at hippocampal Schaffer collateral synapses in both tottering (tg, alph

113 long-term potentiation (LTP) at hippocampal Schaffer collateral-->CA1 synapses in an activity- and t

114 phase of NMDAR-dependent LTP at hippocampal Schaffer collateral-CA1 (Sch-CA1) synapses.

115 ssion and synaptic plasticity at hippocampal Schaffer collateral-CA1 synapses in adult and juvenile m

116 At hippocampal Schaffer collateral-CA1 synapses, LTD was significantly

117 on and long-term potentiation at hippocampal Schaffer collateral-CA1 synapses.

118 els of long-term potentiation at hippocampal Schaffer collateral-CA1 synapses.

119 paired long-term potentiation at hippocampal Schaffer collateral-CA1 synapses.

120 egulating synaptic plasticity at hippocampal Schaffer collateral-cornu ammonis 1 (SC-CA1) synapses ha

121 ding NMDAR-mediated responses at hippocampal Schaffer collaterals (SC)-CA1 and medial perforant path-

122 contrast, we show that at mature hippocampal Schaffer collateral synapses the magnitudes of Ca2+ tran

123 oach, induced different types of hippocampal Schaffer collateral (SC) to CA1 synaptic plasticity, dep

124 paired cAMP-dependent LTP in the hippocampal Schaffer collateral pathway induced by either forskolin

125 Using a computer model of the hippocampal Schaffer collateral pathway that incorporates evidence f

126 of synaptic transmission in the hippocampal Schaffer collateral pathway.

127 ional knockout (cKO) mice in the hippocampal Schaffer collateral pathway.

128 n the functional dynamics of the hippocampal Schaffer collateral synapse by using data-driven nonpara

129 were obtained with regard to the hippocampal Schaffer collateral synapse.

130 s of long-term depression at the hippocampal Schaffer collateral-CA1 synapse.

131 Field recordings in the hippocampal Schaffer collaterals (SC) pathway revealed significantly

132 s in brain slices by transecting hippocampal Schaffer collaterals and cortical layer I axons.

133 ked in CA1 minislices by sub-threshold 35 Hz Schaffer collateral stimulation that activated fast-spik

134 ombine optogenetic stimulation of identified Schaffer collateral axons with two-photon imaging of pos

135 ey provide a measure of axonal conduction in Schaffer-collateral fibers.

136 -type potassium channels that are present in Schaffer collateral axons and terminals.

137 s is actually decreased despite reduction in Schaffer-evoked inhibition.

138 rus synapses, but not in commonly studied in Schaffer collateral-CA1 synapses.

139 ium Green AM to measure Ca(2+) transients in Schaffer collateral presynaptic terminals simultaneously

140 n with bursts preceding the seizures (intact Schaffer collateral tract).

141 CA3 waves lock to the ipsilateral Schaffer potentials, although bilateral coherence was we

142 In contrast, AMPAR-mediated input at local Schaffer-collateral synapses on neurogliaform cells rema

143 nd persistent potentiation of AMPAR-mediated Schaffer collateral (SC)-CA1 fEPSPs in slices derived fr

144 ippocampus of symptomatic Mecp2(308/Y) mice, Schaffer-collateral synapses exhibited enhanced basal sy

145 n, whereas the release of NPY that modulates Schaffer collateral synapses requires integration of bot

146 ptors (5-HT(1B)Rs), without affecting nearby Schaffer collateral synapses.

147 iated excitation at TA-CA1 synapses, but not Schaffer collateral-CA1 synapses, after CUS, with a corr

148 ired synaptic inputs from entorhinal but not Schaffer-collateral afferents.

149 Notably, Schaffer activity may run laterally, as seen after the d

150 rons depends on the coincident activation of Schaffer collateral and temporoammonic inputs at the dis

151 utamate released by repetitive activation of Schaffer collaterals activates group I metabotropic glut

152 t can be facilitated by modest activation of Schaffer-collateral synapses in the upper apical dendrit

153 odependent mechanism involving activation of Schaffer-specific NMDA receptors and presynaptic inhibit

154 Variance-mean analyses of Schaffer collateral release probability (P(r)) at varyin

155 forming synapses with the impinging axons of Schaffer collateral.

156 tential-evoked [Ca(2+)](i) in the boutons of Schaffer collaterals.

