ライフサイエンスコーパス: TG neuron

1 T-ORF63, but not RNA 63, expression in human TG neurons.

2 xpression in subsets of TRPV1(+) and CGRP(+) TG neurons.

3 diate reactivation from latency in PT versus TG neurons.

4 n of viral progeny in SCG neurons but not in TG neurons.

5 ty with unique characteristics of subsets of TG neurons.

6 lized with these receptors in SCG but not in TG neurons.

7 s were able to drive transgene expression in TG neurons.

8 more highly attenuated in MRC5 cells than in TG neurons.

9 capsaicin responses in both male and female TG neurons.

10 ng that tonic NO levels inhibit M-current in TG neurons.

11 he mutation resulted in hyperexcitability of TG neurons.

12 +) channels (VGCCs) in 23% of small-diameter TG neurons.

13 l differences in the translatomes of DRG and TG neurons.

14 PRLRs), short and long, were also present in TG neurons.

15 n gene-related peptide release from cultured TG neurons.

16 , the truncated promoters did not express in TG neurons.

17 e PCR assay for the viral genome on purified TG neurons.

18 alpha 4 and beta 2) were localized in intact TG neurons.

19 but no significant differences were found in TG neurons.

20 mc) encoding genes are almost exclusively in TG neurons.

21 ected calves increased the number of E2F2(+) TG neurons.

22 ial, and increases evoked firing activity in TG neurons.

23 e-gated K(+) currents (IA currents) in these TG neurons.

24 spontaneous neuronal activity in nociceptive TG neurons.

25 HEK293T cells and mouse trigeminal ganglion (TG) neurons.

26 se (PI3K) pathways in female rat trigeminal (TG) neurons.

27 spine density in Mecp2(WT_EGFP) transgenic (TG) neurons.

28 er of BMP4 signaling in trigeminal ganglion (TG) neurons.

29 establishes latency in trigeminal ganglion (TG) neurons.

30 Compared with non-Tg neurons, 3xTg-AD neurons had more mitochondria/neuron

31 h reduced beta-catenin protein expression in TG neurons 6 h after dexamethasone treatment.

32 ome P450 isozymes are rapidly upregulated in TG neurons after orofacial inflammation and increase the

33 k of IL-10R1 in DRG and trigeminal ganglion (TG) neurons also increased circulating and DRG levels of

34 v4.3 channels amplified IA currents in these TG neurons and alleviated orofacial cold hypersensitivit

35 By 24 h, staining was also seen in a few TG neurons and by 96 h their number had greatly increase

36 We found that CCL2 was mainly expressed in TG neurons and cells associated with dura blood vessels,

37 The number of TG neurons and cells in pharyngeal tonsil expressing the

38 fected calves increased the number of Sp1(+) TG neurons and cells in pharyngeal tonsil indicating tha

39 ed selective expression of these hormones in TG neurons and dural nerves; and showed GH expression in

40 8, and 9 trafficked from the whiskerpad into TG neurons and expressed transgenes within cell bodies a

41 phrixotoxin-2 inhibited IA-currents in these TG neurons and induced orofacial cold hypersensitivity.

42 expressed mutant TRESK subunits in cultured TG neurons and observed a significant decrease in the la

43 PRL expression was restricted to TG neurons and was highly overlapped with transient pote

44 discharges from injured trigeminal ganglion (TG) neurons and thalamocortical reorganization are possi

45 activity in dental pulp, trigeminal ganglia (TG) neurons, and their nerve fibers.

46 g patterns in the IAN and V2 branches of the TG neurons; and (3) the receptive field expanded, the mo

47 gly, VP16 protein expression was detected in TG neurons at 8 hours after explant whereas infected-cel

48 In TG neurons, bICP0 and the viral tegument protein VP16 ar

49 sed green fluorescent protein efficiently in TG neurons but did not induce HSV-1 reactivation.

50 multiple transgenes could be co-delivered to TG neurons by separate AAV vectors.

51 Taken together, these data show that adult TG neurons can mount an effective antiviral response onl

52 anscript (LAT)-positive trigeminal ganglion (TG) neurons coexpressed SSEA3, 71% coexpressed Trk(A) (t

53 female GR(S229A) mice contained fewer VP16 + TG neurons compared to male GR(S229A) mice or wild-type

54 These data reveal that rat TG neurons contain the entire spectrum of mammalian NnAC

55 stochemical studies revealed that almost all TG neurons contained alpha 7-LI and alpha 4-LI, and that

56 tribution of LAT-positive, latently infected TG neurons contrasted sharply with (i) the overall distr

57 ught to further investigate this response in TG neurons cultured from adult mice deficient in a varie

58 mouse, may be such an intrinsic modulator of TG neuron development.

