ライフサイエンスコーパス: T

1 T cell expansion and differentiation are critically depe

2 T cell reactivity against SARS-CoV-2 was observed in une

3 T cells can sometimes acquire properties of a memory cel

4 T cells from infant mice were predominantly immature, in

5 T cells were sampled for up to 11 weeks to capture stead

6 T(H)17 cells are believed to orchestrate MS pathology, i

7 T-IFTA was similarly associated with decreased DC-GS.

8 es were nearly 40% at 1.5 T and < 12% at 3.0 T.

9 I(2) was 98% at 1.5 T and 3.0 T.

10 nging efficacy studies were performed on a 1 T MRI scanner using a transgenic APP/PSEN1 mouse model o

11 quid with a magnetic field B(S) = 10(12+/-1) T.

12 Analysis of 13 T-lineage acute lymphoblastic leukemias identified a rec

13 pared to previously reported pure Ti(3) C(2) T(x) films.

14 Here, it is reported on Ti(3) C(2) T(x) MXene microstrip transmission lines with low-energy

15 tiple sclerosis directly after autopsy, at 3 T, using T1 and proton-density/T2-weighted, as well as F

16 rapy or craniotomy who underwent 1.5-T and 3-T same-plane T1-weighted MRI (in any order).

17 three-dimensionally printed molds by using 3-T MRI with DR-CSI and were then sliced to create coregis

18 ancreatic T(2) values were nearly 40% at 1.5 T and < 12% at 3.0 T.

19 I(2) was 98% at 1.5 T and 3.0 T.

20 erwent transpedal MR lymphangiography at 1.5 T with T1-weighted imaging after interstitial pedal of g

21 V using the MOLLI T1 mapping sequence at 1.5 T.Supplemental material is available for this article.(C

22 tion therapy or craniotomy who underwent 1.5-T and 3-T same-plane T1-weighted MRI (in any order).

23 MRI, collected within an ultra-high-field (7 T) scanner, we found that the extent of vertical asymmet

24 Using high-resolution 7 T time-of-flight angiography we manually classified hipp

25 Gal-9(+) T cells exhibited the phenotype characteristics of effec

26 ch with experiments that probe proteins in a T-dependent fashion, e.g., for assessing the stability o

27 eat me" signal for macrophages) and PD-L1 (a T-cell inactivator) on their surface.

28 MD received anti-VEGF therapy according to a T&E (n = 163) or PRN (n = 101) regimen.

29 When a T cell and an antigen-presenting cell form an immunologi

30 thymidine analogs along with the natural A, T, G and C bases during DNA synthesis, which allows for

31 ssible to defeat this mechanism and activate T cells with solution ligands by cross-linking pMHC or u

32 Nuclear Factor of Activated T cells 5 (NFAT5) is a transcription factor (TF) that me

33 comorbidity had larger numbers of activated T cells compared with patients who had fewer risk factor

34 tages of tumor inception to subvert adaptive T cell immunity.

35 f transiently inhibiting LDH during adoptive T cell-based immunotherapy, with an unanticipated cooper

36 The application of adoptive T cell therapies, including those using chimeric antigen

37 e not inert or less potent versions of adult T cells but instead are a broadly reactive layer of T ce

38 nd that the immunodominance of high-affinity T cell clones declined during the chronic infection phas

39 To understand how these compounds alter T cell function, we assessed their therapeutic activity

40 Although T cell migration is most frequently defined in the conte

41 type 2 innate lymphoid cell activation, and T(h)2 cell differentiation were found in gut mucosa of m

42 Ankara vector to induce HBV-specific B- and T-cell responses.

43 te Notch, a critical regulator of B cell and T cell development.

44 response eQTLs (reQTLs) in myeloid cells and T cells, respectively.

45 development of T follicular helper cells and T follicular regulatory (Tfr) cells that regulate germin

46 ting both conventional T cells (T(conv)) and T(regs), subsequently followed by more rapid rebound of

47 c predisposition, epidermal dysfunction, and T-cell driven inflammation.

