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1                                              TMR and TMR genetic risk scores are significantly associ
2                                              TMR causes decreased myocardial HED uptake in most patie
3                                              TMR did not significantly affect resting or stress myoca
4                                              TMR is a nerve transfer procedure developed for prosthes
5                                              TMR is an alternative surgical technique for the treatme
6                                              TMR provide key ecosystem services to hundreds of millio
7                                              TMR studies hypothesize that this method taps into norma
8                                              TMR was used to measure ventricular repolarization dynam
9                                              TMR with CO(2) laser as sole therapy for severe disablin
10                                              TMR(rec) was also associated with all-cause mortality (h
11                                              TMR-D readily diffused transsclerally and dispersed thro
12                                              TMR-D was also observed in the retinal and optic nerve v
13 MR (Galbeta1-4GlcNAcbeta(CH2)8CONH(CH2)2NHCO-TMR)), a fluorescently labeled model substrate for glyca
14 nalogs LacdiNAc-TMR (GalNAcbeta1-4GlcNAcbeta-TMR), LacNAc-TMR, and LacNAcbeta1-6LacNAcbeta-TMR.
15  the beta1-6 bond in LacNAcbeta1-6LacNAcbeta-TMR, an activity that is of potential importance in rela
16 MR), LacNAc-TMR, and LacNAcbeta1-6LacNAcbeta-TMR.
17 redefines mechanisms of ERK2 regulation by 7-TMR signaling in Dictyostelium and establishes new impli
18  controlling seven-transmembrane receptor (7-TMR) ERK2 activation/deactivation function independently
19              Seven-transmembrane receptor (7-TMR)-G protein networks are molecular sensors of extrace
20  nanoparticles, estimated to contain 700-900 TMRs per ca. 23 nm particle, were surface modified with
21 ible MTJs are fully functional, exhibiting a TMR ratio as high as 190% under bending radii as small a
22  acids adjacent to acylation sites or with a TMR cysteine shifted to a cytoplasmic location to identi
23 nctionalized azidobutyl-Q7 and NHS-activated TMR.
24                                 In addition, TMR patients used the same control system to operate adv
25 hedrine (HED) was performed before and after TMR in 8 patients with class IV angina ineligible for CA
26                             Six months after TMR, there was a significant increase over baseline in r
27 ciation with regional ischemia is seen after TMR with a holmium:YAG laser.
28 e 6 h post-CO2 TMR, p = 0.03) was seen after TMR with both lasers.
29  prospectively collected up to 7 years after TMR.
30  of the patients had no angina 5 years after TMR.
31  postamputation pain in major limb amputees, TMR improved PLP and trended toward improved residual li
32 (16.9+/-6.5 U to 25.9+/-14.4 U, P=0.002) and TMR (0.51+/-0.14 to 0.79+/-0.32 mm Hg x mL(-1) x min(-1)
33  well as the % change in IMR (147+/-66%) and TMR (159+/-105%, P=NS versus IMR % change), increased si
34    Overall mortality rates for TMR-alone and TMR + CABG were 6.4% and 4.2%, respectively.
35  stress test had a cardiovascular event, and TMR(rec) was significantly associated with cardiovascula
36 ome-wide association studies for TMR(ex) and TMR(rec) were performed, and genetic risk scores were de
37 ied 12 genetic loci in total for TMR(ex) and TMR(rec), of which 9 are associated with another ECG mar
38  logistic regression model combining GTV and TMR(max), were assessed and compared with a previous tra
39 poxic volume, and the combination of GTV and TMR(max), were not able to stratify patient groups accor
40 significant correlation between mean IMR and TMR values, as well as between the % change in IMR and %
41    Such high values of spin polarization and TMR in readily manufactureable and highly thermally stab
42                                      TMR and TMR genetic risk scores are significantly associated wit
43                          In the aortic arch, TMR-D labeled large and small vagal afferent axons (axon
44   All other investigated parameters, such as TMR(max), GTV, relative hypoxic volume, and the combinat
45 eptor dye pairs (TMR-ATTO647N, Cy3-ATTO647N, TMR-Cy5, and Cy3-Cy5), we find that this phenomenon depe
46 vary (CHO) cells were pulse-labeled with BAC-TMR-dextran by fluid-phase endocytosis.
