ライフサイエンスコーパス: Toxoplasma gondii

1 sential enzyme in the opportunistic pathogen Toxoplasma gondii.

2 vel tool for visualising F-actin dynamics in Toxoplasma gondii.

3 arasitic pathogens Plasmodium falciparum and Toxoplasma gondii.

4 niche for apicomplexan parasites, including Toxoplasma gondii.

5 cycle of the eukaryotic single-cell parasite Toxoplasma gondii.

6 with the brain-dwelling, protozoan parasite, Toxoplasma gondii.

7 parasitic infections in the world, caused by Toxoplasma gondii.

8 s a disease caused by the protozoan parasite Toxoplasma gondii.

9 pathogens, including the protozoan parasite Toxoplasma gondii.

10 ration of TH1 cells following infection with Toxoplasma gondii.

11 ium tuberculosis, Pneumocystis jirovecii, or Toxoplasma gondii.

12 or the lytic cycle of the protozoan parasite Toxoplasma gondii.

13 long immunity to virulent acute infection by Toxoplasma gondii.

14 omposition and function in the centrosome of Toxoplasma gondii.

15 a parasite Plasmodium falciparum and related Toxoplasma gondii.

16 obligate intracellular apicomplexan parasite Toxoplasma gondii.

17 es (ROPs) are essential virulence factors of Toxoplasma gondii.

18 onmentally driven infectious disease such as Toxoplasma gondii.

19 autophagy related 5 (Atg5) in resistance to Toxoplasma gondii.

20 s gliding motility and host cell invasion by Toxoplasma gondii.

21 es of this cytokine following challenge with Toxoplasma gondii.

22 the immune system responds to infection with Toxoplasma gondii.

23 (ROP5) expressed by the protozoan parasite, Toxoplasma gondii.

24 we identified candidates for APT activity in Toxoplasma gondii.

25 tion by the pervasive intracellular parasite Toxoplasma gondii.

26 y with the SRS-domain containing proteins of Toxoplasma gondii.

27 the globally prevalent apicomplexan parasite Toxoplasma gondii.

28 etroviruses, Mycobacterium tuberculosis, and Toxoplasma gondii.

29 nvasion and egress by the protozoan parasite Toxoplasma gondii.

30 ded creature can be an intermediate host for Toxoplasma gondii.

31 ogens such as Mycobacterium tuberculosis and Toxoplasma gondii.

32 re infected with the intracellular protozoan Toxoplasma gondii(1).

33 derstanding how immunity is elicited against Toxoplasma gondii - a complex pathogen with multiple mec

34 Toxoplasma gondii, a common neurotropic parasite, is inc

35 Intriguingly, Toxoplasma gondii, a highly successful apicomplexan para

36 switch between the lytic and latent forms of Toxoplasma gondii, a parasite that causes a persistent b

37 in CNS infections, including infection with Toxoplasma gondii, a protozoan parasite that naturally e

38 Toxoplasma gondii, a protozoan parasite, undergoes a com

39 ook advantage of the genetic tractability of Toxoplasma gondii, a related parasite that shows moderat

40 , deletion of the dpy19 gene in the parasite Toxoplasma gondii abolished C-mannosyltransferase activi

41 The obligate intracellular protozoan Toxoplasma gondii actively invades mammalian cells and,

42 al pH, was cryotrapped in the active site of Toxoplasma gondii aldolase crystals to high resolution.

43 Toxoplasma gondii, an apicomplexan parasite prevalent in

44 The cytoskeleton of Toxoplasma gondii, an important human parasite, contains

45 Toxoplasma gondii, an obligate intracellular protozoan p

46 ocytosis operates during gliding motility in Toxoplasma gondii and appears to be crucial for the esta

47 dence of primary gestational infections with Toxoplasma gondii and congenital toxoplasmosis in Austri

48 target of actinonin in the related parasite Toxoplasma gondii and implicated P. falciparum FtsH1 as

49 ch as Plasmodium species that cause malaria, Toxoplasma gondii and kinetoplastid protozoa, including

50 hree protozoan parasites (Trypanosoma cruzi, Toxoplasma gondii and Leishmania major), in which the Gz

51 a-mediated elimination of pathogens, such as Toxoplasma gondii and murine norovirus.

