ライフサイエンスコーパス: TMV

1 TMV additionally emerged as a prototypic model to invest

2 TMV also exemplifies how a model system furthers novel,

3 TMV CP affects the display of several specific virus and

4 TMV first accumulated in abaxial or external phloem-asso

5 TMV infection of transgenic BY-CP(T42W) protoplasts resu

6 TMV movement protein (MP) and replicase colocalized with

7 TMV MP and PAPK1 are colocalized within cross-walls in a

8 TMV particles collected from the filter could be used fo

9 TMV presentation improved titer and avidity of repeat-sp

10 TMV RNAs initially appeared as single-stranded threads o

11 TMV that carried epitope 31D from the RV nucleoprotein d

12 TMV VLPs are high surface area macromolecules with nanor

13 TMV was simulated with 1.5 diopters (D) of anisometropia

14 TMV-CP(T42W):GFP failed to produce visible infection sit

15 TMV-induced fluorescent lesions on Rca- or AtpC-silenced

16 TMV-p50 effector disrupts the N-UBR7 interaction and rel

17 ys-118 (TMIII), Cys-251 (TMVI), and Cys-202 (TMV)).

18 4.52) (TMIV), F150(4.56) (TMIV), F184(5.40) (TMV), and Y188(5.44) (TMV) was observed away from the li

19 6) (TMIV), F184(5.40) (TMV), and Y188(5.44) (TMV) was observed away from the ligand-binding pocket, b

20 A TMV helicase polypeptide also was capable of unwinding d

21 rotoplasts and in planta when supported by a TMV mutant that expressed the 183-kDa protein but not th

22 thogenicity, the replication and spread of a TMV mutant with a reduced ability to interact with speci

23 is interaction in the display of symptoms, a TMV mutant defective in the PAP1 interaction was identif

24 s of the LRR, is more prevalent 4-8 hr after TMV infection.

25 amatically suppressed in sun1-1 plants after TMV treatment compared to wild-type plants.

26 coded proteins in a defense response against TMV.

27 he induction of resistance responses against TMV.

28 the use of not only AiV-1 and PMMoV but also TMV as indicators of reductions in viral levels can be a

29 partners for Tyr(187) in TMIV (Phe(171)) and TMV (Trp(194)).

30 virus (MHV) and giving rise to TMV-5B19 and TMV-5B19L, respectively.

31 Closer examination of the CMV and TMV populations revealed biases for particular types of

32 for a few final effluents for crAssphage and TMV.

33 type CP, the resulting viruses TMV-RB19E and TMV-4D:31D infected N. tabacum Xanthi-nn plants and BY2

34 is the unproven assumption that the TVCV and TMV life cycles are identical.

35 As TMV poorly infects Arabidopsis thaliana, Turnip vein cle

36 virus (TVCV), a virus from the same genus as TMV, did not require intact microfilaments for normal sp

37 Because TMV has a wide host range, expressing TMV-TMOF in plants

38 The interaction between TMV MP and calreticulin was confirmed in vivo and in vit

39 tion to isolate a tobacco protein that binds TMV MP and identified it as calreticulin.

40 t 200 fold 4 d after infection (dai) by both TMV and TRV.

41 s report that SIPK and WIPK are activated by TMV in a gene-for-gene-dependent manner, we conclude tha

42 Like the HR elicited by TMV infection, transgenic expression of p50 induces a te

43 lowest among the tested viruses, followed by TMV (2.0 +/- 0.3 log(10)) and PMMoV (2.0 +/- 0.4 log(10)

44 Interestingly, induction of both genes by TMV infection was still observed in resistant tobacco pl

45 ic plants confers resistance to infection by TMV and related tobamoviruses.

46 in plants confers resistance to infection by TMV and related tobamoviruses.

47 On plants expressing CP(T42W), infection by TMV-CP:GFP or TMV-MP:GFP-CP produced infection sites of

48 4 genes could be rapidly induced not only by TMV infection but also by SA or its biologically active

49 inability of dRNAs that can be replicated by TMV to move in plants.

50 unctional relationship between calreticulin, TMV MP, and viral cell-to-cell movement.

51 sidue plus the 5B19 epitope fused to the CP (TMV-RB19), resulted in the induction of necrotic local l

52 o mosaic virus (TMV) in place of the TMV CP [TMV(ORF3)], in infected cells it interacted with the TMV

53 Finally, we developed TMV and SNP formulations loaded with the chemotherapeuti

54 CMV had the highest levels of diversity, TMV had an intermediate level of diversity, and CCMV had

55 as very efficient with a 35S promoter-driven TMV replicon that lacked the TMV coat protein gene seque

56 versity of these guilds being unique to each TMV.

