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1 al and dorsocentral subdivisions and nucleus taenia); (2) other diencephalic nuclei (centroposterior,
2 a, hyperstriatum, archistriatum/nucleus (n.) taenia (amygdala), medial part of the LSO, organum vascu
3 ei of the ventral telencephalon, the nucleus taenia, and the medial region of the posterior nucleus o
4           Taenia saginata, Taenia solium and Taenia asiatica (beef, pork and Asian tapeworms, respect
5  asiaticus Eom et al., which is a homonym of Taenia asiatica von Linstow.
6 ominis is proposed as a replacement name for Taenia asiaticus Eom et al., which is a homonym of Taeni
7 were minor, hypoxic relaxation in guinea-pig taenia caeci appears to be directly related to energy li
8 ately 10% of the control value in guinea-pig taenia caeci.
9  +/- 1% (n = 31) and in the duodenum, ileum, taenia coli, proximal colon, and distal colon by 42%-55%
10 trol fEPSP amplitude in the duodenum, ileum, taenia coli, proximal colon, and distal colon by 71%-84%
11  we found that products excreted/secreted by Taenia crassiceps (TcES) triggered cRAF phosphorylation
12 d that the larval homogenate of the cestodes Taenia crassiceps and Taenia solium contained high conce
13 sus and Echinococcus multilocularis and with Taenia crassiceps antigen.
14                                              Taenia crassiceps cysticercosis results in an impressive
15  the peritoneal cavity of mice infected with Taenia crassiceps cysts and examined for cytokine messag
16                 Immune destruction of larval Taenia crassiceps was examined by first injecting BALB/c
17 e origin of AAMvarphis during infection with Taenia crassiceps, and their disease-modulating activity
18 ng as larvae in the body of a host following taenia egg ingestion.
19 a provide new insights into the evolution of Taenia parasites, particularly the recent speciation of
20 cture of an sHsp from the parasitic flatworm Taenia saginata Tsp36, the first metazoan crystal struct
21                                              Taenia saginata, Taenia solium and Taenia asiatica (beef
22 f the nervous system by the cystic larvae of Taenia solium (neurocysticercosis) is a frequent cause o
23                                              Taenia solium (the pork tapeworm) is present in most dev
24  causes of FBD deaths were Salmonella Typhi, Taenia solium and hepatitis A virus.
25                             Taenia saginata, Taenia solium and Taenia asiatica (beef, pork and Asian
26  Echinococcus multilocularis, E. granulosus, Taenia solium and the laboratory model Hymenolepis micro
27 nsmitter glutamate by the parasitic tapeworm Taenia solium appears to be implicated in the pathophysi
28 e to cure (r = -0.559; P = .027), and a high Taenia solium burden (by quantitative polymerase chain r
29 genate of the cestodes Taenia crassiceps and Taenia solium contained high concentrations of the amino
30 in-bound glycoproteins (LLGP) extracted from Taenia solium cysticerci.
31 ree parasitic infections - cerebral malaria, Taenia solium cysticercosis and onchocerciasis - have an
32 current antiparasitic treatment for cerebral Taenia solium cysticercosis with either albendazole (ABZ
33  transmitted through the direct ingestion of Taenia solium eggs, which differentially disrupts the ba
34                                          The Taenia solium genome was mined and primers and probes we
35              Neurocysticercosis is caused by Taenia solium infecting the central nervous system and i
36                         Cysticercosis due to Taenia solium is a cause of adult-acquired seizures and
37                             Taeniasis due to Taenia solium is a disease with important public health
38 ral nervous system (CNS) infection caused by Taenia solium metacestodes.
39                                 Infection by Taenia solium poses a major burden across endemic countr
40          Infection by the causative parasite Taenia solium requires transmission between humans and p
41 tal tool for detecting and treating cases of Taenia solium taeniasis.
42 d, in 2012, for a validated strategy towards Taenia solium taeniasis/cysticercosis control and elimin
43                                          The Taenia solium tapeworm is responsible for cysticercosis,
44 ) antigens extracted from the metacestode of Taenia solium, has been the "gold standard" serodiagnost
45 f the human central nervous system caused by Taenia solium, is a leading cause of seizures.
46                Neurocysticercosis, caused by Taenia solium, is one of the most common causes of seizu
47 d by the larval stages of the pork tapeworm, Taenia solium.
48 sed by the metacestode stage of the parasite Taenia solium.
49 used by the metacestode form of the helminth Taenia solium.
50 the brain by larvae of the parasitic cestode Taenia solium.
51 tors for the spread of the zoonotic parasite Taenia solium.
52 mmary of these changes is provided here, and Taenia suihominis is proposed as a replacement name for
53 bic seizures had somatic degeneration in the taenia tecta within 3 days of amphetamine exposure.
54 also abundant in anterior olfactory nucleus, taenia tecta, and lateral septum.
55 organum vasculosum of the lamina terminalis, taenia tecta, nucleus accumbens, lateral septum, endopir
56 ngulate, and ventrolateral orbital cortices, taenia tecta, nucleus accumbens, paraventricular nucleus
57 factory nucleus; in the induseum griseum and taenia tecta; in the olfactory tubercle; in CA1-CA3, the
58 o propose the use of AMV (instead of nucleus taenia/TnA), AMD, AD, and AI as initial steps toward a u
59 tionally uniform populations of Trichinella, Taenia, Toxoplasma and Sarcocystis, indicating a fruitfu
60 ent of human taeniasis (presence of an adult taenia worm in the human intestine) to NCC mitigation.