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1 0C of Streptomyces coelicolor and SmAA10A of Serratia marcescens.
2 system on intrinsic multidrug resistance in Serratia marcescens.
3 ative bacteria, including the human pathogen Serratia marcescens.
4 monas aeruginosa, Staphylococcus aureus, and Serratia marcescens.
5 between closely related clinical isolates of Serratia marcescens.
6 One example is serratin, isolated from Serratia marcescens.
7 study of the processive chitinase ChiA from Serratia marcescens.
8 ypocrea jecorina and the chitinase ChiA from Serratia marcescens.
9 ost resistance test using the live bacterium Serratia marcescens.
10 controls, including Clostridium species and Serratia marcescens.
11 T6SS from the opportunistic human pathogen, Serratia marcescens.
12 y 18 nonprocessive endochitinase, ChiC, from Serratia marcescens.
13 display a TagJ homologue as shown here with Serratia marcescens.
14 nce to infection with the bacterial pathogen Serratia marcescens.
15 infection by a Gram-negative entomopathogen, Serratia marcescens.
16 acter freundii group, Enterobacter spp., and Serratia marcescens.
17 em receptors from Pseudomonas aeruginosa and Serratia marcescens.
18 bacter species (3%), Proteus mirabilis (2%), Serratia marcescens (0.6%), and Pseudomonas aeruginosa (
19 abilis (9), Pseudomonas aeruginosa (10), and Serratia marcescens (1), were included; and 123 (98.4%)
21 umoniae, 21 Enterobacter cloacae complex, 18 Serratia marcescens, 12 Proteus mirabilis, 10 Citrobacte
22 the clearance of a bacterial infection with Serratia marcescens, 3 Acps significantly reduced the ba
23 r cloacae (9.1%), Acinetobacter spp. (6.2%), Serratia marcescens (5.5%), Enterobacter aerogenes (4.4%
27 structure of anthranilate synthase (AS) from Serratia marcescens, a mesophilic bacterium, has been so
28 luding yeast histone acetyltransferase 1 and Serratia marcescens aminoglycoside 3-N-acetyltransferase
29 cal GCN5-related N-acetyltransferase (GNAT), Serratia marcescens aminoglycoside 3-N-acetyltransferase
30 east histone acetyltransferase 1) and SmAAT (Serratia marcescens aminoglycoside 3-N-acetyltransferase
31 sin, a microbial pigment, was produced using Serratia marcescens and encapsulated with beta-cyclodext
32 neumoniae, Acinetobacter baumannii, E. coli, Serratia marcescens and Enterobacter cloacae complex.
33 sequences of the cheA loci from isolates of Serratia marcescens and Enterobacter cloacae, demonstrat
34 the aspartate transcarbamoylases (ATCase) of Serratia marcescens and Escherichia coli differ in both
35 the aspartate transcarbamoylases (ATCase) of Serratia marcescens and Escherichia coli have distinct a
36 entical to the natural product isolated from Serratia marcescens and from overexpression of the biosy
37 sfu and hit operons previously reported for Serratia marcescens and Haemophilus influenzae, respecti
38 ence time of TLM on the ecFabB homologues in Serratia marcescens and Klebsiella pneumonia is an impor
39 obacter hormaechei, Acinetobacter baumannii, Serratia marcescens and Leclercia adecarboxylata are dom
41 rval stages and in the ants of two bacteria, Serratia marcescens and S. entomophila, which are involv
42 Ds accumulate in midgut cells in response to Serratia marcescens and Sindbis virus or when the native
43 ditis elegans against the bacterial parasite Serratia marcescens and tested for a correlation between
44 nces were noted among Acinetobacter spp. and Serratia marcescens and, to a lesser extent, with Strept
45 e, Proteus spp., Pseudomonas aeruginosa, and Serratia marcescens) and 6 antimicrobial resistance dete
46 colonization by a focal non-core bacterium (Serratia marcescens) and its consequences for bee health
47 roPhenoloxidase activity, resistance against Serratia marcescens), and for the life history traits, a
48 ntoea agglomerance., Microbacterium sp., and Serratia marcescens), and their nine mixture treatments
49 no known homologues, a homologue of OmpF of Serratia marcescens, and a locus (designated rscBAC) wit
51 revealed eradication of Pseudomonas species, Serratia marcescens, and Enterobacter aerogenes in most
53 trobacter freundii, Yersinia enterocolitica, Serratia marcescens, and Morganella morganii) and two no
54 (Neisseria gonorrhoeae and N. meningitidis), Serratia marcescens, and other gram-negative bacteria ut
56 rium tumefaciens, Agrobacterium radiobacter, Serratia marcescens, and Pseudomonas aureofaciens) and f
57 st similar to biotin synthases from E. coli, Serratia marcescens, and Saccharomyces cerevisiae (about
58 inst Burkholderia cepacia, Escherichia coli, Serratia marcescens, and Stenotrophomonas maltophilia is
59 in antibiotic-treated mosquitoes identified Serratia marcescens as a commensal bacterium critical fo
61 we identify a common fecal enterobacterium, Serratia marcescens, as the causal agent of white pox.