48 y epithelial activation, ILC2 expansion, and T(H) 2 differentiation.

49 s mTOR activation in IL-17(+) gammadelta and T(H)17 cells.

50 of Tilia cordata Mill, T.x europaea L., and T. platyphyllos Scop.

51 ignificantly increased GM-CSF production and T(H)2 cell differentiation.

52 responses through altering LN structures and T cell behaviors.

53 activation thresholds (CCL1/3/4/5/XCL1); and T(M) chemokine profiles modulated by persisting viral Ag

54 Anti-T(H) 2 therapies have the potential to effectively reduc

55 xes cannot compete with Gd(III) complexes as T(1) MRI contrast agents.

56 ppress the activity of pancreas autoreactive T cells in newly hyperglycemic non-obese diabetic (NOD)

57 B, T, and myeloid cells were analyzed before anti-CD20 admi

58 However, Batf3 (-/-) T cells underwent increased apoptosis during contraction

59 RT) was associated with significantly better T scores on GP-NDH, WAIS-IIIDS, Stroop Color-Naming; bet

60 findings define AP-1 as the key link between T cell activation and chromatin remodeling.

61 ositive viral test with a cycle threshold (C(T) ) of <35 or seroconverted during the follow-up period

62 air of recurrent cortico-thalamo-cortical (C-T-C) loops.

63 rforin contributed to both CD8+ and CD4+ CAR T cell cytotoxicity but was not required for in vitro or

64 discuss the innovative designs of novel CAR T cell products that are being developed to increase and

65 -presentation, that CD40L-overexpressing CAR T cells elicit an impaired antitumor response in the abs

66 activity of chimeric antigen receptor (CAR) T cells.

67 generation Chimeric Antigen Receptors (CAR) T cells demonstrating specific cytolytic activity.

68 CAR-T cells have shown encouraging activity against recurren

69 These drawbacks can be circumvented by CAR-T cells targeting tumour-specific driver gene mutations,

70 imeric antigen receptor-modified T-cell (CAR-T-cell) therapy are limited.

71 proach is often limited by the extent of CAR-T cell expansion in vivo.

72 ich are then eradicated by CD19-specific CAR-T cells in immunodeficient and immunocompetent mouse mod

73 n have been identified by DFT and DLPNO-CCSD(T) calculations.

74 cific cytokines produced by autoreactive CD4 T cells contribute to the pathogenesis of MS.

75 We have examined the priming of naive CD4 T cells in vitro at fever temperatures, and we report no

76 ive range, multifunctional CD8(+) and CD4(+) T cell responses with S protein-specific killing activit

77 can be found in the peripheral blood CD4(+) T cells of patients at all stages of HIV-1 infection.

78 IV DNA isolated from peripheral blood CD4(+) T-cells at weeks 16 and 18 after randomisation.

79 not promote resurrection of exhausted CD4(+) T-cell memory in chronic infection.

80 th factor-beta receptor 2 (TGFBR2) in CD4(+) T cells, but not CD8(+) T cells, halts cancer progressio

81 entially enhance our understanding of CD4(+) T cell recognition.

82 The discovery of CD4(+) T cell subset-defining master transcription factors and

83 Quiescence is a hallmark of CD4(+) T cells latently infected with human immunodeficiency vi

84 In support, adoptive transfer of old CD4(+) T cells that were transfected with a lentiviral vector i

85 NOD-PerIg CD8(+) T cells but required CD4(+) T cells.

86 mary, the current study suggests that CD4(+) T cells are critical for controlling acute-stage poliomy

87 g of the Th1/Th2 paradigm ignited the CD4(+) T cell field.

88 ifies highly polyfunctional CD8(+) and CD4(+)T(M) subsets; long-term CD8(+)T(M) maintenance is associ

89 CD4+ T cells derived from individuals with latent Mtb infecti

90 Although CD4+ T cells are implicated in MS pathogenesis and have been

91 opic cytokine produced predominantly by CD4+ T cells.

