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1 hat of Enterobacteria (mainly Thorsellia and Serratia).
2 reduced the abundance of the basal resource (Serratia).
3 s of metabolic and anatomical integration of Serratia.
4 strains of the opportunistic human pathogen, Serratia.
5 emergence of clinical and other lineages of Serratia.
6 e largest conjugative plasmids described for Serratia.
7 n pathways in Buchnera lead to dependence on Serratia.
8 years; Nocardia, 0.81 per 100 patient-years; Serratia, 0.98 per 100 patient-years, and severe Staphyl
9 y transcriptional activator CarR(39006) from Serratia 39006 has no detectable affinity for cognate AH
12 ver the first year were associated with high Serratia abundance, pro-inflammatory innate cytokines, a
13 toxin, showing a remarkable predominance of Serratia and Clostridium species, which switched from as
18 ers of two genera of Gram-negative bacteria, Serratia and Erwinia, produce a beta-lactam antibiotic,
19 d the distribution of T6SSs across the genus Serratia and observed that a highly conserved antibacter
20 convenient spectrophotometric assay, by the Serratia and Pseudomonas approximately 50-kDa extracellu
22 s such as the non-specific endonuclease from Serratia and the sequence-specific His-Cys box homing en
24 ogenomic analysis of 3 genera, Enterobacter, Serratia, and Elizabethkingia, reveal lineages of mosqui
25 genomic, phenotypic and plasmid diversity of Serratia, and provide evidence of different patterns of
26 . subtilis colonies, swarming by Proteus and Serratia, and spatially organized interspecific metaboli
31 However, estimated rates of encounter with Serratia based on these modifications were higher for in
34 cosmid containing approximately 35 kb of the Serratia chromosome encodes synthesis of the pigment in
35 We report the isolation of a third locus in Serratia, containing convergently transcribed genes, sma
37 tom are conserved in nucleases homologous to Serratia endonuclease, suggesting that the water cluster
41 rom other insect-associated bacteria such as Serratia entomophila, an insect pathogen, and Yersinia p
43 ures of the class A carbapenemase SFC-1 from Serratia fonticola and of complexes of its Ser70 Ala (Mi
44 comprised of (i) a bacterial basal resource (Serratia fonticola), (ii) an intermediate consumer (Para
46 ing to the genera Bacillus, Macrococcus, and Serratia genera were depleted in Salmonella inoculated m
47 monella, Escherichia, Bacillus, Pseudomonas, Serratia, Hafnia, Enterobacter, Citrobacter, and Lactoba
49 al ABC iron transporters that include Sfu of Serratia, Hit of Haemophilus, and Yfu of Yersinia entero
50 stems: I-F (Pseudomonas, Pectobacterium, and Serratia), I-E (Pseudomonas and Serratia), and I-C (Pseu
51 espite a poorly conserved sequence, which in Serratia includes a cysteine bridge thought to play a re
52 pithelial cell and prior to internalization, Serratia induces an early autophagic response that is en
57 Providencia alcalifaciens, P. rustigianii, Serratia liquefaciens and S. plymuthica strains were stu
58 wth curves of two hypopiezotolerant strains, Serratia liquefaciens and Trichococcus pasteurii, were p
61 ble protease Ser2 is secreted by the species Serratia liquefaciens, a psychrotrophic bacteria frequen
65 bacter species (3%), Proteus mirabilis (2%), Serratia marcescens (0.6%), and Pseudomonas aeruginosa (
66 abilis (9), Pseudomonas aeruginosa (10), and Serratia marcescens (1), were included; and 123 (98.4%)
68 r cloacae (9.1%), Acinetobacter spp. (6.2%), Serratia marcescens (5.5%), Enterobacter aerogenes (4.4%
70 was the main cause of neonatal sepsis, with Serratia marcescens (n = 151), Klebsiella michiganensis
71 loacae (n = 23), Klebsiella oxytoca (n = 8), Serratia marcescens (n = 6), Citrobacter freundii (n = 4
72 domonas aeruginosa (n = 2 and n = 5), two of Serratia marcescens (n = 9 and n = 7), five of Staphyloc
74 ins using a targeted protease, Enhancin from Serratia marcescens (SmE), with ultraviolet photodissoci
75 east histone acetyltransferase 1) and SmAAT (Serratia marcescens aminoglycoside 3-N-acetyltransferase
76 luding yeast histone acetyltransferase 1 and Serratia marcescens aminoglycoside 3-N-acetyltransferase
77 cal GCN5-related N-acetyltransferase (GNAT), Serratia marcescens aminoglycoside 3-N-acetyltransferase
78 sin, a microbial pigment, was produced using Serratia marcescens and encapsulated with beta-cyclodext
79 neumoniae, Acinetobacter baumannii, E. coli, Serratia marcescens and Enterobacter cloacae complex.
