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1 ediated by the interaction of flagellin with Toll-like receptor 5.
2 independently of flagellin identification by Toll-like receptor 5.
3 n of bacterial flagellins that interact with Toll-like receptor 5.
4 nal effectively through both human and mouse Toll-like receptor 5.
5 e to flagellin is likely driven, in part, by Toll-like receptor 5.
6 triggers innate immune responses mediated by Toll-like receptor 5.
10 innate immune responses by signaling through Toll-like receptor 5 and is a potential vaccine adjuvant
11 of C. difficile flagellin FliC in activating Toll-like receptor 5 and triggering nuclear factor-kappa
13 n some genetic backgrounds dominant-negative Toll-like receptor 5 associated negatively with Crohn's
14 response induced by flagellin activation of Toll-like Receptor 5 cell signalling is augmented follow
15 ablated in DCs from NLRC4(-/-) mice but not Toll-like receptor 5-deficient (TLR5(-/-)) mice, indicat
16 gellate Salmonella were highly attenuated in Toll-like receptor 5-deficient (TLR5(-/-)) mice, indicat
19 nduces caspase-1 activation independently of Toll-like receptor 5 in salmonella-infected and lipopoly
20 ellin:allergen fusion protein containing the Toll-like receptor 5 ligand flagellin A from Listeria mo
22 ave designed a membrane-anchored form of the Toll-like receptor 5 ligand flagellin, the major proinfl
23 into the portal circulation, with flagellin (Toll-like receptor 5 ligand) being the most plentiful an
24 rane of the intestinal epithelium, activates Toll-like receptor 5-mediated innate immune signaling pa
26 nduction was also noted after stimulation of Toll-like receptor 5 or 7, but not of other pattern reco
28 s population receives peripheral inputs from Toll-like receptor 5-positive (TLR5(+)) Abeta low-thresh
29 ivation of caspase-1 that was independent of Toll-like receptor 5, required for recognition of extrac
33 cognition site of the innate immune receptor Toll-like receptor 5, three of four FliC epitopes recogn
34 ther such antibodies might negatively impact Toll-like receptor 5 (TLR5) activation, an important com
37 of purified Salmonella-derived flagellin, a Toll-like receptor 5 (TLR5) agonist, protects mice from
40 ived from Salmonella flagellin that binds to Toll-like receptor 5 (TLR5) and activates nuclear factor
41 e I and II) and required flagellin receptors Toll-like receptor 5 (TLR5) and NOD-like receptor C4 (NL
42 rized one candidate nonsynonymous variant in Toll-like receptor 5 (TLR5) and show that it leads to al
43 in the sensing of flagellins; these involve toll-like receptor 5 (TLR5) and the cytosolic proteins B
46 lin revealed that cell surface expression of Toll-like receptor 5 (TLR5) conferred NF-kappaB gene exp
48 ind that mice lacking the flagellin receptor Toll-like receptor 5 (TLR5) exhibit a profound loss of f
51 unifying feature across phyla(6)-stimulates Toll-like receptor 5 (TLR5) in peptide YY (PYY)-labelled
55 t of a decrease in the steady-state level of toll-like receptor 5 (TLR5) or interleukin-1 receptor as
61 bacterial flagella, is a potent activator of toll-like receptor 5 (TLR5) signaling and is a major pro
66 , we show that mice genetically deficient in Toll-like receptor 5 (TLR5), a component of the innate i
67 ecules known to activate innate immunity via Toll-like receptor 5 (TLR5), and critical targets of the
69 agellin, a bacterial protein that stimulates Toll-like receptor 5 (TLR5), induces epithelial expressi
73 raction between bacterial flagellin and host Toll-like receptor 5 (TLR5), which may shed light on the
74 d not elicit antibodies that neutralized the Toll-like receptor 5 (TLR5)-activating activity of flage
75 ve bacteria activate inflammatory cells by a toll-like receptor 5 (TLR5)-dependent signaling pathway.
84 tigate whether flagellin, the sole ligand of Toll-like receptor-5 (TLR5), induces an innate defense t
85 ractions revealed flagellin, via ligation of Toll-like receptor 5, to be a major means of activating
86 supports the evolution of host control, via toll-like receptor 5, which limits symbiont counter evol