92 plication-competent virus cultured from CD4+ T cells.

93 The Matrix-M1 adjuvant induced CD4+ T-cell responses that were biased toward a Th1 phenotype

94 ing the risk of progression using naive CD4+ T-cells was predictive of progression along the whole IA

95 sing macrophage-specific (CD14) but not CD4+ T cell-specific (CD3) antibodies, suggesting that M-trop

96 regnancy is associated with recovery of CD4+ T cell immunity.

97 us and influences the susceptibility of CD4+ T cells to HIV-1 replication.

98 ements drive gene expression in primary CD4+ T cells.

99 ngs demonstrate that STAT1 signaling and CD8 T cells concomitantly act to mitigate MuPyV-encephalopat

100 icrobiome shifts and enhanced intestinal CD8 T cell responses.

101 Resting CMV-specific CD8 T cells were terminally differentiated and expressed hig

102 B*57:01-restricted, HIV epitope-specific CD8 T-cell responses showed beneficial functional patterns a

103 I and III responses, early CD4(+) and CD8(+) T cell activation, and counterregulation by the co-recep

104 elicit broadly protective CD4(+) and CD8(+) T cell responses.

105 ssing and priming for both CD4(+) and CD8(+) T cells and of the direct orchestration of their cross-t

106 Activation of cytotoxic CD8(+) T cells by cross-priming DC contributes to exacerbation

107 n contrast, increased frequency of EM CD8(+) T cells associated with reduced risk of graft failure.

108 e cancer- and virus-induced exhausted CD8(+) T cells, by enhancing the quality and survival of immune

109 PD-L1 pathway reinvigorates exhausted CD8(+) T cells, it fails to restore T cell repertoire diversity

110 is in Fcgr2b(+), but not Fcgr2b(-/-), CD8(+) T cells.

111 Using G9Calpha(-/-)CD8(+) T cells specific for proinsulin, we studied the mechanis

112 ell killing by freshly isolated human CD8(+) T cells, which represent a challenging but valuable mode

113 Inhibition of NOX4 increased CD8(+) T cells and restored responsiveness to immune therapy, s

114 indicated that CD20(+) B lymphocytes, CD8(+) T lymphocytes, and CD11c(+) cells are susceptible to IAV

115 me, phenotype, and function of memory CD8(+) T cells, sharing the same HSV-1 epitope-specificities, f

116 2 (TGFBR2) in CD4(+) T cells, but not CD8(+) T cells, halts cancer progression as a result of tissue

117 ld and a conceptually simple model of CD8(+) T cell Ag recognition, in which Ag dose and affinity do

118 nd neoantigen load) and the degree of CD8(+) T cell infiltration were not associated with clinical re

119 ctional consequence of LEC priming of CD8(+) T cells is unknown.

120 developed in the absence of NOD-PerIg CD8(+) T cells but required CD4(+) T cells.

121 pands the proportion of proliferating CD8(+) T cells in the tumor with enhanced cytolytic potential a

122 cells and the induction of protective CD8(+) T cell responses.

123 R signal strength is able to regulate CD8(+) T cell effector cytokine R production independent of TCR

124 abacavir/abacavir analogue-responsive CD8(+) T-cell clones was measured using IFN-gamma ELIspot.

125 cDC1s in expansion of tumor-specific CD8(+) T cells remains unclear.

126 epertoire diversity of virus-specific CD8(+) T cells.

