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1 pxB abundance in Streptococcus sanguinis and Streptococcus gordonii.
2 n the binding of Streptococcus anginosus and Streptococcus gordonii.
3 al transformation of Streptococcus mitis and Streptococcus gordonii.
4 the surface of the human commensal bacterium Streptococcus gordonii.
5 re supernatants of Staphylococcus aureus and Streptococcus gordonii.
6 the presence of antecedent organisms such as Streptococcus gordonii.
7 s on rates of hydrogen peroxide synthesis by Streptococcus gordonii.
8 by adhering to early plaque bacteria such as Streptococcus gordonii.
9 commensal species Streptococcus sanguinis or Streptococcus gordonii.
10 with the antecedent oral biofilm constituent Streptococcus gordonii.
11 attaches and forms biofilms on substrata of Streptococcus gordonii.
12 ggregation receptor polysaccharides (RPS) of Streptococcus gordonii 38 and Streptococcus oralis J22 w
16 dominant salivary enzyme in humans, binds to Streptococcus gordonii, a primary colonizer of the tooth
17 oped in this study assessed a multi-species (Streptococcus gordonii, Actinobacillus actinomycetemcomi
18 ultispecies bacteria (Enterococcus faecalis, Streptococcus gordonii, Actinomyces naeslundii, and Lact
21 equired for SRR glycoprotein export, Asp2 of Streptococcus gordonii also functions as an O-acetyltran
22 an important determinant of colonization by Streptococcus gordonii, an oral commensal and opportunis
23 nvestigated its role in biofilm formation by Streptococcus gordonii, an organism that colonizes human
24 erence of early colonizing organisms such as Streptococcus gordonii and Actinomyces naeslundii to the
25 en paired with two other initial colonizers, Streptococcus gordonii and Actinomyces oris, as well as
26 Structural studies of the type-C PPases from Streptococcus gordonii and Bacillus subtilis reveal a ho
28 nge between two bacterial species, commensal Streptococcus gordonii and pathogenic Streptococcus muta
33 the human oral flora (Streptococcus mutans, Streptococcus gordonii and Streptococcus sanguinis) to d
35 igher, respectively, in the S-ECC group, and Streptococcus gordonii and Streptococcus sanguinis, whic
37 dulated by coculture with the oral commensal Streptococcus gordonii and the opportunistic commensal F
38 ction in vivo, mice were first infected with Streptococcus gordonii and then challenged with P. gingi
39 C, showed an increased ability to adhere to Streptococcus gordonii and to invade primary cultures of
42 two key initial colonizers of dental plaque, Streptococcus gordonii and Veillonella parvula, on gene
43 l was synthesized, and Streptococcus mutans, Streptococcus gordonii, and Streptococcus sanguinis were
51 omyces naeslundii, Tannerella forsythia, and Streptococcus gordonii) associated with dysbiosis of the
56 velops biofilm microcolonies on substrata of Streptococcus gordonii but not on Streptococcus mutans.
57 of PMNs or HL-60 cells abolished adhesion of Streptococcus gordonii but was required for adhesion of
58 pathogen Porphyromonas gingivalis adheres to Streptococcus gordonii by interacting with a specific re
64 a 585-bp gene was cloned and sequenced from Streptococcus gordonii Challis encoding a 20.5-kDa amyla
67 tococcus pyogenes) exposed on the surface of Streptococcus gordonii commensal bacterial vectors: (i)
68 produced by the glucosyltransferase (GTF) of Streptococcus gordonii confer a hard, cohesive phenotype
69 nsortium members Fusobacterium nucleatum and Streptococcus gordonii confirmed the presence of all thr
70 tion with P. gingivalis, whereas noninvasive Streptococcus gordonii did not have a significant effect
71 acid-containing receptors is associated with Streptococcus gordonii DL1 (Challis) but not with a spon
75 ageneric coaggregation, streptococci such as Streptococcus gordonii DL1 recognize receptor polysaccha
76 ne cluster and used to transform E. coli and Streptococcus gordonii DL1, a nonureolytic, dental plaqu
78 ating early colonizers of the tooth surface (Streptococcus gordonii DL1, Streptococcus oralis 34, and
79 he scaCBA operon in the human oral bacterium Streptococcus gordonii encodes the components of an ABC-
81 pV, a dipeptidase found in culture fluids of Streptococcus gordonii FSS2, was purified and characteri
82 polypeptide (259 kDa) in the oral bacterium Streptococcus gordonii, generates mutants that are marke
87 Previous studies of GspB (the SRR adhesin of Streptococcus gordonii) have shown that a glycine-rich m
88 mbriae by reducing P. gingivalis adhesion to Streptococcus gordonii in a dual-species biofilm model.
