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1             Parasitic nematodes of the genus Trichinella cause significant food-borne illness and occ
2 t time, a phylogeny and biogeography for the Trichinella complex, and show that encapsulated and non-
3 isease of humans caused by roundworms of the Trichinella complex.
4 g the Paleozoic, all contemporary species of Trichinella diversified within the last 20 million years
5 loides, filariasis, cysticercosis, fasciola, trichinella, echinococcosis) were excluded based on bloo
6                                              Trichinella-induced basophil responses were IL-3-IL-3R i
7 ial MMCs express integrin-alpha(E)beta(7) in Trichinella-infected BALB/c and S129 mice.
8       Selective depletion of basophils after Trichinella infection impaired infection-induced CD4(+)
9 k for understanding the history of the genus Trichinella is needed to maximize its utility.
10                                              Trichinella larvae colonize the diaphragm in large numbe
11                                       Viable Trichinella larvae were recovered from frozen cougar tis
12 and have not been previously associated with Trichinella nativa infection.
13  (iv) early hominids may have first acquired Trichinella on the African savannah several million year
14 nellosis and the finding of freeze-resistant Trichinella organisms in wildlife in Idaho extends the r
15 parasitic nematodes Trichinella spiralis and Trichinella pseudospiralis.
16                                              Trichinella serology on patient sera as well as polymera
17 draft genomes representing all 12 recognized Trichinella species and genotypes, define protein-coding
18                                              Trichinella species serology was performed on patient an
19     Serum samples were tested by an in-house Trichinella-specific enzyme-linked innunosorbent assay.
20 ng with myalgia and/or periorbital edema and Trichinella-specific immunoglobulin M and immunoglobulin
21 ated with significantly delayed expulsion of Trichinella spiralis and increased deposition of muscle
22 pulsion of two other gut-dwelling nematodes (Trichinella spiralis and Nippostrongylus brasiliensis).
23 due were detected in the parasitic nematodes Trichinella spiralis and Trichinella pseudospiralis.
24 tant strains) and the muscle-stage larvae of Trichinella spiralis as a representative parasitic nemat
25 is provided here that the parasitic nematode Trichinella spiralis can catalyze the conversion and thu
26  Although the forthcoming genome sequence of Trichinella spiralis can provide invaluable comparative
27 ection of mammalian skeletal muscle cells by Trichinella spiralis causes host nuclei to become polypl
28                                              Trichinella spiralis creates a unique intracellular habi
29       The L1 stage of the parasitic nematode Trichinella spiralis displays on its surface glycoprotei
30                   Because mice infected with Trichinella spiralis experience a pronounced, but transi
31 hown to have a critical role in clearance of Trichinella spiralis from the intestinal tract.
32 le for the expulsion of the related nematode Trichinella spiralis from these animals.
33 at wild boar meat products contaminated with Trichinella spiralis had entered the food chain in Germa
34 te mitochondrial DNA (mtDNA) of the nematode Trichinella spiralis has been amplified in four overlapp
35     The antibody response to the L1 stage of Trichinella spiralis has been described as biphasic.
36 ed in the survival of the parasitic nematode Trichinella spiralis in an intracellular environment are
37 on triggered by the intraepithelial nematode Trichinella spiralis in jejunal epithelium from BALB/c m
38  of acute inflammation during development of Trichinella spiralis in the muscle.
39 Infection of mice with the nematode parasite Trichinella spiralis induces changes in the proteome of
40 anthelmintic properties were evaluated using Trichinella spiralis infected mice.
41                                              Trichinella spiralis infection elicits a vigorous IgE re
42 hat robust induction of IL4 responses during Trichinella spiralis infection enhance the presence of n
43                                              Trichinella spiralis infection induces a strong type 2 c
44              We previously demonstrated that Trichinella spiralis infection inhibits host inducible N
45                                        After Trichinella spiralis infection of mice, we show that bas
46 e mast cell responses and inflammation after Trichinella spiralis infection or the induction of food
47                                              Trichinella spiralis is a highly destructive parasitic n
48                                              Trichinella spiralis is an obligate parasite of animals
49   Expulsion of the gastrointestinal nematode Trichinella spiralis is associated with pronounced masto
50        Infection with the parasitic nematode Trichinella spiralis is initiated when the L1 larva inva
51 inophils protect intracellular, muscle-stage Trichinella spiralis larvae against NO-mediated killing.