127 gamma-delta T cells (CD3(+)TCRgd(+)), CD8(+) T cells (CD3(+)CD8(+)CD161(+)PD1(+)), and memory B cells

128 A distinct TEMRA CD8(+) T cell subpopulation was identified that was characteriz

129 onclusion, we show that MEKi leads to CD8(+) T cell reprogramming into T(SCM) that acts as a reservoi

130 tably, constitutive CCL5 expression by CD8(+)T(M) serves as a unique functional imprint of prior anti

131 8(+) and CD4(+)T(M) subsets; long-term CD8(+)T(M) maintenance is associated with a pronounced increas

132 es dominate the earliest stages of the CD8(+)T(M) recall response because of expeditious synthesis/se

133 The number of intravascular CD3+CD8+ T cells was influenced by CM status (CM+ > CM-, P = 0.00

134 rectly or indirectly excluding effector CD8+ T cells from the tumor microenvironment.

135 m patients with MS points to a role for CD8+ T cells in disease pathogenesis.

136 en-specific progeny of individual naive CD8+ T cells to the T effector (TEFF), T circulating memory (

137 alization are important determinants of CD8+ T cell-mediated efficacy against SIV.

138 letion, impacting both conventional T cells (T(conv)) and T(regs), subsequently followed by more rapi

139 to MCC cells with restored STING, cocultured T cells expressing MCPyV-specific T cell receptors (TCRs

140 -cell depletion, impacting both conventional T cells (T(conv)) and T(regs), subsequently followed by

141 nctional pathway alpha-diversity between CTX-T and CTX-N infants.

142 on the advantages and limitations of current T(1) contrast agents and the potential of IONPs to serve

143 ctor b (P-TEFb), composed of CDK9 and cyclin T, stimulates transcriptional elongation by RNA polymera

144 Cytotoxic T cells play a key role in adaptive immunity by killing

145 Cytotoxic T lymphocytes (T) and natural killer cells are the main

146 nate immune responses and adaptive cytotoxic T cell responses.

147 for activation and persistence of cytotoxic T lymphocytes.

148 use line is an ideal tool to study cytotoxic T lymphocyte biology and to optimize personalized immuno

149 is shows that the thermophoretic mobility (D(T)) is thermophobic in sign and increases linearly with

150 ith lower levels of Th1 cytokines, decreased T cell infiltration, increased B cell numbers, and decre

151 Subsets of gamma-delta T cells (CD3(+)TCRgd(+)), CD8(+) T cells (CD3(+)CD8(+)CD

152 Noninvasive strategies detecting T-cell activation would allow for early diagnosis and po

153 )CD8(-)TCRalphabeta(+), double-negative (DN) T cells, in mouse secondary lymphoid organs.

154 tion of host T(conv) and host T(regs), donor T(regs) failed to engraft even with interleukin-2 (IL-2)

155 We first analyze the E(T(1)), E(S(1)), and E(T(2)) of benzene and cyclobutadiene (CBD) as excited-sta

156 We first analyze the E(T(1)), E(S(1)), and E(T(2)) of benzene and cyclobutadien

157 atory objectives included tracking of edited T cells.

158 T(SCM) that acts as a reservoir for effector T cells with potent therapeutic characteristics.

159 ed the phenotype characteristics of effector T cells (CD45RA(+), CD45RO-/lo, CD62L(-), CD27lo) with h

160 Loss of Yap in T cells results in enhanced T-cell activation, differentiation, and function, which

161 Ex-T(RM) cells, former intestinal T(RM) cells that rejoined

162 This can complement existing T cell-directed immunotherapy, providing a promising app

163 activity is linked to T-bet in Ag-expCD4(+) T cells but that reduction in mTOR activity may not dire

164 Follicular T helper (TFH) cells provide B-cell help and are crucial

165 ranslates in vivo to an improved ability for T cells to infiltrate and repress tumors.

166 munotherapy and has general implications for T cell-based immunotherapies.

167 a(v)3.1 and Ca(v)3.2 and produced functional T-type VGCC currents when patch clamped.

168 The wG-T->G-T* tautomerization is predicted to be endoergic in aqueo

169 3 providing electrostatic stabilization of G-T*.

170 ucture of PDE6 complexed to GTP-bound Galpha(T).

