ライフサイエンスコーパス: T wave inversion

1 ge reduction (QS in V1-V3) and inferolateral T-wave inversion.

2 tall ECG R waves with ischemic-looking deep T-wave inversion.

3 rdiography revealed sinus rhythm and diffuse T-wave inversion.

4 diography revealed sinus rhythm, and diffuse T-wave inversion.

5 e cohort), largely accounted for by abnormal T-wave inversions.

6 y 12%), ST-segment depressions combined with T-wave inversions (16%), or neither (approximately 41%).

7 chest pain with ST-segment elevation and/or T-wave inversion; (2) absence of significant coronary ar

8 Abnormal ECG findings included T-wave inversions (3 athletes [1.7%]), Q waves (2 athlet

9 Of 158 children with T-wave inversion, 4 (2.5%) had a diagnosis of cardiomyop

10 ECG criteria for ischemia (ST depression or T-wave inversion), 40% and 97% for peak troponin-I, and

11 hypertrophy voltage criteria, long QTc, and T-wave inversion, all P<0.05) and predicted clinical wor

12 red with 6.8% of patients with new, isolated T wave inversion and 8.2% of those with no ECG changes (

13 nomenon characterized by diffuse symmetrical T wave inversion and QT prolongation after recovery from

14 t nonischemic pulmonary edema may cause deep T wave inversion and QT prolongation after resolution of

15 Diffuse T-wave inversion and a prolonged QT interval occurred in

16 In acute MI, T-wave inversion and Brugada phenocopy were explained by

17 Most athletes with HCM (96%) exhibited T-wave inversion and had milder LVH (15.8+/-3.4 mm versu

18 gram, as manifested by the strain pattern of T-wave inversion and STdep, are markers for LVH and adve

19 Inferior T-wave inversions and sum of R waves (mm) in V1 to V3 we

20 ular complexes in 24 h, number of leads with T-wave inversion, and right and left ventricular ejectio

21 the 19 surviving patients, 16 (84%) exhibit T-wave inversions, and 10 (53%) have transient QT prolon

22 Electrocardiography revealed nonspecific T-wave inversions, and a series of cardiac biomarkers we

23 ST-segment depressions (approximately 31%), T-wave inversions (approximately 12%), ST-segment depres

24 strain pattern of lateral ST depression and T-wave inversion at baseline has been associated with an

25 of children with postpubertal persistence of T-wave inversion at preparticipation screening is warran

26 Anterior T-wave inversion (ATWI) on electrocardiography (ECG) in

27 e majority (52%) of group 2 changes, whereas T-wave inversions constituted 11%.

28 The prevalence of right precordial T-wave inversion decreased significantly with increasing

29 The prevalence of T-wave inversion decreases significantly after puberty.

30 Right precordial T-wave inversions did not predict increased mortality (n

31 or LBBB identify high risk patients, whereas T wave inversion does not add to the clinical history in

32 n of APD gradients across the heart, and ECG T-wave inversion during closely coupled premature stimul

33 igh-risk features including QRS widening and T-wave inversions for RV dysfunction/dilation.

34 onfidence interval, 2.8-22.5; P<0.001), >/=3 T-wave inversions (hazard ratio, 4.2; 95% confidence int

35 pe (HR: 1.81; P=0.040), number of leads with T wave inversion (HR: 1.17; P=0.039), low QRS voltage (H

36 n 14.3% of 1,416 enrolled patients, isolated T wave inversion in 21.9% and left bundle branch block (

37 T wave inversion in infero-lateral and left precordial l

38 ATWI was defined as T-wave inversion in >/=2 contiguous anterior leads (V1 t

39 32), while for HF, they were the presence of T-wave inversion in 3+ electrocardiogram leads (aHR 2.03

40 ned ventricular tachycardia or sustained VA, T-wave inversion in 3+ leads on electrocardiogram, LVEF

41 e relation, and underlying cardiomyopathy of T-wave inversion in children undergoing preparticipation

42 Prolonging FRPs in the same area caused T-wave inversion in lead X and increased T amplitude in

43 e of ECG left ventricular strain (defined as T-wave inversion in leads V(4) through V(6)) and LVH, as

44 T-wave inversion in right precordial leads V(1) to V(3)

45 block pattern with ST-segment elevation and T-wave inversion in the right precordial leads.