89 parasanguinis, Streptococcus sanguinis, and Streptococcus gordonii, inhibit the growth of P. aerugin
91 omonas gingivalis and the accessory pathogen Streptococcus gordonii interact to form communities in v
93 ed by early dental plaque colonizers such as Streptococcus gordonii interfere with the subsequent col
95 eptococcal coaggregation regulator (ScaR) of Streptococcus gordonii is a manganese-dependent transcri
107 ort, we show that the A regions from the two Streptococcus gordonii M5 antigen I/II proteins (SspA an
108 large cell-surface glycoprotein expressed by Streptococcus gordonii M99 that mediates binding of this
110 he cytosolic O-glycosyltransferase GtfA/B of Streptococcus gordonii modifies the Ser/Thr-rich repeats
113 ed with PAAP(+) S. sanguis than with PAAP(-) Streptococcus gordonii or type II collagen, suggesting a
115 p38 and then stimulated with oral commensal Streptococcus gordonii, oral pathogens Porphyromonas gin
116 r example, more Streptococcus sanguinis than Streptococcus gordonii organisms are consistently isolat
118 oral commensals Streptococcus sanguinis and Streptococcus gordonii release DNA in a process induced
121 nalling between Porphyromonas gingivalis and Streptococcus gordonii serves to constrain development o
122 ion of hydrogen peroxide in solution above a Streptococcus gordonii (Sg) bacterial biofilm was studie
123 could detect 65 +/- 10 muM H2O2 produced by Streptococcus gordonii (Sg) in a simulated biofilm at 50
124 monstrated that a peptide (BAR) derived from Streptococcus gordonii (Sg) potently inhibited adherence
125 species, namely Streptococcus mutants (SM), Streptococcus gordonii (SG), Moraxella catarrhalis (MC),
129 uence motif within the C-terminal portion of Streptococcus gordonii SspB (AgI/II) is bound by Porphyr
130 tion between Streptococcus mutans AgI/II and Streptococcus gordonii SspB in their interaction with th
131 icroorganisms, e.g., Veillonella parvula and Streptococcus gordonii, stimulated higher levels of ROS
132 ns (SLBRs) from two strains of streptococci, Streptococcus gordonii strain Challis (SLBR(Hsa)) and St
139 ologous serine-rich surface glycoproteins of Streptococcus gordonii strains M99 and Challis, respecti
140 nces in virulence among seven representative Streptococcus gordonii strains were observed by using th
141 infantis group, Corynebacterium matruchotii, Streptococcus gordonii, Streptococcus cristatus, Capnocy
142 (Streptococcus mitis, Streptococcus oralis, Streptococcus gordonii, Streptococcus cristatus, Strepto
143 t AgI/II family polypeptides from strains of Streptococcus gordonii, Streptococcus intermedius and St
144 omologous enzymes in Streptococcus pyogenes, Streptococcus gordonii, Streptococcus mutans, Staphyloco
145 phylococcus aureus, Streptococcus sanguinis, Streptococcus gordonii, Streptococcus oralis, and Strept
146 nt understanding of accessory Sec systems in Streptococcus gordonii, Streptococcus parasanguinis, Myc
147 racterized and closely related oral species, Streptococcus gordonii, Streptococcus sanguinis, and car
148 -seq) on cocultures of S. mutans with either Streptococcus gordonii, Streptococcus sanguinis, or Stre
149 lizes lactate produced by the oral bacterium Streptococcus gordonii, suggesting the potential for cro
150 Hsa are homologous surface glycoproteins of Streptococcus gordonii that bind sialic acid moieties on
151 (30-day) colonization of smooth surfaces by Streptococcus gordonii that incorporates the nutrient fl
152 spB is a serine-rich glycoprotein adhesin of Streptococcus gordonii that is exported to the bacterial
155 ococcus mutans, Streptococcus sanguinis, and Streptococcus gordonii, they exhibited antibacterial eff
156 ies have shown that P. gingivalis adheres to Streptococcus gordonii through interaction of the minor
157 SspB (antigen I/II family proteins) can bind Streptococcus gordonii to other oral bacteria and also t
161 erobes, including aerotolerant ones, such as Streptococcus gordonii, use pyruvate dehydrogenase to de
163 The amylase-binding protein A (AbpA) of Streptococcus gordonii was found to be undetectable in s
164 tA gene in the human oral commensal organism Streptococcus gordonii was insertionally inactivated.
165 tococcus oralis, Streptococcus sanguinis, or Streptococcus gordonii was investigated using flow cell
166 gnated atlS, encoding a major autolysin from Streptococcus gordonii, was identified and characterized
167 To investigate disulfide bond formation in Streptococcus gordonii, we identified five putative TDOR
168 enes encoding adhesins of the oral bacterium Streptococcus gordonii were differentially expressed dep
171 yces gereneseriae, Actinomyces israelli, and Streptococcus gordonii when compared with Group EP.