171 late and coordinate alphabeta and gammadelta T cell responses remains unknown.

172 Activation of gammadelta T cells can be elicited by butyrophilin and butyrophilin

173 role of the different subsets of gammadelta T-cells detected in the skin in steady-state, psoriasis,

174 tion of T(h)1 gene expression profiles in GC T(fh) cells.

175 while conjugation of long-chain Vi generates T-dependent antigens, the conjugates also retain T-indep

176 ons/glia for the brain data and granulocytes/T cells/B cells/monocytes for the blood data.

177 ition to suppressing viremia, bNAbs may have T cell immunomodulatory effects as seen for other forms

178 rized population of extrafollicular B helper T cells, which produced IL-10 and could play a prominent

179 ), CD45RO-/lo, CD62L(-), CD27lo) with higher T-bet expression.

180 te robust depletion of host T(conv) and host T(regs), donor T(regs) failed to engraft even with inter

181 of T(regs) Despite robust depletion of host T(conv) and host T(regs), donor T(regs) failed to engraf

182 NINJA will enable studies of how T cells respond to defined neoantigens in the context of

183 Here we show that primary mouse and human T cells engage in macropinocytosis that increases in mag

184 a, we develop a quantitative theory of human T cell dynamics compatible with the statistical laws of

185 apicoplast has a key role in heme biology in T. gondii and is important for both mitochondrial and ge

186 ll, we find that sex-specific differences in T(reg) cells from VAT are determined by the tissue niche

187 Finally, deletion of Dot1L in T cells resulted in an impaired immune response.

188 The highest thymol (66%) was found in T. migricus exposed to blue light, while the least (1.69

189 ght, while the least (1.69%) was observed in T. kotschyanus grown under red-blue light.

190 ifarnib as a potential therapeutic option in T-cell leukemia and TCL.

191 actor RASGRP1 is frequently overexpressed in T-ALL patients.

192 signaling, could have a suppressive role in T-ALL.

193 ions harboring genes with prominent roles in T cell development in both strains.

194 rtance of inappropriate NOTCH1 signalling in T-cell acute lymphoblastic leukaemia (T-ALL), and the in

195 l persistence is particularly significant in T(EM) cells.

196 Loss of Yap in T cells results in enhanced T-cell activation, different

197 on in complex biological settings, including T cell immunology.

198 ts an important role for B cells in indirect T cell priming and further emphasizes the advantage of c

199 The vaccine-induced T cells had a cytotoxic phenotype and were capable of tr

200 in Tph1 deficient ILC2s including inducible T cell co-stimulator (Icos).

201 Ex-T(RM) cells, former intestinal T(RM) cells that rejoined the circulating pool, heritabl

202 heightened capacity to redifferentiate into T(RM) cells.

203 Ki leads to CD8(+) T cell reprogramming into T(SCM) that acts as a reservoir for effector T cells wit

204 Mucosal-associated invariant T (MAIT) cells are important for immune responses agains

205 ing in T-cell acute lymphoblastic leukaemia (T-ALL), and the involvement of BCL6 in other types of le

206 ering-related genes, such as FLOWERING LOCUS T (FT), FLOWERING LOCUS C (FLC), AGAMOUS (AG) and APETAL

207 ng an anomalous peak in specific heat at low T, magnetic phase transitions, and no mixed valency), Yb

208 xBox(4+) for the population of the low-lying T(1) state.

209 Cytotoxic T lymphocytes (T) and natural killer cells are the main cytotoxic kille

210 CD29 also marked T cells with cytotoxic gene expression from different ti

211 rain has the unique ability to infect mature T cells.

212 r vaccinia virus (VV) stimulation to measure T cells elicited after childhood smallpox vaccination.