63 ybrid microswimmer system driven by multiple Serratia marcescens bacteria, we quantify the chemotacti
66 idguts after they fed on the insect pathogen Serratia marcescens but not after feeding on the Leishma
67 resistant species like B. thailandensis and Serratia marcescens, but also a majority of Gram-negativ
70 functional antibiotic resistance enzyme from Serratia marcescens catalyzes adenylation and acetylatio
71 egative bacterium and opportunistic pathogen Serratia marcescens causes ocular infections in healthy
74 , sensitive detection of Escherichia coli or Serratia marcescens cultures from 1 to 10(3) CFU mL(-1).
78 the enteric bacteria Klebsiella pneumoniae, Serratia marcescens, Erwinia carotovora, and Proteus vul
79 of the enterobacteria Klebsiella pneumoniae, Serratia marcescens, Erwinia carotovora, and Proteus vul
80 nt algorithms, especially in differentiating Serratia marcescens, Escherichia coli, and Yersinia ente
84 phore secreted by the Gram-negative bacteria Serratia marcescens, extracts heme from host hemoprotein
85 Klebsiella pneumoniae, Escherichia coli, and Serratia marcescens, for which the trend is >= 30% and t
87 ns of well-known chitinases and an LPMO from Serratia marcescens Importantly, comparison of the chiti
89 Klebsiella pneumoniae, Escherichia coli, and Serratia marcescens, in a further nine of 11 (81%) cases
90 alf-site pairs of the trpEDCBA operator from Serratia marcescens indicated an obligate hierarchy of s
91 eudo-outbreaks of Pseudomonas aeruginosa and Serratia marcescens infections associated with bronchosc
93 ucts generated by the opportunistic pathogen Serratia marcescens involved in activation of autophagy.
111 ered by a T6SS of the opportunistic pathogen Serratia marcescens, is a toxin that forms ion-selective
113 olates, 6 Pseudomonas aeruginosa isolates, 1 Serratia marcescens isolate, 1 Aeromonas hydrophila isol
115 to 0.5 nM alpha-thrombin by only 10% whereas Serratia marcescens metalloprotease reduced the Ca2+ res
116 film formation in the opportunistic pathogen Serratia marcescens, mutations in an oxyR homolog and pr
117 was the main cause of neonatal sepsis, with Serratia marcescens (n = 151), Klebsiella michiganensis
118 loacae (n = 23), Klebsiella oxytoca (n = 8), Serratia marcescens (n = 6), Citrobacter freundii (n = 4
119 domonas aeruginosa (n = 2 and n = 5), two of Serratia marcescens (n = 9 and n = 7), five of Staphyloc
120 ion of extracellular nuclease (Nuc) from the Serratia marcescens nucA chromosomal locus is inhibited
125 that contained group B Streptococcus (GBS), Serratia marcescens, or Escherichia coli before their se
126 is (20%, 3 of 15), and Enterobacter cloacae, Serratia marcescens, Pneumocystis carinii pneumonia, and
127 monas aeruginosa PAO1, Proteus mirabilis and Serratia marcescens, possibly by interfering with their
128 e that is located upstream of NucC-dependent Serratia marcescens promoters and the late promoters of
129 gens (Escherichia coli, Salmonella muenchen, Serratia marcescens, Proteus mirabilis, and Proteus vulg
130 sented here in complex with chitinase B from Serratia marcescens provide further insight into the mec
131 gations, we discover that the E2 enzyme from Serratia marcescens regulates cGAS by imitating the ubiq
132 that coevolution with a bacterial pathogen (Serratia marcescens) resulted in significantly more outc
135 li, Salmonella enterica serovar Typhimurium, Serratia marcescens, Shigella flexneri, Enterobacter aer
136 and hhdB, which, based on their homology to Serratia marcescens shlA and shlB genes, are believed to
137 ins using a targeted protease, Enhancin from Serratia marcescens (SmE), with ultraviolet photodissoci
138 ebsiella pneumoniae, Pseudomonas aeruginosa, Serratia marcescens, Staphylococcus aureus, and Stenotro
139 ith exogenous bacteria (Enterobacter sp. and Serratia marcescens strain Db11) and parasitic African t
143 caused by ingesting the pathogenic bacteria Serratia marcescens, suggesting that subdued has novel f
144 d the same set of D. melanogaster lines with Serratia marcescens, the bacterium used in the previous
145 nsect pathogens (the gram-negative bacterium Serratia marcescens, the gram-positive bacterium Bacillu
146 rate that Rhs1 effectors from two strains of Serratia marcescens, the model strain Db10 and clinical
149 lated data and experimental Tn-Seq data from Serratia marcescens transposon mutant library used to id
150 instance, the chitinase secretion pathway of Serratia marcescens uses an endopeptidase to facilitate
151 coli, Pseudomonas spp., Salmonella enterica, Serratia marcescens, Vibrio vulnificus and Enterobacter
152 ce of infection with Burkholderia cepacia or Serratia marcescens was caused by a new strain in 9 of 1
153 ely 10,000 nM), and Enterobacter cloacae and Serratia marcescens were highly resistant (IC(50), >10,0
154 to consume the virulent bacterial parasite, Serratia marcescens, when given a choice between the par
155 t the lipases produced by P. fluorescens and Serratia marcescens, which comprise a second sequence fa
156 e describe the structure of chitinase B from Serratia marcescens, which consists of a catalytic domai
157 FLP analysis except for Escherichia coli and Serratia marcescens, which could not be interdifferentia
158 ana, we isolated the Gram-negative bacterium Serratia marcescens, which is a potent entomopathogen th