52 hallenge with the common coccidian parasites Toxoplasma gondii and Neospora caninum activated GABAerg

53 zes to the apical pole in invasive stages of Toxoplasma gondii and Plasmodium berghei, and apical pos

54 comparative activities of ADF proteins from Toxoplasma gondii and Plasmodium falciparum, the human m

55 is review primarily focuses on studies using Toxoplasma gondii and Plasmodium spp. as the best studie

56 Apicomplexan parasites Toxoplasma gondii and Plasmodium spp. use latent stages

57 How the protozoan pathogen Toxoplasma gondii and related parasites shuttle proteins

58 Using Toxoplasma gondii and Salmonella enterica Typhimurium we

59 The apical complex of Toxoplasma gondii and some other apicomplexans includes

60 Upon infections with Toxoplasma gondii and vaccinia virus, mice with stabiliz

61 ith potential pathogens (West Nile virus and Toxoplasma gondii) and one with Streptococcus species wi

62 morbillivirus, herpesvirus, West Nile virus, Toxoplasma gondii, and Brucella spp.

63 Th17 cell responses to herpes simplex virus, Toxoplasma gondii, and Citrobacter rodentium infections.

64 thogen challenges, including H1N1 influenza, Toxoplasma gondii, and Ebola virus.

65 esistance against a prototypic Th1 pathogen, Toxoplasma gondii, and for the generation of pathogen-sp

66 c cells (DCs) are critical for resistance to Toxoplasma gondii, and infection with this pathogen lead

67 ovirus, herpes simplex virus, rubella virus, Toxoplasma gondii, and Zika virus.

68 expansion microscopy (U-ExM) to localize the Toxoplasma gondii Apical Cap protein 9 (AC9) and its par

69 Here we tested this model using the Toxoplasma gondii apical membrane protein 1 (TgAMA1), wh

70 ost effects caused by the protozoan parasite Toxoplasma gondii are poorly understood.

71 Plasmodium falciparum and Toxoplasma gondii are widely studied parasites in phylum

72 Here, we use Toxoplasma gondii as a model system to functionally char

73 Here we establish that Toxoplasma gondii aspartyl protease 3 (ASP3) resides in

74 lium discoideum, and the protozoan parasite, Toxoplasma gondii, both lacking HIF.

75 After acute oral infection with Toxoplasma gondii, both WT and Crtam(-/-) mice mounted a

76 rmation of naturally occurring peptides from Toxoplasma gondii bound by HLA-A*02:01.

77 t defense against the intracellular pathogen Toxoplasma gondii, but prior infection with Toxoplasma c

78 function of the motility apparatus of living Toxoplasma gondii by adhering a microsphere to the surfa

79 tance to the intracellular vacuolar pathogen Toxoplasma gondii by inducing the destruction of the par

80 tent and selective inhibitor (compound 1) of Toxoplasma gondii calcium-dependent protein kinase 1 (Tg

81 ) have been shown to be potent inhibitors of Toxoplasma gondii calcium-dependent protein kinase 1.

82 Reactivation of chronic infection with Toxoplasma gondii can cause life-threatening toxoplasmic

83 It has been proposed that Toxoplasma gondii can cross biological barriers as a mot

84 the asexual cycle of the protistan parasite Toxoplasma gondii can occur in any warm-blooded mammal,

85 omplexan parasites Plasmodium falciparum and Toxoplasma gondii cause host cell cytolysis to facilitat

86 hronic infection with the protozoan parasite Toxoplasma gondii causes a nonresolving Th1 myositis wit

87 The apicomplexan parasite Toxoplasma gondii causes macrophage death through uniden

88 oplasmosis, pyrazolopyrimidine inhibitors of Toxoplasma gondii CDPK1 demonstrated in vitro and in viv

89 at removal of the autoinhibitory domain from Toxoplasma gondii CDPK1 is not sufficient for kinase act

90 Toxoplasma gondii chronically infects a quarter of the w

91 Upon infection, the intracellular parasite Toxoplasma gondii co-opts critical functions of its host

92 s left eye due to varicella-zoster virus and Toxoplasma gondii coinfection documented by polymerase c

93 itis secondary to varicella-zoster virus and Toxoplasma gondii coinfection in a male patient in Bogot

94 Toxoplasma gondii contains a limited subset of actin bin

95 inst intracellular pathogens, especially for Toxoplasma gondii control.