213 role for LDH in modulating cytokine-mediated T cell differentiation and underscore the therapeutic po

214 ntiation programs in the human CD8(+) memory T cell pool, with potentially broad implications for the

215 iated with potent antiviral function: memory T cells secreted cytokines and expanded upon antigen re-

216 Human skin contains a population of memory T cells that supports tissue homeostasis and provides pr

217 transfer studies indicate that these memory T cells develop in a cell-intrinsic manner following thy

218 Total memory T-cell responses were measured after anti-CD3 or vaccini

219 hould include flowers of Tilia cordata Mill, T.x europaea L., and T. platyphyllos Scop.

220 ing chimeric antigen receptor (CAR)-modified T cells, to solid tumors requires combinatorial strategi

221 Is) after chimeric antigen receptor-modified T-cell (CAR-T-cell) therapy are limited.

222 pic model of TCR signaling in which multiple T cell responses share a common rate-limiting threshold

223 hlight new evidence indicating that neonatal T cells are not inert or less potent versions of adult T

224 s could benefit from such products, since no T cells recognizing any EBV-derived peptide in this comm

225 lobal transcriptome reversion and normalized T(H)17 cell/IL23 signaling, whereas dupilumab led to a s

226 matical modeling and statistical analyses of T cell receptor sequencing data, we develop a quantitati

227 Bcl6 is required for the development of T follicular helper cells and T follicular regulatory (T

228 whilst much is known about the existence of T-R conflicts, our understanding of the genetic and temp

229 ssue and is related to the B7/CD28 family of T-cell immune checkpoint markers.

230 rly clonal dynamics imprint the hierarchy of T cell clone sizes with implications for pathogen defens

231 In tracking the journey of T cells traversing from the thymus to the periphery and

232 but instead are a broadly reactive layer of T cells poised to quickly develop into regulatory or eff

233 eir therapeutic activity in a mouse model of T cell-mediated autoimmunity that mimics multiple sclero

234 r virus (EBV) is associated with a number of T-cell diseases, including some peripheral T-cell lympho

235 with a substantial increase in the number of T-cell receptor (TCR) sequences and their cognate antige

236 ns of GRA12 to the molecular pathogenesis of T. gondii infection were examined in vitro and in vivo.

237 s had significantly increased percentages of T lymphocytes and higher levels of a wide array of infla

238 ted GC B cell responses and the promotion of T(h)1 gene expression profiles in GC T(fh) cells.

239 bsequently followed by more rapid rebound of T(regs) Despite robust depletion of host T(conv) and hos

240 in ligase Peli1 as an important regulator of T cell metabolism and antitumor immunity.

241 ved drug FTY720 increased the sensitivity of T cells to the cytokine interleukin-2 (IL-2) through a p

242 s study is important to our understanding of T-ALL.

243 tion of CD68/CD206 on MNPs and CD69/CD103 on T cells.

244 e achieved with immunotherapy that relies on T lymphocyte-mediated recognition of tumor antigens.

245 In conclusion, orchestrated T cells are able to regulate osteoclasts at the early st

246 For these organs, the mean pancreatic T(2) values were nearly 40% at 1.5 T and < 12% at 3.0 T.

247 f T-cell diseases, including some peripheral T-cell lymphomas, hemophagocytic lymphohistiocytosis, an

248 rticular, ET were enriched in polyfunctional T cells.

249 rnatives on how to establish tau positivity (T+) for multiple tau-imaging tracers in order to reach a

250 We have recently reported that the putative T. gondii CGL gene encodes a functional enzyme.

251 ursors of M2 macrophages, DCs and regulatory T cells.

252 CR5(-) memory Th cells as well as regulatory T and B cells were increased.

253 or the function of thymus-derived regulatory T (Treg) cells (ie, FOXP3), resulting in impaired Treg f

254 PI3Kdelta is a key regulator of regulatory T (Treg) cell function.

255 g memory, all animals performed a reinforced T-maze alternation task, then a more challenging version

256 cells for metabolic resources often renders T cells dysfunctional.

257 ntigen-presenting cells (aAPCs) and reporter T cells.

