ライフサイエンスコーパス: Streptococcus mutans

1 ence factors of the cavity-causing bacterium Streptococcus mutans.

2 P1 is an adhesin on the surface of Streptococcus mutans.

3 l for biofilm formation by the oral pathogen Streptococcus mutans.

4 Composites were inoculated with Streptococcus mutans.

5 events it from forming protein aggregates in Streptococcus mutans.

6 conditions to inhibit competing species like Streptococcus mutans.

7 were assayed by 74 DNA probes and by PCR to Streptococcus mutans.

8 commensal S. gordonii and the oral pathogen Streptococcus mutans.

9 ression of important virulence attributes of Streptococcus mutans.

10 r the production of the alarmone (p)ppGpp in Streptococcus mutans.

11 e of repressing mutacin I gene expression in Streptococcus mutans.

12 y Actinobacillus actinomycetemcomitans), and Streptococcus mutans.

13 nii, Streptococcus sanguinis, and cariogenic Streptococcus mutans.

14 d functional maturation of the adhesin P1 of Streptococcus mutans.

15 in Gram-positive bacteria does not apply to Streptococcus mutans.

16 bstrata of Streptococcus gordonii but not on Streptococcus mutans.

17 siologic functions and virulence factors for Streptococcus mutans.

18 peratively on the expression of virulence of Streptococcus mutans.

19 ce development for genetic transformation in Streptococcus mutans.

20 sensing is involved in biofilm formation of Streptococcus mutans.

21 eptococcal species Streptococcus sanguis and Streptococcus mutans.

22 d is produced by the Gram-positive bacterium Streptococcus mutans.

23 bohydrate metabolized by the dental pathogen Streptococcus mutans.

24 mensal Streptococcus gordonii and pathogenic Streptococcus mutans.

25 factor is cariogenic microorganisms such as Streptococcus mutans.

26 ding for a PDE promoted biofilm formation in Streptococcus mutans.

27 rmus thermophilus, Synechocystis PCC6803 and Streptococcus mutans.

28 is closely associated with the virulence of Streptococcus mutans.

29 e, mediated by the cariogenic oral bacterium Streptococcus mutans.

30 t of genetic competence in the oral pathogen Streptococcus mutans.

31 ells versus 8.0 x 10(5) cells; P <0.05), and Streptococcus mutans (6.2 x 10(5) cells versus 2.0 x 10(

32 significant antibiofilm bioactivity against Streptococcus mutans, a causative agent of human dental

33 timated timing of a demographic expansion in Streptococcus mutans, a causative agent of human dental

34 Streptococcus mutans, a dental caries pathogen, also cau

35 Streptococcus mutans, a dental pathogen, secretes differ

36 modulation of CtsR activity is different in Streptococcus mutans, a dental pathogen.

37 Streptococcus mutans, a Gram-positive odontopathogen tha

38 Streptococcus mutans, a gram-positive pathogen considere

39 Streptococcus mutans, a major etiological agent of human

40 Streptococcus mutans, a major oral pathogen responsible

41 There are suggestions that the phylogeny of Streptococcus mutans, a member of the human indigenous b

42 Streptococcus mutans, a member of the human oral flora,

43 prevalent infectious disease associated with Streptococcus mutans, a pathogen also linked to endocard

44 Streptococcus mutans, a primary agent of dental caries,

45 Streptococcus mutans, a principal causative agent of den

46 Streptococcus mutans, a principal causative agent of den

47 at inhibits the glucosyltransferase (GTF) of Streptococcus mutans, a virulence enzyme involved in ora

48 Deletion of comR or comS in Streptococcus mutans abolished transformability, as did

49 inked properties of oral pathogens including Streptococcus mutans, Actinomyces naeslundii and Prevote

50 ctivities against 3 single-species biofilms: Streptococcus mutans, Actinomyces naeslundii, and Entero

51 ecies-specific adherence/aggregation between Streptococcus mutans AgI/II and Streptococcus gordonii S

52 e with the surface adhesin protein AgI/II of Streptococcus mutans alone or in combination with LT-IIa

53 e with the surface adhesin protein AgI/II of Streptococcus mutans alone or in combination with LT-IIa

54 Introducing the same mutation in Streptococcus mutans also provided a phage resistance ph

55 Since Streptococcus mutans also releases Gtf enzymes that coul

56 racterized for only select organisms, namely Streptococcus mutans and Aggregatibacter actinomycetemco

57 showed a significant microbicidal effect for Streptococcus mutans and an unencapsulated strain of Por

58 In early childhood caries, Streptococcus mutans and Candida albicans are often co-i

59 ractions of the response regulator ComE from Streptococcus mutans and DNA binding sites through DNase

60 ocalized on the surface of the oral pathogen Streptococcus mutans and facilitates an interaction with

61 independent of total bacteria and levels of Streptococcus mutans and Lactobacilli.

