ライフサイエンスコーパス: TG

1 TG and AG can be further investigated as potential bioma

2 TG and PG parameters from baboon and rhesus macaque plas

3 TG and PG parameters from Guinea pig samples were extrem

4 TG and PG were investigated in male and female donor pla

5 TG explants from latently infected, but not uninfected,

6 TG Therapeutics, Leukemia and Lymphoma Society Therapy A

7 uence contexts contained 85-90% AT and 5-10% TG.

8 The collective impact of abnormal TG and PG in HFD-fed mice produced normal fibrin formati

9 n inhibition alone was sufficient to abolish TG in FXII- or FXI-deficient plasma.

10 nutes or pretreatment with trypsin abolished TG, suggesting the presence of MV-associated proteins th

11 Additionally, TG-LASSO identified genes associated with the drug respo

12 low for EtioG (6.2 uM) as compared with AG, TG, and DHTG (46.2, 56.7, and 71.3 uM, respectively), wh

13 residual acid hydrolytic activities against TG and RE.

14 The abnormal rates of HDL-C and TG increased as the students maturated through the early

15 n (AMI) patients, higher admission LDL-C and TG levels have been shown to be associated with better c

16 ) with ~6M variants separately for LDL-C and TG with weights from a UK Biobank-based genome-wide asso

17 total B-12 with serum TC, LDL-C, HDL-C, and TG concentrations across trimesters.

18 sociated with serum levels of TC, LDL-C, and TG, but inversely associated with serum levels of HDL-C

19 .1(+) T cells was observed in the cornea and TG of SPP-inducible knockout mice compared to that in co

20 1B3-mediated uptake for EtioG, AG, DHTG, and TG were 19.8, 29.3, 69.6, and 110.4 uM, respectively.

21 Percoll-gradient enriched DRG and TG neuronal fractions produced distinct sex-dependent DE

22 asts, carried spontaneous apurinic sites and TG sequence lesions more frequent than ultraviolet (UV)-

23 Compared to ST and TG groups, optimal neural vessel density and branching i

24 th in SU and STG groups compared with ST and TG groups.

25 At longer times both TAC and TG mice progressed to overt HF (at 45 days and 11-13 wee

26 00 mg/dL, HDL <40 mg/dL, LDL >130 mg/dL, and TGs >150 mg/dL were 1.66 (95% CI: 1.52-1.82), 1.45 (95%

27 1.7 IU/mL and 6 IU/mL for anti-TPO and anti-TG, respectively.

28 en demonstrated for anti-thyroglobulin (anti-TG) and anti-thyroid peroxidase (anti-TPO) as models.

29 cant with the exceptions of human PG, baboon TG, rat TG and Guinea pig PG.

30 tes an energy-consuming futile cycle between TG hydrolysis and resynthesis, leading to inhibition of

31 irms several findings of differences between TG and DRG nociceptors described in the literature but a

32 clearance contributes to the decreased blood TG concentrations in ppHF dams.

33 s increased, suggesting that increased blood TG clearance contributes to the decreased blood TG conce

34 Obesity impairs the rise of maternal blood TG concentrations by reducing ANGPTL4 expression in mice

35 ys a vital role in increasing maternal blood TG concentrations during pregnancy.

36 estingly, a lower increase in maternal blood TG concentrations has been observed in some obese mother

37 We found not only that maternal blood TG concentrations in ppHF dams were remarkably lower tha

38 xpression of ANGPTL4 restored maternal blood TG concentrations in ppHF dams.

39 egnancy significantly reduces maternal blood TG levels.

40 ate if and how obesity alters maternal blood TG levels.

41 er, the pregnancy-induced elevation of blood TG was almost abolished in Angptl4 (-/-) dams.

42 ms, Ucp1 gene deletion did not restore blood TG concentrations in ppHF dams.

