ライフサイエンスコーパス: TCR gene rearrangement

1 se mice are determined by factors other than TCR gene rearrangement.

2 tment is determined before or independent of TCR gene rearrangement.

3 YDbSmcy share an invariant Vbeta8.2-Jbeta2.3 TCR gene rearrangement.

4 by measuring the excisional DNA products of TCR-gene rearrangement.

5 circles created by alphabeta and gammadelta TCR gene rearrangements.

6 well as analysis of several sequential human TCR gene rearrangements.

7 vival of TN cells and its role in regulating TCR gene rearrangements.

8 me of jawed vertebrates that were capable of TCR gene rearrangements.

9 urface antigens, and clonal T-cell receptor (TCR) gene rearrangements.

10 81/CD42, and all had clonal T-cell receptor (TCR) gene rearrangements.

11 TCR gene rearrangement and expression are central to the

12 nt as indicated by positive results for both TCR gene rearrangement and flow cytometry was associated

13 erogeneous in developmental potential before TCR gene rearrangement and suggest that in some precurso

14 T cells are a common and natural product of TCR gene rearrangement and thymocyte development.

15 previously shown to impair T cell receptor (TCR) gene rearrangement and to cause a partial block in

16 based on immunoglobulin/T-cell receptor (Ig/TCR) gene rearrangements and with quantification of IKZF

17 The latter involves RAG re-expression, TCR gene rearrangement, and expression of a novel TCR.

18 by polymerase chain reaction analysis of Ig/TCR gene rearrangements, and patients were assigned to a

19 Further, because TCR gene rearrangements are generally limited to T linea

20 of immunoglobulin (Ig) and T-cell receptor (TCR) gene rearrangement are reflected in the accessibili

21 that differ from the mouse are the status of TCR gene rearrangements at the nonexpressed loci, the ti

22 cal Ig heavy chain (IgH) or T-cell receptor (TCR) gene rearrangement at initial diagnosis and subsequ

23 Progenitor cells undergo T cell receptor (TCR) gene rearrangements during their intrathymic differ

24 Quantitative RT-PCR assessment of different TCR gene rearrangement events revealed lower levels in M

25 itored indirectly by measuring the levels of TCR gene rearrangement excision circles in peripheral T

26 p study of the junctional diversity of these TCR gene rearrangements focuses on characterization of t

27 studies that have assessed T-cell receptor (TCR) gene rearrangements (GRs) present at different anat

28 e negative, DN) thymocytes is independent of TCR gene rearrangement; however, induction of CD5 surfac

29 cular, we now have a better understanding of TCR gene rearrangement in endomysial T cells, regulation

30 flow cytometry and by the presence of clonal TCR gene rearrangements in four patients' posttreatment

31 Analysis and interpretation of Ig and TCR gene rearrangements in the conventional, low-through

32 flow cytometry, and clonal T-cell receptor (TCR) gene rearrangements in two of two pretreatment bloo

33 tailed, comprehensive computer simulation of TCR gene rearrangement, incorporating the interaction of

34 , these findings demonstrate that productive TCR gene rearrangement is associated with events that ca

35 No TCR gene rearrangement is observed in G/M cells from non

36 el of lineage commitment in which sequential TCR gene rearrangements may influence alphabeta/gammadel

37 sity, as detected by Ig and T-cell receptor (TCR) gene rearrangements, may represent a very useful pr

38 own-regulation of the protein RAG2, on which TCR gene rearrangement obligatorily depends.

39 We analyzed the sequences of Ig and TCR gene rearrangements obtained at presentation and rel

40 Immunoglobulin (Ig) and T-cell receptor (TCR) gene rearrangements provide clonal markers useful f

41 pment of T cell precursors in the absence of TCR gene rearrangement, recombinase-activating gene-defi

42 tromal signals that induce functions such as TCR gene rearrangement reside mainly in the outer half o

43 ised DNA products of baboon T-cell receptor (TCR) gene rearrangement (signal-joining TCR excision cir

44 excisional DNA products of T-cell-receptor (TCR) gene rearrangement to measure thymic output directl

45 in all cases and an identical clonal IgH or TCR gene rearrangement was found on PCR analysis of DNA

46 ment expressing a repertoire biased to early TCR gene rearrangements, we developed a mouse model in w

47 ifs created by quasi-random T cell receptor (TCR) gene rearrangements, with diversity being highest f