47                                      On-bead TMR-labeled alpha-synuclein and aggregated Cy5-labeled a
48 ing perfusion defects in all patients before TMR (34.6+/-27.3% for HED versus 9.4+/-10.8% for NH3, P=
49 t upon photoconversion, the distance between TMR (donor) bound to a cysteine in the C-terminal domain
50       An intricate connection exists between TMR ratio, RA value and the bandgap and crystal structur
51          We microinjected micelles of Bodipy TMR-PIP(2) into cells, and we measured D on the inner le
52 led with BODIPY-tetramethylrhodamine (BODIPY(TMR)).
53 -tetramethylrhodamine-(+/-)CGP 12177 (BODIPY-TMR-CGP)] at the human beta1-adrenoceptor expressed in C
54 scence resonance energy transfer from Bodipy-TMR-PIP2 to Texas Red MARCKS(151-175) adsorbed to large
55 olipid substrates, GM3-BODIPY-FL, GM1-BODIPY-TMR, and lactosylceramide-BODIPY-650/665.
56         The dissociation rate of 3 nM BODIPY-TMR-CGP was 0.09 +/- 0.01 min(-1) in the absence of comp
57        Second, we detect quenching of Bodipy-TMR-PIP2 in large unilamellar vesicles when unlabeled MA
58 e-resolved fluorescence spectroscopy, Bodipy-TMR-X-labeled 4R1, 4BB, and 4alphaE quenched fluorescenc
59                  These effects on the BODIPY-TMR-CGP dissociation rate were markedly enhanced in beta
60  cleavage lies relatively far from the bound TMR ( approximately 15 A).
61                             Beads with bound TMR-labeled alpha-synuclein are then incubated with a re
62 hanges in this cleavage, are not affected by TMR-labeling of actin.
63                                          C2A-TMR assembled into oligomers via a novel N-terminal olig
64 es the membrane-spanning and C2A domain (C2A-TMR) into proteoliposomes.
65 uced disturbances, including climate change, TMR can be used as natural model systems to examine the
66 = 0.05; 206+/-36.7% of baseline 6 h post-CO2 TMR, p = 0.03) was seen after TMR with both lasers.
67 y underway to assess the results of combined TMR and CABG [4].
68               Filaments of cofilin-decorated TMR-actin and unlabeled actin are indistinguishable, as
69 374) with tethramethylrhodamine derivatives (TMR-actin) has been widely used for direct observation o
70  loads, e.g., tetramethyl rhodamine-dextran (TMR-D) and CdSe/ZnS quantum dots (QDs), are loaded in th
71  of the tracer tetramethylrhodamine dextran (TMR-D) into the nodose ganglion to label vagal aortic af
72 f 0.25% 70-kDa tetramethylrhodamine-dextran (TMR-D).
73 oads (tetramethylrhodamine-modified dextran, TMR-D; microperoxidase-11, MP-11; CdSe/ZnS quantum dots;
74                             TAC-Lime-dextran-TMR fluorescence was K(+)-selective, increasing >4-fold
75 tran to measure ASL [K(+)], TAC-Lime-dextran-TMR, consisting of a green-fluorescing triazacryptand K(
76                                    Efficient TMR trafficking is critical for regulation of several pr
77                              Silica-embedded TMR retains very high quantum yield, is resistant to que
78 ynamics, exposed in this cohort by exercise (TMR during exercise, TMR(ex)) and recovery from exercise
79 ercise, TMR(ex)) and recovery from exercise (TMR during recovery, TMR(rec)).
80 his cohort by exercise (TMR during exercise, TMR(ex)) and recovery from exercise (TMR during recovery
81 tunnel barriers and CoFe electrodes, exhibit TMR values of up to approximately 220% at room temperatu
82 luding 3 crossover patients, results favored TMR over standard treatment.