43 of HSV-1 in the lip propagates virus to both TG, but with delay in reaching the TG contralateral to t

44 suggesting that MTTP recognizes PRAP1-bound TG as a cargo and transfers TG along with PRAP1 to lipid

45 g of pairs of tyrosines, and is completed by TG proteolysis(3).

46 in-containing protein 3 (TIM-3) expressed by TG infiltrating gB-specific CD8(+) T cells from the HSV-

47 Moreover, genes identified by TG-LASSO for multiple drugs in a tissue were associated

48 t that 5-8% of the LD surface is occupied by TG molecules, a number that exceeds the maximal solubili

49 Then, we developed a new algorithm called TG-LASSO that explicitly integrates information on sampl

50 CaMKIIdelta TG mice lacking deltaB exhibited more severe HF, eccentr

51 were entirely mimicked in young CaMKIIdelta TG mice (6-8 weeks) where no overt cardiac dysfunction w

52 In CatA-TG mice, LV interstitial fibrosis formation was enhanced

53 Cardiac remodeling in CatA-TG was accompanied by an increased LV weight/body weight

54 activity in the LV of transgenic mice (CatA-TG) reduced EC-SOD protein levels by 43%.

55 radicals (WT, 4.54 mumol/mg tissue/min; CatA-TG, 8.62 mumol/mg tissue/min), increased inflammation, m

56 LV end diastolic volume (WT, 50.8 mul; CatA-TG, 61.9 mul).

57 ion, myocyte hypertrophy (WT, 19.8 mum; CatA-TG, 21.9 mum), cellular apoptosis, and elevated mRNA exp

58 ective role for CC to limit ROS and cellular TG accumulation, and to alter hepatic energy metabolism

59 reased ROS and marginally decreased cellular TG.

60 pplied for the quantification of most common TGs in almond, tiger nut, and argan oil.

61 The degree of interdigitation caused by CORE-TG is stable and determines the width of the TG-PL overl

62 tercalate just into the PL tail region (CORE-TG) are disordered and increase the amount of PL packing

63 disease) were included, with a median daily TG dose of 20 mg (range: 20-40 mg), a median treatment d

64 Decreasing TG synthesis by deleting neuronal diglyceride acyltransf

65 he long-term efficacy and safety of low-dose TG therapy in IBD patients failing AZA and MP.

66 ing and cooling processes, using TG/DTG/DTA, TG-MS, DSC, hot stage microscopy and DRX analysis.

67 An elevated TG level was not protective after adjustment.

68 erexpression of ANGPTL8 resulted in elevated TG levels in lean mice.

69 ified FAHFA levels, demonstrating that FAHFA-TGs breakdown is a regulator of cellular FAHFA levels.

70 GWAS of fasting TG and postprandial serum TG at 150 minutes resulted in

71 randial TG metabolism independent of fasting TG concentrations, resulting in smaller increases of HDL

72 on of postprandial TG independent of fasting TG, we calculated the TG response at 150 minutes by the

73 d TG at 150 minutes as a function of fasting TG.

74 TC, 120 for LDL-C, 47 for HDL-C, and 139 for TG; no strong trends.

75 ] for LDL-C and 1.24 [95% CI, 1.13-1.36] for TG PRS).

76 No significant effects were observed for TG, HbA1c, CRP, ALT, and AST.

77 d may be considered as cell-free therapy for TG nerve repair.

78 exceeds the maximal solubility reported for TGs in PL bilayers (2.8%).

79 directing neuronal lipid synthesis away from TG synthesis and toward PL synthesis may promote axon re

80 We transferred the reaction sites from TG to an engineered tyrosine donor-acceptor pair in the

81 Hormone synthesis from TG occurs in the thyroid gland via the iodination and co

82 of wild-type (WT) superinfecting virus from TG during the latent stage.

83 g acute infection in the trigeminal ganglia (TG) and brain stem compared to the control-vaccinated gr

84 on was quantified in the trigeminal ganglia (TG) and the brain stem from the same latently infected a

85 , sensory neurons within trigeminal ganglia (TG) are an important site for latency.