62 tro wear, and antibacterial activity against Streptococcus mutans and Lactobacillus casei (in both pl

63 Lack of binding activity was noted with Streptococcus mutans and Lactobacillus casei.

64 firmed the association of the acid producers Streptococcus mutans and Lactobacillus spp. with childho

65 e complexes (IC) of the cariogenic bacterium Streptococcus mutans and mAbs against its surface adhesi

66 occus pyogenes (Group A Streptococcus; GAS), Streptococcus mutans and Mycobacterium tuberculosis.

67 the oral cavity with the cariogenic pathogen Streptococcus mutans and other bacteria.

68 mechanisms of AgD from H. pylori as well as Streptococcus mutans and Porphyromonas gingivalis.

69 against the oral specific bacterial species, Streptococcus mutans and Porphyromonas gingivalis.

70 us actinomycetemcomitans) and dental caries (Streptococcus mutans and S. sobrinus).

71 t defect observed for orthologous mutants of Streptococcus mutans and Streptococcus agalactiae.

72 hen tested against the gram-positive species Streptococcus mutans and Streptococcus mitis, however, l

73 led a high degree of synteny with operons in Streptococcus mutans and Streptococcus pneumoniae.

74 Oral bacteria, including Streptococcus mutans and Streptococcus salivarius, contr

75 s study, we used two oral bacterial species, Streptococcus mutans and Streptococcus sanguinis (former

76 In contrast, Streptococcus mutans and Streptococcus sanguinis, caries

77 n reaction (PCR)/quantitative PCR (qPCR) for Streptococcus mutans and Streptococcus sobrinus.

78 Streptococcus mutans and sucrose are key modulators asso

79 oxidase is involved in both competition with Streptococcus mutans and virulence for infective endocar

80 , adjusted for age, sex, and the presence of Streptococcus mutans) and SM surfaces (1,004 participant

81 terococcus faecalis, Actinomyces naeslundii, Streptococcus mutans, and Aggregatibacter actinomycetemc

82 cterium nucleatum, Porphyromonas gingivalis, Streptococcus mutans, and Campylobacter rectus are also

83 , including oral streptococci, lactobacilli, Streptococcus mutans, and Candida, in saliva than did HI

84 good dental health, whereas Prevotella spp., Streptococcus mutans, and Human herpesvirus 4 (Epstein-B

85 rial load was calculated for total bacteria, Streptococcus mutans, and Lactobacilli.

86 ive group were Actinomyces sp. strain B19SC, Streptococcus mutans, and Lactobacillus spp., which exhi

87 s including those of Salmonella typhimurium, Streptococcus mutans, and Thermus aquaticus encode a ded

88 The Streptococcus mutans antigen I/II (AgI/II) is a cell sur

89 Streptococcus mutans antigen I/II (AgI/II) is a cell sur

90 Streptococcus mutans antigen I/II (AgI/II) protein was o

91 ssing the saliva-binding region (SBR) of the Streptococcus mutans antigen I/II adhesin, either alone

92 cterial species, including caries-associated Streptococcus mutans as well as several periodontal path

93 cidal effect was observed against cariogenic Streptococcus mutans at pH 7.4, even when using NO-relea

94 The Streptococcus mutans atlA gene encodes an autolysin requ

95 Streptococcus gordonii and Streptococcus mutans avidly colonize teeth.

96 th resin-only composites (53 +/- 6 um) after Streptococcus mutans bacteria were grown for 3 d in a ba

97 Streptococcus mutans belongs to the viridans group of or

98 oups (N-G, N-H, A-G and A-H) were exposed to Streptococcus mutans biofilm for 4, 8, 15, 20 or 25 days

99 Streptococcus mutans biofilm was disrupted from sandblas

100 o mediate photodynamic therapy (PDT) against Streptococcus mutans biofilms.