52                                              Trichinella spiralis larvae were identified in wild boar
53 ccompanying the expulsion of the GI parasite Trichinella spiralis may be dependent on IL-4 and mediat
54                               Infection with Trichinella spiralis rarely leads to significant morbidi
55 metabolizing enzymes in secreted products of Trichinella spiralis suggests that endoparasites use sim
56           We have used the parasite helminth Trichinella spiralis to study the generation and differe
57          Infection of mice with the nematode Trichinella spiralis triggers recruitment and differenti
58 binant human chymase efficiently degrade the Trichinella spiralis virulence factor heat shock protein
59 n the larval stage of the parasitic nematode Trichinella spiralis was shown to invade epithelial cell
60  MC in the jejunum of BALB/c mice exposed to Trichinella spiralis were assessed during the course of
61 polygyrus, Nippostrongylus brasiliensis, and Trichinella spiralis) alters intestinal epithelial cell
62         We report a draft genome sequence of Trichinella spiralis, a food-borne zoonotic parasite, wh
63              The mouse is a natural host for Trichinella spiralis, a worm that establishes chronic in
64 p necrotizing hepatitis after infection with Trichinella spiralis, and inflammation is dependent on t
65            We used a natural mouse parasite, Trichinella spiralis, and multipoint intravital time-lap
66 MMC) recruitment coincides with expulsion of Trichinella spiralis, at a time when the majority of the
67 pond vigorously to intestinal infection with Trichinella spiralis, eliminating a role for T-bet in MC
68 rimary infection with the parasitic nematode Trichinella spiralis, eosinophils play an important immu
69 hanced expulsion of the intestinal nematode, Trichinella spiralis, from the small intestine.
70           Studies with rodents infected with Trichinella spiralis, Heligmosomoides polygyrus, Nippost
71 tinal infection with the parasitic nematode, Trichinella spiralis, induces a pronounced eosinophilia
72  parasites, Nippostrongylus brasiliensis and Trichinella spiralis, is similar in that both require IL
73 olate-induced peritonitis and infection with Trichinella spiralis, stimulated similar responses in Ga
74                  In parasitic infection with Trichinella spiralis, the immune response incorporates b
75 y response caused by an intestinal parasite, Trichinella spiralis, to study the relationship between
76              We have surveyed the genomes of Trichinella spiralis, Trichuris muris, and Romanomermis
77 ulsion of another gastrointestinal nematode, Trichinella spiralis, unlike N. brasiliensis expulsion,
78 t it does not contribute to immunity against Trichinella spiralis, which lives within IEC.
79 in were observed in the jejunal submucosa of Trichinella spiralis-infected BALB/c mice.
80               Jejunal segments isolated from Trichinella spiralis-infected mice were used to assess 5
81  mice and in mesenteric lymph nodes (mLN) of Trichinella spiralis-infected mice.
82                 Previous work has shown that Trichinella spiralis-infected rats transport IgE from pl
83 ctor phase of a secondary immune response to Trichinella spiralis.
84 zing hepatitis following oral infection with Trichinella spiralis.
85 racterize cellular responses to muscle-stage Trichinella spiralis.
86 yperplasia in the mouse after infection with Trichinella spiralis.
87 the course (0 to 289 h) of an infection with Trichinella spiralis.
88  harbor exceptionally uniform populations of Trichinella, Taenia, Toxoplasma and Sarcocystis, indicat
89  show that encapsulated and non-encapsulated Trichinella taxa diverged from their most recent common
90 cal and genetic variability within and among Trichinella taxa, and major controversy surrounds the sy
91 ly, since even frozen meat may harbor viable Trichinella that can cause illness.
92 ults consistent with genotypes T. nativa and Trichinella type T6.
93 f the phylogeny and biogeographic history of Trichinella using the variation in three genes (nuclear