157 dendrites that receive a uniform density of Schaffer collateral input (approximately 100-250 microme

158 tial strength and the short-term dynamics of Schaffer collateral excitatory synapses are regulated by

159 stigate in detail the short-term dynamics of Schaffer collateral excitatory synapses onto CA1 stratum

160 of estradiol on the functional integrity of Schaffer collateral to CA1 (Sch-CA1) pyramidal cell syna

161 B, we measured long-term depression (LTD) of Schaffer-collateral synapses in the hippocampus of c-Rel

162 c deletion of TRPA1 channels affected LTP of Schaffer collateral to CA1 pyramidal neuron synapses.

163 induction of long-term potentiation (LTP) of Schaffer collateral inputs to hippocampal CA1 pyramidal

164 CRS reduced long-term potentiation (LTP) of Schaffer collateral/commisural-CA1 pathway, phospho-alph

165 bserved that stress reduced the magnitude of Schaffer collateral/commissural-Cornu Ammonis field 1 lo

166 tion (LTP) induced by theta-burst pairing of Schaffer collateral inputs and postsynaptic firing is as

167 ally depotentiates long-term potentiation of Schaffer collateral (SC) synapses.

168 on markedly impairs synaptic potentiation of Schaffer collaterals and commissural inputs to the CA1 a

169 ther increase or decrease the probability of Schaffer-collateral (SC)-evoked CA1 spikes.

170 in the fundamental presynaptic properties of Schaffer collateral synapses that could account for dist

171 Prolonged 1 Hz stimulation (900 pulses) of Schaffer collaterals, which normally depresses CA3/CA1 s

172 Eliprodil markedly improved the recovery of Schaffer collateral-CA1 excitatory postsynaptic potentia

173 slices, we find that short trains (2-3 s) of Schaffer collateral fiber stimulation delivered at 5 Hz

174 production of distance-dependent scaling of Schaffer collateral synapses is an elevated postsynaptic

175 nt role in the distance-dependent scaling of Schaffer collateral synapses.

176 pocampal slices after tetanic stimulation of Schaffer collateral synapses.

177 We find that stimulation of Schaffer collateral/commissural fibers in hippocampal sl

178 TP elicited by either tetanic stimulation of Schaffer collaterals or a pairing protocol, and persiste

179 LTP) evoked by high-frequency stimulation of Schaffer collaterals, and that CN2097 attenuates this LT

180 re also evident after tetanic stimulation of Schaffer collaterals.

181 mostly representing excitatory terminals of Schaffer collaterals.

182 ivity at various times after tetanization of Schaffer collateral/commissural-CA1 synapses.

183 The basal synaptic transmission of Schaffer collateral-CA1 neurons was normal but long-term

184 Building on Schaffer's finite population model, we define the large

185 eduction of O-GlcNAc levels had no effect on Schaffer collateral CA1 basal hippocampal synaptic trans

186 the amygdala and examining stress effects on Schaffer collateral/commissural-CA1 LTP and spatial memo

187 CB1Rs are located on Schaffer collateral (Sc) axon terminals in the hippocamp

188 WT mice, but not in Prnp(0/0) mice, pairing Schaffer collateral stimulation with depolarization of C

189 In presynaptic Schaffer collateral-CA1 terminals of control hippocampal

190 ices in which the cell bodies of presynaptic Schaffer collateral/commissural fibres are removed.

191 (basket cells) or dendritically projecting (Schaffer collateral-associated cells) interneurons and p

192 s long-term potentiation at the CA1 proximal Schaffer collateral synapses when the two inputs are pai

193 as little effect on LTP at the more proximal Schaffer collateral inputs.

194 e magnitude of NMDAR-NR2B conductance at rat Schaffer collateral-CA1 synapses in vitro.

195 ross the range of apical dendrites receiving Schaffer collateral input.

196 Moreover, TRPV1 knockouts have reduced Schaffer collateral LTP, which is rescued by activating

197 TORC1-mediated late-LTP in SOM-INs regulated Schaffer collateral pathway LTP in pyramidal neurons.

198 lecular composition between commonly studied Schaffer collateral synapses and perforant path-dentate

199 theta cycle interval by proximally targeted Schaffer collateral activity, temporoammonic EPSPs propa

200 We found that Schaffer-specific LFPs are composed of a regular success

201 eurons containing somatostatin, we show that Schaffer collateral synapses onto the EGFP-expressing so

202 The Schaffer collateral pathway provides hippocampal CA1 pyr

203 in two separate afferent pathways among the Schaffer collaterals during intracellular recording of C

204 llel fiber to Purkinje cell synapse, and the Schaffer collateral to CA1 pyramidal cell synapse.