101 In Streptococcus mutans, both competence and bacteriocin pr

102 lood mononuclear cells (PBMC) after engaging Streptococcus mutans, but monocytes in developing endoca

103 Carbohydrate catabolite repression (CCR) in Streptococcus mutans can be independent of catabolite co

104 Streptococcus mutans can produce exopolysaccharides (EPS

105 Actinomyces israelii, Streptococcus sanguis, Streptococcus mutans, Candida tropicalis, Candida paraps

106 Addition of tears to late-exponential-phase Streptococcus mutans cells resulted in time- and dose-de

107 saccharides (EPS) by a cariogenic bacterium, Streptococcus mutans (Cheng et al., 2016).

108 The Streptococcus mutans Cid/Lrg system represents an ideal

109 that the underlying mechanisms by which the Streptococcus mutans ClpXP protease affects virulence tr

110 ously that mucosal immunization of mice with Streptococcus mutans coated with the monoclonal antibody

111 In subjects positive for Streptococcus mutans, collagen binding activity was posi

112 that examines whether HIV infection affects Streptococcus mutans colonization in the oral cavity.

113 ns, all Caucasians had significantly greater Streptococcus mutans colonization, but only Db-negative

114 bsp. equisimilis (Group G Streptococcus), or Streptococcus mutans complemented the GAC biosynthesis p

115 The ciaRH operon in Streptococcus mutans contains 3 contiguous genes, ciaXRH

116 en salivary agglutinin and the adhesin P1 of Streptococcus mutans contribute to bacterial aggregation

117 In this study, we found that in Streptococcus mutans CSP induced co-ordinated expression

118 eractions between sucrose- (and starch-) and Streptococcus mutans-derived exoenzymes present in the p

119 Streptococcus mutans develops competence for genetic tra

120 mRNA (irvA) from the dental caries pathogen Streptococcus mutans directly modulates target mRNA (gbp

121 Streptococcus mutans displays complex regulation of gene

122 parable genotyping results for investigating Streptococcus mutans diversity and transmission.

123 An insertionally inactivated fabM strain of Streptococcus mutans does not produce unsaturated membra

124 Streptococcus mutans employs a key virulence factor, thr

125 The oral pathogen Streptococcus mutans employs a variety of mechanisms to

126 The cariogenic bacterium Streptococcus mutans employs so-called competence-stimul

127 Streptococcus mutans employs the secreted peptides CSP a

128 The fruA gene of Streptococcus mutans encodes for a secreted fructan hydr

129 s in humans, and the primary caries pathogen Streptococcus mutans encodes multiple enzymes involved i

130 In a Streptococcus mutans endocarditis rat model, we identifi

131 ce TLR2 was associated with poor response to Streptococcus mutans, Enterococcus faecalis, and Lactoba

132 In Streptococcus mutans, enzyme II(scr) and sucrose-6-phosp

133 We previously demonstrated that Streptococcus mutans expresses a functional agmatine dei

134 Streptococcus mutans F-ATPase, the major component of th

135 frame (ORF) encoding a protein homologous to Streptococcus mutans FruK.

136 Active immunization with Streptococcus mutans glucan binding protein B (GBP-B) ha

137 Of these, Streptococcus mutans glucan binding protein B (GbpB) is

138 which are derived from functional domains of Streptococcus mutans glucosyltransferases (GTF) have bee

139 Streptococcus mutans glucosyltransferases form extracell

140 Recently, Streptococcus mutans has been found with high frequency

141 inding region (SBR) of the adhesin AgI/II of Streptococcus mutans has been shown to induce a mixed Th

142 Glucan-binding protein B (GbpB) from Streptococcus mutans has been shown to induce protective

143 The adhesin P1 of Streptococcus mutans has been studied as an anticaries v

144 Streptococcus mutans has two YidC paralogs.

145 Using the Streptococcus mutans HdrRM LRS as a model, we demonstrat

146 The Streptococcus mutans hdrRM operon encodes a novel two-ge

147 y partially characterized the dnaK operon of Streptococcus mutans (hrcA-grpE-dnaK) and demonstrated t

148 We found that inactivation of Streptococcus mutans htrA by gene-replacement also resul

149 ically the acidophilic and caries-associated Streptococcus mutans in 17-year old Swedish adolescents

150 pathogen, is frequently found together with Streptococcus mutans in dental biofilms associated with