80 sequences of the cheA loci from isolates of Serratia marcescens and Enterobacter cloacae, demonstrat
81 the aspartate transcarbamoylases (ATCase) of Serratia marcescens and Escherichia coli differ in both
82 the aspartate transcarbamoylases (ATCase) of Serratia marcescens and Escherichia coli have distinct a
83 entical to the natural product isolated from Serratia marcescens and from overexpression of the biosy
84 sfu and hit operons previously reported for Serratia marcescens and Haemophilus influenzae, respecti
85 ence time of TLM on the ecFabB homologues in Serratia marcescens and Klebsiella pneumonia is an impor
86 obacter hormaechei, Acinetobacter baumannii, Serratia marcescens and Leclercia adecarboxylata are dom
88 rval stages and in the ants of two bacteria, Serratia marcescens and S. entomophila, which are involv
89 Ds accumulate in midgut cells in response to Serratia marcescens and Sindbis virus or when the native
90 ditis elegans against the bacterial parasite Serratia marcescens and tested for a correlation between
91 nces were noted among Acinetobacter spp. and Serratia marcescens and, to a lesser extent, with Strept
92 in antibiotic-treated mosquitoes identified Serratia marcescens as a commensal bacterium critical fo
95 ybrid microswimmer system driven by multiple Serratia marcescens bacteria, we quantify the chemotacti
98 idguts after they fed on the insect pathogen Serratia marcescens but not after feeding on the Leishma
100 functional antibiotic resistance enzyme from Serratia marcescens catalyzes adenylation and acetylatio
101 egative bacterium and opportunistic pathogen Serratia marcescens causes ocular infections in healthy
104 , sensitive detection of Escherichia coli or Serratia marcescens cultures from 1 to 10(3) CFU mL(-1).
110 ns of well-known chitinases and an LPMO from Serratia marcescens Importantly, comparison of the chiti
112 alf-site pairs of the trpEDCBA operator from Serratia marcescens indicated an obligate hierarchy of s
113 eudo-outbreaks of Pseudomonas aeruginosa and Serratia marcescens infections associated with bronchosc
115 ucts generated by the opportunistic pathogen Serratia marcescens involved in activation of autophagy.