86 sory ganglia such as the trigeminal ganglia (TG) is influenced by virus-specific CD8(+) T cells that

87 1) leads to infection of trigeminal ganglia (TG), typically followed by the establishment of latency

88 rally VZV-infected human trigeminal ganglia (TG).

89 s sensory neurons within trigeminal ganglia (TG).

90 d ganglion types [DRG vs trigeminal ganglia (TG)].

91 iptome signature in the trigeminal ganglion (TG) that includes Rictor, the rapamycin-insensitive comp

92 ters latency within the trigeminal ganglion (TG), from which it can reactivate throughout the life of

93 minescence (PL) lifetime enables time-gated (TG) detection without autofluorescence background.

94 Simultaneously, the thrombin generation (TG) assay was used to assess their ability to initiate c

95 d PG, HFD also enhanced thrombin generation (TG).

96 GG/TG genotypes for rs7905446 and female gender were associ

97 The GG/TG genotypes were consistently associated with response

98 Transplant glomerulopathy (TG) is a major cause of late allograft loss.

99 EC-Glrx TG mice showed higher levels of VEGF-A in the tumors, in

100 Despite reduced angiogenesis, EC-Glrx TG mice unexpectedly developed larger tumors compared wi

101 Transection (ST), Simple Transection & Glue (TG), Stepwise Transection and Sutures (SU), and Stepwise

102 ) after periodontal debridement (test group [TG]1), or omega-3 PUFA + ASA (3 g of fish oil/d + 100 mg

103 h SCP were treated with aPDT+ST (test group, TG) or ST only (control group, CG), in a split-mouth des

104 On the other hand, TG-LASSO improved the predictions and distinguished resi

105 HCR-TG-FRET provided washing-free nucleic acid quantificatio

106 tions, resulting in smaller increases of HDL-TG and VLDL subparticles.

107 (high-density lipoprotein) subparticles (HDL-TG), a smaller decrease of HDL diameter and smaller incr

108 Hepatic TG production and intestinal TG absorption were unchange

109 remodeling may underlie the altered hepatic TG secretion we observed.

110 Liver size and hepatic TG content were significantly reduced, but plasma TG was

111 D development in GSD 1a by balancing hepatic TG production and secretion.

112 expression of PNPLA3(148M) increased hepatic TG levels in WT, but not in Cgi-58 KO mice.

113 as well as a novel mechanism whereby hepatic TG storage is promoted in response to lipogenic stimulat

114 Relative to non-significantly heritable TGs, heritable TGs had a greater number of associations

115 o non-significantly heritable TGs, heritable TGs had a greater number of associations with gene trans

116 ated risk of experiencing low HDL-C and high TG (all p < 0.05).

117 h LDL-C, 48% for low HDL-C, and 21% for high TG; no strong trends.

118 A 1 SD higher TG and LDL-C level caused a 0.062 (95% CI 0.040, 0.083)

119 d fat-free masses (0.35-0.49 kg), but higher TGs (11-13%).

120 T-ORF63, but not RNA 63, expression in human TG neurons.

121 vitro hormone-production assays using human TG expressed in HEK293T cells.

122 Intestinal mucus layers from IDO1-TG mice were 2-fold thicker than mucus layers from contr

123 here were significantly fewer CD8 T cells in TG from day 10 to day 25 but that after that the differe

124 ulatory protein VP16 was readily detected in TG neurons prior to infected-cell protein 0 (ICP0) and I

125 e neuro-inflammation severity, especially in TG.

126 l (CI): 0.46, 0.86) and 10 mg/dL increase in TG change associated with increased odds of LGA (OR = 1.

127 to be associated with a smaller increase in TG concentrations at 150 minutes (beta=-0.11; P-value=5.

128 emia zinc finger), and KLF15, are induced in TG neurons early during dexamethasone-induced reactivati

129 e mechanisms that lead to podocyte injury in TG remain unclear.

