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1 cture or bacteremia with Escherichia coli or Streptococcus pneumoniae infection).
2 esponses of AMs and exaggerated responses to Streptococcus pneumoniae infection.
3  survival in a mouse model of NLRP3-mediated Streptococcus pneumoniae infection.
4 ysaccharide and an impaired host response to Streptococcus pneumoniae infection.
5 ase that serves as a virulence factor during Streptococcus pneumoniae infection.
6  epithelium might increase susceptibility to Streptococcus pneumoniae infection.
7 s, CIA, or SLE but increased mortality after Streptococcus pneumoniae infection.
8 ) mice were more susceptible to acute lethal Streptococcus pneumoniae infection.
9 e has been shown to protect mice from lethal Streptococcus pneumoniae infection.
10 , CD127(+) MAIT cells protected mice against Streptococcus pneumoniae infection.
11 hways in influenza-induced susceptibility to Streptococcus pneumoniae infection.
12 ssociated with virulence in a mouse model of Streptococcus pneumoniae infection.
13  The numerators were the numbers of invasive Streptococcus pneumoniae infections among persons 18-64
14                  Here, using mouse models of Streptococcus pneumoniae infection and Alternaria altern
15 eptor) are significantly more susceptible to Streptococcus pneumoniae infection and fail to clear S.
16                                              Streptococcus pneumoniae infections are an important cau
17                                              Streptococcus pneumoniae infections arising in hospitali
18 Measures of the contribution of influenza to Streptococcus pneumoniae infections, both in the seasona
19 o regulate the pathogen-host response during Streptococcus pneumoniae infection, but the role Chil1 p
20 onsible for thrombocytopenia during systemic Streptococcus pneumoniae infection by eliminating platel
21                                              Streptococcus pneumoniae infection can result in bactere
22 d PCV13) induce immunological memory against Streptococcus pneumoniae infections caused by vaccine se
23 es in cerebrospinal fluid from children with Streptococcus pneumoniae infection, compared with childr
24 th influenza, mice are better protected from Streptococcus pneumoniae infection due to a population o
25 tion disorders were significantly higher for Streptococcus pneumoniae infection (eg, 7.89 [95% CI, 5.
26                CFTR was down-regulated after Streptococcus pneumoniae infection ex vivo or in vivo in
27 lex real-time PCR for the rapid diagnosis of Streptococcus pneumoniae infection from culture-negative
28 improved survival and clearance of secondary Streptococcus pneumoniae infection from the lungs and bl
29 on-based laboratory surveillance of invasive Streptococcus pneumoniae infections from January 1, 1995
30                              Since secondary Streptococcus pneumoniae infections greatly increase the
31       In 1994, 24 (21%) of 113 patients with Streptococcus pneumoniae infections had PRP; 13 PRP isol
32                       Likewise, in pulmonary Streptococcus pneumoniae infection, HVEM is also require
33                                       During Streptococcus pneumoniae infection, ILC3 rapidly accumul
34  provide protective mucosal immunity against Streptococcus pneumoniae infection in aged mice.
35    We have developed a model of low-inoculum Streptococcus pneumoniae infection in infant rats.
36  the mechanistic effects of these factors on Streptococcus pneumoniae infection in mice.
37 xpression and its effect on inflammation and Streptococcus pneumoniae infection in mice.
38  cortical impact model followed by secondary Streptococcus pneumoniae infection in mice.
39 ke receptor 2 (TLR2) in host defense against Streptococcus pneumoniae infection in the middle ear, wi
40 A (PspA) has been shown to be protective for Streptococcus pneumoniae infections in mice.
41                                              Streptococcus pneumoniae infections in Taiwan mostly occ
42 iator of host resistance in murine models of Streptococcus pneumoniae infection; in humans, anti-TNF
43                                              Streptococcus pneumoniae infections induce inflammatory
44                                              Streptococcus pneumoniae infection is a frequent cause o
45                                    Secondary Streptococcus pneumoniae infection is a significant caus
46 tein (CRP) in protecting mice against lethal Streptococcus pneumoniae infection is unknown.
47 es in protecting elderly individuals against Streptococcus pneumoniae infections is not clear.
48    How mucin is induced in upper respiratory Streptococcus pneumoniae infections is unknown.
49 100A8/A9 involvement in the host response to Streptococcus pneumoniae infection making use of S100a9(
50 ential component of the host defense against Streptococcus pneumoniae infection of the lungs.
51                             Life-threatening Streptococcus pneumoniae infections often occur after he
52                      The diagnosis of severe Streptococcus pneumoniae infection relies heavily on ins
53                                              Streptococcus pneumoniae infections remain a major cause
54 MP), and generated better protection against Streptococcus pneumoniae infection than 2'3'-cGAMP adjuv
55 re, we used a murine model of intrapulmonary Streptococcus pneumoniae infection to investigate the ro
56                                              Streptococcus pneumoniae infection was more severe in FX
57         Using two different animal models of Streptococcus pneumoniae infection, we have demonstrated
58 tive protein (CRP) protects mice from lethal Streptococcus pneumoniae infection when injected into mi