151 Streptococcus mutans in dental plaque biofilms play a ro

152 ndida albicans cells are often detected with Streptococcus mutans in plaque biofilms from children af

153 frequently detected with heavy infection of Streptococcus mutans in plaque-biofilms from children af

154 In this study, the role of luxS of Streptococcus mutans in the regulation of traits crucial

155 efore investigated the roles of cnm-positive Streptococcus mutans in this single hospital-based, obse

156 or age, education group, and the presence of Streptococcus mutans) in self-reported whites (ages 14 t

157 Escherichia coli and Streptococcus mutans induced lesser effects.

158 expression of the fructanase gene (fruA) of Streptococcus mutans: induction by levan, inulin, or suc

159 Strategies aimed at the prevention of Streptococcus mutans infection and dental caries include

160 Streptococcus mutans is a biofilm-forming bacterium that

161 Streptococcus mutans is a biofilm-forming oral pathogen

162 Streptococcus mutans is a cariogenic oral pathogen whose

163 Streptococcus mutans is a commensal member of the health

164 Streptococcus mutans is a facultative member of the oral

165 The biofilm-forming Streptococcus mutans is a gram-positive bacterium that r

166 Streptococcus mutans is a key contributor to the formati

167 Streptococcus mutans is a major etiologic agent of denta

168 Streptococcus mutans is a primary pathogen for dental ca

169 The transport of carbohydrates by Streptococcus mutans is accomplished by the phosphoenolp

170 that the membrane fatty acid composition of Streptococcus mutans is affected by growth pH.

171 Streptococcus mutans is an etiologic agent of human dent

172 environmental stress, especially low pH, by Streptococcus mutans is central to the virulence of this

173 Streptococcus mutans is considered the primary etiologic

174 Streptococcus mutans is considered the principal carioge

175 Streptococcus mutans is considered to be the major etiol

176 urface Cnm protein expressed on cnm-positive Streptococcus mutans is involved in the development of C

177 The quorum-sensing system of Streptococcus mutans is mediated by the competence-stimu

178 Streptococcus mutans is often cited as the main bacteria

179 Streptococcus mutans is one of several members of the or

180 Dental caries induced by Streptococcus mutans is one of the most prevalent chroni

181 Streptococcus mutans is particularly well adapted for hi

182 Streptococcus mutans is prominently linked to dental car

183 Streptococcus mutans is the etiological agent of dental

184 Streptococcus mutans is the leading cause of dental cari

185 Streptococcus mutans is the leading cause of dental cari

186 Streptococcus mutans is the primary cariogen that produc

187 The gram-positive bacterium Streptococcus mutans is the primary causative agent in t

188 Streptococcus mutans is the primary etiological agent of

189 Streptococcus mutans is the principal acidogenic compone

190 When Streptococcus mutans is transferred from a preferred car

191 The cnm gene in Streptococcus mutans isolated from saliva was screened u

192 Strains of Streptococcus mutans lacking DnaK or GroEL appear not to

193 3 bp region (igr66) between grpE and dnaK of Streptococcus mutans lacks a promoter but is required fo

194 The primary structure of the Streptococcus mutans lantibiotic mutacin 1140 was elucid

195 ith special emphasis on the immunobiology of Streptococcus mutans, leading to active and passive vacc

196 The cariogenic bacterial species Streptococcus mutans metabolizes sucrose to initiate bio

197 omologous with transcriptional regulators of Streptococcus mutans (MetR), Streptococcus iniae (CpsY),

198 l interaction, while the cariogenic pathogen Streptococcus mutans (mutans group) interacts with the f

199 ed a previously constructed stress-sensitive Streptococcus mutans mutant Tn-1 strain resulting from d

200 jor cell-surface adhesion protein SA I/II of Streptococcus mutans, one of the major causative agents

201 essential for in vitro biofilm formation by Streptococcus mutans or Streptococcus gordonii grown in

202 causative agent of dental caries in humans, Streptococcus mutans, outcompetes other bacterial specie

203 Streptococcus mutans (p < 0.005), Streptococcus sobrinus

204 bacterium Cluster 1 (p = 0.11), and by qPCR, Streptococcus mutans (p = 0.008) and Scardovia wiggsiae

205 In Streptococcus mutans, (p)ppGpp synthesis is catalyzed by

206 tivity against periodontal pathogens such as Streptococcus mutans, Porphyromonas gingivalis, Fusobact

207 The presence of bacterial DNA from Streptococcus mutans, Porphyromonas gingivalis, Tannerel

208 The oral pathogen, Streptococcus mutans, possesses inducible DNA repair def

209 Many clinical isolates of Streptococcus mutans produce peptide antibiotics called

210 Streptococcus mutans proliferates as a biofilm on the to

211 Streptococcus mutans regulates genetic competence throug

212 many gram-positive bacteria, but its role in Streptococcus mutans remains enigmatic.