130 Increased urine podocin/creatinine ratio in TG signifies accelerated podocyte loss.

131 We observed a significant reduction in TG (p = 0.001), HbA1c (p = 0.019) and fasting plasma glu

132 02) but also the most pronounced increase in TGs (P = 0.03).

133 via LXRalpha and required for the increased TG accumulation in liver.

134 f LDs, LD-associated proteins, and increased TGs.

135 was necessary for abolishing RBC-MV-induced TG in normal pooled plasma, whereas kallikrein inhibitio

136 n on CD8(+) T cells in the latently infected TG is influenced by viral gene expression, suggests that

137 ne expression in the HSV-1 latently infected TG.

138 -specific CD8(+) T cells within the infected TG have been shown to play a crucial role in inhibiting

139 ikrein activated FIX in buffer and initiated TG in normal pooled plasma, as well as FXII- or FXI-defi

140 RBC-MVs initiated TG in normal pooled plasma and in FXII- or FXI-deficient

141 Integrating TG and PG measurements may define a prothrombotic risk f

142 Hepatic TG production and intestinal TG absorption were unchanged in ppHF dams, but systemic

143 in the distal intestine of VDR null mice (KO/TG mice) results in the normalization of serum calcium a

144 ,25(OH)(2)D(3) in the distal intestine of KO/TG mice.

145 ysis, which adjusted for the effects of LDL, TGs, body mass index (BMI), and age at menarche, corrobo

146 ing approaches were applied to the rat liver TG-GATEs dataset to develop feature selection and predic

147 Median TG was 1.19 (95% CI, 1.18-1.20) mmol/L, ranging from 0.9

148 ine the causal role of Trib1 in BBR-mediated TG lowering independent of LDLR regulation.

149 n of the VLDL lipidation proteins microsomal TG transfer protein and transmembrane 6 superfamily memb

150 MIOX-TG mice had worsening renal functions with kidneys havin

151 nerated MIOX-overexpressing transgenic (MIOX-TG) and MIOX knockout (MIOX-KO) mice.

152 For example, Akt3 is detected in more TG neurons during BoHV-1 latency than in reactivation an

153 signal-to-background ratios compared to non-TG imaging.

154 pid groups, for example, in males, 17.04% of TG lipid species decline with age whilst 37.86% increase

155 put, resulting in the excess accumulation of TG.

156 that chromatin obtained from the adrenals of TG mice containing the intron conversion binds more stro

157 Overall, application of TG can rationally modify the functionality and digestibi

158 In case of TG, when using the full datasets, an increased risk for

159 Two distinct classes of TG molecules that interact with the LD monolayer are fou

160 digestion showed decreased digestibility of TG-crosslinked chickpea-stabilized emulsions, while prot

161 for HSK but that while initial infection of TG is greater in CD28(-/-) mice, this begins to normaliz

162 inetic parameters to calculate the metric of TG/PG ratio revealed a quantifiable net shift toward a p

163 olated luteal LDs were composed primarily of TG, with lesser amounts of cholesteryl esters, diglyceri

164 the lack of a three-dimensional structure of TG has prevented mechanistic understanding(4).

165 ion with an efficiency comparable to that of TG.

166 hniques and paves the way for further use of TG-SERS and SPR in EV studies.

167 The hydrolytic cleavage of TGs generates free fatty acids (FFAs), which can serve a

168 drive adipocyte death and saponification of TGs.

169 rate at 12 months was 65 and 54% remained on TG until the end of follow-up.

170 sociated with statin use, but not the HDL or TG.

171 ntent were significantly reduced, but plasma TG was elevated in KO mice.

172 les, which correlated negatively with plasma TG and positively correlated with bacterial genera enric

173 LysoPE species and total CB polyunsaturated TGs.

174 o identify genetic variation of postprandial TG independent of fasting TG, we calculated the TG respo

175 o elucidate the genetics of the postprandial TG response through genome-wide association studies (GWA

176 r LIPC as a main contributor to postprandial TG metabolism independent of fasting TG concentrations,

177 als of a nonlinear regression that predicted TG at 150 minutes as a function of fasting TG.