57 n increased amount of N protein and enhanced TMV resistance.

58 ecause TMV has a wide host range, expressing TMV-TMOF in plants can be used as a general method to pr

59 tural stability with the largest length, for TMV VLP to date.

60 l replicase- and VRC-enriched fractions from TMV-infected Nicotiana tabacum plants.

61 tein and the homologous 125-kDa protein from TMV and TVCV, respectively.

62 Furthermore, TMV encoding the MP mutant mimicking phosphorylation was

63 used to the green fluorescent protein (GFP) (TMV-CP:GFP, TMV-CP(T42W):GFP) and clones harboring TMV m

64 green fluorescent protein (GFP) (TMV-CP:GFP, TMV-CP(T42W):GFP) and clones harboring TMV movement prot

65 :GFP, TMV-CP(T42W):GFP) and clones harboring TMV movement protein (MP):GFP were followed in nontransg

66 However, TMV vectors are, in general, not efficiently delivered t

67 Hybrid TMV-5B19, administered by subcutaneous injections, elici

68 tibody, showed specific decoration of hybrid TMV particles, confirming the expression and display of

69 Plants that were infected with the hybrid TMV RNA accumulated TMOF to levels of 1.3% of total solu

70 In TMV-infected tobacco, these studies revealed that critic

71 Furthermore, in TMV-infected plant tissues overexpressing calreticulin,

72 monstrated that expression of these genes in TMV-inoculated leaves is mediated via an SA-independent

73 However, in TMV-infected tissues, PAP1 failed to accumulate to signi

74 sion size upon TMV infection, as observed in TMV-inoculated N gene-containing NahG tobacco plants.

75 d-type tobacco, suggesting a role for PME in TMV systemic infection.

76 vestigate the role of these host proteins in TMV accumulation and plant defense, we used a Tobacco ra

77 nspiration rate was significantly reduced in TMV infected N. tabacum.

78 recognized by N-containing tobacco to induce TMV resistance.

79 cing of NbBRI1 and NbBSK1 blocked BR-induced TMV resistance, and silencing of NbBES1/BZR1 blocked Bik

80 ortance of specific domains of N in inducing TMV resistance, by examining various N deletion and poin

81 IAA-interacting protein was found to inhibit TMV accumulation and phloem loading selectively in flowe

82 subviral replication complexes that initiate TMV replication immediately after entry to adjacent cell

83 When the biosensor was infiltrated into TMV-inoculated leaves displaying HR lesions at 90 and 16

84 tapeptide sequence TLIAHPQ was inserted into TMV coat protein near the C end.

85 howing that non-viral expression of a 50 kDa TMV helicase fragment (p50) is sufficient to induce the

86 In AtpC- and Rca-silenced leaves TMV accumulation and pathogenicity were greatly enhanced

87 SIPK/WIPK pathway attenuated N gene-mediated TMV resistance.

88 proteins play a critical role in N-mediated TMV resistance.

89 Genetically modified TMV VLPs express both surface attachment-promoting cyste

90 plasm and have documented associations of MP(TMV) with endoplasmic reticulum (ER) membrane, microtubu

91 obacco mosaic virus (TMV) 30-kDa protein (MP(TMV)).

92 d program averages intensities from multiple TMV molecules for accurate standard determination, makes

93 Native TMV forms a hollow, high aspect-ratio nanotube measuring

94 culated leaves because of the lack of native TMV coat protein.

95 and the poly(A) tail but not with the native TMV 3'-UTR which contains an independent translational e

96 Comparing antigenicity of TMV displaying 3 to 20 copies of NPNA revealed that low

97 bicin, and we demonstrate the application of TMV rods and spheres for chemotherapy delivery targeting

98 TMV by temporal and quantitative control of TMV Cg CP (CgCP) gene expression using a simple, methoxy

99 The dependence of TMV, PVX, and TBSV on intact microfilaments for intercel

100 irmed in rhesus macaques where a low dose of TMV-NPNAx5 elicited Abs that persisted at functional lev

101 f vein classes in source leaves for entry of TMV, and the lack of equivalence of vein classes in sink

102 ta presented here supports the evaluation of TMV-NPNAx5/ALFQ in human trials.