46 of derived T waves in the X lead and caused T-wave inversion in the Z lead.

47 wed sinus rhythm, right bundle branch block, T-wave inversion in V6, and evidence of right atrial dil

48 onstrating convex ST elevation combined with T-wave inversions in leads V1 to V4 (6 of 12 athletes [5

49 All 5 TRDN-null patients displayed extensive T-wave inversions in precordial leads V1 through V4, wit

50 T-wave inversions in right precordial leads are relative

51 T-wave inversions in right precordial leads V(1) to V(3)

52 a with a heart rate of 102 beats per minute, T-wave inversions in the inferior leads, left axis devia

53 he prevalence and prognostic significance of T-wave inversions in the middle-aged general population

54 potentially lethal disease characterized by T-wave inversions in the precordial leads, transient QT

55 T-wave inversions in V1 through V3 were observed in 85%

56 ular arrhythmias before ICD implantation and T-wave inversions inferiorly.

57 However, T-wave inversion is a common ECG abnormality of cardiomy

58 The ECG strain pattern of ST depression and T-wave inversion is strongly associated with left ventri

59 lassic ECG strain pattern, ST depression and T-wave inversion, is a marker for left ventricular hyper

60 e number of anterior and inferior leads with T-wave inversion, left and right ventricular ejection fr

61 e number of anterior and inferior leads with T-wave inversion, left and right ventricular ejection fr

62 High-risk ECG features include lateral T-wave inversion (LV dysfunction), deep S waves in V1 an

63 ed with ST segment elevation (n = 19) and/or T wave inversion (n = 20) on admission ECG.

64 This may explain the T-wave inversion observed and will have implications for

65 y independent predictor for right precordial T-wave inversion (odds ratio, 3.6; 95% confidence interv

66 nction had higher odds of lateral precordial T-wave inversions (odds ratio, 18.4; 95% confidence inte

67 l, 1.21-4.01; P=0.01) and lateral precordial T-wave inversions (odds ratio, 9.87; 95% confidence inte

68 T-wave inversion on a 12-lead ECG is usually dismissed i

69 right ventricular dysfunction and number of T-wave inversions on electrocardiogram were unchanged.

70 ardia, premature ventricular complex burden, T-wave inversions on electrocardiogram, cardiac syncope,

71 gment depressions, ST-segment elevations, or T-wave inversions on the presenting ECG.

72 ncy departments in Ontario, Canada, Q-waves, T-wave inversion, or ST-depression were present in 51.8%

73 ST-segment elevation, ST-segment depression, T-wave inversion, or the presence of confounding factors

74 The older patient had anterior T wave inversions, prolonged terminal activation duratio

75 Pathological T-wave inversion (PTWI) is rarely observed on the ECG of

76 lows discrimination from ischemic precordial T-wave inversions regardless of the coronary artery invo

77 tablished ECG features (low QRS voltages and T-wave inversions), specified these features (eg, R- and

78 ith presence of MAD, leaflet redundancy, and T-wave inversion/ST-segment depression (all p < 0.0001)

79 left ventricular end-systolic diameter, and T-wave inversion/ST-segment depression (all p <= 0.001).

80 T-wave inversion through V(3) demonstrated optimal sensi

81 ameters that differed were the prevalence of T-wave inversion through V(4) (59% versus 12%, respectiv

82 ts, TKOS has been characterized by extensive T-wave inversions, transient QT prolongation, and severe

83 Postpacing precordial T-wave inversion (TWI), known as cardiac memory (CM), mi

84 ercise, including biventricular dilation and T-wave inversion (TWI), may create diagnostic overlap wi

85 Anterior T-wave inversion was more common in females than males (

86 ked RV enlargement with concomitant anterior T-wave inversion was observed in 3.0% of BAs versus 0.3%

87 T-wave inversion was predominantly confined to leads V1

88 Anterior T-wave inversion was present in 14.3% of BAs versus 3.7%

89 T-wave inversion was recorded in 158 children (5.7%) and

90 T-wave inversions were the most sensitive predictor of L

91 ular complex count, and number of leads with T-wave inversion) were associated with LTVA.

92 ischemic heart disease, new large or global T wave inversion with QT prolongation was observed after

93 DLTs included reversible, asymptomatic T-wave inversions, without any associated changes in tro