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1 mmonly has been used to distinguish GAS from Streptococcus agalactiae.
2 rial pathogens: Streptococcus pneumoniae and Streptococcus agalactiae.
3 -positive bacteria Staphylococcus aureus and Streptococcus agalactiae.
4 d to detect 2/3 clinical blood cultures with Streptococcus agalactiae.
5 function was conserved in Gap homologs from Streptococcus agalactiae.
6 hologous mutants of Streptococcus mutans and Streptococcus agalactiae.
7 rot broth culture alone for the detection of Streptococcus agalactiae.
9 atus (1), the Streptococcus bovis group (5), Streptococcus agalactiae (9), the Streptococcus anginosu
10 streptococcal enzymes, from S.pneumoniae and Streptococcus agalactiae, allowed for insights into this
13 cus pneumoniae, vancomycin, and ceftriaxone, Streptococcus agalactiae, ampicillin, and cefotaxime, Es
14 s multiplexed detection of Candida albicans, Streptococcus agalactiae and Chlamydia trachomatis with
16 GacI homologs perform a similar function in Streptococcus agalactiae and Enterococcus faecalis In co
17 monstrated the presence of a supragenome for Streptococcus agalactiae and Haemophilus influenzae, it
19 coccus pyogenes), group B streptococci (GBS; Streptococcus agalactiae), and Streptococcus pneumoniae
20 idermidis, 10 Staphylococcus lugdunensis, 10 Streptococcus agalactiae, and 10 Enterococcus faecalis)
21 illus plantarum, Neisseria meningitidis, and Streptococcus agalactiae, and 3% of the residues in its
22 nterococcus faecium, Streptococcus pyogenes, Streptococcus agalactiae, and Streptococcus pneumoniae w
23 terococcus faecalis, Streptococcus pyogenes, Streptococcus agalactiae, and viridans streptococci.
28 ens, such as the group B streptococcus (GBS) Streptococcus agalactiae, are an important cause of syst
33 esent report, we show that crude extracts of Streptococcus agalactiae catalyze the gamma-GCS and GS r
37 , Mycoplasma hominis, Neisseria gonorrhoeae, Streptococcus agalactiae, Chlamydia trachomatis, Trichom
39 model of Saccharomyces cerevisiae and one of Streptococcus agalactiae constructed using the KEGG data
40 eria meningitidis, Streptococcus pneumoniae, Streptococcus agalactiae, cytomegalovirus, enterovirus,
41 aks of invasive group B streptococcal (iGBS; Streptococcus agalactiae) disease in infants is unknown.
43 vo growth assays with Staphylococcus aureus, Streptococcus agalactiae, Escherichia coli and Klebsiell
44 ) values of 100% for Neisseria meningitidis, Streptococcus agalactiae, Escherichia coli K1, Listeria
45 tococcus pneumoniae, Streptococcus pyogenes, Streptococcus agalactiae, Escherichia coli, Klebsiella p
46 Enterococcus faecalis, Enterococcus faecium, Streptococcus agalactiae, Escherichia coli, Klebsiella p
48 ine and human macrophages induced by group B Streptococcus agalactiae (GBS) is likely an important vi
51 A streptococci), and recombinant SCPB, from Streptococcus agalactiae (group B streptococci), were co
52 ave previously shown that the human pathogen Streptococcus agalactiae (Group B Streptococci, GBS) enc
57 this study, we characterized 31 isolates of Streptococcus agalactiae (group B Streptococcus [GBS]) f
68 on of phoZ in Escherichia coli, E. faecalis, Streptococcus agalactiae (group B streptococcus [GBS]),
74 pyogenes gacB with the homologous gene from Streptococcus agalactiae (Group B Streptococcus), Strept
81 nthesized in high abundance by the bacterium Streptococcus agalactiae (Group B Streptococcus, GBS).
86 e is presented by the Gram-positive bacteria Streptococcus agalactiae (Group B Streptococcus; GBS) ty
87 rations with potentially pathogenic bacteria Streptococcus agalactiae (Group-B-Streptococci; GBS) and
93 cterium tuberculosis (also using GeneXpert), Streptococcus agalactiae, herpes simplex virus (types 1
96 cated in gastritis and peptic ulcer disease, Streptococcus agalactiae, implicated in neonatal meningi
97 perinatal treatment of women colonized with Streptococcus agalactiae include vancomycin prophylaxis
101 apsulated, intact Neisseria meningitidis nor Streptococcus agalactiae inhibited the OVA-specific IgG
108 Streptococcus pyogenes (</=0.12 microg/mL), Streptococcus agalactiae (</=0.12 microg/mL), Streptococ
110 tans (MetR), Streptococcus iniae (CpsY), and Streptococcus agalactiae (MtaR) that regulate methionine
115 sPCR fragment H10, similar to an unknown Streptococcus agalactiae protein, was present in 31% of
116 eudomonas aeruginosa, Klebsiella pneumoniae, Streptococcus agalactiae, Proteus, Enterococcus and Stap
119 ntly described the crystal structures of the Streptococcus agalactiae SAG2603 V/R sortase SrtC1 in tw
121 he whole-genome shotgun draft sequence for a Streptococcus agalactiae strain representing multilocus
123 Listeria monocytogenes, Streptococcus spp., Streptococcus agalactiae, Streptococcus anginosus group,
124 -species recombination in the pan-genomes of Streptococcus agalactiae, Streptococcus pyogenes and Str
125 ts, Staphylococcus aureus in 8 patients, and Streptococcus agalactiae, Streptococcus pyogenes, and St
127 the five major disease-causing serotypes of Streptococcus agalactiae, the main cause of neonatal inf
129 scherichia coli, Fusobacterium nucleatum and Streptococcus agalactiae to oligomannose N-glycans, gala
130 ed a mutant strain of group B Streptococcus (Streptococcus agalactiae) type III (GBS-III) that expres
132 nce and transmission of antibiotic-resistant Streptococcus agalactiae, we compared phenotypic and gen
134 corresponding region of a GspB homologue of Streptococcus agalactiae, which is acidic rather than ba