178 ociated with higher systolic blood pressure, TGs, lipopolysaccharide-binding protein, and lower HDL c

179 ed for untargeted lipidomics and to quantify TG.

180 h the exceptions of human PG, baboon TG, rat TG and Guinea pig PG.

181 egrowth induced by lipin1 depletion required TG hydrolysis and PL synthesis.

182 uantify the lipoprotein profile (e.g., serum TG, LDL-C, and HDL-C concentrations).

183 a 40% (+/-12%; p < 0.05) reduction in serum TG concentration, a 28% (+/-21%; p < 0.05) increase in H

184 GWAS of fasting TG and postprandial serum TG at 150 minutes resulted in completely overlapping loc

185 GWAS of fasting and postprandial serum TG at 150 minutes were performed.

186 , swine and rat plasmas demonstrated similar TG, but swine plasmas did not generate plasmin.

187 In this study, we performed simultaneous TG and PG analysis in blood plasma samples from humans a

188 e-gated surface-enhanced Raman spectroscopy (TG-SERS) and monitoring the kinetics and amount of cellu

189 e TG-PL overlap, whereas that caused by SURF-TG fluctuates and is highly correlated with the area per

190 hus, when the supply of PLs is limited, SURF-TG may reduce surface tension by behaving as a secondary

191 Those at the monolayer surface (SURF-TG) are ordered like PLs with the glycerol moiety expose

192 ue transglutaminase immunoglobulin A (anti-t-TG-IgA) titer was assessed.

193 rt kinetics of glucuronides of testosterone (TG), dihydrotestosterone (DHTG), androsterone (AG), and

194 ride (TG) input in the liver is greater than TG output, resulting in the excess accumulation of TG.

195 We found that TG mice containing the intron conversion have (a) increa

196 Findings of this study indicate that TG can be used safely and the occurrence of hepatotoxici

197 From these results, it can be inferred that TG itself is not a causal risk factor for CAD, but it's

198 t contain shared and unique SNPs showed that TG is not a risk factor for CAD.

199 Long-term follow-up suggests that TG can be an effective and well-tolerated therapy in mor

200 The fact that TGs and SEs are the typical cargo of lipid droplets sugg

201 lls that infiltrated both the cornea and the TG.

202 independent of fasting TG, we calculated the TG response at 150 minutes by the residuals of a nonline

203 L-C PRS and 1.31 (95% CI, 1.19-1.43) for the TG PRS.

204 lation in Scn10a-positive nociceptors in the TG and DRG of male and female mice.

205 fficiency of gB-CD8s and their levels in the TG at early times.

206 that behavioral responses are greater in the TG region and this effect is completely reversible with

207 tivation was detected more frequently in the TG than in the brain stem and, in all but one case, the

208 amount of virus recovered was greater in the TG than that detected in the brain stem.

209 nt wild-type flank infection infiltrated the TG and were retained long term, suggesting that latent e

210 GWAS of the TG response identified a variant near LIPC as a main con

211 From GWAS of the TG response, we identified rs7350789-A (allele frequency

212 .34x10(-3)), mainly smaller increases of the TG-component in almost all HDL (high-density lipoprotein

213 o mCherry demonstrate the versatility of the TG-FRET nanoprobes and the possibility of in vivo biocon

214 TG is stable and determines the width of the TG-PL overlap, whereas that caused by SURF-TG fluctuates

215 s to both TG, but with delay in reaching the TG contralateral to the side of lip infection.

216 lower than in control dams but also that the TG peak occurred earlier during gestation.

217 to 1.55 (95% CI, 1.48-1.61) mmol/L with the TG PRS.