134 olates, 6 Pseudomonas aeruginosa isolates, 1 Serratia marcescens isolate, 1 Aeromonas hydrophila isol
136 to 0.5 nM alpha-thrombin by only 10% whereas Serratia marcescens metalloprotease reduced the Ca2+ res
137 ion of extracellular nuclease (Nuc) from the Serratia marcescens nucA chromosomal locus is inhibited
141 e that is located upstream of NucC-dependent Serratia marcescens promoters and the late promoters of
142 sented here in complex with chitinase B from Serratia marcescens provide further insight into the mec
143 gations, we discover that the E2 enzyme from Serratia marcescens regulates cGAS by imitating the ubiq
145 and hhdB, which, based on their homology to Serratia marcescens shlA and shlB genes, are believed to
146 ith exogenous bacteria (Enterobacter sp. and Serratia marcescens strain Db11) and parasitic African t
152 lated data and experimental Tn-Seq data from Serratia marcescens transposon mutant library used to id
153 instance, the chitinase secretion pathway of Serratia marcescens uses an endopeptidase to facilitate
154 ce of infection with Burkholderia cepacia or Serratia marcescens was caused by a new strain in 9 of 1
155 ely 10,000 nM), and Enterobacter cloacae and Serratia marcescens were highly resistant (IC(50), >10,0
156 e, Proteus spp., Pseudomonas aeruginosa, and Serratia marcescens) and 6 antimicrobial resistance dete
157 colonization by a focal non-core bacterium (Serratia marcescens) and its consequences for bee health
158 that coevolution with a bacterial pathogen (Serratia marcescens) resulted in significantly more outc
159 roPhenoloxidase activity, resistance against Serratia marcescens), and for the life history traits, a
160 ntoea agglomerance., Microbacterium sp., and Serratia marcescens), and their nine mixture treatments
161 umoniae, 21 Enterobacter cloacae complex, 18 Serratia marcescens, 12 Proteus mirabilis, 10 Citrobacte
162 the clearance of a bacterial infection with Serratia marcescens, 3 Acps significantly reduced the ba
166 structure of anthranilate synthase (AS) from Serratia marcescens, a mesophilic bacterium, has been so
167 no known homologues, a homologue of OmpF of Serratia marcescens, and a locus (designated rscBAC) wit
169 revealed eradication of Pseudomonas species, Serratia marcescens, and Enterobacter aerogenes in most
171 trobacter freundii, Yersinia enterocolitica, Serratia marcescens, and Morganella morganii) and two no
172 (Neisseria gonorrhoeae and N. meningitidis), Serratia marcescens, and other gram-negative bacteria ut
174 rium tumefaciens, Agrobacterium radiobacter, Serratia marcescens, and Pseudomonas aureofaciens) and f
175 st similar to biotin synthases from E. coli, Serratia marcescens, and Saccharomyces cerevisiae (about
176 inst Burkholderia cepacia, Escherichia coli, Serratia marcescens, and Stenotrophomonas maltophilia is
177 we identify a common fecal enterobacterium, Serratia marcescens, as the causal agent of white pox.
178 resistant species like B. thailandensis and Serratia marcescens, but also a majority of Gram-negativ
179 ngs were observed with another CGD pathogen, Serratia marcescens, but not with Escherichia coli.
181 of the enterobacteria Klebsiella pneumoniae, Serratia marcescens, Erwinia carotovora, and Proteus vul
182 the enteric bacteria Klebsiella pneumoniae, Serratia marcescens, Erwinia carotovora, and Proteus vul
183 nt algorithms, especially in differentiating Serratia marcescens, Escherichia coli, and Yersinia ente
184 phore secreted by the Gram-negative bacteria Serratia marcescens, extracts heme from host hemoprotein
185 Klebsiella pneumoniae, Escherichia coli, and Serratia marcescens, for which the trend is >= 30% and t
186 Klebsiella pneumoniae, Escherichia coli, and Serratia marcescens, in a further nine of 11 (81%) cases
187 ered by a T6SS of the opportunistic pathogen Serratia marcescens, is a toxin that forms ion-selective
188 film formation in the opportunistic pathogen Serratia marcescens, mutations in an oxyR homolog and pr
190 that contained group B Streptococcus (GBS), Serratia marcescens, or Escherichia coli before their se
191 is (20%, 3 of 15), and Enterobacter cloacae, Serratia marcescens, Pneumocystis carinii pneumonia, and
192 monas aeruginosa PAO1, Proteus mirabilis and Serratia marcescens, possibly by interfering with their
193 gens (Escherichia coli, Salmonella muenchen, Serratia marcescens, Proteus mirabilis, and Proteus vulg