96 The intracellular parasite Toxoplasma gondii converts from a rapidly replicating ta

97 ior exposure to the pathogens Brucella spp., Toxoplasma gondii, Coxiella burnetii, Francisella tulare

98 tumoral injection of an attenuated strain of Toxoplasma gondii (cps) that cannot replicate in vivo an

99 The obligate intracellular parasite Toxoplasma gondii critically relies on host cell invasio

100 The Toxoplasma gondii cyst stage is resistant to drug therap

101 ssion of the obligate intracellular parasite Toxoplasma gondii depends on its ability to switch betwe

102 The protozoan parasite Toxoplasma gondii develops within a parasitophorous vacu

103 In Toxoplasma gondii, disruption of the first, fifth, or si

104 Toxoplasma gondii DNA detection is essential to antenata

105 ng fluorescent proteins or targeted genes of Toxoplasma gondii, driven by N. caninum promoters, have

106 contribution of each gene from the parasite Toxoplasma gondii during infection of human fibroblasts.

107 A particular emphasis is placed on Toxoplasma gondii, during both its acute and latent stag

108 ts of the Ca(2+) signaling pathway dictating Toxoplasma gondii egress have been identified, whether t

109 When Toxoplasma gondii egresses from the host cell, glycerald

110 Toxoplasma gondii encodes three protein kinase A catalyt

111 The genome of Toxoplasma gondii encodes ubiquitination machinery, but

112 The intracellular parasite Toxoplasma gondii enjoys a wide host range and is adept

113 cellular pathogen, the apicomplexan parasite Toxoplasma gondii evades immune system-mediated clearanc

114 In Toxoplasma gondii, extracellular potassium levels and ot

115 In Toxoplasma gondii, fatty acid synthesis via the apicopla

116 n of subtelomeric virulence genes, while the Toxoplasma gondii genome was dominated by clustering of

117 ing a synthetic phosphoglycan portion of the Toxoplasma gondii glycosylphosphatidylinositol (GPI1) fo

118 uous supply of potent killer T cells to curb Toxoplasma gondii growth during latency.

119 Here, we demonstrate that the apicomplexan Toxoplasma gondii harbors homologues of proteins from al

120 Toxoplasma gondii has been taken as an example for this

121 Traditionally, the protozoan parasite Toxoplasma gondii has been thought of as relevant to pub

122 The intracellular parasite Toxoplasma gondii has multiple strategies to alter host

123 A chronic infection with the parasite Toxoplasma gondii has previously been shown to protect m

124 The obligate intracellular parasite Toxoplasma gondii has unusually stable cortical microtub

125 Apicomplexan parasites like Toxoplasma gondii have a specialized cilium-like structu

126 Waterborne outbreaks of Toxoplasma gondii have focused attention on the importan

127 ROP16 is a protein kinase of Toxoplasma gondii identified in the mouse model as a vir

128 study evaluated the usefulness of adding the Toxoplasma gondii IgA antibody enzyme-linked immunosorbe

129 ly intrinsically disordered component of the Toxoplasma gondii IMC, as essential for apical complex d

130 sive neurocognitive assessments and had anti-Toxoplasma gondii immunoglobulin G (anti-Toxo IgG) measu

131 Using the zoonotic parasite Toxoplasma gondii in California, USA as a model for coas

132 e Ca(2+) signaling in the model apicomplexan Toxoplasma gondii In doing so, we took advantage of the

133 that produce strong immune responses against Toxoplasma gondii in HLA supermotif, transgenic mice.

134 lator of autophagy and autophagic killing of Toxoplasma gondii in host cells.

135 Brain infection by the parasite Toxoplasma gondii in mice is thought to generate vulnera

136 elements for detection and immune control of Toxoplasma gondii in mice, but not in humans.

137 Studies in Plasmodium spp. and Toxoplasma gondii in particular have revealed that these

138 ter, cytomegalovirus, Epstein-Barr virus and Toxoplasma gondii in patients with uveitis.

139 intain the latency of chronic infection with Toxoplasma gondii in the brain.

140 e knockout screens in Plasmodium berghei and Toxoplasma gondii, in which pooled transfections of mult

141 nd that the intracellular protozoan parasite Toxoplasma gondii induced an early IL-1beta response (wi

142 Chronic infection with Toxoplasma gondii induces a potent resistance against re

143 is essential for CD8+ T-cell recognition of Toxoplasma gondii infected cells.

144 f immune responses that limit cyst burden in Toxoplasma gondii-infected mice.

