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1 minths (Heligmosomoides polygyrus bakeri and Trichuris muris).
2  and mice infected with the enteric parasite Trichuris muris.
3 ris trichiura and the mouse laboratory model Trichuris muris.
4  with the gastrointestinal nematode parasite Trichuris muris.
5 e large intestine dwelling helminth parasite Trichuris muris.
6 sing the murine model of whipworm infection, Trichuris muris.
7 table and widely-used model of trichuriasis, Trichuris muris.
8 phiregulin delayed expulsion of the nematode Trichuris muris.
9 sponse against the gastrointestinal helminth Trichuris muris.
10 impaired immunity to the intestinal helminth Trichuris muris.
11 o infection by the gastrointestinal nematode Trichuris muris.
12 tion with the gut-dwelling helminth parasite Trichuris muris.
13 -scale metabolic model of the mouse whipworm Trichuris muris.
14 oor enclosure and infected with the parasite Trichuris muris.
15 ed in AKR/J mice using a parasite infection, Trichuris muris.
16 ion of the caecal epithelium by the parasite Trichuris muris allowed us to live image syncytial tunne
17 atory conditions in intestines infected with Trichuris muris and within the tumor microenvironment (B
18 urveyed the genomes of Trichinella spiralis, Trichuris muris, and Romanomermis culicivorax and identi
19 lminth parasites, Heligmosomoides polygyrus, Trichuris muris, and Schistosoma mansoni.
20             In studies of mice infected with Trichuris muris, blocking B7 ligand interactions inhibit
21  infected with the gastrointestinal helminth Trichuris muris displayed accelerated expulsion of paras
22                                Additionally, Trichuris muris eggs displayed defective hatching in the
23 treated mice infected with enteric parasite, Trichuris muris, exhibited attenuated 5-HT production, c
24 taneous peristaltic movement and facilitated Trichuris muris expulsion.
25 polygyrus, Nippostronglyus brasiliensis, and Trichuris muris have provided considerable information a
26 on during murine infection with the helminth Trichuris muris However, the mechanisms required for bas
27 infection with the gastrointestinal helminth Trichuris muris, immunity to which is critically depende
28 c infection by the gastrointestinal nematode Trichuris muris in susceptible AKR mice, which mount a T
29 ngruent infection with the nematode parasite Trichuris muris in the large intestine around the time o
30 the chronically infecting parasitic nematode Trichuris muris in the large intestine of mice is depend
31                           Here, we show that Trichuris muris infection in mice increases coinfection
32        We used the well-established model of Trichuris muris infection to investigate the innate resp
33 is induced in the cecum of mice resistant to Trichuris muris infection.
34 with the gastrointestinal toxin piroxicam or Trichuris muris infection.
35 ation in MLN and mount Th1 cell responses to Trichuris muris infection.
36 nvestigate early infection events using both Trichuris muris infections of mice and murine caecaloids
37  infection with the caecum-dwelling helminth Trichuris muris is dependent on interleukin (IL)-4 and I
38   Expulsion of the gastrointestinal nematode Trichuris muris is mediated by a T helper (Th) 2 type re
39 infection with the gastrointestinal parasite Trichuris muris, memory CD4+ T cells persist in the drai
40                The p43 (Tm-DLP-1) protein of Trichuris muris of mice comprises 95% of the protein sec
41  Th cells, ICOS(-/-) mice were infected with Trichuris muris or Toxoplasma gondii.
42                                              Trichuris muris resides in intimate contact with its hos
43 nfection with the gastrointestinal helminth, Trichuris muris, revealing roles for miRNAs in fibrosis
44 okines in immunity to the parasitic helminth Trichuris muris, the local effector mechanism culminatin
45 n infection with the cecum-dwelling nematode Trichuris muris, the majority of inbred strains of mice
46 IL-12-, IL-4-, and IL-12-deficient mice with Trichuris muris to determine whether IL-10 contributes t
47   In this study, we used the murine whipworm Trichuris muris to investigate the effect of the ferment
48  used the gastrointestinal helminth pathogen Trichuris muris to test the hypothesis that a chronic sy
49                      The intestinal nematode Trichuris muris was recently demonstrated to utilise mic
50 nic infection with gastrointestinal helminth Trichuris muris, we identify dual functions for RELMbeta
51 le to infection with the intestinal nematode Trichuris muris, whereas their wild-type littermates wer
52 ection with the intestinal helminth parasite Trichuris muris, which yields a chronic infection becaus
53  and activity against evolutionarily distant Trichuris muris whipworm adults.
54 e show that intestinal Th2 responses against Trichuris muris worms and Schistosoma mansoni eggs do no