218 ul in identifying activated cells within the TG during HSV-1 infection.IMPORTANCE Without an effectiv

219 In contrast, the TGs that intercalate just into the PL tail region (CORE-

220 Isolated LDs contained nearly all of the TGs and cholesteryl esters present in luteal tissue.

221 gnificantly increased genome levels in their TG but many fewer LAT-positive cells than wild-type mice

222 These TG mice exhibited a physiologic-like cardiac hypertrophy

223 Thioguanine (TG) is a thiopurine which has been used for patients wit

224 Simultaneous thrombin (TG) and plasmin (PG) generation is useful to assessing c

225 Thyroglobulin (TG) is the protein precursor of thyroid hormones, which

226 mall-molecule inhibitor of GSK3, tideglusib (TG), not only normalizes the GSK3beta-CUGBP1 pathway but

227 he ATP noncompetitive inhibitor, Tideglusib (TG), can equally enhance reparative dentine formation to

228 We observe that PRAP1 directly binds to TG and facilitates MTTP-mediated lipid transfer.

229 ime-gated Forster resonance energy transfer (TG-FRET) between terbium donors and dye acceptors into H

230 izes PRAP1-bound TG as a cargo and transfers TG along with PRAP1 to lipid acceptors.

231 ype (WT), Fgf15(-/-) , and Fgf15 transgenic (TG) mice with BA levels modulated by feeding cholestyram

232 We generated humanized transgenic (TG) mice containing all the introns, exons, and 5'- and

233 ) with/without addition of transglutaminase (TG).

234 also the main components of triacyglycerols (TGs), which enable energy storage in lipid droplets.

235 of neutral lipids, such as triacylglycerol (TG) and sterol esters, surrounded by a phospholipid (PL)

236 including LDL cholesterol, triacylglycerol (TG), fasting glucose (FG), glycated hemoglobin (HbA1c),

237 gates the ambient aging of triacylglycerols (TGs) and other lipids in latent fingerprint residue util

238 patomegaly due to glycogen and triglyceride (TG) accumulation in the liver.

239 ensity lipoprotein (LDL-C) and triglyceride (TG) levels form the cornerstone approach of cardiovascul

240 l lipid supply, maternal blood triglyceride (TG) concentrations are robustly elevated during pregnanc

241 oxygen species (ROS), cellular triglyceride (TG), and glucose and B-hydroxybutyrate (BHB) export were

242 Serum lipid levels, especially triglyceride (TG) levels, were higher in conventional mice exposed to

243 ng MR, we inferred that higher triglyceride (TG) and LDL cholesterol (LDL-C) levels caused elevated A

244 on of gene modules involved in triglyceride (TG) synthesis and adipogenesis decreased, and this was a

245 tor of ATGL, the rate-limiting triglyceride (TG) lipase in many cell types.

246 ibited higher in vivo rates of triglyceride (TG) turnover (representing both lipolysis and synthesis

247 the assembly and secretion of triglyceride (TG)-rich, apoB-containing lipoproteins.

248 effect of that accumulation on triglyceride (TG) hydrolysis by adipose triglyceride lipase (ATGL), th

249 rotein) cholesterol (LDL-C) or triglyceride (TG)-increasing variants associates with increased CAD ri

250 the CNS by favorably producing triglyceride (TG) storage lipids rather than phospholipid (PL) membran

251 The increase in serum triglyceride (TG) concentrations in response to a meal is considered a

252 Serum triglyceride (TG), total cholesterol (TC), high-density lipoprotein ch

253 es, CTRP2 treatment suppressed triglyceride (TG) hydrolysis, and its deficiency enhanced agonist-indu

254 ad significantly elevated VLDL-triglyceride (TG) and strikingly impaired lipid clearance from circula

255 lin resistance, manifests when triglyceride (TG) input in the liver is greater than TG output, result

256 otein cholesterol (HDL-c) and triglycerides (TG) from two independent GWAS datasets.

257 otein cholesterol (LDL-C) and triglycerides (TG) levels.

258 tein cholesterol (HDL-C), and triglycerides (TG) were evaluated preconception and throughout pregnanc

259 included ceramides (Cer) and triglycerides (TG).

