1                                       Higher TWEAKR/TNFRSF12A [OR 2.25 (95% CI 1.15, 4.77)] and MBL/M

2               Finally, the downregulation of TWEAKR by miR-K10a in primary human endothelial cells re

3 endothelial cell line, the downregulation of TWEAKR by miR-K10a resulted in reduced levels of TWEAK-i

4                            The expression of TWEAKR was downregulated in cells transfected with miR-K

5 egion (UTR) of TWEAKR suggest a targeting of TWEAKR by miR-K10a.

6 -K10a recognition sites within the 3' UTR of TWEAKR completely disrupts inhibition by miR-K10a.

7 ntaining the 3' untranslated region (UTR) of TWEAKR suggest a targeting of TWEAKR by miR-K10a.

8                              TWEAK receptor (TWEAKR/Fn14) is identified as a novel direct target of m

9  weak inducer of apoptosis (TWEAK) receptor (TWEAKR) was among the most consistently and robustly dow