103 e of vein classes in sink leaves for exit of TMV.

104 the deposited model for the helical form of TMV at the lower-radius region (residues 88 to 109).

105 The history of TMV illustrates how pragmatic strategies to control an e

106 verexpressing calreticulin, the inability of TMV MP to reach plasmodesmata substantially impaired cel

107 th wild-type TMV infections: an inability of TMV to support dRNAs that can move in plants and the ina

108 E-binding region resulted in inactivation of TMV cell-to-cell movement.

109 provide evidence that a wild-type isolate of TMV is able to enter C71 cells grown in liquid medium, r

110 eral SIS genes in the uninoculated leaves of TMV-infected NahG plants was delayed and/or reduced, rai

111 the density map to build an atomic model of TMV.

112 Mutants of TMV lacking functional MP accumulated vRNA, but the dist

113 The use of amino acid deletion mutants of TMV MP showed that its domain was necessary and sufficie

114 tein, directed by the subgenomic promoter of TMV coat protein in Av, supported systemic infection wit

115 To improve these properties of TMV vectors, the gene encoding the 30-kDa movement prote

116 The host range of TMV was extended to include spinach as a permissive host

117 Here we report a helical reconstruction of TMV in its calcium-free, metastable assembling state at

118 ction is delayed, and cell-to-cell spread of TMV and CaLCuV movement proteins is inhibited.

119 Previously, the structure of TMV was solved at atomic detail by X-ray fiber diffracti

120 lind Franklin, who had taken up the study of TMV.

121 ere successfully displayed on the surface of TMV, and viruses accumulated to high levels in infected

122 o display foreign peptides on the surface of TMV.

123 ositively charged epitopes on the surface of TMV.

124 ly interfered with plasmodesmal targeting of TMV MP, which, instead, was redirected to the microtubul

125 ne, and inhibits cell-to-cell trafficking of TMV and CaLCuV movement proteins, when tested in an Agro

126 ork, infection induced by RNA transcripts of TMV clones that contain wt CP or mutant CP(T42W) fused t

127 Systemic transport of TMV in these plants, however, was substantially delayed

128 h agrees with available experimental data on TMV reconstruction.

129 but not three other myosins, inhibited only TMV movement.

130 ressing CP(T42W), infection by TMV-CP:GFP or TMV-MP:GFP-CP produced infection sites of smaller size t

131 0.5 D, where it improved in MMV by 19% over TMV.

132 icacy of Tobacco mosaic virus-like particle (TMV VLP) sensing probes using an impedimetric microsenso

133 eover, Arabidopsis PAPK1 also phosphorylates TMV MP in vitro at its C terminus.

134 n nontransgenic and wt CP transgenic plants, TMV-CP:GFP produced expanding, highly fluorescent disk-s

135 ith podocalyxin and ezrin, where it promotes TMV release.

136 -cell tobacco mosaic virus movement protein (TMV MP) mediates viral spread between the host cells thr

137 es of tobacco mosaic virus movement protein (TMV MP); this posttranslational modification has been sh

138 Mice that received TMV-5B19L intranasally developed serum IgG and IgA speci

139 Recombinant TMV functioned as an epitope tag recognizing streptavidi

140 Recombinant TMV was left on the filter.

141 Recombinant TMV-streptavidin complex was dissociated with 0.2M aceti

142 With a similar approach, a recombinant TMV vector carrying a gene for the ectopic expression of

143 The streptavidin-specific recombinant TMV system was applied successfully for purification of

144 streptavidin were incubated with recombinant TMV virions.

145 ncing of NbBES1/BZR1 blocked Bikinin-reduced TMV resistance.

146 We were able to obtain discrete rod-shaped TMV@MOF core-shell hybrids with good uniformity, and the

147 mers to the capsid exterior, organic-soluble TMV rods have been prepared.