213 ed evidence that the oral cariogenic species Streptococcus mutans remains viable but physiologically

214 n of the agmatine deiminase system (AgDS) of Streptococcus mutans requires agmatine and is optimal at

215 ormerly designated salivary agglutinin) with Streptococcus mutans requires an alanine-rich repetitive

216 demonstrated that competence development in Streptococcus mutans requires the type II ComRS quorum-s

217 Streptococcus mutans resides in the oral polymicrobial b

218 ccus faecalis, Cardiobacterium valvarum, and Streptococcus mutans, respectively.

219 though some studies focused on understanding Streptococcus mutans' response to fluoride, the mechanis

220 secretion and acid tolerance (sat) operon of Streptococcus mutans resulted in an acid-sensitive pheno

221 Inactivation of the Smu0630 gene of Streptococcus mutans resulted in dramatic decreases in b

222 to measure the local glucose consumption of Streptococcus mutans (S. mutans) biofilms.

223 t shows homology to the N-terminal domain of Streptococcus mutans SagA protein (42% similarity), prev

224 ll-known acidogenic/aciduric species such as Streptococcus mutans, Scardovia wiggsiae, Parascardovia

225 species associated with severe ECC included Streptococcus mutans, Scardovia wiggsiae, Veillonella pa

226 source of CO(2), buffers acid production by Streptococcus mutans (Sm), a key organism associated wit

227 ated, infected with Actinomyces viscosus and Streptococcus mutans (sobrinus) 6715, and fed a cariogen

228 ported identification of two Spx proteins in Streptococcus mutans - SpxA1 was the primary activator o

229 eptococcus pyogenes, Streptococcus gordonii, Streptococcus mutans, Staphylococcus aureus, and Enteroc

230 tococcus pyogenes, Streptococcus pneumoniae, Streptococcus mutans, Staphylococcus aureus, and Lactoco

231 Streptococcus mutans strain GS-5 produces a two-peptide

232 T MAP constructs, followed by infection with Streptococcus mutans strain SJr.

233 Using Streptococcus mutans strain UA159, we demonstrate that t

234 Eleven cases showed Streptococcus mutans strains positive for cnm.

235 Streptococcus mutans strains were isolated from cohorts

236 colonizing species of the human oral flora (Streptococcus mutans, Streptococcus gordonii and Strepto

237 Antibacterial material was synthesized, and Streptococcus mutans, Streptococcus gordonii, and Strept

238 Streptococcus mitis, Streptococcus sobrinus, Streptococcus mutans, Streptococcus oralis, and Candida

239 imum bactericidal concentration test against Streptococcus mutans, Streptococcus sanguinis, and Strep

240 The salivary bacterial levels evaluated were Streptococcus mutans, Streptococcus sobrinus, Streptococ

241 In Streptococcus mutans, Streptococcus suis, and species of

242 ecombinant DNA methods were used to make the Streptococcus mutans supercolonizing strain, JH1140, lac

243 clonal antibody (MAb) 6-11A directed against Streptococcus mutans surface adhesin P1 was shown previo

244 Allelic replacement of the C terminus of a Streptococcus mutans surface protein affects murein hydr

245 ne segments were identified from a strain of Streptococcus mutans that was isolated from a patient wi

246 In Streptococcus mutans, the alternative sigma factor ComX

247 Among these is Streptococcus mutans, the causative agent of dental cari

248 ccus gordonii, Streptococcus intermedius and Streptococcus mutans, the genes were cloned and expresse

249 In Streptococcus mutans, the global response regulator CovR

250 d the metabolism of lactose and galactose by Streptococcus mutans, the major etiological agent of hum

251 he facultative anaerobic human oral pathogen Streptococcus mutans, the mechanisms used to protect aga

252 hment to the dental surfaces was studied for Streptococcus mutans, the most abundant cariogenic bacte

