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1  rejection more effectively than control Th2/Tc2 cells.
2 ificantly higher levels on Tc1 cells than on Tc2 cells.
3 t in 10-fold higher numbers than control Th2/Tc2 cells.
4 ogression than that of functionally distinct Tc2 cells.
5 pe and function from T cytotoxic 1 (Tc1) and Tc2 cells.
6 utic efficiency when compared with wild-type Tc2 cells.
7 ibuted to TCR-dependent PGD(2) production in Tc2 cells.
8 ive CD8(+) T cell differentiation to Tc17 or Tc2 cells.
9 ively transferred ovalbumin-specific Tc1 and Tc2 cells accumulated at the tumor site by day 2 after t
10                                              Tc2 cells achieved a comparable reduction in lung tumor
11 (2) receptor 2 (DP2) play important roles in Tc2 cell activation, including migration, cytokine produ
12 ) T cells differentiate into IL-13-producing Tc2 cells and play a major role in a bleomycin-induced m
13  underwent more efficient expansion than did Tc2 cells, and only Tc1 cells were detected at the site
14 Type 2 immunity consists of GATA-3(+) ILC2s, TC2 cells, and TH2 cells producing IL-4, IL-5, and IL-13
15                    Human type 2 cytotoxic T (Tc2) cells are enriched in severe eosinophilic asthma an
16 egulation of pulmonary fibrosis and identify Tc2 cells as key mediators of fibrogenesis.
17                                 Both Tc1 and Tc2 cells can mediate murine cardiac allograft rejection
18 ed cytokine secretion did not inhibit Tc1 or Tc2 cell cytolytic function.
19                                 In contrast, Tc2 cells derived from select cytokine gene-deficient mi
20                    In contrast, both Tc1 and Tc2 cells, derived from either FasL or TNF-alpha/lymphot
21 arily vascularized allograft, and if Tcl and Tc2 cells differ in their ability to mediate rejection.
22 T-cytotoxic 1 (Tc1) cells, but not by Th2 or Tc2 cells, establishing Bonzo as a differential marker o
23  type 2 T helper (Th2) and type 2 cytotoxic (Tc2) cell frequencies, skewing ILC2 towards a CD117(low)
24 vely transferred allo-(H-2d)-reactive Tcl or Tc2 cells from H-2b mice into each recipient.
25               Transfer of perforin-deficient Tc2 cells generated from perforin gene knockout mice sho
26 h1 cytotoxic (Tc1) cytokines, but not Th2 or Tc2, cell generation.
27                                      Tc1 and Tc2 cells had similar adenosine signaling, as measured b
28                                              Tc2 cell IL-4 and IL-5 secretion was not reduced by CGS,
29                     Differentiation of these Tc2 cells in the lung requires IL-21, and bleomycin trea
30 ovided a better understanding of the role of Tc2 cells in the pathogenesis of asthma.
31 convert from IFN-gamma(+) (Tc1) to IL-13(+) (Tc2) cells in the presence of IL-4.
32  adoptively transferred OVA-specific Tc1 and Tc2 cells induce considerable suppression, but not cure,
33                  CGS greatly reduced Tc1 and Tc2 cell interleukin 2 (IL-2) and tumor necrosis factor
34                                  Transfer of Tc2 cells into IFN-gamma-deficient tumor-bearing mice wa
35  speculate that the effectiveness of Tc1 and Tc2 cells may depend on different mechanisms.
36 stay and may play a protective role, whereas Tc2 cells may play a previously underappreciated role in
37                            Conversely, donor Tc2 cell numbers markedly diminished at later times, sug
38                 Tc1 cells persisted, whereas Tc2 cell numbers progressively diminished over time.
39                             We conclude that Tc2 cells potently inhibit marrow graft rejection withou
40 graft vasculitis, although IL-4high IL-5high Tc2 cells promote recruitment of secondary effectors lik
41 n-2/interferon-gamma (IL-2/IFN-gamma) or TH2/TC2 cells secreting IL-4/IL-5/IL-10.
42 leens of tumor-bearing mice receiving Tc1 or Tc2 cells showed markedly enhanced tumor Ag-specific cyt
43 omplexes were not expressed by either Tc1 or Tc2 cells, suggesting that CD49d is solely expressed in
44 fferential expression of VLA-4 on Tc1 versus Tc2 cells, Tc1 cells alone were competent to adhere to p
45 pothesized that rapamycin would generate Th2/Tc2 cells (Th2/Tc2.R cells) that abrogate fully MHC-disp
46 the expression of tissue-remodeling genes in Tc2 cells that enhanced the fibroblast proliferation and
47 gressive response and that non-host-reactive Tc2 cells therefore facilitate engraftment across geneti
48 owledge, functions of the PGD(2)/DP2 axis in Tc2 cells to induce tissue-remodeling effects and IgE-in
49                            PGD(2) stimulated Tc2 cells to produce PGD(2) using the routine PGD(2) syn
50      At 650 cGy irradiation, the addition of Tc2 cells to the F1 marrow resulted in extensive F1 chim
51 ignature with high CD62L expression, and Th2/Tc2 cells towards a CD45RA(-)CD62L(+) central memory phe
52 mmatory cytokine production, and survival of Tc2 cells triggered by PGD(2) but also attenuated the ti
53                          VLA-4 expression on Tc2 cells was down-regulated in an interleukin (IL)-4 do
54 f IFN-gammahigh Tc1, but not of IFN-gammalow Tc2 cells was followed by the development of graft vascu
55 on of a panel of homing receptors on Tc1 and Tc2 cells, we found that very late antigen (VLA)-4 (a he
56                                Donor Tc1 and Tc2 cells were generated that preferentially secreted ty
57 type 1 cytotoxic T lymphocytes (Tc1) and Th2/Tc2 cells were generated using an antigen-presenting cel
58                      Rapamycin-generated Th2/Tc2 cells were less likely to die after adoptive transfe
59 ts, IL-5-producing CD8(+) T cells, so-called Tc2 cells, which in healthy donors can only be detected
60  CXCR4 in ILC2, and GPR183 in ILC2, Th2, and Tc2 cells while enhancing their type 2 cytokine producin
61 he frequencies of circulating ILC2, Th2, and Tc2 cells, with reduced tissue homing receptor expressio