194 li, Salmonella enterica serovar Typhimurium, Serratia marcescens, Shigella flexneri, Enterobacter aer
195 ebsiella pneumoniae, Pseudomonas aeruginosa, Serratia marcescens, Staphylococcus aureus, and Stenotro
196 caused by ingesting the pathogenic bacteria Serratia marcescens, suggesting that subdued has novel f
197 d the same set of D. melanogaster lines with Serratia marcescens, the bacterium used in the previous
198 nsect pathogens (the gram-negative bacterium Serratia marcescens, the gram-positive bacterium Bacillu
199 rate that Rhs1 effectors from two strains of Serratia marcescens, the model strain Db10 and clinical
200 coli, Pseudomonas spp., Salmonella enterica, Serratia marcescens, Vibrio vulnificus and Enterobacter
201 to consume the virulent bacterial parasite, Serratia marcescens, when given a choice between the par
202 t the lipases produced by P. fluorescens and Serratia marcescens, which comprise a second sequence fa
203 e describe the structure of chitinase B from Serratia marcescens, which consists of a catalytic domai
204 FLP analysis except for Escherichia coli and Serratia marcescens, which could not be interdifferentia
205 ana, we isolated the Gram-negative bacterium Serratia marcescens, which is a potent entomopathogen th
224 se structures suggests that the magnesium of Serratia nuclease participates in catalysis via an inner
225 analog for which structural data exist, the Serratia nuclease, indicates several interesting differe
226 On the genus level, FF mice had increased Serratia (P < 0.001) and Lactococcus (P < 0.05) whereas
229 elin, a siderophore previously identified in Serratia plymuthica Serratiochelin-producing mutants als
230 irulent bacteriophage (PhiMAM1) that infects Serratia plymuthica was isolated from the natural enviro
231 hin a community dominated by a nearly clonal Serratia population and harboring a lower abundance Ente
234 hibitor recently discovered in the bacterium Serratia proteamaculans and the prototype of a new famil
236 ble to protease degradation and suggest that Serratia protease is able to differentiate the GPIb-medi
241 a high-resolution crystal structure for the Serratia sp. ATCC 39006 carbapenem resistance protein Ca
246 enterobacterium Erwinia (Pectobacterium) and Serratia sp. ATCC 39006, intrinsic resistance to the car
249 he co-culture exhibits bimodal growth with a Serratia sp. fermenting pyruvate followed by hydrogenotr
250 ponsible for prodigiosin biosynthesis in two Serratia sp. In this article we report the creation of i
251 gmented antibiotic, prodigiosin, produced by Serratia sp. is known to involve separate pathways for t
253 ic activity is essential for cytotoxicity in Serratia sp. SCBI and that its regulation appears to be
254 ion of predicted protease genes in wild-type Serratia sp. SCBI, the highest mRNA levels for the alkal
257 ntial for extracellular protease activity in Serratia sp. strain SCBI and to determine what role prot
259 l interactions, with the pyruvate fermenting Serratia sp. supplying amino acids as essential growth f
260 host-associated Gammaproteobacteria species (Serratia sp.) that was absent from soil yet observed in
263 coli less common in COVID-19 patients, while Serratia species was more often identified in late VA-LR
266 nal fluid cultures growing Enterobacter spp, Serratia spp, or Citrobacter spp were evaluated using th
267 uginosa (n = 14), Proteus mirabilis (n = 3), Serratia spp. (n = 10), Stenotrophomonas maltophilia (n
268 terococcus spp., Pseudomonas aeruginosa, and Serratia spp. were recovered from infected devices, whil
269 ingle patient isolates of Enterobacter spp., Serratia spp., Citrobacter spp., and Pseudomonas aerugin
270 differentiate Klebsiella, Enterobacter, and Serratia spp., enteric pathogens were identified only by
274 ions (q-value < 0.05) between Acinetobacter, Serratia, Streptococcus and Bacillus inferred abundances
275 osing bees with disrupted gut microbiomes to Serratia strongly reduced lifespan and, as a result, als
276 tative symbionts, especially the presence of Serratia symbiotica (Enterobacterales: Yersiniaceae).
277 in aphids, involving Buchnera aphidicola and Serratia symbiotica in the Lachninae subfamily [7-9].
279 nts (Arsenophonus sp., Hamiltonella defensa, Serratia symbiotica, and Regiella insecticola), in black
280 catalytic residue corresponding to Arg57 in Serratia, the structure determined here indicates that A
282 f a Plasmodium-blocking symbiotic bacterium, Serratia ureilytica Su_YN1, isolated from the midgut of