83 e increased severing of copolymer filaments, TMR-actin accelerates the polymerization of unlabeled ac
84 cal improvement in angina pectoris following TMR.
85                                          For TMR patients, the mean motion selection and motion compl
86 al (aCI) -0.1 to 6.9, adjusted P = 0.06) for TMR and standard treatment, respectively.
87 isk-adjusted mortality was also compared for TMR + CABG relative to CABG only in patients not amenabl
88 e also identified important risk factors for TMR and compared outcomes of TMR combined with coronary
89 tion in residual limb pain was favorable for TMR (P = 0.10).
90                  Overall mortality rates for TMR-alone and TMR + CABG were 6.4% and 4.2%, respectivel
91          Genome-wide association studies for TMR(ex) and TMR(rec) were performed, and genetic risk sc
92   We identified 12 genetic loci in total for TMR(ex) and TMR(rec), of which 9 are associated with ano
93 dels are consistent with the requirement for TMRs in viral fusion proteins.
94  protein conformation and the distances from TMR to the carotenoid throughout the photocycle.
95  binding to DNA, the total fluorescence from TMR-Tc3 increases by three- to fourfold.
96       Moreover, we also show that functional TMR-cytochrome c undergoes a response of identical magni
97  by the metabolic products produced from GM1-TMR and the other monitored the metabolic products produ
98 thylrhodamine-labeled glycosphingolipid (GM1-TMR) was used as a substrate.
99 olipid tetramethylrhodamine labeled GM1 (GM1-TMR) produced by single cells isolated from the rat cere
100 n with tetramethylrhodamine-labeled GM1 (GM1-TMR).
101 ntrol in eight transhumeral amputees who had TMR in a balanced randomized cross-over study.
102 l virus-host model, we show that herpesvirus TMRs facilitate viral genome integration into host telom
103                   Consistent with these high TMR values, superconducting tunnelling spectroscopy expe
104                                  Much higher TMR values have been theoretically predicted for perfect
105                                   Homologous TMR motifs are often found as multiple repeats in quite
106 F-actin that substitute for disrupted (as in TMR-actin and mutant actin) or weakened (as in ADP-actin
107  between the % change in IMR and % change in TMR.
108 tCS) during sleep can also be used to induce TMR, resulting in the facilitation of high-level cogniti
109 rescently labeled substrate analogs LacdiNAc-TMR (GalNAcbeta1-4GlcNAcbeta-TMR), LacNAc-TMR, and LacNA
110 1 and lipid-anchored synaptobrevin-2 lacking TMRs efficiently promoted spontaneous and Ca(2+)-trigger
111  al. show that lipid-anchored SNAREs lacking TMRs can support neurotransmitter release, suggesting th
112 ta1-4GlcNAcbeta-tetramethylrhodamine (LacNAc-TMR (Galbeta1-4GlcNAcbeta(CH2)8CONH(CH2)2NHCO-TMR)), a f
113 Ac-TMR (GalNAcbeta1-4GlcNAcbeta-TMR), LacNAc-TMR, and LacNAcbeta1-6LacNAcbeta-TMR.
114  whereas they were less active toward LacNAc-TMR conjugates.
115 = 12) or carbon dioxide (CO2) (n = 12) laser TMR.
116  These changes were not seen after CO2 laser TMR.
117 stimulated nostril) was improved after local TMR during sleep.
118                              Moreover, local TMR improved phase-amplitude coupling (PAC) between slow
119 lateral stimulus processing to perform local TMR in one brain hemisphere.
120 tion switching and tunnel magnetoresistance (TMR) in perpendicularly magnetized CoFeB/MgO/CoFeB MTJs
121  interplay between tunnel magnetoresistance (TMR) ratio and the product of resistance and junction ar
122 nal bias-dependent tunnel magnetoresistance (TMR) response is observed in Sm0.75 Sr0.25 MnO3 -based n
123 om-temperature tunnelling magnetoresistance (TMR) of technologically relevant MTJs.