258 th enhanced cytolytic potential and requires T cell migration from lymph nodes for therapeutic effica

259 tection of atabecestat metabolite-responsive T-cell clones activated via a pharmacological interactio

260 xhausted CD8(+) T cells, it fails to restore T cell repertoire diversity.IMPORTANCE Checkpoint inhibi

261 pendent antigens, the conjugates also retain T-independent properties, leading to detrimental effects

262 SETBP1 in aCML, and SF3B1-JAK2 in MDS/MPN-RS-T).

263 SARS-CoV-2-specific T cell responses were driven by TCR clusters shared betw

264 We were able to detect SARS-CoV-2-specific T cells in 10 of 10 COVID-19 patients with mild symptoms

265 enance of extraordinarily large CMV-specific T cell populations.

266 ffinity analyses of cytomegalovirus-specific T cells, and through the generation and in vivo monitori

267 sed to measure the frequency of EBV-specific T-cell responses between groups following stimulation wi

268 cocultured T cells expressing MCPyV-specific T cell receptors (TCRs) show increased cytokine producti

269 pitopes targeted by clusters of Mtb-specific T cells, we carried out a screen of 3,724 distinct prote

270 Total and spike-specific T cell responses correlated with spike-specific antibody

271 ly, we discovered that G. stearothermophilus T-1 can also utilize lactose and galactosyl-glycerol via

272 Cytokines that stimulate T cell proliferation, such as interleukin (IL)-15, have

273 rate that alphaCD3 alone induced substantial T-cell depletion, impacting both conventional T cells (T

274 p Color-Naming; better motor and SIP summary T scores.

275 magnitude upon T cell activation to support T cell growth even under amino acid (AA) replete conditi

276 iming and effector phases, provokes systemic T cell responses against dominant and subdominant neoant

277 naive CD8+ T cells to the T effector (TEFF), T circulating memory (TCIRCM), and TRM pools by lineage-

278 the superconducting transition temperature, T(c), near to optimal doping that sheds light on the nat

279 The resultant radiance bound by the T(4) law limits the ability to regulate radiative heat.

280 ng through multiple receptors, including the T-cell receptor (TCR), co-receptors, and cytokine recept

281 Activation of the T cell receptor (TCR) results in binding of the adapter

282 ation resolution, including dampening of the T helper 1 response, alternative activation of macrophag

283 e reasons, a cataloging and appraisal of the T-cell epitopes targeted in type 1 diabetes was complete

284 we report a chromosome-scale assembly of the T. sinense genome.

285 geny of individual naive CD8+ T cells to the T effector (TEFF), T circulating memory (TCIRCM), and TR

286 Thioflavin T fluorimetry estimates rapid and near-stoichiometric co

287 data suggest that mTOR activity is linked to T-bet in Ag-expCD4(+) T cells but that reduction in mTOR

288 nd ecosystem respiration (RE) in response to T(a) and EF anomalies were compared for different forest

289 regulation of 12-OPDA and JA in response to T. virens colonization results in ISR induction.

290 G), grape seed and olive (O) or grape total (T), called ESG, ESO and EST, respectively.

291 lation between IVIG dose and toxin-triggered T-cell proliferation (r = -.67, P < .0001).

292 pinocytosis that increases in magnitude upon T cell activation to support T cell growth even under am

293 [(11)C]UCB-J V(T) was significantly lower in the frontal and anterior c

294 gen-mediated signals to human Vgamma9Vdelta2 T cells.

295 The wG-T->G-T* tautomerization is predicted to be endoergic in

296 However, whether T cells induced by one viral species cross-react with ot

297 In those whose T cells had the capacity to respond, older patients with

298 vident in immunodeficient mice infected with T. gondii, as associated with high expression level (P <

299 with this hypothesis, mice transplanted with T-cells co-expressing NOTCH1 and NRARP develop leukemia

300 p leukemia later than mice transplanted with T-NOTCH1 cells.