145 re we show that GCs are induced during acute Toxoplasma gondii infection and directly control the T c

146 Toxoplasma gondii infection causes substantial morbidity

147 Here, we describe Toxoplasma gondii infection converts NK cells into ILC1-

148 Toxoplasma gondii infection has been described previousl

149 Toxoplasma gondii infection in mice provides an excellen

150 The course of Toxoplasma gondii infection in rats closely resembles th

151 Toxoplasma gondii infection is an asymptomatic disease,

152 n, although their role in protection against Toxoplasma gondii infection is not thoroughly understood

153 Intrinsic to Toxoplasma gondii infection is the parasite-induced modu

154 An early hallmark of Toxoplasma gondii infection is the rapid control of the

155 er they develop these characteristics during Toxoplasma gondii infection is unknown.

156 Toxoplasma gondii infection occurs through the oral rout

157 the epigenomic and transcriptomic effects of Toxoplasma gondii infection on human host cells and demo

158 in NK cells both in vitro and in a model of Toxoplasma gondii infection revealed de novo chromatin a

159 rns of hydrocephalus secondary to congenital Toxoplasma gondii infection were identified and characte

160 of processes related to the establishment of Toxoplasma gondii infection, such as the formation of th

161 new insights into the roles of CDPKs during Toxoplasma gondii infection.

162 responses using a mouse model for persistent Toxoplasma gondii infection.

163 immunity exacerbates ileitis induced by oral Toxoplasma gondii infection.

164 osis Study (NCCCTS) have a high incidence of Toxoplasma gondii infection.

165 August 2010 for serologic evidence of recent Toxoplasma gondii infection.

166 f granulocyte responses and pathology during Toxoplasma gondii infection.

167 The most common method to diagnose Toxoplasma gondii infections is the serological determin

168 The intracellular parasite Toxoplasma gondii infects nucleated cells in virtually a

169 into host cells through endocytic pathways, Toxoplasma gondii initiates infection at the cell surfac

170 The Toxoplasma gondii inner membrane complex (IMC) is an imp

171 ined the effects of endothelial cell CD40 on Toxoplasma gondii invasion of the retina and brain, orga

172 Toxoplasma gondii is a classic model for studying obliga

173 Toxoplasma gondii is a common parasite of humans and ani

174 Toxoplasma gondii is a common protozoan parasite that in

175 Coastal habitat contamination with Toxoplasma gondii is a health risk to humans and marine

176 Toxoplasma gondii is a highly prevalent obligate intrace

177 d control of HIV type 1 replication, whereas Toxoplasma gondii is a life-threatening opportunistic in

178 The intracellular protozoan parasite Toxoplasma gondii is a major food-borne illness and oppo

179 Infection with the protozoan parasite Toxoplasma gondii is a major health risk owing to birth

180 Toxoplasma gondii is a protist parasite of warm-blooded

181 Toxoplasma gondii is a protozoan pathogen in the phylum

182 Toxoplasma gondii is a remarkably successful protozoan p

183 Toxoplasma gondii is a ubiquitous, obligate intracellula

184 Toxoplasma gondii is a widespread parasite of warm-blood

185 Toxoplasma gondii is a widespread parasite with consider

186 Toxoplasma gondii is a widespread parasitic pathogen tha

187 Toxoplasma gondii is a widespread protozoan parasite inf

188 The obligate intracellular parasite Toxoplasma gondii is able to infect a broad range of hos

189 The obligate intracellular parasite Toxoplasma gondii is able to infect nearly all nucleated

190 The propagation of Toxoplasma gondii is accomplished by repeated lytic cycl

191 Infection with Toxoplasma gondii is acquired through consumption of und

192 Toxoplasma gondii is among the most prevalent parasites

193 Toxoplasma gondii is an apicomplexan parasite that cause

194 The parasite Toxoplasma gondii is an environmentally persistent patho

195 Toxoplasma gondii is an incredibly successful parasite o

196 Toxoplasma gondii is an intracellular parasite that caus

197 Toxoplasma gondii is an intracellular parasite that caus

198 Toxoplasma gondii is an intracellular parasite that is h

199 Toxoplasma gondii is an obligate intracellular parasite

200 Toxoplasma gondii is an obligate intracellular parasite

201 Toxoplasma gondii is an obligate intracellular parasite

202 Toxoplasma gondii is an obligate intracellular parasite

203 Toxoplasma gondii is an obligate intracellular parasite

204 Toxoplasma gondii is an obligate intracellular parasite

205 Toxoplasma gondii is an obligate intracellular protozoan

206 Toxoplasma gondii is an obligate protozoan parasite that

207 Toxoplasma gondii is an obligate, intracellular eukaryot

208 Toxoplasma gondii is an opportunistic infection that can

209 Toxoplasma gondii is associated with physiological and p

210 Toxoplasma gondii is associated with physiological effec

211 Toxoplasma gondii is considered to be one of the most su

212 Cerebral infection with the parasite Toxoplasma gondii is followed by activation of resident

213 ted target of actinonin in P. falciparum and Toxoplasma gondii is FtsH1, a homolog of a bacterial mem