124 teine with tetramethylrhodamine-5-maleimide (TMR), a fluorophore with specific brightnesses that we a
125 ocystis with tetramethylrhodamine-maleimide (TMR) and obtained a photoactive OCP-TMR complex, the flu
126  and by transverse digital microradiography (TMR) for mineral content.
127 eased markedly, driven predominately by more TMR + CABG cases.
128                    Immobilized ribosome.mRNA.TMR-Met-tRNAMetf complexes form peptide bonds with purom
129 ual assumption of complete quenching for NBD-TMR pairs in the dimer could not be quantitatively recon
130 leimide (TMR) and obtained a photoactive OCP-TMR complex, the fluorescence of which was highly sensit
131                                      The OCP-TMR complex was sensitive to the light intensity, temper
132 nal insights into the mechanism of action of TMR, which remains the Achilles' heel of this procedure.
133                             The advantage of TMR-actin is that it does not lock actin in filaments (a
134  MgZnO barriers allow effective balancing of TMR ratio and RA value.
135                             The challenge of TMR is related to improvement in perioperative outcomes,
136     The destabilizing and severing effect of TMR-actin is countered by filament stabilizing factors,
137                               The effects of TMR may be enhanced when paired with pattern recognition
138 lts show an absence of allosteric effects of TMR on subdomain 2, while confirming ATP/ADP-dependent c
139 s point to an analogy between the effects of TMR-actin and severing proteins on F-actin, and imply th
140                    The long-term efficacy of TMR persists for >/=5 years.
141                          The fluorescence of TMR covalently linked to Y290C, T287C, and H83C decrease
142 e specificity constant within the library of TMR analogues.
143              Single molecule measurements of TMR-Tc3 bound to DNA shows that this complex also fluctu
144  ring as a function of the average number of TMR molecules per ring.
145  us to compute the number and orientation of TMR dimers per ring as a function of the average number
146 isk factors for TMR and compared outcomes of TMR combined with coronary artery bypass graft surgery (
147 ponses depend strongly on the molar ratio of TMR used to label cytochrome c.
148          In the recently solved structure of TMR-modified ADP-G-actin, the nucleotide cleft is in a c
149                           Further studies of TMR + CABG are needed given its growing use and unclear
150                          Clinical studies of TMR have noted a significant incidence of cardiac compli
151 ies, the FDA granted approval for the use of TMR as a sole therapy.
152                                   The use of TMR, and in particular, TMR + CABG, is expanding in comm
153  for cardiovascular risk and the validity of TMR as a cardiovascular risk predictor.
154 luate the cardiovascular prognostic value of TMR in a UK middle-aged population and identify any gene
155 ration is poorly understood, the presence of TMRs in a number of herpesviruses suggests it is their d
156              Our results and the presence of TMRs in many herpesviruses suggest that integration medi
157 , there are limited data on the outcomes of (TMR + CABG).
158                   HCF145 represents the only TMR protein found in vascular plants.
159 l mixed ration (TMR) (Control, C; n = 40) or TMR + alfalfa green feed (30% of diet) (Treated, T; n =
160  pain were assigned to standard treatment or TMR.
161 rogrammed ribosomes with TMR-Met-tRNAMetf or TMR-Met-Phe-tRNAPhe are immobilized on mica and observed
162                            While the overall TMR is slower than predicted based on concurrent regiona
163 ing four different donor-acceptor dye pairs (TMR-ATTO647N, Cy3-ATTO647N, TMR-Cy5, and Cy3-Cy5), we fi
164           The use of TMR, and in particular, TMR + CABG, is expanding in community practice.
165 2001, the number of STS hospitals performing TMR and total procedural counts increased markedly, driv
166 er equimolar mismatch DNA for each polyamide-TMR conjugate.
167 ned from 2.4+/-1.6 pre-TMR to 1.7+/-2.0 post-TMR (P=0.01).
168 at 1 h, p = 0.02; 18.4% decrease at 6 h post-TMR, p = 0.01).