205 t path (PP) of the entorhinal cortex and the Schaffer collaterals (SCs) from individual CA3 pyramidal

206 ly mGlu7 is expressed presynaptically at the Schaffer collateral (SC)-CA1 synapse in the hippocampus

207 forms of long-term potentiation (LTP) at the Schaffer collateral CA1 synapse require stimulation of b

208 receptors modulate synaptic function at the Schaffer collateral input to CA1 pyramidal cells, thereb

209 report similar changes in plasticity at the Schaffer collateral pathway in alert behaving mice.

210 icited by such heterosynaptic pairing at the Schaffer collateral synapse in mice.

211 ng-term synaptic potentiation (L-LTP) at the Schaffer collateral synapse of the hippocampus is an exp

212 activity induces synaptic plasticity at the Schaffer collateral synapse onto CA1 pyramidal neurones.

213 tential diversity of nAChR influences at the Schaffer collateral synapse onto CA1 pyramidal neurons.

214 N-type voltage-gated Ca(2+) channels at the Schaffer collateral than perforant path synapses.

215 tate gyrus as well as decremental LTP at the Schaffer collateral-->CA1 pyramidal cell synapse.

216 PG-induced inhibition of transmission at the Schaffer collateral-->CA1 synapse.

217 F-dependent long-lasting potentiation at the Schaffer collateral-CA1 synapse in hippocampus.

218 iation (LTP) of synaptic transmission at the Schaffer collateral-CA1 synapse in the hippocampus is su

219 Long-term potentiation (LTP) at the Schaffer collateral-CA1 synapse involves interacting sig

220 e to block agonist-mediated responses at the Schaffer collateral-CA1 synapse, a location at which neu

221 nhibition of group III mGluR function at the Schaffer collateral-CA1 synapse.

222 We examined this question at the Schaffer collateral-CA1 synapses in slices of mouse hipp

223 z/1 s) in control slices elicited LTP at the Schaffer collateral-CA1 synapses, but neither LTP nor LT

224 ion and the isolated NMDAR potentials at the Schaffer collateral-CA1 synapses, but without affecting

225 remental long-term potentiation (LTP) at the Schaffer collateral-CA1 synapses.

226 Long-term potentiation (LTP) at the Schaffer collateral-commissural synapses in the CA1 area

227 otentiation of long-term potentiation at the Schaffer collateral/cornu ammonis 1 synapse in the dorsa

228 ials and long-term potentiation (LTP) at the Schaffer collaterals-CA1 synapse.

229 synaptic plasticity was investigated at the Schaffer-collateral-CA1 pyramidal cell synapse of mouse

230 ctionally important interactions between the Schaffer collateral and perforant pathways have been hyp

231 al synapses requires integration of both the Schaffer collateral and temporoammonic pathways.

232 gh CA3 pyramidal cells give rise to both the Schaffer collateral fiber synapses in CA1 and the assoc-

233 opment, basic synaptic transmission from the Schaffer collateral pathway to CA1 pyramidal neurons rem

234 ly established long-term potentiation in the Schaffer collateral (SC) pathway through complex signali

235 P16-CREB, enhanced in vivo LTP evoked in the Schaffer collateral pathway and caused significant chang

236 Long-term potentiation (LTP) in the Schaffer collateral pathway from the CA3 to the CA1 regi

237 ction of long-term potentiation (LTP) in the Schaffer collateral pathway of CA1 pyramidal neurons and

238 Neuronal stimulation in the Schaffer collateral pathway promotes pS857 dephosphoryla

239 rine) on input-output (I-O) relations in the Schaffer collateral pathway to CA1 (SC-CA1) and mossy fi

240 y-phase LTP to late-phase LTP (L-LTP) in the Schaffer collateral pathway, likely as a result of incre

241 ircuit-specific and were not observed in the Schaffer collateral pathway-associated inhibitory synaps

242 circuit-specific and are not observed in the Schaffer collateral pathway-associated inhibitory synaps

243 ase of long-term potentiation (L-LTP) in the Schaffer collateral pathway.

244 ase of long-term potentiation (L-LTP) in the Schaffer collateral pathway.

245 phase of long-term potentiation (LTP) in the Schaffer collateral pathway.

246 ession and facilitation were apparent in the Schaffer collateral synapse.

247 d that deletion of Cdc42 impaired LTP in the Schaffer collateral synapses and postsynaptic structural

248 be related to impairment of the E-LTP in the Schaffer collateral synapses but not of that of the perf

249 and -independent forms of LTP induced in the Schaffer collateral to CA1 synapses.