260 rotein cholesterol (HDLc) and triglycerides (TG)].

261 M1 PUFA containing triglycerides (TG) and phospholipids were correlated with CB LysoPC and

262 tic lipid scores specific for triglycerides (TG), high density lipoprotein cholesterol (HDL), low den

263 ficant age-related changes in triglycerides (TG), diglycerides (DG), phosphatidylcholine, phosphatidy

264 with total cholesterol (TC), triglycerides (TG), and lipoprotein concentrations is unknown.

265 L, 2,756 subjects (7.0%); and triglycerides (TGs) >150 mg/dL, 2,881 subjects (7.3%).

266 and insulin, cholesterol, and triglycerides (TGs), and further measured blood pressure.

267 image neutral lipids, such as triglycerides (TGs).

268 or nonmelanizing conditions; triglycerides (TGs), sterol esters (SEs), and polyisoprenoids (PPs) wer

269 e cell membranes, hydrolyzing triglycerides (TGs), or inducing apoptosis.

270 Luteal tissue was enriched in triglycerides (TGs) compared to other tissues, except for adipose tissu

271 eting ATGL (shATGL) increased triglycerides (TGs) and the number and size of LDs, indicating that ATG

272 rder in which accumulation of triglycerides (TGs) in the liver can lead to inflammation, fibrosis, an

273 onsible for the catabolism of triglycerides (TGs) that is complemented by lipophagy as the autophagic

274 studies; 86,854 people], and triglycerides [TG; 84 studies; 121,009 people]) levels and prevalences

275 y lipoproteins [LDL and HDL], triglycerides [TGs], and glycated haemoglobin [HbA1c]).

276 low-density lipoprotein, LDL; triglycerides, TGs) with risk for BC using Mendelian randomization (MR)

277 ime that calibration curves for the ABC type TGs are reported.

278 kout), CaMKIIdeltaC transgenic in wild-type (TG), or KO background, and wild-type mice exposed to TAC

279 from latently infected, but not uninfected, TG contained significantly more GR-positive neurons foll

280 Unsaturated TGs were found to undergo ambient ozonolysis resulting i

281 ds related to the degradation of unsaturated TGs due to ambient ozonolysis emerged with time and were

282 Tracking the degradation of unsaturated TGs over time proved to be relatively reproducible in mu

283 vior on heating and cooling processes, using TG/DTG/DTA, TG-MS, DSC, hot stage microscopy and DRX ana

284 ysis and very low density lipoprotein (VLDL)-TG secretion assays revealed that hepatic ChREBP knockdo

285 e of "old fat." Interestingly, enhanced VLDL-TG secretion in shSCR-treated L-G6pc(-/-) mice associate

286 lt of reduced glycolysis and suppressed VLDL-TG secretion.

287 y low-density lipoprotein triglyceride (VLDL-TG) palmitate.

288 CG + GG, p = 0.0003; and rs12255372- TT vs. TG + GG, p = 0.003.

289 sting despite adjusting for baseline weight, TG, HDL-C and HbA1c (p = 0.002).

290 sported by both OATP1B1 and OATP1B3, whereas TG and AG were preferentially (>68%) transported by OATP

291 d (c) increased blood pressure compared with TG mice containing WT intron 2.

292 AP1's ability to form a ternary complex with TG and MTTP, as well as impairs its ability to facilitat

293 and that the correction of this pathway with TG increases postnatal survival and improves growth and

294 4 is increased in podocytes of patients with TG but not in those without TG despite other forms of re

295 e-damaging effects of IgG from patients with TG.

296 t IgG from kidney transplant recipients with TG, but not from those without TG, cause a reduction in

297 crolimus) and were subsequently treated with TG as rescue monotherapy between 2003 and 2019 at three

298 Tyrosine proximity within TG is thought to enable the coupling reaction but hormon

299 of patients with TG but not in those without TG despite other forms of renal dysfunction.

300 cipients with TG, but not from those without TG, cause a reduction in the expression of nephrin, sign