148 low sulfate TMVs, while the highest sulfate TMV harboured 16S rRNA phylotypes associated with sulfur

149 Significantly, the T103C-TMV-E50C-A74C shows the highest robustness in assembly c

150 Detailed studies demonstrate that TMV multiplies exclusively in the cytoplasm and have doc

151 icating vascular tissues, we determined that TMV could enter minor, major or transport veins directly

152 Combined, these findings indicate that TMV directs the reprogramming of auxin-regulated gene ex

153 These studies show that TMV can be an effective vaccine delivery vehicle for par

154 Combined, these findings suggest that TMV replicase-Aux/IAA interactions selectively enhance v

155 n in the PME antisense plants suggested that TMV systemic movement may be a polar process in which th

156 The TMV coat protein (CP) was fused to TMOF at the C terminu

157 The TMV hybrids were propagated in tobacco plants, and the v

158 The TMV-based vector also enabled C. acutatum to transiently

159 Although promoters containing the TMV Omega sequence exhibited slightly increased basal ex

160 t a regulatory mechanism for controlling the TMV MP-plasmodesmata interactions in a host-dependent fa

161 gA specific for the 5B19 epitope and for the TMV coat protein.

162 promoter-driven TMV replicon that lacked the TMV coat protein gene sequence.

163 mistries have been established to modify the TMV rod, such methods have not yet been described for th

164 m Tobacco mosaic virus (TMV) in place of the TMV CP [TMV(ORF3)], in infected cells it interacted with

165 Thus, both the structure and assembly of the TMV CP function as determinants in the induction of dise

166 des covering the interacting portions of the TMV helicase domain were expressed and purified.

167 These results suggest that features of the TMV helicase domain, independent of its enzymatic activi

168 hese host genes altered the phenotype of the TMV infection foci and VRCs, yielding foci with concentr

169 Introduction of the TMV Omega sequence into the 5'UTR resulted in a further

170 nted with alterations in at least one of the TMV parameters.

171 Transfer of the TMV Pumilio recognition sequences into the genome of pot

172 evious study indicated that a portion of the TMV replicase containing a putative helicase domain is i

173 The ability of the TMV replicase to interact with Aux/IAA proteins from div

174 pathogens, the helicase domain (p50) of the TMV replicase, the avirulence gene of N, was linked to s

175 ethyltransferase and helicase domains of the TMV replicase.

176 lay of the MHV epitope on the surface of the TMV.

177 Based on the TMV classification, bone turnover was normal in 48%, hig

178 NPNAx5 antigen displayed as a loop on the TMV particle was found to be most optimal and its effica

179 en together, these data demonstrate that the TMV helicase domain interacts with itself to produce hex

180 Da proteins, these findings suggest that the TMV replicase may form a similar oligomer.

181 Combined, these data indicate that the TMV replicase protein interferes with the plant's auxin

182 The authors believe that the TMV-based affinity system can also be used for the purif

183 d/or synthesis inhibitors indicated that the TMV-induced expression of several SIS genes is independe

184 ucleus, suggesting that interaction with the TMV replicase protein disrupts PAP1 localization.

185 )], in infected cells it interacted with the TMV RNA to form filamentous ribonucleoprotein (RNP) part

186 odern usage of the word "virus." Since then, TMV has been acknowledged as a preferred didactic model

187 The robust nature of this TMV VLP allows for reducer-free synthesis of excellent e

188 Using this TMV expression vector, some foreign proteins were expres

189 pression studies show that in mature tissues TMV 126/183-kDa-interacting Aux/IAAs predominantly expre

190 In addition to TMV MP, PME is recognized by MPs of turnip vein clearing

191 Heat-transformation can be applied to TMV yielding spherical nanoparticles (SNPs) measuring ~5

192 ignificantly reduced with MMV as compared to TMV only at intermediate object distances, however was u

193 ines exhibit hypersensitive response (HR) to TMV and restrict virus spread to the inoculated site.

194 pleted in InsP6 and were hypersusceptible to TMV, turnip mosaic virus, cucumber mosaic virus and caul

195 promote oncogenic signaling, which leads to TMV release while inhibiting invadopodium formation.

196 assays, treatment with Intrepid-2F prior to TMV infection resulted in high levels of viral resistanc

197 Rar1- like gene for N-mediated resistance to TMV and some powdery mildew resistance genes in barley p

198 Here, we examined resistance to TMV by temporal and quantitative control of TMV Cg CP (C

199 the tobacco N gene can confer resistance to TMV in heterologous N. benthamiana.

200 results in very high levels of resistance to TMV infection in plants containing CP(T42W).

201 NIM1- like genes in N-mediated resistance to TMV using a TRV based VIGS approach.

202 d in attenuation of N-mediated resistance to TMV, indicating that these miRNAs have functional roles

203 ired for N to provide complete resistance to TMV.

204 K1/NQK1, attenuated N-mediated resistance to TMV.

205 me also compromises N-mediated resistance to TMV.

206 e necessary to confer complete resistance to TMV.