214 Unlike its animal counterparts, Skp1 from Toxoplasma gondii is hydroxylated by an O2-dependent pro

215 CD40-mediated autophagic killing of Toxoplasma gondii is known to require TNF-alpha.

216 Toxoplasma gondii is sensitive to bulky pyrazolo [3,4-d]

217 The protozoan parasite Toxoplasma gondii is thought to exploit monocyte traffic

218 gMyoA, a class XIVa myosin from the parasite Toxoplasma gondii, is required for the parasite to effic

219 In mice, infection with Toxoplasma gondii leads to a Th1-polarized parasite-spec

220 Infection by Toxoplasma gondii leads to massive changes to the host c

221 Toxoplasma gondii, like all apicomplexan parasites, uses

222 defense against the intracellular pathogens Toxoplasma gondii, Listeria monocytogenes, and Mycobacte

223 nfections with pathogens such as Zika virus, Toxoplasma gondii, Listeria monocytogenes, Treponema pal

224 pathogens, including the protozoan parasite Toxoplasma gondii, live inside a vacuole that resides in

225 The protozoan parasite Toxoplasma gondii lives inside a vacuole in the host cyt

226 ve genome-scale metabolic model (GEM) of the Toxoplasma gondii metabolic network that incorporates ge

227 , including Bacillus anthracis lethal toxin, Toxoplasma gondii, muramyl dipeptide, and host intracell

228 he founding member of the class XIVa family, Toxoplasma gondii myosin A (TgMyoA), is a monomeric unco

229 ases of invasive protozoal infections due to Toxoplasma gondii (n = 3), Trypanosoma cruzi, and Leishm

230 ed to functions within pathways/networks for Toxoplasma gondii, Neospora caninum, Cryptosporidium and

231 Indeed, in mice challenged with the parasite Toxoplasma gondii, NK and T cell responses are character

232 Horizontal transmission of Toxoplasma gondii occurs primarily via ingestion of envi

233 deliver a double punch that can destroy the Toxoplasma gondii parasite and its niche inside cells.

234 Toxoplasma gondii parasites must actively invade host ce

235 Toxoplasma gondii parasites rapidly exit their host cell

236 Bradyzoite forms of Toxoplasma gondii persist in tissue cysts for the lifeti

237 medical and veterinary importance, including Toxoplasma gondii, Plasmodium falciparum, and C. parvum

238 Apicomplexan parasites such as Toxoplasma gondii possess an unusual heme biosynthesis p

239 Toxoplasma gondii possesses a limited set of actin-regul

240 The intracellular parasite Toxoplasma gondii possesses three distinct Drps.

241 The Toxoplasma gondii PV is filled with a network of tubulat

242 Toxoplasma gondii reaches the CNS by circulating in bloo

243 Apicomplexan parasites such as Toxoplasma gondii rely on a unique form of locomotion kn

244 Toxoplasma gondii replicates asexually by a unique inter

245 The obligate intracellular parasite Toxoplasma gondii replicates in an unusual process, desc

246 ability, the obligate intracellular parasite Toxoplasma gondii reprograms its metabolism.

247 l of chronic CNS infection with the parasite Toxoplasma gondii requires ongoing T cell responses in t

248 TRIM21 as a previously unknown modulator of Toxoplasma gondii resistance in vivo thereby extending h

249 mucosal responses to the protozoan parasite Toxoplasma gondii resulted in dysbiosis and the eliminat

250 ted in a 100% reduction in the recurrence of Toxoplasma gondii retinochoroiditis over 1 year of treat

251 from Campinas, Brazil, with active recurrent Toxoplasma gondii retinochoroiditis were included.