169 01)Tl imaging at any temporal end point post-TMR.
170 an anginal class improved from 3.5+/-0.5 pre-TMR to 2.8+/-0.7 and 2.5+/-0.7 at 3 and 6 months, respec
171  unstable angina declined from 2.4+/-1.6 pre-TMR to 1.7+/-2.0 post-TMR (P=0.01).
172                            Their average pre-TMR angina class was 3.7+/-0.4.
173  with nitrilotriacetic acid (NTA), producing TMR-SiO2-NTA-Ni2+.
174 red with those from six published randomized TMR trials.
175 orresponds to a mean thermal migration rate (TMR) of 0.0065 degrees C yr(-1) (95% CI = 0.0005-0.0132
176 f thermophilization [thermal migration rate (TMR), degrees C y(-1)] across all censuses were 0.011 de
177 8)F-FMISO and maximum tumor-to-muscle ratio (TMR(max)) at 4 h after injection, gross tumor volume (GT
178     Buffaloes received a total mixed ration (TMR) (Control, C; n = 40) or TMR + alfalfa green feed (3
179                Targeted memory reactivation (TMR) allows the manipulation of sleep-dependent consolid
180  method called targeted memory reactivation (TMR) for selectively strengthening memories during sleep
181 ved to rely on targeted memory reactivation (TMR) induced by the sensory stimulation.
182 ermine whether targeted memory reactivation (TMR) of specific memory traces during slow-wave sleep pr
183 e promoted via targeted memory reactivation (TMR) that re-presents training cues or context during sl
184 his technique, targeted memory reactivation (TMR), selectively enhances memory consolidation.
185  performance in a control group who received TMR before sleep.
186       We identified 3,717 patients receiving TMR at 173 U.S. hospitals participating in the Society o
187 er internalization, transmembrane receptors (TMRs) are typically recycled back to the cell surface or
188 recovery from exercise (TMR during recovery, TMR(rec)).
189 rane helices forming a transmembrane region (TMR), a large periplasmic (PP) domain, and a cytoplasmic
190 oned at the end of the transmembrane region (TMR).
191 otif, and a C-terminal transmembrane region (TMR).
192 hat each contains six transmembrane regions (TMRs) localized near the carboxyl terminus.
193 fusion by pulling the transmembrane regions (TMRs) of SNARE proteins together, thus allowing their TM
194 been proposed for the transmembrane regions (TMRs) of SNARE proteins, including formation of channel-
195 repeats, four central transmembrane regions (TMRs), and a cytoplasmic tail.
196               Targeted Muscle Reinnervation (TMR) is a nerve transfer procedure that creates addition
197 hnique called targeted muscle reinnervation (TMR) transfers residual arm nerves to alternative muscle
198 ignated the transcript binding motif repeat (TMR) domains, confer sequence-specific RNA binding capab
199 her herpesviruses, harbor telomeric repeats (TMRs) identical to host telomeres at either end of their
200 her herpesviruses harbors telomeric repeats (TMRs) that are identical to host telomere sequences (TTA
201 hy that when compared to previously reported TMR analogues, subtle modification of the TMR scaffold c
202  we examined whether synaptic SNAREs require TMRs for catalysis of synaptic vesicle fusion, which was
203 istance (TER) and tunnel magneto resistance (TMR) states in the junction.
204 elate with true microcirculatory resistance (TMR), defined as the distal left anterior descending (LA
205           The T-wave morphology restitution (TMR), an ECG marker quantifying ventricular repolarizati
206           Transmyocardial revascularisation (TMR) is an operative treatment for refractory angina pec
207 ter transmyocardial laser revascularization (TMR) and to identify predictors of these adverse clinica
208 ing transmyocardial laser revascularization (TMR) as the sole operative therapy for patients with sev
209 ith transmyocardial laser revascularization (TMR) at our institution.