207 transcripts, exhibit complete resistance to TMV.

208 tein, fail to exhibit complete resistance to TMV.

209 g transcriptional alterations in response to TMV contain multiple auxin response promoter elements.

210 is transcriptionally induced in response to TMV infection, and its overexpression significantly redu

211 ts undergoing the hypersensitive response to TMV infection.

212 In response to TMV infections, ATAF2 and PR1 display increased transcri

213 ole in the N-mediated resistance response to TMV.

214 ine hepatitis virus (MHV) and giving rise to TMV-5B19 and TMV-5B19L, respectively.

215 InsP6 levels and enhanced susceptibility to TMV and to virulent and avirulent strains of the bacteri

216 -1 plants exhibit enhanced susceptibility to TMV, as well as to virulent pathogens.

217 roperties of the tobacco mosaic tobamovirus (TMV) coat protein (CP) make it possible to display forei

218 Today, TMV is used as a tool to study host-pathogen interaction

219 ' native aberrations and induce traditional (TMV) and modified (MMV) monovision corrections.

220 ccurring dRNAs in association with wild-type TMV infections: an inability of TMV to support dRNAs tha

221 Unlike TMV, the PVX RNA was concentrated in distinctive 'whorls

222 ene N induces a hypersensitive response upon TMV infection and protects tobacco against systemic spre

223 ypersensitive response (HR) lesion size upon TMV infection, as observed in TMV-inoculated N gene-cont

224 helicase domain of the Tobacco mosaic virus (TMV) 126- and 183-kDa replicase proteins was previously

225 helicase domain of the Tobacco mosaic virus (TMV) 126- and/or 183-kDa replicase protein(s) was found

226 helicase domain of the Tobacco mosaic virus (TMV) 126-/183-kDa replicase protein(s) and the Arabidops

227 hese activities is the Tobacco mosaic virus (TMV) 30-kDa protein (MP(TMV)).

228 confers resistance to tobacco mosaic virus (TMV) and encodes a Toll-interleukin-1 receptor/nucleotid

229 confers resistance to Tobacco mosaic virus (TMV) and encodes a toll-interleukin-1 receptor/nucleotid

230 replication protein of tobacco mosaic virus (TMV) and phloem-specific auxin/indole acetic acid (Aux/I

231 to this approach using Tobacco Mosaic Virus (TMV) as a test specimen and obtained a map from 210,000

232 tle virus (PMMoV), and tobacco mosaic virus (TMV) as indicators of the reduction of human enteric vir

233 url virus (CaLCuV) and Tobacco mosaic virus (TMV) cell-to-cell movement proteins.

234 ical properties of the Tobacco mosaic virus (TMV) coat protein (CP) are addressed in relation to its

235 Expression of tobacco mosaic virus (TMV) coat protein (CP) in plants confers resistance to i

236 ion of a gene encoding tobacco mosaic virus (TMV) coat protein (CP) in transgenic plants confers resi

237 as optimized using the tobacco mosaic virus (TMV) display platform.

238 e replicase protein of Tobacco mosaic virus (TMV) disrupts the localization and stability of interact

239 The genome of tobacco mosaic virus (TMV) encodes replicase protein(s), movement protein (MP)

240 Tobacco mosaic virus (TMV) entry into minor, major and transport veins from no

241 Tobacco mosaic virus (TMV) forms dense cytoplasmic bodies containing replicati

242 ackled the assembly of Tobacco mosaic virus (TMV) from its constituent RNA and protein subunits.

243 ence (called Omega) of tobacco mosaic virus (TMV) functions as a translational enhancer in plants.

244 n vectors based on the tobacco mosaic virus (TMV) genome are powerful tools for foreign gene expressi

245 Tobacco mosaic virus (TMV) has had an illustrious history for more than 100 ye

246 plant defense against Tobacco Mosaic Virus (TMV) in Nicotiana benthamiana.

247 ein was expressed from Tobacco mosaic virus (TMV) in place of the TMV CP [TMV(ORF3)], in infected cel

248 ory activities against tobacco mosaic virus (TMV) in vivo and in vitro.

249 ore and for 3 hr after tobacco mosaic virus (TMV) infection.

250 e protein shell of the tobacco mosaic virus (TMV) provides a robust and practical tubelike scaffold f

251 the helper virus (HV) Tobacco mosaic virus (TMV) replicase.