252 A recent study in Toxoplasma gondii revealed a unique bipartite structure

253 The reduction in invasiveness of the Toxoplasma gondii RH-AMA1 knockout (RH-AMA1(KO)) tachyzo

254 GPI1 in combination with SAG1 may strengthen Toxoplasma gondii serology, in particular in seroepidemi

255 nd/or CARD8, including anthrax lethal toxin, Toxoplasma gondii, Shigella flexneri and the small molec

256 Transmissible stages of Toxoplasma gondii store energy in the form of the carboh

257 iments performed on the rat pineal gland and Toxoplasma gondii, successfully detecting known and prev

258 tudies have reported elevated levels of anti-Toxoplasma gondii (T. gondii) antibodies in patients wit

259 positive Goldmann-Witmer coefficient (GWC), Toxoplasma gondii (T. gondii) immunoblot, or T. gondii-s

260 Toxoplasma gondii (T. gondii) is a protozoan parasite th

261 oites of the apicomplexan protozoan parasite Toxoplasma gondii (T. gondii).

262 y Pneumocystis jirovecii (P. jirovecii, pj), Toxoplasma gondii (T. gondii, tg), and Mycobacterium avi

263 c cells infected with the protozoan parasite Toxoplasma gondii (Tg).

264 stigate the ATP4 protein of the apicomplexan Toxoplasma gondii, TgATP4.

265 e function of a large coiled-coil protein in Toxoplasma gondii, TgCep250, in connecting the two centr

266 3 in the parasites of this phylum, including Toxoplasma gondii (TgElp3), possesses a unique C-termina

267 olar proton pyrophosphatase (H(+) -PPase) of Toxoplasma gondii (TgVP1), a membrane proton pump, local

268 n infection caused by the protozoan parasite Toxoplasma gondii that can lead to severe sequelae in th

269 social amoeba Dictyostelium and the parasite Toxoplasma gondii The full effect of hydroxylation requi

270 In Dictyostelium (a social amoeba), Toxoplasma gondii (the agent for human toxoplasmosis), a

271 In apicomplexan parasites such as Toxoplasma gondii, the apical complex includes a spiral

272 In Toxoplasma gondii, the apical complex is a central site

273 Toxoplasma gondii, the causative agent of toxoplasmosis,

274 Toxoplasma gondii, the causative agent of toxoplasmosis,

275 The protozoan parasite Toxoplasma gondii, the causative agent of toxoplasmosis,

276 Toxoplasma gondii, the most common parasitic infection o

277 As a major natural host for Toxoplasma gondii, the mouse is widely used for the stud

278 In the ubiquitous protozoan parasite Toxoplasma gondii, the oocyst stage possesses a bilayere

279 -scale CRISPR screens recently developed for Toxoplasma gondii to discover sensitizing and desensitiz

280 Rodents are critical for the transmission of Toxoplasma gondii to the definitive feline host via pred

281 use pathogens, Heligmosomoides polygyrus and Toxoplasma gondii, to investigate the negative impact of

282 use pathogens, Heligsomosoides polygyrus and Toxoplasma gondii, to investigate the negative impact of

283 f intracellular proteins, the human pathogen Toxoplasma gondii transfers a different sugar, fucose, t

284 It is believed that infection by Toxoplasma gondii triggers a lifelong protective immunit

285 d that systemic infection with the protozoan Toxoplasma gondii triggers not only a transient increase

286 complexa phylum, such as Plasmodium spp. and Toxoplasma gondii, undergo complex life cycles involving

287 ne the polymerization properties of actin in Toxoplasma gondii, unexpectedly finding that it exhibits

288 e to reactivation of cerebral infection with Toxoplasma gondii using a murine model.

289 re we describe the myristoylated proteome of Toxoplasma gondii using chemoproteomic methods and show

290 pes 1 and 2, Mycobacterium tuberculosis, and Toxoplasma gondii via real-time polymerase chain reactio

291 The Toxoplasma gondii virulence factors ROP5 and ROP18 both

292 Toxoplasma gondii was not detected in field-collected sn

293 ng pathways that regulate these processes in Toxoplasma gondii We found that microneme secretion was

294 Salmonella typhimurium or Toxoplasma gondii were administered to knockout (KO) mic

295 ded in apicomplexan parasites, especially in Toxoplasma gondii where 14 separate genes encoding these

296 ed how CD8(+) T cells remove tissue cysts of Toxoplasma gondii, which can grow to the size of >50 mum

297 trol infection by the intracellular parasite Toxoplasma gondii, which corresponded to defects in mono

298 mice responded to the intracellular parasite Toxoplasma gondii While the loss of IL-27p28 and its ove

299 sion pathway analogous to that described for Toxoplasma gondii, with infectious stages traveling in f

300 The survival of Toxoplasma gondii within its host cell requires protein