210 ith transmyocardial laser revascularization (TMR) for induction of therapeutic angiogenesis and arter
211 ugh transmyocardial laser revascularization (TMR) has provided symptomatic relief of angina over the
212  of transmyocardial laser revascularization (TMR) on regional left ventricular systolic and diastolic
213 ter transmyocardial laser revascularization (TMR).
214 tcomes of transmyocardial revascularization (TMR) in community practice.
215 ents receiving incomplete revascularization, TMR + CABG was not associated with decreased mortality r
216 assembly incompetent tethramethyl rhodamine (TMR)-actin and T203C/C374S yeast mutant actin.
217 irpin polyamides with tetramethyl rhodamine (TMR) attached to an internal pyrrole ring were synthesiz
218 cent signal, only the tetramethyl rhodamine (TMR) HALO ligand provides sufficient signal for detectio
219 e in target tissue, a tetramethyl rhodamine (TMR)-labeled ribozyme was administered as a single SC or
220                     Tropical montane rivers (TMR) are born in tropical mountains, descend through mon
221 not be accurately determined, yet these same TMR analogues promoted ATP occlusion at relatively low c
222              Herein, we characterize several TMR analogues that elicit modest turnover ( k cat <or= 1
223                          The fifth and sixth TMRs contain numerous beta-branched amino acids that may
224               However, the role of the SNARE TMRs in synaptic vesicle fusion has not yet been tested
225 d with coronary artery bypass graft surgery (TMR + CABG) versus bypass alone in patients receiving in
226                        Tetramethylrhodamine (TMR)-doped silica nanoparticles, estimated to contain 70
227 nsensitive chromophore tetramethylrhodamine (TMR).
228    The fluorescent dye tetramethylrhodamine (TMR) was conjugated to a synthetic peptide containing th
229 to the fluorescent dye tetramethylrhodamine (TMR), and the characterization of its optical and guest-
230 arger fluorescent dye, tetramethylrhodamine (TMR).
231  N-termini with either tetramethylrhodamine (TMR) or 7-nitrobenz-2-oxa-1,3-diazole (NBD) also indicat
232 s with the fluorophore tetramethylrhodamine (TMR).
233 cent rhodamines [e.g., tetramethylrhodamine (TMR)-thiols].
234 olecular weight marker tetramethylrhodamine (TMR)-dextran and its blockage by the Na(+)/H(+) exchange
235 oncovalently "stacked" tetramethylrhodamine (TMR) dimers have been used to both report and perturb th
236 ling with a green dye (tetramethylrhodamine, TMR) and attachment to microbeads.
237 a synthetic library of tetramethylrosamine ( TMR) analogues, we observed significant variation in ATP
238                         Tetramethylrosamine (TMR) displays a relatively high affinity for Pgp when co
239 of the aptamer bound to tetramethylrosamine (TMR), a high-affinity MG analog.
240 arge corpus of studies now demonstrates that TMR is both reliable and effective.
241 ed by several new studies demonstrating that TMR upregulates the reactivation of cued memories, and t
242              Although it is established that TMR-actin alone is polymerization incompetent, the impac
243 ow-oscillation upstate, we hypothesized that TMR stimulation effects would depend on the phase of the
244 severing proteins on F-actin, and imply that TMR-actin may be inappropriate for investigations of act
245                  In this study, we show that TMR-actin perturbs the filaments structure when copolyme
246 binding site in the apical domain, such that TMR molecules on adjacent subunits are able to form dime
247                  These findings suggest that TMR during sleep can alter memory representations and pr
248                                          The TMR dimers also serve as a surrogate for substrate prote
249                                          The TMR dimers thus serve as quantitative reporter of the al
250                                          The TMR modification inhibits nucleotide exchange, but has n
251                                          The TMR patients were able to repeatedly perform 10 differen
252 his review, background information about the TMR procedure will be discussed along with an analysis o
253 ise distance between the SNARE motif and the TMR was critical for fusion.
254 ssential for fusion, the SNARE motif and the TMR, and these domains have to be closely coupled to fun
255 e the folding of loop nucleotides around the TMR.