253 However, the function of this molecule in Streptococcus mutans, the primary aetiological agent of

254 Streptococcus mutans, the primary causative agent of hum

255 Streptococcus mutans, the primary etiological agent of d

256 h-coverage genome sequence of 57 isolates of Streptococcus mutans, the primary etiological agent of h

257 Streptococcus mutans, the primary etiological agent of h

258 Streptococcus mutans, the primary etiological agent of h

259 trigger factor homologue, was identified in Streptococcus mutans, the primary etiological agent of h

260 Streptococcus mutans, the principal causative agent of d

261 sives and the DOX-containing eluates against Streptococcus mutans through agar diffusion assays.

262 films by an important opportunistic pathogen Streptococcus mutans through the action of a family of 3

263 licated in the ability of certain strains of Streptococcus mutans to bind to collagen and to invade h

264 ugh substantial epidemiologic evidence links Streptococcus mutans to caries, the pathobiology of cari

265 The ability of Streptococcus mutans to catabolize cellobiose, a beta-li

266 ace and competes with oral pathogens such as Streptococcus mutans to maintain oral health.

267 Recently, we showed that exposure of Streptococcus mutans to oxygen strongly inhibits biofilm

268 The ability of Streptococcus mutans to produce and tolerate organic aci

269 -containing enzyme used by the oral pathogen Streptococcus mutans to reduce diatomic oxygen to water

270 he tooth by recruiting the pioneer bacterium Streptococcus mutans to the surface.

271 In Streptococcus mutans, two Spx homologues, SpxA1 and SpxA

272 Here, we use the facultative anaerobe Streptococcus mutans UA159 as a heterologous host for th

273 elocity water microdrop on the detachment of Streptococcus mutans UA159 biofilms from the interproxim

274 The production of (p)ppGpp by Streptococcus mutans UA159 is catalyzed by three gene pr

275 o remove biofilms of the cariogenic pathogen Streptococcus mutans UA159, as well as Actinomyces naesl

276 been identified in the cariogenic bacterium Streptococcus mutans UA159.

277 being upregulated in a proteome analysis of Streptococcus mutans under acid stress conditions.

278 t was found to fail to inhibit the growth of Streptococcus mutans under microaerobic conditions.

279 The cariogenic bacterium Streptococcus mutans uses adhesin P1 to adhere to tooth

280 caries (ECC), while strongly associated with Streptococcus mutans using selective detection (culture,

281 e human oral cavity plays a putative role in Streptococcus mutans virulence gene expression and in ap

282 The presence of cnm-positive Streptococcus mutans was significantly associated with I

283 ), which is an important virulence factor of Streptococcus mutans, was recombinantly expressed in the

284 Using an oral bacterium (Streptococcus mutans), we find that microbial cells sett

285 In order to examine gene function in Streptococcus mutans, we have recently initiated an anti

286 Extremely high levels of Streptococcus mutans were detected in the S-ECC group.

287 implicated in the acid tolerance response of Streptococcus mutans when a mutation in its gene resulte

288 lular polysaccharide (IPS) is accumulated by Streptococcus mutans when the bacteria are grown in exce

289 reptococcus mitis, Streptococcus oralis, and Streptococcus mutans, whereas dentate individuals had hi

290 However, it does not adhere to Streptococcus mutans, which expresses SpaP, a highly con

291 the basis of these findings, we propose that Streptococcus mutans, which resides in a multispecies or

292 The mannose-PTS of Streptococcus mutans, which transports mannose and gluco

293 ified enolase as a cell surface component of Streptococcus mutans, which was confirmed by enzyme-link

294 en I/II) is a sucrose-independent adhesin of Streptococcus mutans whose functional architecture on th

295 (p < 0.001), Scardovia wiggsiae (p = 0.003), Streptococcus mutans with bifidobacteria (p < 0.001), an

296 able to selectively kill cariogenic pathogen Streptococcus mutans with high efficacy within a human s

297 h for noninvasive treatment based on killing Streptococcus mutans with high-frequency microwave energ

298 target the EPS-producing cariogenic bacteria Streptococcus mutans with higher killing specificity (ve

299 We previously showed that oral Streptococcus mutans with the cnm gene encoding a collag

300 r the function of the Escherichia coli YidC, Streptococcus mutans YidC2, and the chloroplast Arabidop