59 Cultured PTPRO(-/-) TG neurons display enhanced axonal outgrowth and branchi

60 For example, Akt3 is detected in more TG neurons during BoHV-1 latency than in reactivation an

61 y expressed in the same trigeminal ganglion (TG) neuron during reactivation and cooperatively stimula

62 emia zinc finger), and KLF15, are induced in TG neurons early during dexamethasone-induced reactivati

63 Direct mechanical stimulation of duck TG neurons evokes high-amplitude depolarizing current wi

64 Subsets of TG neurons express different receptors for growth factor

65 We further showed that these small-sized TG neurons expressed Kv4.3 voltage-gated K(+) channels,

66 hibition in vivo increases the percentage of TG neurons expressing deltaR on the surface and allows e

67 The number of TG neurons expressing E2F2, a transcription factor and c

68 diated GR activation increased the number of TG neurons expressing viral regulatory proteins, which e

69 root ganglion (DRG) and trigeminal ganglion (TG) neurons expressing the cold-sensitive TRPM8 channel

70 mbrane currents evoked in piperine-sensitive TG neurons far exceeded the algebraic sum of the respons

71 ling in a heterologous expression system and TG neurons from PRL receptor (PRLR)-null mutant mice by

72 In PTPRO mutant mice, subsets of TG neurons grow longer and more elaborate axonal branche

73 n part to a dramatic increase in the loss of TG neurons in animals infected with the LAT mutants.

74 he rabbit efficiently transduced >70% of the TG neurons in the optic tract.

75 frequencies from latently infected explanted TG neurons in the presence or absence of CD45(+) cells.

76 nd hypersensitization of trigeminal ganglia (TG) neurons in alcohol-withdrawal WT mice, but not in al

77 n anatomically relevant trigeminal ganglion (TG) neurons in both the xenograft and a carcinogen (4-ni

78 ndings support the conclusion that injury to TG neurons is more likely to cause chronic pain and chro

79 RPM8 antagonists, and were absent in DRG and TG neurons isolated from Trpm8(-/-) mice.

80 the state of persisting HSV genomes in some TG neurons may be more dynamic and more easily activated

81 Furthermore, the majority of BK-responsive TG neurons may have a potential to become responsive to

82 The predominant PRLR form in TG neurons/nerves from rats and humans is PRLR-S.

83 C/EBP-alpha protein expression is induced in TG neurons of infected calves and after dexamethasone-in

84 About 1.8% of the TG neurons of mice infected with 17/tBTK(-) harbored the

85 spontaneous activities, first in the injured TG neurons of the IAN (2-30 d), followed by uninjured V2

86 ifferences in the growth rates of TMN and DM-TG neurons on L1.

87 d WIN 55,212-2 (WIN) to cultured trigeminal (TG) neurons or isolated skin biopsies rapidly and signif

88 ulatory protein VP16 was readily detected in TG neurons prior to infected-cell protein 0 (ICP0) and I

89 nfection with HSV and antiviral signaling in TG neurons produce an unorthodox autophagic response.

90 These studies indicate that SP production in TG neurons projecting to the nasal epithelium is transie

91 Despite these findings, TG neurons responded to IFN-beta pretreatment with STAT1

92 We report that ~50% of trigeminal ganglion (TG) neurons retrogradely labeled from the DP express TRP

93 TG neurons showed decreased basal cytosolic calcium leve

94 f the trigeminocervical complex (TCC), where TG neurons terminate, and C-FOS expression was reversed

95 re pervasive latent HSV-1 infection of human TG neurons than originally thought.

96 additional genes, many within the subset of TG neurons that express TRPV1.

97 dicate GR activation increases the number of TG neurons that express viral regulatory proteins during

98 The percent of retrograde labeled TG neurons that respond to TRPV1 agonist, capsaicin, is

99 d NTG administration increased the number of TG neurons that responded to calcitonin-gene-related pep

100 e immunosurveillance of trigeminal ganglion (TG) neurons that harbor latent HSV-1.

101 tability of small-sized trigeminal ganglion (TG) neurons that innervated orofacial regions.

102 In contrast to latently infected TG neurons, the HSV-1 latency-associated transcript was

103 nd L1, were markedly different on TMN and DM-TG neurons, these differences were not sufficient to cau

104 al inflammation and increase the capacity of TG neurons to generate OLAMs.

105 in cGMP in the TG in vivo Exposing isolated TG neurons to GTN caused a rightward shift in the voltag

106 Capsazepine blocked the response of TG neurons to piperine at both physiological and acidic

107 The relative permissiveness of TG neurons to viral gene expression near the joint regio

108 In this study, HSV-1 infection of murine TG neurons triggered unusual clusters of autophagosomes,

109 y enhances both CGRP and PACAP signalling in TG neurons, ultimately leading to persistent neuronal se

110 phages and T cells in TG and dura but not in TG neurons under both control and disease states.

111 els in nociceptive-like trigeminal ganglion (TG) neurons underlies the trigeminal neuropathic pain ma

112 levels and redox state in nontransgenic (non-Tg) neurons until middle age, followed by a decline in o

113 (MRC5) and adult murine trigeminal ganglion (TG) neurons using the Illumina platform for cDNA sequenc

114 d on cultured adult rat trigeminal ganglion (TG) neurons voltage-clamped near their resting membrane

115 nt sites established in trigeminal ganglion (TG) neurons was determined using a single-cell quantitat

116 ne abundantly expressed in latently infected TG neurons, was not detected in PT during latency.

117 issociated adult murine trigeminal ganglion (TG) neurons were assessed for relative permissiveness fo

118 e of spread from the olfactory epithelium to TG neurons, while SSMs blocked systemic spread.

119 Treatment of TG neurons with epileptogenic compound-PTZ led to a mark

120 genetically manipulate trigeminal ganglion (TG) neurons would be useful in the study of the craniofa