256 by substrate protein is also mimicked by the TMR dimers.
257                                 Finally, the TMR analogues facilitated uptake of calcein-AM into CR1R
258 inases examined hydrolyzed LacdiNAc from the TMR aglycone to various degrees, whereas they were less
259 t CCSA score was II or lower in 47.8% in the TMR group compared with 14.3% in the medication-only gro
260 res for PLP was significantly greater in the TMR group compared with standard treatment [mean (aCI) =
261                                       In the TMR group, the entire LCx region was treated with transm
262 l vascular density was increased only in the TMR-treated regions six months postoperatively.
263                                Moreover, the TMR analogues competitively inhibited VER-dependent ATPa
264  within the first transmembrane helix of the TMR and deletions within the PP domain both resulted in
265 netics were identical in the presence of the TMR derivatives, VER, or in the absence of drug, suggest
266         The free energy of unstacking of the TMR dimers was estimated at 2.6 +/- 1.0 kJ/mol dimer.
267 ng sites, with concomitant unstacking of the TMR dimers.
268 re raised regarding the possible role of the TMR label on Cys(374) in determining these aspects of G-
269 ed TMR analogues, subtle modification of the TMR scaffold can confer large differences in ATP turnove
270            Individuals in the top 20% of the TMR(rec) genetic risk score were significantly more like
271                        Our reanalysis of the TMR-modified actin structures suggests that the nucleoti
272                    In contrast, shifting the TMR cysteine to a cytoplasmic location virtually elimina
273       Evidence is presented to show that the TMR-Tc3 conjugate exists in at least two conformational
274 however, for larger barrier thicknesses, the TMR-bias dependence is more complex and reverses sign at
275 ion of a library of compounds based upon the TMR scaffold and use this set to assess the determinants
276 most stable conformation is one in which the TMR fluorescence is quenched.
277 tom magnetic layers is observed in which the TMR ratio of the MTJs is almost unmeasurable.
278  to the membrane and/or sequences within the TMR and PP domain.
279                      The conservation of the TMRs in all SCAMPs with the variable presence of N-termi
280 se-related motifs support association of the TMRs to the psaA RNA, presumably pointing to a regulator
281 SNARE proteins together, thus allowing their TMRs to form a fusion pore.
282 nction in membrane traffic mediated by their TMRs.
283                                        Thus, TMR in human sleep transcends global action by selective
284                            ERalpha, bound to TMR-SiO2-NTA-Ni++ beads in a site-specific manner, exhib
285                          This dual-mode type TMR-SiO2-NTA-Ni2+ system represents a powerful combinati
286 retained excellent performance and unaltered TMR ratio after over 1000 bending cycles.
287                       Animals then underwent TMR with a holmium:yttrium-aluminum-garnet (holmium:YAG)
288 on or transhumeral amputations who underwent TMR surgery between February 2002 and October 2006 and 5
289 s suggest that integration mediated by viral TMRs is a conserved mechanism, which ensures faithful vi
290           At least in the case of MDV, viral TMRs facilitate integration into host telomeres.
291                              We observe weak TMR fluorescence in the absence of DNA.
292 efficient of extremely high molecular weight TMR-Dextran (MW 2 000 000), which rises linearly with th
293                                      Whether TMR acts locally or globally on cortical sleep oscillati
294                   Mice treated SC or IV with TMR-labeled ribozyme had positive fluorescence in the li
295 A GroEL mutant (K242C) has been labeled with TMR, close to the peptide-binding site in the apical dom
296 ify clinical risk factors for mortality with TMR.
297  complexes of mRNA-programmed ribosomes with TMR-Met-tRNAMetf or TMR-Met-Phe-tRNAPhe are immobilized
298 0 cells were used that had been treated with TMR-G(M1).
299 7.4+/-44.2% of baseline 6 h post-holmium:YAG TMR, p = 0.05; 206+/-36.7% of baseline 6 h post-CO2 TMR,
300 .4% of baseline, p = 0.02) after holmium:YAG TMR.

 
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