250 erm potentiation (LTP) can be induced in the Schaffer collateral-->CA1 synapse of hippocampus by stim

251 sion and long-term potentiation (LTP) in the Schaffer Collateral-CA1 and the mossy fiber-CA3 pathways

252 as enhanced paired-pulse facilitation in the Schaffer Collateral-CA1 glutamatergic synapses of the cb

253 implanted with stimulating electrodes in the Schaffer collaterals and with recording electrodes in th

254 gh and low levels of nAChR activation in the Schaffer collaterals inhibit and facilitate, respectivel

255 pecifically and quickly to affect LTP in the Schaffer collaterals of the hippocampus.

256 at hippocampal long-term potentiation in the Schaffer collaterals was identical in homozygous, hetero

257 very different time-varying responses in the Schaffer-commissural projections than in the lateral per

258 One week after transection of the Schaffer collateral and temporoammonic afferents to CA1

259 PSPs evoked by electrical stimulation of the Schaffer collateral inputs.

260 Here we show that tetanic stimulation of the Schaffer collateral pathway causes an increase in the co

261 roscopy revealed that LTP stimulation of the Schaffer collateral pathway promoted MAP2 labeling in sp

262 tratum radiatum following stimulation of the Schaffer collateral pathway.

263 y processes or long-term potentiation of the Schaffer collateral pathway.

264 requency electrical stimulation (LFS) of the Schaffer collateral pathway.

265 esynaptic and postsynaptic components of the Schaffer collateral synapse.

266 These results suggest that part of the Schaffer collateral synaptic response has pharmacologica

267 h that under intact pharmacology, LTP of the Schaffer collateral to CA1 pyramidal neuron synapses wil

268 Through electrophysiological analysis of the Schaffer collateral-CA1 synapse in dorsal hippocampus, w

269 Brief stimulation of the Schaffer collaterals elicited an alkaline pH(e) transien

270 egion of the hippocampus, stimulation of the Schaffer collaterals elicits an alkaline pH(e) transient

271 We found that orthodromic stimulation of the Schaffer collaterals for 10 min reduces the amplitude of

272 In hippocampus, activation of the Schaffer collaterals generates an extracellular alkaline

273 SCs) evoked by electrical stimulation of the Schaffer collaterals in CA1 hippocampal pyramidal cells.

274 sociated with the axons and terminals of the Schaffer collaterals.

275 rea CA1 following tetanic stimulation of the Schaffer collaterals.

276 tion spike by orthodromic stimulation of the Schaffer collaterals.

277 ippocampus in response to stimulation of the Schaffer collaterals.

278 in response to electrical stimulation of the Schaffer collaterals.

279 no expression within the perforant path, the Schaffer collaterals, or neuronal cell bodies.

280 were determined in acute brain sections; the Schaffer collateral pathway was stimulated and the field

281 We stimulated the Schaffer collateral/commissural fibers in acute hippocam

282 CA or sham-operated rats by stimulating the Schaffer collaterals and recording in the CA1 pyramidal

283 ed before (control) and after tetanizing the Schaffer/collateral commissural pathway.

284 gic and has a larger NMDA component than the Schaffer collateral (sc) input.

285 of the normal CA3 outflow tract through the Schaffer collaterals are well known, their aberrant reor

286 paired-pulse stimulation was applied to the Schaffer collaterals and population spikes were monitore

287 oammonic (TA) pathway and indirectly via the Schaffer collateral (SC) pathway from CA3.

288 nged in epileptic animals, and input via the Schaffer collaterals is actually decreased despite reduc

289 the perforant path (PP) and from CA3 through Schaffer collaterals (SC).

290 e perforant path (PP) and indirectly through Schaffer collaterals (SC).

291 m potentiation (LTP) when locally applied to Schaffer collateral-CA1 synapses.

292 Evoked responses of CA1 neurons to Schaffer collateral stimulation changed over the cycle,

293 atio onto CA1 pyramidal cells in response to Schaffer collateral stimulation in slices from young adu

294 ratio of CA1 pyramidal cells in response to Schaffer collateral stimulation.

295 udies revealed that the amplitude of unitary Schaffer collateral inputs increases with distance from

296 The present findings show that, using Schaffer collateral stimulation, the effects of SNX and

297 mission and plasticity at dorsal and ventral Schaffer collateral (SC) synapses in the mouse hippocamp

298 e reported conflicting results as to whether Schaffer collateral axons have target-cell specific shor

299 ine (10 microM) enhanced NMDAR currents with Schaffer collateral stimulation in WT mice but not HZ mi

300 g of back-propagating action potentials with Schaffer collateral EPSPs was accompanied by an overall