252 The dominant tobacco mosaic virus (TMV) resistance gene N induces a hypersensitive response

253 lectivity studies with tobacco mosaic virus (TMV) showed an excellent specificity for the targeted TN

254 age of a deconstructed tobacco mosaic virus (TMV) system, where the capsid protein (CP) gene is repla

255 Using tobacco mosaic virus (TMV) tagged with green fluorescent protein to follow inf

256 licase (p50) domain of Tobacco mosaic virus (TMV) through its TIR domain.

257 Cell-to-cell spread of tobacco mosaic virus (TMV) through plant intercellular connections, the plasmo

258 cell-to-cell spread of tobacco mosaic virus (TMV) through plant intercellular connections, the plasmo

259 of the surface of the tobacco mosaic virus (TMV) virion with a mosquito decapeptide hormone, trypsin

260 ovalent network within tobacco mosaic virus (TMV) virus-like particles (VLPs).

261 irst discovered virus, tobacco mosaic virus (TMV) was at the center of virus research.

262 ast, susceptibility to tobacco mosaic virus (TMV) was not altered in CSN-silenced plants.

263 Recombinant tobacco mosaic virus (TMV) was used as an affinity matrix for isolation and pu

264 Hybrids of tobacco mosaic virus (TMV) were constructed with the use of fusion to the coat

265 ective RNAs (dRNAs) of Tobacco mosaic virus (TMV) were examined in planta with helper viruses that ex

266 e RNA from the helical tobacco mosaic virus (TMV) were extracted using phenol/chloroform.

267 confers resistance to tobacco mosaic virus (TMV), and leads to induction of standard defense and res

268 bust protein template, tobacco mosaic virus (TMV), can be used to regulate the size and shape of as-f

269 is-like plant viruses, Tobacco mosaic virus (TMV), Cucumber mosaic virus (CMV), and Cowpea chlorotic

270 ll-to-cell movement of tobacco mosaic virus (TMV), is also required for the systemic spread of viral

271 ses is highlighted for tobacco mosaic virus (TMV), M13 bacteriophage, cowpea chlorotic mottle virus (

272 of known MPL, such as Tobacco Mosaic Virus (TMV), MPL of the fibrils in question can be determined.

273 from different genera [tobacco mosaic virus (TMV), potato virus X (PVX), tomato bushy stunt virus (TB

274 Infection with Tobacco mosaic virus (TMV), which is known to activate the SIPK cascade and in

275 Tobacco mosaic virus (TMV)-based transient expression vectors can express very

276 A tobacco mosaic virus (TMV)-based vector carrying such inverted-repeats, homolo

277 ing SAR development in tobacco mosaic virus (TMV)-infected tobacco and Pseudomonas syringae-infected

278 in the genomic RNA of tobacco mosaic virus (TMV).

279 the virulent pathogen tobacco mosaic virus (TMV).

280 leaves inoculated with tobacco mosaic virus (TMV).

281 confers resistance to Tobacco mosaic virus (TMV).

282 confers resistance to tobacco mosaic virus (TMV).

283 is work focuses on the tobacco mosaic virus (TMV).

284 nts after infection by tobacco mosaic virus (TMV).

285 resisting infection by tobacco mosaic virus (TMV).

286 confers resistance to tobacco mosaic virus (TMV).

287 ed on the plant virus, tobacco mosaic virus (TMV).

288 confers resistance to tobacco mosaic virus (TMV).

289 that of wild-type CP, the resulting viruses TMV-RB19E and TMV-4D:31D infected N. tabacum Xanthi-nn p

290 The molecular pathway by which TMV MP interacts with the host cell is largely unknown.

291 a benthamiana plants prior to challenge with TMV expressing green fluorescent protein.

292 ns was 3.5 +/- 0.5 D (mean) as compared with TMV (2.7 +/- 0.3 D).

293 1.0 D) than with negative SA, compared with TMV.

294 act with TMV MP, none of them coresides with TMV MP within plasmodesmata.

295 ions were isolated from plants infected with TMV(ORF3) or with GRV itself.

296 he effect in NN tobacco plants infected with TMV.

297 ost factors have been shown to interact with TMV MP, none of them coresides with TMV MP within plasmo

298 otiana glutinosa and Chenopodium quinoa with TMV).

299 eptible host systems (Nicotiana tabacum with TMV (Tobacco mosaic virus), and Arabidopsis thaliana wit

300 ble from that of the wild-type N plants, yet TMV was able to move systemically, causing a systemic hy